Research Topics
Species | Hareesh B NairSummaryAffiliation: University of Texas Health Science Center Country: USA Publications
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Detail Information
Publications
BU-32: a novel proteasome inhibitor for breast cancerJoseph K Agyin
Department of Biochemistry, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA
Breast Cancer Res 11:R74. 2009..This underscores the need to find new and more efficacious proteasome inhibitors. In this study, we evaluate the efficacy of the novel proteasome inhibitor BU-32 (NSC D750499-S) using in vitro and in vivo breast cancer models...
Delivery of antiinflammatory nutraceuticals by nanoparticles for the prevention and treatment of cancerHareesh B Nair
Department of Obstetrics and Gynecology, The University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229 3900, USA
Biochem Pharmacol 80:1833-43. 2010....
Induction of aromatase expression in cervical carcinomas: effects of endogenous estrogen on cervical cancer cell proliferationHareesh B Nair
Department of Gynecology and Obstetrics, University of Texas Health Science Center at San Antonio, San Antonio, Texas 78229, USA
Cancer Res 65:11164-73. 2005....
Modulation of in situ estrogen synthesis by proline-, glutamic acid-, and leucine-rich protein-1: potential estrogen receptor autocrine signaling loop in breast cancer cellsRajib Rajhans
Department of Obstetrics and Gynecology, University of Texas Health Science Center, Floyd Curl Drive, San Antonio, Texas 78229 3900, USA
Mol Endocrinol 22:649-64. 2008....
HER-2/neu x aromatase double transgenic mice model: the effects of aromatase overexpression on mammary tumorigenesisRajeshwar Rao Tekmal
Department of Obstetrics and Gynecology, The University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA
J Steroid Biochem Mol Biol 106:111-8. 2007....
Estrogen receptor alpha is required for mammary development and the induction of mammary hyperplasia and epigenetic alterations in the aromatase transgenic miceRajeshwar Rao Tekmal
Department of Obstetrics and Gynecology, The University of Texas Health Science Center at San Antonio, MSC7836, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA
J Steroid Biochem Mol Biol 95:9-15. 2005....
Overexpression of the colony-stimulating factor (CSF-1) and/or its receptor c-fms in mammary glands of transgenic mice results in hyperplasia and tumor formationNameer Kirma
Department of Obstetrics and Gynecology, The University of Texas Health Science Center at San Antonio, 78229, USA
Cancer Res 64:4162-70. 2004..These findings, along with the histological data, support the hypothesis that CSF-1 and its receptor are involved in the etiology of breast cancer...
Elevated expression of the oncogene c-fms and its ligand, the macrophage colony-stimulating factor-1, in cervical cancer and the role of transforming growth factor-beta1 in inducing c-fms expressionNameer Kirma
Department of Obstetrics and Gynecology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA
Cancer Res 67:1918-26. 2007..Additionally, our data suggest that transforming growth factor-beta1 may be a factor in inducing the expression of c-fms in cervical cancer cells. The data suggest that c-fms may be a valuable therapeutic target in cervical cancer...
Induction of endometrial epithelial cell invasion and c-fms expression by transforming growth factor betaYa Guang Liu
Department of Obstetrics and Gynecology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA
Mol Hum Reprod 15:665-73. 2009....
Elevated aromatase expression correlates with cervical carcinoma progressionPoornachand Veerapaneni
Department of Obstetrics and Gynecology, University of Texas Health Science Center at San Antonio, San Antonio, TX 78229, USA
Gynecol Oncol 114:496-500. 2009..g., IL-6 and TNFalpha) may correlate with aromatase expression...
