Robert E Schifferle

Summary

Affiliation: University at Buffalo
Country: USA

Publications

  1. ncbi request reprint Nutrition and periodontal disease
    Robert E Schifferle
    Department of Periodontics and Endodontics, School of Dental Medicine, University at Buffalo, State University of New York, 318 Foster Hall, 3435 Main Street, Buffalo, NY 14214 3092, USA
    Dent Clin North Am 49:595-610, vii. 2005
  2. pmc Dependence of bacterial protein adhesins on toll-like receptors for proinflammatory cytokine induction
    George Hajishengallis
    Department of Oral Biology, State University of New York at Buffalo, Buffalo, New York 14214, USA
    Clin Diagn Lab Immunol 9:403-11. 2002
  3. pmc Counteracting interactions between lipopolysaccharide molecules with differential activation of toll-like receptors
    George Hajishengallis
    Department of Oral Biology, State University of New York at Buffalo, 14214, USA
    Infect Immun 70:6658-64. 2002
  4. ncbi request reprint Role of the phosphatidylinositol 3 kinase-Akt pathway in the regulation of IL-10 and IL-12 by Porphyromonas gingivalis lipopolysaccharide
    Michael Martin
    Department of Microbiology, University of Alabama, Birmingham, AL 35294, USA
    J Immunol 171:717-25. 2003
  5. ncbi request reprint Differential interactions of fimbriae and lipopolysaccharide from Porphyromonas gingivalis with the Toll-like receptor 2-centred pattern recognition apparatus
    George Hajishengallis
    Center for Oral Health and Systemic Disease and Department of Periodontics Endodontics, University of Louisville Health Sciences Center, Louisville, KY 40292, USA
    Cell Microbiol 8:1557-70. 2006
  6. ncbi request reprint The effects of Porphyromonas gingivalis LPS and Actinobacillus actinomycetemcomitans LPS on human dendritic cells in vitro, and in a mouse model in vivo
    Rangsini Mahanonda
    Department of Periodontology, Faculty of Dentistry, Chulalongkorn University, Bangkok, Thailand
    Asian Pac J Allergy Immunol 24:223-8. 2006
  7. ncbi request reprint Lipopolysaccharides from atherosclerosis-associated bacteria antagonize TLR4, induce formation of TLR2/1/CD36 complexes in lipid rafts and trigger TLR2-induced inflammatory responses in human vascular endothelial cells
    Martha Triantafilou
    Infection and Immunity Group, School of Life Sciences, University of Sussex, Falmer, Brighton, BN1 9QG, UK
    Cell Microbiol 9:2030-9. 2007
  8. ncbi request reprint Serum IgG to heat shock proteins and Porphyromonas gingivalis antigens in diabetic patients with periodontitis
    Tom J Sims
    Department of Oral Medicine, University of Washington, Seattle WA 98195, USA
    J Clin Periodontol 29:551-62. 2002

Collaborators

Detail Information

Publications8

  1. ncbi request reprint Nutrition and periodontal disease
    Robert E Schifferle
    Department of Periodontics and Endodontics, School of Dental Medicine, University at Buffalo, State University of New York, 318 Foster Hall, 3435 Main Street, Buffalo, NY 14214 3092, USA
    Dent Clin North Am 49:595-610, vii. 2005
    ..This article discusses general concepts of nutrition and provides an overview of the current understanding of the relationship between nutrition and periodontal disease...
  2. pmc Dependence of bacterial protein adhesins on toll-like receptors for proinflammatory cytokine induction
    George Hajishengallis
    Department of Oral Biology, State University of New York at Buffalo, Buffalo, New York 14214, USA
    Clin Diagn Lab Immunol 9:403-11. 2002
    ..Our data suggest that the CD14-TLR2/4 system is involved in cytokine production and tolerance induction upon interaction with certain proinflammatory bacterial protein adhesins...
  3. pmc Counteracting interactions between lipopolysaccharide molecules with differential activation of toll-like receptors
    George Hajishengallis
    Department of Oral Biology, State University of New York at Buffalo, 14214, USA
    Infect Immun 70:6658-64. 2002
    ..Interactions between TLR2 and TLR4 agonists may be important in the regulation of inflammatory reactions...
  4. ncbi request reprint Role of the phosphatidylinositol 3 kinase-Akt pathway in the regulation of IL-10 and IL-12 by Porphyromonas gingivalis lipopolysaccharide
    Michael Martin
    Department of Microbiology, University of Alabama, Birmingham, AL 35294, USA
    J Immunol 171:717-25. 2003
    ..gingivalis LPS affects the induction of key immunoregulatory cytokines that control both qualitative and quantitative aspects of innate and adaptive immunity...
  5. ncbi request reprint Differential interactions of fimbriae and lipopolysaccharide from Porphyromonas gingivalis with the Toll-like receptor 2-centred pattern recognition apparatus
    George Hajishengallis
    Center for Oral Health and Systemic Disease and Department of Periodontics Endodontics, University of Louisville Health Sciences Center, Louisville, KY 40292, USA
    Cell Microbiol 8:1557-70. 2006
    ..In summary, TLR2 activation by P. gingivalis LPS or fimbriae involves differential dependence on accessory signalling or ligand-binding receptors, which may differentially influence innate immune responses...
  6. ncbi request reprint The effects of Porphyromonas gingivalis LPS and Actinobacillus actinomycetemcomitans LPS on human dendritic cells in vitro, and in a mouse model in vivo
    Rangsini Mahanonda
    Department of Periodontology, Faculty of Dentistry, Chulalongkorn University, Bangkok, Thailand
    Asian Pac J Allergy Immunol 24:223-8. 2006
    ..In conclusion, stimulation by P. gingivalis LPS and A. actinomycetemcomitans LPS appears to bias Mo-DCs towards Th2 production...
  7. ncbi request reprint Lipopolysaccharides from atherosclerosis-associated bacteria antagonize TLR4, induce formation of TLR2/1/CD36 complexes in lipid rafts and trigger TLR2-induced inflammatory responses in human vascular endothelial cells
    Martha Triantafilou
    Infection and Immunity Group, School of Life Sciences, University of Sussex, Falmer, Brighton, BN1 9QG, UK
    Cell Microbiol 9:2030-9. 2007
    ..This antagonistic activity of H. pylori or P. gingivalis LPS, as well as their TLR2 activation capability may be associated with their ability to contribute to atherosclerosis...
  8. ncbi request reprint Serum IgG to heat shock proteins and Porphyromonas gingivalis antigens in diabetic patients with periodontitis
    Tom J Sims
    Department of Oral Medicine, University of Washington, Seattle WA 98195, USA
    J Clin Periodontol 29:551-62. 2002
    ..We have therefore hypothesized that profiles of serum autoantibody levels and IgG titers, to P. gingivalis-specific antigens may be useful in assessing risk for refractory periodontitis in such patients...