Deborah A Rice
Affiliation: University of Southern Maine
- Consequences of exposure from persistent organic pollutants (POPs): Session X summary and research needsDeborah A Rice
Maine Department of Environmental Protection, Bureau of Remediation and Waste Management, State House Station 17, Augusta, ME 04333 0017, USA
Neurotoxicology 25:521-3. 2004
- The US EPA reference dose for methylmercury: sources of uncertaintyDeborah C Rice
National Center for Environmental Assessment Office of Research and Development, US Environmental Protection Agency, Washington, DC 20460, USA
Environ Res 95:406-13. 2004..In addition, it may be argued that derivation of a RfD for methylmercury is inappropriate, given that there does not appear to be a threshold for adverse neuropsychological effects based on available data...
- Assessing the effects of environmental toxicant exposure in developmental epidemiological studies: issues for risk assessmentDeborah C Rice
Environmental Health Unit, Maine Bureau of Health, State House Station 11, Augusta, ME 04333, USA
Neurotoxicology 26:483-9. 2005..Opportunity for discussion among investigators, statisticians, and risk assessors potentially would result in human developmental toxicity studies being maximally useful for public health decisions...
- Methods and rationale for derivation of a reference dose for methylmercury by the U.S. EPADeborah C Rice
National Center for Environmental Assessment, Office of Research and Development, U S Environmental Protection Agency, Washington, DC 20460, USA
Risk Anal 23:107-15. 2003..Other areas that EPA identified as significant areas requiring further attention are cardiovascular consequences of methylmercury exposure and delayed neurotoxicity during aging as a result of previous developmental or adult exposure...
- Maternal seafood consumption and children's developmentAlan H Stern
Lancet 370:217-8; author reply 218. 2007
- Evaluation of the U.S. EPA/OSWER preliminary remediation goal for perchlorate in groundwater: focus on exposure to nursing infantsGary L Ginsberg
Connecticut Department of Public Health, Hartford, Connecticut 06134, USA
Environ Health Perspect 115:361-9. 2007..This does not account for the greater exposure that is possible in nursing infants or for the relative source contribution (RSC), a factor normally used to lower the PRG due to nonwater exposures...
- Prenatal methylmercury exposure affects spatial vision in adult monkeysThomas M Burbacher
Department of Environmental and Occupational Health Sciences, Box 357234, University of Washington, Seattle, WA 98195, USA
Toxicol Appl Pharmacol 208:21-8. 2005..The results from this study demonstrate that chronic in utero MeHg exposure, at subclinical levels, is associated with permanent adverse effects on spatial vision in adult monkeys...
- Effects of PCB exposure on neuropsychological function in childrenSusan L Schantz
Department of Veterinary Biosciences, College of Veterinary Medicine, University of Illinois at Urbana Champaign, Urbana, Illinois 61802, USA
Environ Health Perspect 111:357-576. 2003..More complete information on the health effects of various congeners or congener classes would allow more informed scientific and risk assessment decisions...
- Blood lead concentration and delayed puberty in girlsSherry G Selevan
National Center for Environmental Assessment, Office of Research and Development, Environmental Protection Agency, Washington, DC 20460, USA
N Engl J Med 348:1527-36. 2003..Environmental lead exposure has been linked to alterations in growth and endocrine function. It is not known whether such exposure affects pubertal development...
- Alzheimer's disease (AD)-like pathology in aged monkeys after infantile exposure to environmental metal lead (Pb): evidence for a developmental origin and environmental link for ADJinfang Wu
Department of Biomedical and Pharmaceutical Sciences, University of Rhode Island, Kingston, Rhode Island 02881, USA
J Neurosci 28:3-9. 2008..These data suggest that AD pathogenesis is influenced by early life exposures and argue for both an environmental trigger and a developmental origin of AD...