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Genomes and Genes | Yuko OdaSummaryAffiliation: University of California Country: USA Publications
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Publications
Coactivator MED1 ablation in keratinocytes results in hair-cycling defects and epidermal alterationsYuko Oda
Department of Medicine and Endocrinology, University of California, San Francisco and Veterans Affairs Medical Center San Francisco, San Francisco, California 94121, USA
J Invest Dermatol 132:1075-83. 2012..These results indicate that MED1 has a critical role in regulating hair/epidermal proliferation and differentiation...
Differential role of two VDR coactivators, DRIP205 and SRC-3, in keratinocyte proliferation and differentiationYuko Oda
Departments of Medicine and Endocrinology, University of California San Francisco, and Veterans Affairs Medical Center San Francisco, 41150 Clement Street, San Francisco, CA 94121, USA
J Steroid Biochem Mol Biol 103:776-80. 2007..These results support the concept that the selective use of coactivators by VDR underlies the selective regulation of gene expression in keratinocyte proliferation and differentiation...- Vitamin D receptor and coactivators SRC2 and 3 regulate epidermis-specific sphingolipid production and permeability barrier formationYuko Oda
Department of Medicine, University of California, San Francisco, California, USA
J Invest Dermatol 129:1367-78. 2009..In contrast, DRIP silencing had no apparent effect on these processes indicating that the two classes of coactivators are differentially utilized... 
The transcriptional coactivator DRIP/mediator complex is involved in vitamin D receptor function and regulates keratinocyte proliferation and differentiationYuko Oda
Department of Medicine and Endocrinology, University of California, San Francisco and Veterans Affairs Medical Center San Francisco, San Francisco, California 94121, USA
J Invest Dermatol 130:2377-88. 2010..These results show that keratinocytes use the transcriptional coactivator mediator to regulate VDR functions and control keratinocyte proliferation and differentiation...- Two distinct coactivators, DRIP/mediator and SRC/p160, are differentially involved in VDR transactivation during keratinocyte differentiationYuko Oda
Department of Medicine, University of California San Francisco, Veterans Affairs Medical Center San Francisco, 4150 Clement Street, San Francisco, CA 94121, USA
J Steroid Biochem Mol Biol 89:273-6. 2004.... - Regulation of cathelicidin antimicrobial peptide expression by an endoplasmic reticulum (ER) stress signaling, vitamin D receptor-independent pathwayKyungho Park
Department of Dermatology, School of Medicine, University of California, San Francisco, California 94143, USA
J Biol Chem 286:34121-30. 2011..Hence, ER stress increases CAMP expression via NF-κB-C/EBPα activation, independent of VDR, illuminating a novel VDR-independent role for ER stress in stimulating innate immunity... - The role of the calcium-sensing receptor in epidermal differentiationChia Ling Tu
Department of Medicine, Veteran Affairs Medical Center, University of California and Endocrine Unit, San Francisco, CA 94121, USA
Cell Calcium 35:265-73. 2004..These concepts derived from in vitro experiments have been tested and confirmed in two mouse models... - Squamous cell carcinomas fail to respond to the prodifferentiating actions of 1,25(OH)2D: why?Daniel D Bikle
Veterans Affairs Medical Center 111 N, University of California, 4150 Clement Street, San Francisco, CA 94121, USA
Recent Results Cancer Res 164:111-22. 2003.... - Two distinct coactivators, DRIP/mediator and SRC/p160, are differentially involved in vitamin D receptor transactivation during keratinocyte differentiationYuko Oda
Departments of Medicine and Endocrinology University of California San Francisco and Veterans Affairs Medical Center, San Francisco, California 94121, USA
Mol Endocrinol 17:2329-39. 2003.... - Histone acetylation in keratinocytes enables control of the expression of cathelicidin and CD14 by 1,25-dihydroxyvitamin D3Jürgen Schauber
Division of Dermatology, University of California, San Diego and VA San Diego Healthcare System, San Diego, California, USA
J Invest Dermatol 128:816-24. 2008.... - Topical antihistamines display potent anti-inflammatory activity linked in part to enhanced permeability barrier functionTzu Kai Lin
1 Dermatology Service, Department of Veterans Affairs Medical Center, and Department of Dermatology, UCSF, San Francisco, California, USA 2 Department of Dermatology, National Cheng Kung University Hospital, and Graduate Institute of Clinical Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan
J Invest Dermatol 133:469-78. 2013..These results could shift current paradigms of antihistamine utilization from a predominantly systemic to a topical approach... - Quantification of the vitamin D receptor-coregulator interactionArnaud Teichert
Endocrine Unit, University of California, San Francisco, San Francisco, California 94121, USA
Biochemistry 48:1454-61. 2009.... - Hairless suppresses vitamin D receptor transactivation in human keratinocytesZhongjian Xie
Endocrine Unit, Veterans Affairs Medical Center, San Francisco, California 94121, USA
Endocrinology 147:314-23. 2006..These data indicate that Hr functions as a corepressor of VDR to block 1,25(OH)2D3 action on keratinocytes... - Vitamin D and skin cancer: a problem in gene regulationDaniel D Bikle
Endocrine Research Unit, Veterans Affairs Medical Center 111N, University of California, 4150 Clement Street, San Francisco, CA 94121, USA
J Steroid Biochem Mol Biol 97:83-91. 2005.... - The vitamin D receptor: a tumor suppressor in skinDANIEL DAVID BIKLE
Department of Medicine, University of California San Francisco, San Francisco Veterans Affairs Medical Center, 94121, USA
Discov Med 11:7-17. 2011..These results lead us to the hypothesis that the VDR with its ligand 1,25(OH)(2)D(3) functions as a tumor suppressor with respect to epidermal tumor formation in response to UVR by regulating Hh and beta-catenin signaling... - The vitamin D response element of the involucrin gene mediates its regulation by 1,25-dihydroxyvitamin D3Daniel D Bikle
Department of Medicine, Veterans Affairs Medical Center and University of California, San Francisco, USA
J Invest Dermatol 119:1109-13. 2002.... - Epidermal expression of the full-length extracellular calcium-sensing receptor is required for normal keratinocyte differentiationLaszlo Komuves
Department of Dermatology, Veterans Affairs Medical Center and the University of California, San Francisco School of Medicine, San Francisco, California 94121, USA
J Cell Physiol 192:45-54. 2002..Our results demonstrate that the epidermal expression of the full-length CaR is required for the normal terminal differentiation of keratinocytes... - HOXB13 homeodomain protein is cytoplasmic throughout fetal skin developmentLaszlo G Komuves
Department of Dermatology, VA Medical Center and University of California, San Francisco, California, USA
Dev Dyn 227:192-202. 2003..Expression in hyperproliferative skin conditions remained cytoplasmic with the exception of epidermis associated with Kaposi's sarcoma, which showed strong HOXB13 expression that was partially localized to the nucleus... - A topical Chinese herbal mixture improves epidermal permeability barrier function in normal murine skinMona Man
Dermatology Service, VAMC, and Department of Dermatology, University of California San Francisco, San Francisco, CA, USA Department of Dermatology, Yonsei University College of Medicine, Yongdong Severance Hospital, Seoul, Korea Department of Medicine, University of California San Francisco, San Francisco, CA, USA Dermatology, Hospital del Mar, UAB, Barcelone, Spain
Exp Dermatol 20:285-8. 2011.... - Expression of fibroblast growth factors and their receptors during full-thickness skin wound healing in young and aged miceAkiko Komi-Kuramochi
Signaling Molecules Research Laboratory, National Institute of Advanced Industrial Science and Technology (AIST, Higashi, Tsukuba 305-8566, Ibaraki, Japan
J Endocrinol 186:273-89. 2005.... - Defective cyclic guanosine monophosphate-gated calcium channels and the pathogenesis of psoriasisRoddie C McKenzie
Epidermal Inflammation and Protection Group, Department of Dermatology, University of Edinburgh, Edinburgh, Scotland, UK
Acta Derm Venereol 83:414-8. 2003....