Tristano Pancani

Summary

Affiliation: University of Kentucky
Country: USA

Publications

  1. pmc Imaging of a glucose analog, calcium and NADH in neurons and astrocytes: dynamic responses to depolarization and sensitivity to pioglitazone
    Tristano Pancani
    Department of Molecular and Biomedical Pharmacology, University of Kentucky Medical Center, Lexington, United States
    Cell Calcium 50:548-58. 2011
  2. pmc Long-term pioglitazone treatment improves learning and attenuates pathological markers in a mouse model of Alzheimer's disease
    James L Searcy
    Department of Molecular and Biomedical Pharmacology, University of Kentucky Medical Center, Lexington, KY 40536 0084, USA
    J Alzheimers Dis 30:943-61. 2012
  3. pmc Effect of high-fat diet on metabolic indices, cognition, and neuronal physiology in aging F344 rats
    Tristano Pancani
    Department of Molecular and Biomedical Pharmacology, University of Kentucky Medical Center, Lexington, KY 40536 0084, USA
    Neurobiol Aging 34:1977-87. 2013
  4. pmc Effects of long-term pioglitazone treatment on peripheral and central markers of aging
    Eric M Blalock
    Department of Molecular and Biomedical Pharmacology, University of Kentucky Medical Center, Lexington, Kentucky, United States of America
    PLoS ONE 5:e10405. 2010
  5. doi request reprint Distinct modulation of voltage-gated and ligand-gated Ca2+ currents by PPAR-gamma agonists in cultured hippocampal neurons
    Tristano Pancani
    Department of Molecular and Biomedical Pharmacology, University of Kentucky Medical Center, 800 Rose Street, MS 310, Lexington, Kentucky 40536 0298, USA
    J Neurochem 109:1800-11. 2009
  6. pmc Reduction in neuronal L-type calcium channel activity in a double knock-in mouse model of Alzheimer's disease
    Olivier Thibault
    Department of Molecular and Biomedical Pharmacology, University of Kentucky College of Medicine, Lexington, KY 40536, USA
    Biochim Biophys Acta 1822:546-9. 2012

Collaborators

  • Inga Kadish
  • John C Gant
  • Olivier Thibault
  • James L Searcy
  • Eric M Blalock
  • Philip W Landfield
  • Jelena Popovic
  • Nada M Porter
  • Jeremiah T Phelps
  • Kuey Chu Chen
  • Katie L Anderson
  • Christopher M Norris
  • Michael P Murphy
  • Tina L Beckett
  • Don A Cohen
  • Margarita G Avdiushko

Detail Information

Publications6

  1. pmc Imaging of a glucose analog, calcium and NADH in neurons and astrocytes: dynamic responses to depolarization and sensitivity to pioglitazone
    Tristano Pancani
    Department of Molecular and Biomedical Pharmacology, University of Kentucky Medical Center, Lexington, United States
    Cell Calcium 50:548-58. 2011
    ....
  2. pmc Long-term pioglitazone treatment improves learning and attenuates pathological markers in a mouse model of Alzheimer's disease
    James L Searcy
    Department of Molecular and Biomedical Pharmacology, University of Kentucky Medical Center, Lexington, KY 40536 0084, USA
    J Alzheimers Dis 30:943-61. 2012
    ..Taken together, these results confirm prior animal studies showing that TZDs can ameliorate cognitive deficits associated with AD-related pathology, but also extend these findings by pointing to novel molecular targets in the brain...
  3. pmc Effect of high-fat diet on metabolic indices, cognition, and neuronal physiology in aging F344 rats
    Tristano Pancani
    Department of Molecular and Biomedical Pharmacology, University of Kentucky Medical Center, Lexington, KY 40536 0084, USA
    Neurobiol Aging 34:1977-87. 2013
    ..Interestingly, although peripheral glucose regulation was relatively insensitive to HFD, the brain appeared to show greater sensitivity to HFD in F344 rats...
  4. pmc Effects of long-term pioglitazone treatment on peripheral and central markers of aging
    Eric M Blalock
    Department of Molecular and Biomedical Pharmacology, University of Kentucky Medical Center, Lexington, Kentucky, United States of America
    PLoS ONE 5:e10405. 2010
    ..Because aging is a common risk factor for both AD and T2DM, we investigated whether the TZD, pioglitazone could alter brain aging under non-pathological conditions...
  5. doi request reprint Distinct modulation of voltage-gated and ligand-gated Ca2+ currents by PPAR-gamma agonists in cultured hippocampal neurons
    Tristano Pancani
    Department of Molecular and Biomedical Pharmacology, University of Kentucky Medical Center, 800 Rose Street, MS 310, Lexington, Kentucky 40536 0298, USA
    J Neurochem 109:1800-11. 2009
    ....
  6. pmc Reduction in neuronal L-type calcium channel activity in a double knock-in mouse model of Alzheimer's disease
    Olivier Thibault
    Department of Molecular and Biomedical Pharmacology, University of Kentucky College of Medicine, Lexington, KY 40536, USA
    Biochim Biophys Acta 1822:546-9. 2012
    ..The results suggest that mechanisms for Ca(2+) dysregulation can differ substantially between animal models of normal aging and models of pathological aging...