Yuko Oda

Summary

Affiliation: University of California
Country: USA

Publications

  1. pmc Coactivator MED1 ablation in keratinocytes results in hair-cycling defects and epidermal alterations
    Yuko Oda
    Department of Medicine and Endocrinology, University of California, San Francisco and Veterans Affairs Medical Center San Francisco, San Francisco, California 94121, USA
    J Invest Dermatol 132:1075-83. 2012
  2. ncbi request reprint Differential role of two VDR coactivators, DRIP205 and SRC-3, in keratinocyte proliferation and differentiation
    Yuko Oda
    Departments of Medicine and Endocrinology, University of California San Francisco, and Veterans Affairs Medical Center San Francisco, 41150 Clement Street, San Francisco, CA 94121, USA
    J Steroid Biochem Mol Biol 103:776-80. 2007
  3. pmc Vitamin D receptor and coactivators SRC2 and 3 regulate epidermis-specific sphingolipid production and permeability barrier formation
    Yuko Oda
    Department of Medicine, University of California, San Francisco, California, USA
    J Invest Dermatol 129:1367-78. 2009
  4. doi request reprint The transcriptional coactivator DRIP/mediator complex is involved in vitamin D receptor function and regulates keratinocyte proliferation and differentiation
    Yuko Oda
    Department of Medicine and Endocrinology, University of California, San Francisco and Veterans Affairs Medical Center San Francisco, San Francisco, California 94121, USA
    J Invest Dermatol 130:2377-88. 2010
  5. ncbi request reprint Two distinct coactivators, DRIP/mediator and SRC/p160, are differentially involved in VDR transactivation during keratinocyte differentiation
    Yuko Oda
    Department of Medicine, University of California San Francisco, Veterans Affairs Medical Center San Francisco, 4150 Clement Street, San Francisco, CA 94121, USA
    J Steroid Biochem Mol Biol 89:273-6. 2004
  6. pmc Regulation of cathelicidin antimicrobial peptide expression by an endoplasmic reticulum (ER) stress signaling, vitamin D receptor-independent pathway
    Kyungho Park
    Department of Dermatology, School of Medicine, University of California, San Francisco, California 94143, USA
    J Biol Chem 286:34121-30. 2011
  7. ncbi request reprint The role of the calcium-sensing receptor in epidermal differentiation
    Chia Ling Tu
    Department of Medicine, Veteran Affairs Medical Center, University of California and Endocrine Unit, San Francisco, CA 94121, USA
    Cell Calcium 35:265-73. 2004
  8. ncbi request reprint Squamous cell carcinomas fail to respond to the prodifferentiating actions of 1,25(OH)2D: why?
    Daniel D Bikle
    Veterans Affairs Medical Center 111 N, University of California, 4150 Clement Street, San Francisco, CA 94121, USA
    Recent Results Cancer Res 164:111-22. 2003
  9. ncbi request reprint Two distinct coactivators, DRIP/mediator and SRC/p160, are differentially involved in vitamin D receptor transactivation during keratinocyte differentiation
    Yuko Oda
    Departments of Medicine and Endocrinology University of California San Francisco and Veterans Affairs Medical Center, San Francisco, California 94121, USA
    Mol Endocrinol 17:2329-39. 2003
  10. ncbi request reprint Histone acetylation in keratinocytes enables control of the expression of cathelicidin and CD14 by 1,25-dihydroxyvitamin D3
    Jürgen Schauber
    Division of Dermatology, University of California, San Diego and VA San Diego Healthcare System, San Diego, California, USA
    J Invest Dermatol 128:816-24. 2008

Collaborators

Detail Information

Publications22

  1. pmc Coactivator MED1 ablation in keratinocytes results in hair-cycling defects and epidermal alterations
    Yuko Oda
    Department of Medicine and Endocrinology, University of California, San Francisco and Veterans Affairs Medical Center San Francisco, San Francisco, California 94121, USA
    J Invest Dermatol 132:1075-83. 2012
    ..These results indicate that MED1 has a critical role in regulating hair/epidermal proliferation and differentiation...
  2. ncbi request reprint Differential role of two VDR coactivators, DRIP205 and SRC-3, in keratinocyte proliferation and differentiation
    Yuko Oda
    Departments of Medicine and Endocrinology, University of California San Francisco, and Veterans Affairs Medical Center San Francisco, 41150 Clement Street, San Francisco, CA 94121, USA
    J Steroid Biochem Mol Biol 103:776-80. 2007
    ..These results support the concept that the selective use of coactivators by VDR underlies the selective regulation of gene expression in keratinocyte proliferation and differentiation...
  3. pmc Vitamin D receptor and coactivators SRC2 and 3 regulate epidermis-specific sphingolipid production and permeability barrier formation
    Yuko Oda
    Department of Medicine, University of California, San Francisco, California, USA
    J Invest Dermatol 129:1367-78. 2009
    ..In contrast, DRIP silencing had no apparent effect on these processes indicating that the two classes of coactivators are differentially utilized...
  4. doi request reprint The transcriptional coactivator DRIP/mediator complex is involved in vitamin D receptor function and regulates keratinocyte proliferation and differentiation
    Yuko Oda
    Department of Medicine and Endocrinology, University of California, San Francisco and Veterans Affairs Medical Center San Francisco, San Francisco, California 94121, USA
    J Invest Dermatol 130:2377-88. 2010
    ..These results show that keratinocytes use the transcriptional coactivator mediator to regulate VDR functions and control keratinocyte proliferation and differentiation...
  5. ncbi request reprint Two distinct coactivators, DRIP/mediator and SRC/p160, are differentially involved in VDR transactivation during keratinocyte differentiation
    Yuko Oda
    Department of Medicine, University of California San Francisco, Veterans Affairs Medical Center San Francisco, 4150 Clement Street, San Francisco, CA 94121, USA
    J Steroid Biochem Mol Biol 89:273-6. 2004
    ....
  6. pmc Regulation of cathelicidin antimicrobial peptide expression by an endoplasmic reticulum (ER) stress signaling, vitamin D receptor-independent pathway
    Kyungho Park
    Department of Dermatology, School of Medicine, University of California, San Francisco, California 94143, USA
    J Biol Chem 286:34121-30. 2011
    ..Hence, ER stress increases CAMP expression via NF-κB-C/EBPα activation, independent of VDR, illuminating a novel VDR-independent role for ER stress in stimulating innate immunity...
  7. ncbi request reprint The role of the calcium-sensing receptor in epidermal differentiation
    Chia Ling Tu
    Department of Medicine, Veteran Affairs Medical Center, University of California and Endocrine Unit, San Francisco, CA 94121, USA
    Cell Calcium 35:265-73. 2004
    ..These concepts derived from in vitro experiments have been tested and confirmed in two mouse models...
  8. ncbi request reprint Squamous cell carcinomas fail to respond to the prodifferentiating actions of 1,25(OH)2D: why?
    Daniel D Bikle
    Veterans Affairs Medical Center 111 N, University of California, 4150 Clement Street, San Francisco, CA 94121, USA
    Recent Results Cancer Res 164:111-22. 2003
    ....
  9. ncbi request reprint Two distinct coactivators, DRIP/mediator and SRC/p160, are differentially involved in vitamin D receptor transactivation during keratinocyte differentiation
    Yuko Oda
    Departments of Medicine and Endocrinology University of California San Francisco and Veterans Affairs Medical Center, San Francisco, California 94121, USA
    Mol Endocrinol 17:2329-39. 2003
    ....
  10. ncbi request reprint Histone acetylation in keratinocytes enables control of the expression of cathelicidin and CD14 by 1,25-dihydroxyvitamin D3
    Jürgen Schauber
    Division of Dermatology, University of California, San Diego and VA San Diego Healthcare System, San Diego, California, USA
    J Invest Dermatol 128:816-24. 2008
    ....
  11. pmc Topical antihistamines display potent anti-inflammatory activity linked in part to enhanced permeability barrier function
    Tzu Kai Lin
    Dermatology Service, Department of Veterans Affairs Medical Center, and Department of Dermatology, UCSF, San Francisco, California 94121, USA
    J Invest Dermatol 133:469-78. 2013
    ..These results could shift current paradigms of antihistamine utilization from a predominantly systemic to a topical approach...
  12. doi request reprint 1α,25(OH)2-dihydroxyvitamin D3/VDR protects the skin from UVB-induced tumor formation by interacting with the β-catenin pathway
    Yan J Jiang
    Endocrine Unit, Department of Medicine and Dermatology, VA Medical Center and University of California at San Francisco, CA 94121, USA
    J Steroid Biochem Mol Biol 136:229-32. 2013
    ..Further study using VDR or β-catenin single null mice is necessary to compare with the data from double null mice. This article is part of a Special Issue entitled 'Vitamin D Workshop'...
  13. ncbi request reprint Hairless suppresses vitamin D receptor transactivation in human keratinocytes
    Zhongjian Xie
    Endocrine Unit, Veterans Affairs Medical Center, San Francisco, California 94121, USA
    Endocrinology 147:314-23. 2006
    ..These data indicate that Hr functions as a corepressor of VDR to block 1,25(OH)2D3 action on keratinocytes...
  14. pmc Quantification of the vitamin D receptor-coregulator interaction
    Arnaud Teichert
    Endocrine Unit, University of California, San Francisco, San Francisco, California 94121, USA
    Biochemistry 48:1454-61. 2009
    ....
  15. ncbi request reprint Vitamin D and skin cancer: a problem in gene regulation
    Daniel D Bikle
    Endocrine Research Unit, Veterans Affairs Medical Center 111N, University of California, 4150 Clement Street, San Francisco, CA 94121, USA
    J Steroid Biochem Mol Biol 97:83-91. 2005
    ....
  16. ncbi request reprint The vitamin D receptor: a tumor suppressor in skin
    DANIEL DAVID BIKLE
    Department of Medicine, University of California San Francisco, San Francisco Veterans Affairs Medical Center, 94121, USA
    Discov Med 11:7-17. 2011
    ..These results lead us to the hypothesis that the VDR with its ligand 1,25(OH)(2)D(3) functions as a tumor suppressor with respect to epidermal tumor formation in response to UVR by regulating Hh and beta-catenin signaling...
  17. ncbi request reprint The vitamin D response element of the involucrin gene mediates its regulation by 1,25-dihydroxyvitamin D3
    Daniel D Bikle
    Department of Medicine, Veterans Affairs Medical Center and University of California, San Francisco, USA
    J Invest Dermatol 119:1109-13. 2002
    ....
  18. ncbi request reprint Epidermal expression of the full-length extracellular calcium-sensing receptor is required for normal keratinocyte differentiation
    Laszlo Komuves
    Department of Dermatology, Veterans Affairs Medical Center and the University of California, San Francisco School of Medicine, San Francisco, California 94121, USA
    J Cell Physiol 192:45-54. 2002
    ..Our results demonstrate that the epidermal expression of the full-length CaR is required for the normal terminal differentiation of keratinocytes...
  19. ncbi request reprint HOXB13 homeodomain protein is cytoplasmic throughout fetal skin development
    Laszlo G Komuves
    Department of Dermatology, VA Medical Center and University of California, San Francisco, California, USA
    Dev Dyn 227:192-202. 2003
    ..Expression in hyperproliferative skin conditions remained cytoplasmic with the exception of epidermis associated with Kaposi's sarcoma, which showed strong HOXB13 expression that was partially localized to the nucleus...
  20. doi request reprint A topical Chinese herbal mixture improves epidermal permeability barrier function in normal murine skin
    Mona Man
    Dermatology Service, VAMC, and Department of Dermatology, University of California San Francisco, San Francisco, CA, USA Department of Dermatology, Yonsei University College of Medicine, Yongdong Severance Hospital, Seoul, Korea Department of Medicine, University of California San Francisco, San Francisco, CA, USA Dermatology, Hospital del Mar, UAB, Barcelone, Spain
    Exp Dermatol 20:285-8. 2011
    ....
  21. ncbi request reprint Defective cyclic guanosine monophosphate-gated calcium channels and the pathogenesis of psoriasis
    Roddie C McKenzie
    Epidermal Inflammation and Protection Group, Department of Dermatology, University of Edinburgh, Edinburgh, Scotland, UK
    Acta Derm Venereol 83:414-8. 2003
    ....
  22. ncbi request reprint Expression of fibroblast growth factors and their receptors during full-thickness skin wound healing in young and aged mice
    Akiko Komi-Kuramochi
    Signaling Molecules Research Laboratory, National Institute of Advanced Industrial Science and Technology AIST, Higashi, Tsukuba 305 8566, Ibaraki, Japan
    J Endocrinol 186:273-89. 2005
    ....