J Piven

Summary

Affiliation: University of Iowa
Country: USA

Publications

  1. ncbi request reprint Psychiatric disorder and the broad autism phenotype: evidence from a family study of multiple-incidence autism families
    J Piven
    Department of Psychiatry, University of Iowa College of Medicine, Iowa City 52242 1000, USA
    Am J Psychiatry 156:557-63. 1999
  2. ncbi request reprint Examination of AVPR1a as an autism susceptibility gene
    T H Wassink
    Department of Psychiatry, University of Iowa College of Medicine, Iowa City, IA 52242, USA
    Mol Psychiatry 9:968-72. 2004
  3. ncbi request reprint Broader autism phenotype: evidence from a family history study of multiple-incidence autism families
    J Piven
    Department of Psychiatry, University of Iowa College of Medicine, Iowa City, USA
    Am J Psychiatry 154:185-90. 1997
  4. ncbi request reprint Chromosomal abnormalities in a clinic sample of individuals with autistic disorder
    T H Wassink
    Department of Psychiatry, University of Iowa College of Medicine, Iowa City, USA
    Psychiatr Genet 11:57-63. 2001
  5. ncbi request reprint Evidence supporting WNT2 as an autism susceptibility gene
    T H Wassink
    Department of Psychiatry, University of Iowa College of Medicine, Iowa City, Iowa 52242, USA
    Am J Med Genet 105:406-13. 2001
  6. ncbi request reprint The biological basis of autism
    J Piven
    Department of Psychiatry, University of Iowa College of Medicine, Psychiatry Research, Medical Education Building, Room 1 293, Iowa City, Iowa 52242, USA
    Curr Opin Neurobiol 7:708-12. 1997
  7. ncbi request reprint Incorporating language phenotypes strengthens evidence of linkage to autism
    Y Bradford
    Department of Molecular Physiology and Biophysics, Vanderbilt University, Nashville, Tennessee, USA
    Am J Med Genet 105:539-47. 2001
  8. ncbi request reprint An autosomal genomic screen for autism. Collaborative linkage study of autism
    S Barrett
    The Johns Hopkins University School of Medicine, Baltimore, Maryland
    Am J Med Genet 88:609-15. 1999
  9. ncbi request reprint Linkage disequilibrium at the Angelman syndrome gene UBE3A in autism families
    E L Nurmi
    Program in Human Genetics, Department of Molecular Physiology and Biophysics, Vanderbilt University Medical Center, Nashville, Tennessee 37232, USA
    Genomics 77:105-13. 2001
  10. ncbi request reprint The broad autism phenotype: a complementary strategy for molecular genetic studies of autism
    J Piven
    North Carolina Mental Retardation and Developmental Disabilities Research Center, University of North Carolina at Chapel Hill, 27599-7250, USA
    Am J Med Genet 105:34-5. 2001

Collaborators

  • V J Vieland
  • S E Folstein
  • J Huang
  • Thomas Wassink
  • Wei Huang
  • Jacqueline N Crawley
  • SHERYL MOY
  • K Wang
  • Y Chen
  • Y Bradford
  • E L Nurmi
  • S L Santangelo
  • J L Haines
  • V Sheffield
  • M B Gardiner
  • S Barrett
  • B Arnone
  • J Hall
  • M Gardiner
  • H B Hutcheson
  • H Hutcheson
  • S Santangelo
  • V C Sheffield
  • T Braun
  • M A Pericak-Vance
  • T Cassavant
  • J S Sutcliffe
  • J R Gilbert
  • S A Copeland-Yates
  • J Haines
  • R C Michaelis
  • S Slager
  • J C Beck
  • T L Casavant
  • E Bisson
  • T A Braun
  • R Bernier
  • D Y Nishimura
  • D Childress
  • K Hopkins
  • C Searby
  • J Singleton
  • S Gilman
  • J Purdy
  • P Palmer
  • N H Meyer
  • R Landa
  • J A Mullane
  • M Garcia

Detail Information

Publications11

  1. ncbi request reprint Psychiatric disorder and the broad autism phenotype: evidence from a family study of multiple-incidence autism families
    J Piven
    Department of Psychiatry, University of Iowa College of Medicine, Iowa City 52242 1000, USA
    Am J Psychiatry 156:557-63. 1999
    ..In this study the authors examined the rate of axis I psychiatric disorders in nonautistic relatives from multiple-incidence autism families and the possible relationship of these disorders to the broad autism phenotype...
  2. ncbi request reprint Examination of AVPR1a as an autism susceptibility gene
    T H Wassink
    Department of Psychiatry, University of Iowa College of Medicine, Iowa City, IA 52242, USA
    Mol Psychiatry 9:968-72. 2004
    ..Given the emerging biological, animal model, and now genetic data, AVPR1a and genes in the AVP system remain strong candidates for involvement in autism susceptibility and deserve continued scrutiny...
  3. ncbi request reprint Broader autism phenotype: evidence from a family history study of multiple-incidence autism families
    J Piven
    Department of Psychiatry, University of Iowa College of Medicine, Iowa City, USA
    Am J Psychiatry 154:185-90. 1997
    ..The objective of this study was to examine behaviors that may define this broader phenotype in relatives ascertained through two autistic siblings...
  4. ncbi request reprint Chromosomal abnormalities in a clinic sample of individuals with autistic disorder
    T H Wassink
    Department of Psychiatry, University of Iowa College of Medicine, Iowa City, USA
    Psychiatr Genet 11:57-63. 2001
    ..These data support the contribution of chromosomal abnormalities to a small but significant number of cases of autism, and highlight the involvement of chromosome 15 and the sex chromosomes...
  5. ncbi request reprint Evidence supporting WNT2 as an autism susceptibility gene
    T H Wassink
    Department of Psychiatry, University of Iowa College of Medicine, Iowa City, Iowa 52242, USA
    Am J Med Genet 105:406-13. 2001
    ....
  6. ncbi request reprint The biological basis of autism
    J Piven
    Department of Psychiatry, University of Iowa College of Medicine, Psychiatry Research, Medical Education Building, Room 1 293, Iowa City, Iowa 52242, USA
    Curr Opin Neurobiol 7:708-12. 1997
    ..The results of these studies are likely to have a significant impact on our understanding of the pathogenesis of autism...
  7. ncbi request reprint Incorporating language phenotypes strengthens evidence of linkage to autism
    Y Bradford
    Department of Molecular Physiology and Biophysics, Vanderbilt University, Nashville, Tennessee, USA
    Am J Med Genet 105:539-47. 2001
    ..Although based on our data, we were not able to distinguish between epistasis or heterogeneity models, we tentatively concluded that there may be more than one autism susceptibility locus related to language development...
  8. ncbi request reprint An autosomal genomic screen for autism. Collaborative linkage study of autism
    S Barrett
    The Johns Hopkins University School of Medicine, Baltimore, Maryland
    Am J Med Genet 88:609-15. 1999
    ..By comparing results across a number of studies, we expect to be able to narrow our search for autism susceptibility genes to a small number of genomic regions. Am. J. Med. Genet. (Neuropsychiatr. Genet.) 88:609-615, 1999...
  9. ncbi request reprint Linkage disequilibrium at the Angelman syndrome gene UBE3A in autism families
    E L Nurmi
    Program in Human Genetics, Department of Molecular Physiology and Biophysics, Vanderbilt University Medical Center, Nashville, Tennessee 37232, USA
    Genomics 77:105-13. 2001
    ....
  10. ncbi request reprint The broad autism phenotype: a complementary strategy for molecular genetic studies of autism
    J Piven
    North Carolina Mental Retardation and Developmental Disabilities Research Center, University of North Carolina at Chapel Hill, 27599-7250, USA
    Am J Med Genet 105:34-5. 2001
    ..Inclusion of information on the broad autism phenotype in relatives, in linkage studies of autism, may provide a potentially important, complementary approach for detecting the genes causing this condition...
  11. ncbi request reprint Sociability and preference for social novelty in five inbred strains: an approach to assess autistic-like behavior in mice
    S S Moy
    North Carolina STAART Center for Autism Research, Neurodevelopmental Disorders Research Center, University of North Carolina School of Medicine, Chapel Hill, NC 27599, USA
    Genes Brain Behav 3:287-302. 2004
    ..This new standardized procedure to quantitate sociability and preference for social novelty in mice provides a method to assess tendencies for social avoidance in mouse models of autism...