David P Richman

Summary

Affiliation: University of California
Country: USA

Publications

  1. pmc Treatment of myasthenia gravis based on its immunopathogenesis
    Jee Young Kim
    Department of Neurology, Kwandong University College of Medicine, Myongji Hospital, Goyang, Korea
    J Clin Neurol 7:173-83. 2011
  2. ncbi request reprint Animal models of antimuscle-specific kinase myasthenia
    David P Richman
    Department of Neurology, Center for Neuroscience, University of California, Davis, 95616, USA
    Ann N Y Acad Sci 1274:140-7. 2012
  3. pmc Dominantly inherited myotonia congenita resulting from a mutation that increases open probability of the muscle chloride channel CLC-1
    David P Richman
    Department of Neurology and Center for Neuroscience, University of California, Davis, CA 95616, USA
    Neuromolecular Med 14:328-37. 2012
  4. ncbi request reprint Acute severe animal model of anti-muscle-specific kinase myasthenia: combined postsynaptic and presynaptic changes
    David P Richman
    Department of Neurology, University of California Davis, 1515 Newton Ct, Davis, CA 95616, USA
    Arch Neurol 69:453-60. 2012
  5. ncbi request reprint Treatment principles in the management of autoimmune myasthenia gravis
    David P Richman
    Department of Neurology, University of California, Davis, Davis, California 95616, USA
    Ann N Y Acad Sci 998:457-72. 2003
  6. ncbi request reprint Treatment of autoimmune myasthenia gravis
    David P Richman
    University of California, Davis 95616, USA
    Neurology 61:1652-61. 2003
  7. ncbi request reprint Structural characterization of the main immunogenic region of the Torpedo acetylcholine receptor
    Stuart W Morell
    University of California, Davis School of Medicine, Department of Neurology, One Shields Avenue, 1515 Newton Court, Room 510C, Davis, CA 95616, United States Biochemistry, Molecular, Cellular, and Developmental Biology Graduate Group of UC Davis, United States
    Mol Immunol 58:116-31. 2014
  8. ncbi request reprint Agonist-induced transitions of the acetylcholine receptor
    Robert H Fairclough
    Department of Neurology, University of California, Davis, Davis, California 95616, USA
    Ann N Y Acad Sci 998:101-13. 2003
  9. ncbi request reprint Variable phenotypes associated with mutations in DOK7
    Jennifer A Anderson
    Department of Neurology, University of California at Davis, Davis, CA 95618, USA
    Muscle Nerve 37:448-56. 2008
  10. ncbi request reprint Three forms of immune myasthenia
    Mark A Agius
    Department of Neurology, University of California at Davis, Davis, California 95616, USA
    Ann N Y Acad Sci 998:453-6. 2003

Collaborators

Detail Information

Publications11

  1. pmc Treatment of myasthenia gravis based on its immunopathogenesis
    Jee Young Kim
    Department of Neurology, Kwandong University College of Medicine, Myongji Hospital, Goyang, Korea
    J Clin Neurol 7:173-83. 2011
    ..It is hoped that well-organized studies and newer experimental trials will lead to improved treatments...
  2. ncbi request reprint Animal models of antimuscle-specific kinase myasthenia
    David P Richman
    Department of Neurology, Center for Neuroscience, University of California, Davis, 95616, USA
    Ann N Y Acad Sci 1274:140-7. 2012
    ..The observations in this human disease and its animal models demonstrate the role of MuSK not only in the formation of this synapse but also in its maintenance...
  3. pmc Dominantly inherited myotonia congenita resulting from a mutation that increases open probability of the muscle chloride channel CLC-1
    David P Richman
    Department of Neurology and Center for Neuroscience, University of California, Davis, CA 95616, USA
    Neuromolecular Med 14:328-37. 2012
    ..Since expression levels of the mutant and wild-type subunits in artificial model cell systems were unable to explain the disease symptoms, the mechanism leading to dominant inheritance in this family remains to be determined...
  4. ncbi request reprint Acute severe animal model of anti-muscle-specific kinase myasthenia: combined postsynaptic and presynaptic changes
    David P Richman
    Department of Neurology, University of California Davis, 1515 Newton Ct, Davis, CA 95616, USA
    Arch Neurol 69:453-60. 2012
    ..Design, Setting, and..
  5. ncbi request reprint Treatment principles in the management of autoimmune myasthenia gravis
    David P Richman
    Department of Neurology, University of California, Davis, Davis, California 95616, USA
    Ann N Y Acad Sci 998:457-72. 2003
    ..In general, the plan involves an initial forceful attack followed by a slow and measured withdrawal...
  6. ncbi request reprint Treatment of autoimmune myasthenia gravis
    David P Richman
    University of California, Davis 95616, USA
    Neurology 61:1652-61. 2003
    ..The short-term therapies, i.e., IV immunoglobulin or plasmapheresis, may be effective in the early stages of treatment or later during an exacerbation. Steroid-sparing medications are usually added to facilitate the tapering phase...
  7. ncbi request reprint Structural characterization of the main immunogenic region of the Torpedo acetylcholine receptor
    Stuart W Morell
    University of California, Davis School of Medicine, Department of Neurology, One Shields Avenue, 1515 Newton Court, Room 510C, Davis, CA 95616, United States Biochemistry, Molecular, Cellular, and Developmental Biology Graduate Group of UC Davis, United States
    Mol Immunol 58:116-31. 2014
    ..This third segment appears to form a foundation upon which the three-dimensional 132A epitope is anchored. ..
  8. ncbi request reprint Agonist-induced transitions of the acetylcholine receptor
    Robert H Fairclough
    Department of Neurology, University of California, Davis, Davis, California 95616, USA
    Ann N Y Acad Sci 998:101-13. 2003
    ..The carbamylcholine-induced loss of the 383C epitope, the 10-A contraction of the beta-hairpin loop, and the loss of multivalent cations from the channel likely represent the first molecular transitions leading to AChR channel opening...
  9. ncbi request reprint Variable phenotypes associated with mutations in DOK7
    Jennifer A Anderson
    Department of Neurology, University of California at Davis, Davis, CA 95618, USA
    Muscle Nerve 37:448-56. 2008
    ..Overall, our study corroborates the findings of others and provides an additional demonstration of the considerable phenotypic variability associated with CMS due to DOK7 mutations...
  10. ncbi request reprint Three forms of immune myasthenia
    Mark A Agius
    Department of Neurology, University of California at Davis, Davis, California 95616, USA
    Ann N Y Acad Sci 998:453-6. 2003
    ..This classification has advantages over the present classifications as regards homogeneity of groups, etiology, mechanism of disease, and prognosis...
  11. doi request reprint Accelerated central nervous system autoimmunity in BAFF-receptor-deficient mice
    Susan S Kim
    Department of Neurology and Neuroscience Graduate Program, University of California Davis, Davis, CA, USA
    J Neurol Sci 306:9-15. 2011
    ..Thus, BAFF-R may be important in EAE pathogenesis, possibly by influencing macrophage function through a mechanism that involves modulation of BAFF expression...