Research Topics
| G N RaoSummaryAffiliation: University of Texas Medical Branch Country: USA Publications
| Collaborators |
Detail Information
Publications
Differential regulation of p27kip1 levels and CDK activities by hypertrophic and hyperplastic agents in vascular smooth muscle cellsG N Rao
Division of Cardiology, University of Texas Medical Branch, Galveston 77555 1064, USA
Biochim Biophys Acta 1448:525-32. 1999..Together, these findings suggest that Ang II-induced hypertrophy may be due to its failure to activate cellular signaling events required for G1/S transition...
JunB forms the majority of the AP-1 complex and is a target for redox regulation by receptor tyrosine kinase and G protein-coupled receptor agonists in smooth muscle cellsG N Rao
Division of Cardiology, University of Texas Medical Branch, Galveston, Texas 77555, USA
J Biol Chem 274:6003-10. 1999....
A potential role for extracellular signal-regulated kinases in prostaglandin F2alpha-induced protein synthesis in smooth muscle cellsG N Rao
Division of Cardiology, University of Texas Medical Branch, Galveston, Texas 77555, USA
J Biol Chem 274:12925-32. 1999....
Oxidant stress stimulates phosphorylation of eIF4E without an effect on global protein synthesis in smooth muscle cells. Lack of evidence for a role of H202 in angiotensin II-induced hypertrophyG N Rao
Department of Anatomy and Neurosciences, University of Texas Medical Branch, Galveston, Texas 77555, USA
J Biol Chem 275:16993-9. 2000....
