E Albert Reece

Summary

Affiliation: University of Maryland
Country: USA

Publications

  1. doi request reprint Perspectives on obesity, pregnancy and birth outcomes in the United States: the scope of the problem
    E Albert Reece
    Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD 21201, USA
    Am J Obstet Gynecol 198:23-7. 2008
  2. doi request reprint The fetal and maternal consequences of gestational diabetes mellitus
    E Albert Reece
    Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, 655 W Baltimore Street, Room 14 029, Baltimore, MD 21201 1559, USA
    J Matern Fetal Neonatal Med 23:199-203. 2010
  3. doi request reprint The diagnostic criteria for gestational diabetes: to change or not to change?
    E Albert Reece
    University of Maryland School of Medicine, Baltimore, MD 21201, USA
    Am J Obstet Gynecol 208:255-9. 2013
  4. pmc Fully aligned academic health centers: a model for 21st-century job creation and sustainable economic growth
    E Albert Reece
    University of Maryland School of Medicine, Baltimore, MD 21201 1559, USA
    Acad Med 87:982-7. 2012
  5. ncbi request reprint Adapting industry-style business model to academia in a system of Performance-based Incentive Compensation
    E Albert Reece
    University of Maryland, USA
    Acad Med 83:76-84. 2008
  6. ncbi request reprint What's in a name? A paradigm shift for clinical research
    E Albert Reece
    University of Maryland School of Medicine, Baltimore, MD 21201, USA
    J Investig Med 55:220-2. 2007
  7. doi request reprint Obesity, diabetes, and links to congenital defects: a review of the evidence and recommendations for intervention
    E Albert Reece
    University of Maryland School of Medicine, Baltimore, MD 21201, USA
    J Matern Fetal Neonatal Med 21:173-80. 2008
  8. doi request reprint Gestational diabetes: the need for a common ground
    E Albert Reece
    University of Maryland School of Medicine, Department of Obstetrics, Gynecology, and Reproductive Sciences, Baltimore, MD 21201 1559, USA
    Lancet 373:1789-97. 2009
  9. doi request reprint Diabetes-induced birth defects: what do we know? What can we do?
    E Albert Reece
    University of Maryland School of Medicine, 655 West Baltimore Street, Room 14 029, Baltimore, MD 21201 1559, USA
    Curr Diab Rep 12:24-32. 2012
  10. pmc Oxidative stress-induced JNK1/2 activation triggers proapoptotic signaling and apoptosis that leads to diabetic embryopathy
    Xuezheng Li
    Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, Maryland, USA
    Diabetes 61:2084-92. 2012

Research Grants

  1. GENERAL CLINICAL RESEARCH CENTER
    E Reece; Fiscal Year: 2006
  2. GENERAL CLINICAL RESEARCH CENTER
    AUBREY MD HOUGH; Fiscal Year: 2006
  3. GENERAL CLINICAL RESEARCH CENTER
    E Reece; Fiscal Year: 2007
  4. GENERAL CLINICAL RESEARCH CENTER
    E Reece; Fiscal Year: 2009
  5. Mechanisms of Diabetic Embryopathy and Molecular Pathways
    E Reece; Fiscal Year: 2009
  6. Mechanisms of Diabetic Embryopathy and Molecular Pathways
    E Albert Reece; Fiscal Year: 2010
  7. Cancer Center Renovation Grant
    Donald Wilson; Fiscal Year: 2004
  8. RES ANIMAL FACIL: ANIMAL MODEL
    Donald Wilson; Fiscal Year: 2002
  9. CONSTRUCTION OF RESEARCH ANIMAL FACILITY
    Donald Wilson; Fiscal Year: 2002
  10. Web-Based IRB Data Management and Archiving System
    Donald Wilson; Fiscal Year: 2002

Collaborators

Detail Information

Publications53

  1. doi request reprint Perspectives on obesity, pregnancy and birth outcomes in the United States: the scope of the problem
    E Albert Reece
    Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD 21201, USA
    Am J Obstet Gynecol 198:23-7. 2008
    ..Judging by the speed of its progression and the toll it is taking, obesity can truly be called America's newest and fastest-growing epidemic...
  2. doi request reprint The fetal and maternal consequences of gestational diabetes mellitus
    E Albert Reece
    Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, 655 W Baltimore Street, Room 14 029, Baltimore, MD 21201 1559, USA
    J Matern Fetal Neonatal Med 23:199-203. 2010
    ..There remains, however, a great deal of controversy over when to begin screening for hyperglycemia in pregnancy and at what level of hyperglycemia should aggressive intervention be initiated...
  3. doi request reprint The diagnostic criteria for gestational diabetes: to change or not to change?
    E Albert Reece
    University of Maryland School of Medicine, Baltimore, MD 21201, USA
    Am J Obstet Gynecol 208:255-9. 2013
    ..The scientific dilemma to change or not to change, thus, requires a rigorous analysis of the scientific, economic, practice, and legal pros and cons to achieve a satisfactory answer...
  4. pmc Fully aligned academic health centers: a model for 21st-century job creation and sustainable economic growth
    E Albert Reece
    University of Maryland School of Medicine, Baltimore, MD 21201 1559, USA
    Acad Med 87:982-7. 2012
    ..Indeed, UMM, JHM, and other fully aligned AHCs have shown that they can be powerful economic engines and offer a model of job growth and economic development in the 21st century...
  5. ncbi request reprint Adapting industry-style business model to academia in a system of Performance-based Incentive Compensation
    E Albert Reece
    University of Maryland, USA
    Acad Med 83:76-84. 2008
    ..The PBIC program continues to challenge our faculty members to attain their highest potential while rewarding them accordingly...
  6. ncbi request reprint What's in a name? A paradigm shift for clinical research
    E Albert Reece
    University of Maryland School of Medicine, Baltimore, MD 21201, USA
    J Investig Med 55:220-2. 2007
  7. doi request reprint Obesity, diabetes, and links to congenital defects: a review of the evidence and recommendations for intervention
    E Albert Reece
    University of Maryland School of Medicine, Baltimore, MD 21201, USA
    J Matern Fetal Neonatal Med 21:173-80. 2008
    ..In addition, the article offers recommendations for facilitating the broader scale implementation of evidence-based approaches to preventing obesity, particularly among pregnant women...
  8. doi request reprint Gestational diabetes: the need for a common ground
    E Albert Reece
    University of Maryland School of Medicine, Department of Obstetrics, Gynecology, and Reproductive Sciences, Baltimore, MD 21201 1559, USA
    Lancet 373:1789-97. 2009
    ....
  9. doi request reprint Diabetes-induced birth defects: what do we know? What can we do?
    E Albert Reece
    University of Maryland School of Medicine, 655 West Baltimore Street, Room 14 029, Baltimore, MD 21201 1559, USA
    Curr Diab Rep 12:24-32. 2012
    ..Based on what we know about the molecular pathophysiology of diabetic embryopathy, our laboratory and others are developing interventions against to key molecular targets in this multifactorial disease process...
  10. pmc Oxidative stress-induced JNK1/2 activation triggers proapoptotic signaling and apoptosis that leads to diabetic embryopathy
    Xuezheng Li
    Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, Maryland, USA
    Diabetes 61:2084-92. 2012
    ..Our results show that JNK1 and JNK2 are equally involved in diabetic embryopathy and that the oxidative stress-JNK1/2-caspase pathway mediates the proapoptotic signals and the teratogenicity of maternal diabetes...
  11. doi request reprint Maternal hyperglycemia activates an ASK1-FoxO3a-caspase 8 pathway that leads to embryonic neural tube defects
    Peixin Yang
    Department of Obstetrics, University of Maryland School of Medicine, Baltimore, MD 21201, USA
    Sci Signal 6:ra74. 2013
    ..Thus, activation of an ASK1-FoxO3a-TRADD-caspase 8 pathway participates in the development of neural tube defects, which could be prevented by inhibiting intermediates in this cascade. ..
  12. pmc SOD1 suppresses maternal hyperglycemia-increased iNOS expression and consequent nitrosative stress in diabetic embryopathy
    Hongbo Weng
    Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD, USA
    Am J Obstet Gynecol 206:448.e1-7. 2012
    ..Hyperglycemia induces oxidative stress and increases inducible nitric oxide synthase (iNOS) expression. We hypothesized that oxidative stress is responsible for hyperglycemia-induced iNOS expression...
  13. doi request reprint The essential role of protein kinase Cδ in diabetes-induced neural tube defects
    Yuanning Cao
    Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD 21201, USA
    J Matern Fetal Neonatal Med 25:2020-4. 2012
    ..The aims of this study are to investigate the role of proapoptotic PKCδ in NTD formation and the underlying mechanisms...
  14. pmc Superoxide dismutase 1 overexpression in mice abolishes maternal diabetes-induced endoplasmic reticulum stress in diabetic embryopathy
    Fang Wang
    Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD
    Am J Obstet Gynecol 209:345.e1-7. 2013
    ..Both oxidative stress and endoplasmic reticulum stress (ER stress) are causal events in diabetic embryopathy. We tested whether oxidative stress causes ER stress...
  15. pmc Caspase-8: a key role in the pathogenesis of diabetic embryopathy
    Zhiyong Zhao
    Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, 655 West Baltimore Street, Baltimore, MD 21201, USA
    Birth Defects Res B Dev Reprod Toxicol 86:72-7. 2009
    ..These data suggest that caspase-8 may control diabetic embryopathy-associated apoptosis via regulation of the Bid-stimulated mitochondrion/caspase-9 pathway...
  16. ncbi request reprint Prepregnancy care and the prevention of fetal malformations in the pregnancy complicated by diabetes
    E Albert Reece
    Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, Maryland, USA
    Clin Obstet Gynecol 50:990-7. 2007
    ..Therefore, it is recommended that all women with diabetes of childbearing age be advised of the importance of seeking preconception care, which includes attainment of optimal glucose control and the use of contraception...
  17. doi request reprint Demonstration of the essential role of protein kinase C isoforms in hyperglycemia-induced embryonic malformations
    - Zhiyong Zhao
    Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, Maryland 21201, USA
    Reprod Sci 15:349-56. 2008
    ..Activation of cPLA(2) was also decreased, suggesting that PKCs mediate the hyperglycemic effects through the cPLA(2)-phospholipid peroxidation pathway...
  18. doi request reprint Blockade of c-Jun N-terminal kinase activation abrogates hyperglycemia-induced yolk sac vasculopathy in vitro
    Peixin Yang
    Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD, USA
    Am J Obstet Gynecol 198:321.e1-7. 2008
    ..In addition, during embryopathy, teratogen such as hyperglycemia is associated with concomitant injury to the yolk sac and embryo...
  19. pmc Protein kinase Cβ2 inhibition reduces hyperglycemia-induced neural tube defects through suppression of a caspase 8-triggered apoptotic pathway
    Yuanning Cao
    Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD, USA
    Am J Obstet Gynecol 204:226.e1-5. 2011
    ..The apoptosis is triggered by caspase 8, which activates members of the Bcl-2 and caspase families, such as Bid and caspase 3. Whether PKCβ2 regulates caspase 8-induced apoptosis remains to be addressed...
  20. doi request reprint Activation of oxidative stress signaling that is implicated in apoptosis with a mouse model of diabetic embryopathy
    Peixin Yang
    Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD, USA
    Am J Obstet Gynecol 198:130.e1-7. 2008
    ..To establish a link between oxidative stress signaling and diabetic embryopathy, levels of phosphorylated-p66Shc (which is a key signaling molecule that mediates oxidative stress-induced apoptosis) were evaluated...
  21. pmc New concepts in diabetic embryopathy
    Zhiyong Zhao
    Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD 21201, USA
    Clin Lab Med 33:207-33. 2013
    ..Translating advances made in animal studies into clinical applications in humans requires collaborative efforts across the basic research, preclinical, and clinical communities...
  22. pmc Reduction in embryonic malformations and alleviation of endoplasmic reticulum stress by nitric oxide synthase inhibition in diabetic embryopathy
    Zhiyong Zhao
    Department of Obstetrics, Gynecology, and Reproductive Sciences, Department of Biochemistry and Molecular Biology, University of Maryland School of Medicine, Baltimore, MD 21201, USA
    Reprod Sci 19:823-31. 2012
    ..Oral treatment with NOS-2 inhibitor alleviates nitrosative and ER stress, decreases apoptosis, and reduces NTDs in the embryos, providing information for further interventional studies to reduce diabetes-associated birth defects...
  23. ncbi request reprint The impact of self-monitoring of blood glucose on self-efficacy and pregnancy outcomes in women with diet-controlled gestational diabetes
    Carol J Homko
    General Clinical Research Center, Department of Obstetrics, Gynecology, and Reproductive Sciences, Temple University Hospital, 4 West, 3401 North Broad Street, Philadelphia, PA 19140, USA
    Diabetes Educ 28:435-43. 2002
    ..The purpose of this study was to examine the effects of self-monitoring of blood glucose (SMBG) on feelings of self-efficacy, dietary compliance, and pregnancy outcomes in women with diet-controlled gestational diabetes mellitus (GDM)...
  24. ncbi request reprint University of Maryland School of Medicine: 200 years of "firsts"
    E Albert Reece
    University of Maryland, MD, USA
    Md Med 8:19-21. 2007
  25. pmc Role of HIF-1α in maternal hyperglycemia-induced embryonic vasculopathy
    Peixin Yang
    Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD, USA
    Am J Obstet Gynecol 204:332.e1-7. 2011
    ..Maternal diabetes adversely impacts embryonic vasculogenesis, which results in embryonic vasculopathy. The purpose of our study is to determine whether hypoxia inducible factor (HIF)-1α plays a role in diabetic embryonic vasculopathy...
  26. pmc SOD1 overexpression in vivo blocks hyperglycemia-induced specific PKC isoforms: substrate activation and consequent lipid peroxidation in diabetic embryopathy
    Xuezheng Li
    Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD, USA
    Am J Obstet Gynecol 205:84.e1-6. 2011
    ..We tested whether mitigating oxidative stress, using superoxide dismutase 1 (SOD1) transgenic (Tg) mice, would block hyperglycemia-induced specific protein kinase C (PKC) isoform activation and its downstream cascade...
  27. pmc Protein kinase C (PKC) delta suppresses keratinocyte proliferation by increasing p21(Cip1) level by a KLF4 transcription factor-dependent mechanism
    Yap Ching Chew
    Department of Biochemistry and Molecular Biology, University of Maryland School of Medicine, Baltimore, Maryland 21201, USA
    J Biol Chem 286:28772-82. 2011
    ..In addition, KLF4-siRNA inhibits PKCδ-dependent p21(Cip1) promoter activity. PKCδ increases KLF4 expression leading to enhanced KLF4 interaction with the GC-rich elements in the p21(Cip1) promoter to activate transcription...
  28. ncbi request reprint University of Maryland School of Medicine
    E Albert Reece
    University of Maryland School of Medicine, Baltimore, Maryland, USA
    Acad Med 82:1021. 2007
  29. pmc PKC-delta and -eta, MEKK-1, MEK-6, MEK-3, and p38-delta are essential mediators of the response of normal human epidermal keratinocytes to differentiating agents
    Gautam Adhikary
    Department of Biochemistry and Molecular Biology, University of Maryland School of Medicine, Baltimore, Maryland, USA
    J Invest Dermatol 130:2017-30. 2010
    ..These findings are informative as they suggest an essential role for selected PKC and MAPK cascade enzymes in mediating a range of end responses to a range of differentiation stimuli in keratinocytes...
  30. ncbi request reprint Involvement of c-Jun N-terminal kinases activation in diabetic embryopathy
    Peixin Yang
    Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, 655 W Baltimore Street, Baltimore, MD 21201, USA
    Biochem Biophys Res Commun 357:749-54. 2007
    ..JNK activation itself is sufficient to induce embryonic anomalies, and thus mimics the teratogenic effect of hyperglycemia...
  31. ncbi request reprint Global health and the University of Maryland School of Medicine: a legacy of excellence and commitment
    E Albert Reece
    University of Maryland School of Medicine, USA
    Md Med 8:15-6. 2007
  32. doi request reprint In the search for genes responsible for neural tube defects, glucose metabolism matters
    E Albert Reece
    Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, Maryland, USA
    Reprod Sci 15:7-8. 2008
  33. ncbi request reprint Synopsis of the North American Diabetes in Pregnancy Study Group Conference in Little Rock, Arkansas, May 2003
    E Albert Reece
    J Matern Fetal Neonatal Med 15:1-5. 2004
  34. ncbi request reprint Development of early-onset type 2 diabetes in the young: implications for child bearing
    Carol J Homko
    General Clinical Research Center, Temple University School of Medicine, 3401 N Broad Street, 4 West, Philadelphia, PA 19140, USA
    Curr Diab Rep 3:313-8. 2003
    ..This article reviews the existing data on pregnancies complicated by type 2 diabetes and the implications for management and prevention...
  35. ncbi request reprint [MAP kinase signal pathway in hyperglycemia-induced congenital neural tube defects]
    Bi Liang Chen
    Department of Obstetrics and Gynecology, Xijing Hospital, Fourth Military Medical University, Xi an 710 033, China
    Yi Chuan 26:615-9. 2004
    ..The supplementation of polyunsaturated fatty acid arachidonic acid, and antioxidant vitamin E rescued conceptuses from diabetic embryopathy by triggering a restoration of normal membrane signaling pathways...
  36. ncbi request reprint Aberrant patterns of cellular communication in diabetes-induced embryopathy in rats: II, apoptotic pathways
    E Albert Reece
    Department of Obstetrics and Gynecology, College of Medicine, University of Arkansas for Medical Sciences, Little Rock, AR 72205 7199, USA
    Am J Obstet Gynecol 192:967-72. 2005
    ..The objective was to test the hypothesis that hyperglycemia-induced injury of yolk sac cell membranes is associated with disruption of cellular apoptotic signaling pathways...
  37. ncbi request reprint Nicotine-induced embryonic malformations mediated by apoptosis from increasing intracellular calcium and oxidative stress
    Zhiyong Zhao
    Department of Obstetrics and Gynecology, The Arkansas Center for Birth Defects Research and Prevention, College of Medicine, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    Birth Defects Res B Dev Reprod Toxicol 74:383-91. 2005
    ..However, the role of nicotine in embryonic malformations has not been addressed, and the mechanisms by which nicotine affects embryonic development remain to be delineated...
  38. ncbi request reprint Experimental mechanisms of diabetic embryopathy and strategies for developing therapeutic interventions
    Zhiyong Zhao
    Department of Obstetrics and Gynecology, The Arkansas Center for Birth Defects Research and Prevention, Little Rock, Arkansas, USA
    J Soc Gynecol Investig 12:549-57. 2005
    ..The ultimate goal is to develop multi-nutrient dietary supplements to eliminate embryonic abnormalities induced by maternal diabetes...
  39. ncbi request reprint Characterization of differential gene expression profiles in diabetic embryopathy using DNA microarray analysis
    E Albert Reece
    Department of Obstetrics and Gynecology, The Arkansas Center for Birth Defects Research and Prevention, University of Arkansas for Medical Sciences, Little Rock, AR, USA
    Am J Obstet Gynecol 195:1075-80. 2006
    ....
  40. ncbi request reprint Dietary vitamin and lipid therapy rescues aberrant signaling and apoptosis and prevents hyperglycemia-induced diabetic embryopathy in rats
    E Albert Reece
    Department of Obstetrics and Gynecology, College of Medicine, University of Arkansas for Medical Sciences, Little Rock, AR, USA
    Am J Obstet Gynecol 194:580-5. 2006
    ....
  41. ncbi request reprint Periconceptional care of women with diabetes mellitus
    Gustavo Leguizamon
    Department of Obstetrics and Gynecology, Center for Medical Education and Clinical Research C E M I C University, Av Galván 4102, Buenos Aires, Argentina
    Obstet Gynecol Clin North Am 34:225-39, viii. 2007
    ..Because unplanned pregnancy is common among diabetic women, they should be counseled early for the importance of preconception care in the progression of this disease...
  42. doi request reprint Diabetes mellitus and birth defects
    Adolfo Correa
    Division of Birth Defects and Developmental Disabilities, Centers for Disease Control and Prevention, Atlanta, GA 30333, USA
    Am J Obstet Gynecol 199:237.e1-9. 2008
    ..The purpose of this study was to examine associations between diabetes mellitus and 39 birth defects...
  43. ncbi request reprint Meeting the challenges facing clinical research: solutions proposed by leaders of medical specialty and clinical research societies
    Horacio Murillo
    University of Arkansas for Medical Sciences, 4301 W Markham, Slot 550, Little Rock, AR 72205, USA
    Acad Med 81:107-12. 2006
    ..Collectively, the response and proposed strategies can serve as a roadmap to improve clinical research funding and training, evidence-based medical practice, and health care quality...
  44. ncbi request reprint Solutions and strategies from medical and nursing school leadership for the challenges facing the clinical research enterprise
    Horacio Murillo
    Department of Radiology, University of Texas Health Science Center San Antonio, San Antonio, Texas, USA
    Acad Med 82:625-32. 2007
    ..This study, conducted by the Institute of Medicine's Clinical Research Roundtable (CRR), sought to learn about the perceptions by medical and nursing school deans of these challenges or the efforts and strategies needed to address them...
  45. ncbi request reprint Insulins and oral hypoglycemic agents in pregnancy
    Carol J Homko
    General Clinical Research Center, Temple University School of Medicine, Philadelphia, PA 19140, USA
    J Matern Fetal Neonatal Med 19:679-86. 2006
    ..More intensive investigation regarding the safety and feasibility of oral agents in pregnancies complicated by type 2 diabetes is necessary...
  46. ncbi request reprint Correction for insulin-dependent diabetes in maternal serum alpha-fetoprotein testing has outlived its usefulness
    Mark I Evans
    Department of Obstetrics and Gynecology, MCP Hahnemann University, 245 N 15th Street, MS 495, Philadelphia, PA 19102, USA
    Am J Obstet Gynecol 187:1084-6. 2002
    ..With new laboratory methods and improved precision, we sought to re-evaluate the correctness of this approach...
  47. ncbi request reprint Central challenges facing the national clinical research enterprise
    Nancy S Sung
    Burroughs Wellcome Fund, PO Box 13901, 21 T W Alexander Dr, Research Triangle Park, NC 27709, USA
    JAMA 289:1278-87. 2003
    ....
  48. ncbi request reprint A consensus conference of the Diabetes in Pregnancy Study Group of North America
    E Albert Reece
    J Matern Fetal Neonatal Med 12:361. 2002
  49. ncbi request reprint Clinical research in the United States at a crossroads: proposal for a novel public-private partnership to establish a national clinical research enterprise
    William F Crowley
    Harvard Medical School, Massachusetts General Hospital, Boston 02114, USA
    JAMA 291:1120-6. 2004
    ..All participants in the US health care delivery system must come together to focus on system-wide improvements that will benefit the public...
  50. ncbi request reprint Is the intrapartum biophysical profile useful?
    So Young Kim
    Division of Maternal Fetal Medicine, Department of Obstetrics and Gynecology, Temple University School of Medicine, Philadelphia, Pennsylvania 19140, USA
    Obstet Gynecol 102:471-6. 2003
    ..To assess the role of biophysical profile (BPP) during normal labor. The secondary goal was to assess the effect of oxytocics, regional anesthesia, and ruptured membranes on fetal behavior during labor...
  51. ncbi request reprint Preventing diabetes in high-risk individuals
    E Albert Reece
    University of Arkansas College of Medicine, Little Rock, AR 72205 7199, USA
    Curr Diab Rep 6:289-90. 2006
  52. ncbi request reprint A clarion call for translational and collaborative research
    E Albert Reece
    Am J Obstet Gynecol 194:1507-9. 2006
  53. ncbi request reprint ANGELS and University of Arkansas for Medical Sciences paradigm for distant obstetrical care delivery
    Curtis Lowery
    Division of Maternal Fetal Medicine, Department of Obstetrics and Gynecology, College of Medicine, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    Am J Obstet Gynecol 196:534.e1-9. 2007
    ..The described ANGELS model encourages replication among other programs that wish to improve perinatal regionalization attempts in their service areas...

Research Grants25

  1. GENERAL CLINICAL RESEARCH CENTER
    E Reece; Fiscal Year: 2006
    ..During the upcoming grant period, we anticipate the planning and funding of the UM Clinical and Translational Science Center. ..
  2. GENERAL CLINICAL RESEARCH CENTER
    AUBREY MD HOUGH; Fiscal Year: 2006
    ..In this second funding cycle, the UAMS GCRC will continue expanding campus-wide clinical investigation. Support from all three sister institutions will continue to ensure GCRC success. ..
  3. GENERAL CLINICAL RESEARCH CENTER
    E Reece; Fiscal Year: 2007
    ..During the upcoming grant period, we anticipate the planning and funding of the UM Clinical and Translational Science Center. ..
  4. GENERAL CLINICAL RESEARCH CENTER
    E Reece; Fiscal Year: 2009
    ..During the upcoming grant period, we anticipate the planning and funding of the UM Clinical and Translational Science Center. ..
  5. Mechanisms of Diabetic Embryopathy and Molecular Pathways
    E Reece; Fiscal Year: 2009
    ..The results of the study will provide information that will serve as a basis for future development of therapeutic interventions to prevent maternal diabetes-associated birth defects. ..
  6. Mechanisms of Diabetic Embryopathy and Molecular Pathways
    E Albert Reece; Fiscal Year: 2010
    ..The results of the study will provide information that will serve as a basis for future development of therapeutic interventions to prevent maternal diabetes-associated birth defects. ..
  7. Cancer Center Renovation Grant
    Donald Wilson; Fiscal Year: 2004
    ....
  8. RES ANIMAL FACIL: ANIMAL MODEL
    Donald Wilson; Fiscal Year: 2002
    ..The proposed project, which will be co-financed by an $1,165,000 contribution from the State of Maryland, is therefore, considered instrumental for sustaining the stature of the MPRC in schizophrenia researc ..
  9. CONSTRUCTION OF RESEARCH ANIMAL FACILITY
    Donald Wilson; Fiscal Year: 2002
    ..The proposed project, which will be co-financed by contributions from the State of Maryland, therefore, considered instrumental for sustaining the stature of the MPRC in schizophrenia research. ..
  10. Web-Based IRB Data Management and Archiving System
    Donald Wilson; Fiscal Year: 2002
    ..abstract_text> ..
  11. GENERAL CLINICAL RESEARCH CENTER
    E Reece; Fiscal Year: 2003
    ..Biostatisticians will assist investigators in designing clinical studies and analyzing data. Combined, the resources provided through the GCRC will sustain and improve the clinical research efforts at UAMS and the VAMC. ..
  12. EXTRAMURAL RESEARCH FACILITIES IMPROVEMENT PROVEMENT
    Donald Wilson; Fiscal Year: 2003
    ....
  13. Human Subjects Research Electronic Tracking System
    Donald Wilson; Fiscal Year: 2003
    ..WVU has a different organizational structure, a different information technology network, and different human subjects research emphasis from the University of Maryland School of Medicine. ..
  14. Mechanisms of Diabetic Embryopathy and Molecular Pathways
    E Albert Reece; Fiscal Year: 2010
    ..The results of the study will provide information that will serve as a basis for future development of therapeutic interventions to prevent maternal diabetes-associated birth defects. ..