Research Topics
Species | Vasantha PadmanabhanSummaryAffiliation: University of Michigan Country: USA Publications
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Detail Information
Publications
Developmental programming: gestational bisphenol-a treatment alters trajectory of fetal ovarian gene expressionAlmudena Veiga-Lopez
PhD, Department of Pediatrics and Reproductive Sciences Program, University of Michigan, 300 North Ingalls Building, Room 1138, Ann Arbor, Michigan 48109 5404
Endocrinology 154:1873-84. 2013....- Developmental programming: impact of prenatal testosterone excess on insulin sensitivity, adiposity, and free Fatty Acid profile in postpubertal female sheepA Veiga-Lopez
PhD, Department of Pediatrics and Reproductive Sciences Program, University of Michigan, 300 North Ingalls Building, Room 1137, Ann Arbor, Michigan 48109 0404
Endocrinology 154:1731-42. 2013.... 
Delivery type not associated with global methylation at birthShama Virani
Department of Pediatrics, The University of Michigan, Ann Arbor, MI, USA
Clin Epigenetics 4:8. 2012..abstract:..- Contributions of androgen and estrogen to fetal programming of ovarian dysfunctionDavid H Abbott
National Primate Research Center, Department of Ob Gyn, Endocrinology Reproductive Physiology Training Program, University of Wisconsin, Madison, WI 53715, USA
Reprod Biol Endocrinol 4:17. 2006..Understanding the quality of the fetal female sex steroid hormone environment is thus becoming increasingly important in improving our knowledge of mechanisms underlying a variety of female reproductive pathologies... - Prenatal exposure to excess testosterone modifies the developmental trajectory of the insulin-like growth factor system in female sheepErica J Crespi
Reproductive Sciences Program, 300 N Ingalls Bldg, Rm 1109 SW, Ann Arbor, MI 48109 0404, USA
J Physiol 572:119-30. 2006.... - Developmental reprogramming of reproductive and metabolic dysfunction in sheep: native steroids vs. environmental steroid receptor modulatorsV Padmanabhan
Department of Pediatrics and the Reproductive Sciences Program, The University of Michigan, Ann Arbor, MI48109, USA
Int J Androl 33:394-404. 2010..These findings provide evidence in support of developmental origin of adult reproductive and metabolic diseases and highlight the risk posed by exposure to environmental endocrine disrupting chemicals... - Maternal bisphenol-A levels at delivery: a looming problem?V Padmanabhan
Department of Pediatrics, University of Michigan, Ann Arbor, MI 48109 0404, USA
J Perinatol 28:258-63. 2008..The objective was to determine whether bisphenol-A (BPA) is found in maternal circulation of pregnant women in the US population and is related to gestational length and birth weight... - Developmental programming: impact of prenatal testosterone excess and postnatal weight gain on insulin sensitivity index and transfer of traits to offspring of overweight femalesV Padmanabhan
Department of Pediatrics and Reproductive Sciences Program, University of Michigan, 300 North Ingalls Building, Ann Arbor, Michigan 48109 0404, USA
Endocrinology 151:595-605. 2010..The findings are of relevance in the context of developmental programming of insulin resistance by prenatal steroids and excess weight gain... - Developmental origin of reproductive and metabolic dysfunctions: androgenic versus estrogenic reprogrammingVasantha Padmanabhan
Department of Pediatrics, The University of Michigan, Ann Arbor, Michigan, USA
Semin Reprod Med 29:173-86. 2011.... - Developmental programming: gestational testosterone treatment alters fetal ovarian gene expressionLacey J Luense
Department of Molecular and Integrative Physiology, University of Kansas Medical Center, Kansas City, Kansas 66160, USA
Endocrinology 152:4974-83. 2011.... 
Prenatal testosterone excess programs reproductive and metabolic dysfunction in the femaleVasantha Padmanabhan
Department of Pediatrics, University of Michigan, Ann Arbor, MI 48109 0404, USA
Mol Cell Endocrinol 246:165-74. 2006....- Neuroendocrine control of follicle-stimulating hormone (FSH) secretion. I. Direct evidence for separate episodic and basal components of FSH secretionV Padmanabhan
Reproductive Sciences Program, University of Michigan, Ann Arbor 48109 0404, USA
Endocrinology 138:424-32. 1997.... - Neuroendocrine control of follicle-stimulating hormone (FSH) secretion: II. Is follistatin-induced suppression of FSH secretion mediated via changes in activin availability and does it involve changes in gonadotropin-releasing hormone secretion?Vasantha Padmanabhan
Department of Pediatrics, Reproductive Sciences Program, University of Michigan, Ann Arbor, MI 48109 0404, USA
Biol Reprod 66:1395-402. 2002..The slower clearance of total follistatin and the lack of follistatin effects on GnRH secretion suggest that changes in activin bioavailability dictate the level of pituitary FSH secretion and that this is a pituitary-specific effect... - Neuroendocrine control of follicle-stimulating hormone (FSH) secretion: III. Is there a gonadotropin-releasing hormone-independent component of episodic FSH secretion in ovariectomized and luteal phase ewes?Vasantha Padmanabhan
Department of Pediatrics, The University of Michigan, Ann Arbor, Michigan 48109, USA
Endocrinology 144:1380-92. 2003..These studies provide the first direct evidence of the existence of an acute GnRH I-independent component of episodic FSH secretion... - Hypothalamic, pituitary and gonadal regulation of FSHV Padmanabhan
Department of Pediatrics, and the Reproductive Sciences Program, University of Michigan, Ann Arbor, MI 48109, USA
Reprod Suppl 59:67-82. 2002.... - Neuroendocrine vs. paracrine control of follicle-stimulating hormoneV Padmanabhan
Department of Pediatrics and the Reproductive Sciences Program, University of Michigan, Ann Arbor, MI 48109 0404, USA
Arch Med Res 32:533-43. 2001..This review focuses on the recent advances made in the neuroendocrine and paracrine regulation of FSH synthesis/secretion/heterogeneity and pinpoints areas of gaps in our knowledge... - Is there an FSH-releasing factor?V Padmanabhan
Department of Pediatrics and the Reproductive Sciences Program, University of Michigan, Ann Arbor, MI 48109, USA
Reproduction 121:21-30. 2001.... - Developmental programming: impact of prenatal testosterone excess on pre- and postnatal gonadotropin regulation in sheepMohan Manikkam
Department of Pediatrics, the Reproductive Sciences Program, and the Center for Statistical Consultation and Research, University of Michigan, Ann Arbor, Michigan 48109 0404, USA
Biol Reprod 78:648-60. 2008.... - Fetal programming: prenatal testosterone excess leads to fetal growth retardation and postnatal catch-up growth in sheepMohan Manikkam
Department of Pediatrics, University of Michigan, 300 North Ingalls Building, Room 1109 SW, Ann Arbor, MI 48109-0404, USA
Endocrinology 145:790-8. 2004..These findings support the concept that fetal growth retardation and postnatal catch-up growth, early markers of future adult diseases, can also be programmed by prenatal exposure to excess sex steroids... - Neuroendocrine control of FSH secretion: IV. Hypothalamic control of pituitary FSH-regulatory proteins and their relationship to changes in FSH synthesis and secretionTejinder P Sharma
Department of Pediatrics, University of Michigan, Ann Arbor, Michigan 48109 0404, USA
Biol Reprod 86:171. 2012..Perhaps the effects of GnRH pulse frequency on FSH is due to changes in the balance of free versus bound amounts of these FSH regulatory proteins or to the involvement of other regulators not monitored in this study... - Developmental programming: exogenous gonadotropin treatment rescues ovulatory function but does not completely normalize ovarian function in sheep treated prenatally with testosteroneTeresa L Steckler
Departments of Pediatrics, University of Michigan, Ann Arbor, Michigan 48109 0404, USA
Biol Reprod 79:686-95. 2008.... - Developmental programming: excess weight gain amplifies the effects of prenatal testosterone excess on reproductive cyclicity--implication for polycystic ovary syndromeTeresa L Steckler
Department of Pediatrics, University of Michigan, Ann Arbor, Michigan 48109 0404, USA
Endocrinology 150:1456-65. 2009..These findings agree with the increased prevalence of anovulation observed in obese women with polycystic ovary syndrome and indicate that excess postnatal weight gain amplifies reproductive disruptions caused by prenatal T excess... - Developmental programming: postnatal steroids complete prenatal steroid actions to differentially organize the GnRH surge mechanism and reproductive behavior in female sheepLeslie M Jackson
Reproductive Sciences Program, University of Michigan, Ann Arbor, Michigan, USA
Endocrinology 154:1612-23. 2013..These findings support the hypothesis that the feedback controls of the GnRH surge are sensitive to programming by prenatal and postnatal steroids in a precocial species... - Developmental programming: differential effects of prenatal exposure to bisphenol-A or methoxychlor on reproductive functionMozhgan Savabieasfahani
Department of Pediatrics, 300 North Ingalls Building, Room 1109 SW, Ann Arbor, University of Michigan 48109-0404, USA
Endocrinology 147:5956-66. 2006..05) in BPA sheep. These findings suggest that prenatal BPA and MXC exposure have long-term differential effects on a variety of reproductive endocrine parameters that could impact fertility... - Developmental programming: contribution of prenatal androgen and estrogen to estradiol feedback systems and periovulatory hormonal dynamics in sheepAlmudena Veiga-Lopez
Department of Pediatrics, University of Michigan, Ann Arbor, Michigan 48109 0404, USA
Biol Reprod 80:718-25. 2009..The findings may be of translational relevance since daughters of mothers with hyperandrogenism are at risk of increased exposure to androgens... - Developmental programming: prenatal testosterone excess disrupts anti-Müllerian hormone expression in preantral and antral folliclesAlmudena Veiga-Lopez
Department of Pediatrics, University of Michigan, Ann Arbor, Michigan 48109 5404, USA
Fertil Steril 97:748-56. 2012..To investigate the impact of prenatal T excess on the expression of key ovarian regulators implicated in follicular recruitment and persistence using a large animal model of polycystic ovarian syndrome (PCOS)... - Developmental programming: insulin sensitizer treatment improves reproductive function in prenatal testosterone-treated female sheepAlmudena Veiga-Lopez
Department of Pediatrics and Reproductive Sciences Program, University of Michigan, Ann Arbor, Michigan 48109 0404, USA
Endocrinology 151:4007-17. 2010..These findings provide evidence that insulin sensitizer treatment prevents further deterioration of the reproductive axis in prenatal T-treated sheep, a finding of translational relevance to women with polycystic ovary syndrome... - Fetal programming: testosterone exposure of the female sheep during midgestation disrupts the dynamics of its adult gonadotropin secretion during the periovulatory periodMozhgan Savabieasfahani
Department of Pediatrics, University of Michigan, Ann Arbor, Michigan 48109, USA
Biol Reprod 72:221-9. 2005.... - Developmental programming: follicular persistence in prenatal testosterone-treated sheep is not programmed by androgenic actions of testosteroneTeresa Steckler
Department of Pediatrics, University of Michigan, 300 North Ingalls Building, Room 1109, Ann Arbor, Michigan 48109 0404, USA
Endocrinology 148:3532-40. 2007.... - Fetal programming: prenatal testosterone treatment leads to follicular persistence/luteal defects; partial restoration of ovarian function by cyclic progesterone treatmentMohan Manikkam
Department of Pediatrics, University of Michigan, Ann Arbor, 48109-0404, USA
Endocrinology 147:1997-2007. 2006..Cyclic P treatment was able to only partially restore follicular dynamics, but this may be related to the low replacement concentrations of P achieved... - Fetal programming: prenatal androgen disrupts positive feedback actions of estradiol but does not affect timing of puberty in female sheepTejinder Pal Sharma
Department of Pediatrics, University of Michigan, Ann Arbor, MI 48109, USA
Biol Reprod 66:924-33. 2002..The present findings indicate that disruption of the surge system can account for the fertility problems that occur during adulthood in prenatally androgenized sheep... - Developmental programming: prenatal and postnatal contribution of androgens and insulin in the reprogramming of estradiol positive feedback disruptions in prenatal testosterone-treated sheepBachir Abi Salloum
Department of Pediatrics and Reproductive Sciences Program, University of Michigan, Ann Arbor, Michigan 48109, USA
Endocrinology 153:2813-22. 2012.... - Diurnal changes in FSH-regulatory peptides and their relationship to gonadotrophins in pubertal girlsCarol M Foster
Department of Pediatrics Endocrinology and Reproductive Sciences Program, University of Michigan, Ann Arbor, MI 48109 0718, USA
Hum Reprod 20:543-8. 2005..We hypothesized that day/night variations in FSH serum concentrations would be associated with diurnal variation in FSH-regulatory peptides... - GnRH agonist stimulation of the pituitary-gonadal axis in children: age and sex differences in circulating inhibin-B and activin-ADaniel D Elsholz
Department of Pediatrics, Division of Endocrinology, University of Michigan, Ann Arbor, MI 48109-0646, USA
Hum Reprod 19:2748-58. 2004..CONCLUSIONS: An inhibin-B-FSH feedback loop exists prior to the onset of puberty in girls older than 5 years. Sex differences in activin-A and inhibin-B concentrations may be responsible for sex differences in serum FSH concentrations... - Prenatal testosterone and dihydrotestosterone exposure disrupts ovine testicular developmentCharles L Bormann
Department of Obstetrics and Gynecology, University of Michigan, Ann Arbor, Michigan 48109, USA
Reproduction 142:167-73. 2011.... - Developmental programming: impact of excess prenatal testosterone on intrauterine fetal endocrine milieu and growth in sheepAlmudena Veiga-Lopez
Department of Pediatrics, University of Michigan, Ann Arbor, Michigan 48109 5404, USA
Biol Reprod 84:87-96. 2011.... - Fetal programming: excess prenatal testosterone reduces postnatal luteinizing hormone, but not follicle-stimulating hormone responsiveness, to estradiol negative feedback in the femaleHirendra N Sarma
Department of Pediatrics, University of Michigan, Ann Arbor, Michigan 48109, USA
Endocrinology 146:4281-91. 2005..Collectively, these findings support the hypothesis that prenatal exposure to excess T decreases postnatal responsiveness to E2 inhibitory feedback of LH/GnRH secretion to contribute to the development of hypergonadotropism... - Long-term exposure of female sheep to physiologic concentrations of estradiol: effects on the onset and maintenance of reproductive function, pregnancy, and social development in female offspringKarl D Malcolm
Reproductive Sciences Program, University of Michigan, Ann Arbor, Michigan 48109-0404, USA
Biol Reprod 75:844-52. 2006..Despite these disruptive steroid actions, ovarian cyclicity and fertility are not invariably compromised, pointing to an impressive resiliency of the reproductive axis to insult by exogenous estrogenic compounds... - Sex differences in FSH-regulatory peptides in pubertal age boys and girls and effects of sex steroid treatmentCarol M Foster
Department of Pediatrics Endocrinology and the Reproductive Sciences Program, University of Michigan, Ann Arbor, MI 48109, USA
Hum Reprod 19:1668-76. 2004..FSH concentrations are higher in girls than in boys before puberty. We hypothesized that steroid-mediated changes in FSH-regulatory proteins underlie the sex differences in FSH secretion and pubertal timing... - Juvenile rank can predict male-typical adult mating behavior in female sheep treated prenatally with testosteroneEila K Roberts
Department of Psychology, University of Michigan, Ann Arbor, Michigan 48109 1043, USA
Biol Reprod 80:737-42. 2009..We conclude that the effect of prenatal testosterone exposure on adult male-specific but not female-specific mating behavior is modulated by juvenile social experiences... - Programming of GnRH feedback controls timing puberty and adult reproductive activityDouglas L Foster
Reproductive Sciences Program, Departments of Obstetrics and Gynecology, Pediatrics, and Physiology, University of Michigan, Ann Arbor, MI 48109 0404, United States
Mol Cell Endocrinol 254:109-19. 2006.... - Prenatal testosterone treatment alters LH and testosterone responsiveness to GnRH agonist in male sheepSergio E Recabarren
Reproductive Sciences Program, Departments of Obstetrics and Gynecology, Pediatrics and Molecular and Integrative Physiology, University of Michigan, Ann Arbor, MI 48109, USA
Biol Res 40:329-38. 2007..05). Our findings suggest that prenatal T treatment advances the developmental trajectory of gonadal responsiveness to GnRH in male offspring... - Antimüllerian hormone levels are independently related to ovarian hyperandrogenism and polycystic ovariesRobert L Rosenfield
Department of Pediatrics, Pritzker School of Medicine, University of Chicago, Chicago, Illinois, USA
Fertil Steril 98:242-9. 2012..To determine the relationship of antimüllerian hormone (AMH) levels to polycystic ovaries and ovarian androgenic function... - Acidic mix of FSH isoforms are better facilitators of ovarian follicular maturation and E2 production than the less acidicChristine R West
Department of Pediatrics, University of Michigan, Ann Arbor, MI 48109, USA
Endocrinology 143:107-16. 2002..The C-FSH ewes had more estrogenic follicles than the GnRH and N-FSH groups (P < 0.05). Our findings show that C-FSH clears more slowly than N-FSH, and C-FSH is a better facilitator of follicular development and maturation than N-FSH... - Differential effects of aging on activin A and its binding protein, follistatin, across the menopause transitionNancy E Reame
School of Nursing, University of Michigan, Ann Arbor, Michigan, USA
Fertil Steril 88:1003-5. 2007.... - Fetal programming: prenatal testosterone treatment causes intrauterine growth retardation, reduces ovarian reserve and increases ovarian follicular recruitmentTeresa Steckler
Department of Pediatrics, University of Michigan, Ann Arbor, Michigan 48109-0404, USA
Endocrinology 146:3185-93. 2005..The extent to which metabolic changes associated with intrauterine growth retardation contribute toward altered trajectory of ovarian folliculogenesis remains to be determined... - Insight into the neuroendocrine site and cellular mechanism by which cortisol suppresses pituitary responsiveness to gonadotropin-releasing hormoneKellie M Breen
Reproductive Sciences Program, Department of Molecular and Integrative Physiology, University of Michigan, Ann Arbor, MI 48109, USA
Endocrinology 149:767-73. 2008..Collectively, these observations provide evidence that cortisol acts via the type II GR within the pituitary gland to elicit a rapid decrease in responsiveness to GnRH, independent of changes in expression of the GnRH receptor... - Developmental programming: Impact of prenatal exposure to bisphenol-A and methoxychlor on steroid feedbacks in sheepBachir Abi Salloum
Department of Pediatrics, University of Michigan, Ann Arbor, MI 48109, USA
Toxicol Appl Pharmacol 268:300-8. 2013..These findings suggest that the postpubertal reproductive defects seen in these animals may have stemmed from ovarian defects and the steroidal signals emanating from them... - Differential effects of prenatal testosterone timing and duration on phenotypic and behavioral masculinization and defeminization of female sheepEila K Roberts
Department of Psychology, University of Michigan, Ann Arbor, Michigan 48109 1043, USA
Biol Reprod 79:43-50. 2008.... - Developmental programming: differential effects of prenatal testosterone excess on insulin target tissuesShadia E Nada
Department of Pediatrics, University of Michigan, Ann Arbor, Michigan 48109 5404, USA
Endocrinology 151:5165-73. 2010..These changes are consistent with the hypothesis that prenatal T excess disrupts the insulin sensitivity of peripheral tissues, with liver and muscle being insulin resistant and adipose tissue insulin sensitive... - Insulin resistance influences central opioid activity in polycystic ovary syndromeAlison Berent-Spillson
Molecular and Behavioral Neuroscience Institute, University of Michigan, Ann Arbor, Michigan, USA
Fertil Steril 95:2494-8. 2011.... - The role of endogenous growth hormone-releasing hormone in acromegalyEleni V Dimaraki
Department of Internal Medicine, Division of Metabolism, Endocrinology and Diabetes, University of Michigan, Ann Arbor, Michigan 48109, USA
J Clin Endocrinol Metab 91:2185-90. 2006..Some indirect evidence suggests hypothalamic control of GH secretion in acromegaly... - Developmental programming: deficits in reproductive hormone dynamics and ovulatory outcomes in prenatal, testosterone-treated sheepA Veiga-Lopez
Department of Pediatrics, and the Reproductive Sciences Program, University of Michigan, Ann Arbor, Michigan 48109 0404, USA
Biol Reprod 78:636-47. 2008.... - Prepubertal administration of estradiol valerate disrupts cyclicity and leads to cystic ovarian morphology during adult life in the rat: role of sympathetic innervationAlzira Rosa-E-Silva
Department of Physiology, Universidad de Sao Paulo, Brazil
Endocrinology 144:4289-97. 2003..These findings provide evidence that EV exposure during juvenile life leads to long-lasting deleterious reproductive consequences via activation of the sympathetic ovarian nerve... - Postnatal developmental consequences of altered insulin sensitivity in female sheep treated prenatally with testosteroneSergio E Recabarren
Laboratory of Animal Physiology and Endocrinology, Faculty of Veterinary Medicine, Univ of Concepción, Chillan, Chile
Am J Physiol Endocrinol Metab 289:E801-6. 2005..Our findings suggest that prenatal T treatment leads to offspring with reduced birth weight and impaired insulin sensitivity in early postnatal life... - Endocrine antecedents of polycystic ovary syndrome in fetal and infant prenatally androgenized female rhesus monkeysDavid H Abbott
Wisconsin National Primate Research Center, University of Wisconsin, Madison, Wisconsin 53715, USA
Biol Reprod 79:154-63. 2008..Thus, in this nonhuman primate model, differential programming of the fetal hypothalamo-pituitary unit with concomitant hyperandrogenism provides evidence to suggest developmental origins of LH and androgen excess in adulthood... - Developmental origin of health and diseasePeter W Nathanielsz
J Physiol 572:3-4. 2006 - Prenatal programming of reproductive neuroendocrine function: fetal androgen exposure produces progressive disruption of reproductive cycles in sheepRachel A Birch
Laboratory of Neuroendocrinology, Department of Neurobiology, The Babraham Institute, Cambridge CB2 4AT, United Kingdom
Endocrinology 144:1426-34. 2003..Our findings reveal that exposure to androgens in utero causes a progressive loss of cyclic function in adulthood... - Prenatal exposure of the ovine fetus to androgens sexually differentiates the steroid feedback mechanisms that control gonadotropin releasing hormone secretion and disrupts ovarian cyclesJane E Robinson
Laboratory of Neuroendocrinology, Babraham Institute, Babraham, Cambridge, United Kingdom
Arch Sex Behav 31:35-41. 2002..These data indicate that a relatively short exposure to male hormones during in utero life permanently alters the neural mechanisms that control reproduction and leads progressively to a state of infertility... - In utero programming of sexually differentiated gonadotrophin releasing hormone (GnRH) secretionJane E Robinson
Laboratory of Neuroendocrinology, The Babraham Institute, Cambridge CB2 4AT, UK
Domest Anim Endocrinol 23:43-52. 2002..The consequences of in utero androgen exposure are reflected in a progressive and dramatic impairment of fertility in the ovary-intact ewe... - Assessment of ovarian reserve by using the follicle-stimulating hormone isoform distribution pattern to predict the outcome of in vitro fertilizationGerard S Letterie
Northwest Center for Reproductive Sciences, Seattle, Washington 98119, USA
Fertil Steril 86:1547-9. 2006..Changes in FSH isoforms occur at temporally specific times during the menstrual cycle and reproductive lifespan. Changes in ratio in FSH isoforms may provide a sensitive test of ovarian reserve before IVF... - Environment and origin of diseaseVasantha Padmanabhan
Rev Endocr Metab Disord 8:67-9. 2007 - Polycystic ovary syndrome and its developmental originsDaniel A Dumesic
Wisconsin National Primate Research Center, University of Wisconsin, Madison, WI 53715, USA
Rev Endocr Metab Disord 8:127-41. 2007.... - Participation of vasoactive intestinal polypeptide in ovarian steroids production during the rat estrous cycle and in the development of estradiol valerate-induced polycystic ovaryClaudio Parra
Lab Neurobioquímica, Departamento de Bioquimica y Biologia Molecular, Facultad de Ciencias Quimicas y Farmaceuticas, Universidad de Chile, Santiago, Chile
Reproduction 133:147-54. 2007..These results strongly suggest that intraovarian VIP participates in the control of estradiol secretion during the rat estrous cycle and possibly in the maintenance of increased ovarian estradiol secretory activity of EV-treated rats... - Hypertension caused by prenatal testosterone excess in female sheepAndrew J King
Deparetment of Pharmacology and Toxicology, B440 Life Sciences, Michigan State University, East Lansing, MI 48824, USA
Am J Physiol Endocrinol Metab 292:E1837-41. 2007..This first-ever use of radiotelemetric blood pressure recordings in sheep demonstrates that mild hypertension, a risk factor reported in some women with PCOS, is also a feature of the sheep model of PCOS produced by prenatal T treatment...
Research Grants
- Endocrine Disruptors and Fetal DevelopmentVasantha Padmanabhan; Fiscal Year: 2010....
- Bisphenol-A and Reproductive DysfunctionVasantha Padmanabhan; Fiscal Year: 2011..The findings will be of relevance to the threat estrogenic environmental disruptors pose at current exposure levels to reproductive health. ..
- Bisphenol-A and Reproductive DysfunctionVasantha Padmanabhan; Fiscal Year: 2010..The findings will be of relevance to the threat estrogenic environmental disruptors pose at current exposure levels to reproductive health. ..
- Reproductive Consequences of Prenatal AndrogenizationVasantha Padmanabhan; Fiscal Year: 2005..Specifically it will establish that discrete, experimentally induced androgen excess of fetal sheep provides the first clear etiology for hyperandrogenic anovulation in adulthood. ..
- NEUROENDOCRINE CONTROL OF FSHVasantha Padmanabhan; Fiscal Year: 2001....