J J Mulchahey

Summary

Affiliation: University of Cincinnati
Country: USA

Publications

  1. ncbi request reprint Cerebrospinal fluid and plasma testosterone levels in post-traumatic stress disorder and tobacco dependence
    J J Mulchahey
    Department of Psychiatry, University of Cincinnati College of Medicine, Cincinnati, OH 45267, USA
    Psychoneuroendocrinology 26:273-85. 2001
  2. ncbi request reprint Regulation of corticotropin-releasing factor-binding protein expression in amygdalar neuronal cultures
    J W Kasckow
    Cincinnati VAMC, Psychiatry Service, Cincinnati, OH 45220, USA
    J Neuroendocrinol 11:959-66. 1999
  3. ncbi request reprint Coordinate and divergent regulation of corticotropin-releasing factor (CRF) and CRF-binding protein expression in an immortalized amygdalar neuronal cell line
    J J Mulchahey
    Department of Psychiatry, University of Cincinnati College of Medicine, Ohio 45267, USA
    Endocrinology 140:251-9. 1999
  4. ncbi request reprint Changes in brain corticotropin-releasing factor messenger RNA expression in aged Fischer 344 rats
    J W Kasckow
    Cincinnati VAMC Psychiatry Service, 3200 Vine Street, Cincinnati, OH 45220, USA
    Brain Res 822:228-30. 1999
  5. ncbi request reprint Interaction of neuropeptide Y and corticotropin-releasing factor signaling pathways in AR-5 amygdalar cells
    S Sheriff
    University of Cincinnati College of Medicine, Department of Surgery, Cincinnati, OH 45267, USA
    Peptides 22:2083-9. 2001
  6. ncbi request reprint Regulation of corticotropin-releasing hormone in vitro
    M Pisarska
    Cincinnati VAMC, Psychiatry Service, 3200 Vine Street, Cincinnati, OH 45220, USA
    Peptides 22:705-12. 2001
  7. ncbi request reprint Aging associated changes in amygdalar corticotropin-releasing hormone (CRH) and CRH-binding protein in Fischer 344 rats
    C Xiao
    University of Cincinnati Department of Surgery, 231 Bethesda Ave, Cincinnati, OH 45267, USA
    Brain Res 1073:325-31. 2006
  8. ncbi request reprint The effect of feeding on cerebrospinal fluid corticotropin-releasing hormone levels in humans
    J W Kasckow
    Cincinnati VAMC, Psychiatry Service, 3200 Vine St, Cincinnati, OH 45220, USA
    Brain Res 904:218-24. 2001
  9. ncbi request reprint Health-related quality of well-being in chronically hospitalized patients with schizophrenia: comparison with matched outpatients
    J W Kasckow
    VA Cincinnati Healthcare System and University of Cincinnati, 231 Bethesda Ave, Cincinnati, OH 45267 0559, USA
    Psychiatry Res 103:69-78. 2001
  10. ncbi request reprint Corticotropin-releasing hormone (CRH) expression and protein kinase A mediated CRH receptor signalling in an immortalized hypothalamic cell line
    J Kasckow
    Cincinnati VAMC, Psychiatry Service 116A, and School of Medicine, Department of Psychiatry, and Neurosciences Program, University of Cincinnati, OH 45267 0559, USA
    J Neuroendocrinol 15:521-9. 2003

Detail Information

Publications11

  1. ncbi request reprint Cerebrospinal fluid and plasma testosterone levels in post-traumatic stress disorder and tobacco dependence
    J J Mulchahey
    Department of Psychiatry, University of Cincinnati College of Medicine, Cincinnati, OH 45267, USA
    Psychoneuroendocrinology 26:273-85. 2001
    ..The goal of this study was to screen for potential abnormalities in CNS testosterone levels in patients with post-traumatic stress disorder (PTSD) and/or tobacco dependence...
  2. ncbi request reprint Regulation of corticotropin-releasing factor-binding protein expression in amygdalar neuronal cultures
    J W Kasckow
    Cincinnati VAMC, Psychiatry Service, Cincinnati, OH 45220, USA
    J Neuroendocrinol 11:959-66. 1999
    ..05; n=3 for each). The results suggest that CRF-BP in the amygdala is stimulated by numerous pathways which may play a significant role in promoting behavioural changes...
  3. ncbi request reprint Coordinate and divergent regulation of corticotropin-releasing factor (CRF) and CRF-binding protein expression in an immortalized amygdalar neuronal cell line
    J J Mulchahey
    Department of Psychiatry, University of Cincinnati College of Medicine, Ohio 45267, USA
    Endocrinology 140:251-9. 1999
    ..These data also suggest that dexamethasone may decrease the biological availability of CRF in the amygdala by increasing the expression of CRF-BP, rather than by decreasing CRF expression...
  4. ncbi request reprint Changes in brain corticotropin-releasing factor messenger RNA expression in aged Fischer 344 rats
    J W Kasckow
    Cincinnati VAMC Psychiatry Service, 3200 Vine Street, Cincinnati, OH 45220, USA
    Brain Res 822:228-30. 1999
    ..These changes may contribute to impaired adaptations to stress, cognitive decline and other pathophysiological processes during aging...
  5. ncbi request reprint Interaction of neuropeptide Y and corticotropin-releasing factor signaling pathways in AR-5 amygdalar cells
    S Sheriff
    University of Cincinnati College of Medicine, Department of Surgery, Cincinnati, OH 45267, USA
    Peptides 22:2083-9. 2001
    ..These findings suggest that CRF and NPY may counter-regulate each other in amygdalar neurons via reciprocal effects on the protein kinase A pathway...
  6. ncbi request reprint Regulation of corticotropin-releasing hormone in vitro
    M Pisarska
    Cincinnati VAMC, Psychiatry Service, 3200 Vine Street, Cincinnati, OH 45220, USA
    Peptides 22:705-12. 2001
    ..These mechanistic studies have focused on PKA- and glucocorticoid-mediated changes in CRH expression. Clearly much more investigative work in better understanding CRH regulation in vitro is needed...
  7. ncbi request reprint Aging associated changes in amygdalar corticotropin-releasing hormone (CRH) and CRH-binding protein in Fischer 344 rats
    C Xiao
    University of Cincinnati Department of Surgery, 231 Bethesda Ave, Cincinnati, OH 45267, USA
    Brain Res 1073:325-31. 2006
    ..05 via ANOVA and Fisher's PLSD). These changes may contribute to impaired adaptations to stress, cognitive decline, and other pathophysiological processes during aging...
  8. ncbi request reprint The effect of feeding on cerebrospinal fluid corticotropin-releasing hormone levels in humans
    J W Kasckow
    Cincinnati VAMC, Psychiatry Service, 3200 Vine St, Cincinnati, OH 45220, USA
    Brain Res 904:218-24. 2001
    ..Alternatively, our findings could also reflect changes in CRH levels associated with feeding in multiple brain areas and in the spinal cord with the net effect being in the negative direction...
  9. ncbi request reprint Health-related quality of well-being in chronically hospitalized patients with schizophrenia: comparison with matched outpatients
    J W Kasckow
    VA Cincinnati Healthcare System and University of Cincinnati, 231 Bethesda Ave, Cincinnati, OH 45267 0559, USA
    Psychiatry Res 103:69-78. 2001
    ..These findings further support the use of the QWB in severely mentally ill populations; implications for improving health-related quality of life among older patients with schizophrenia are discussed...
  10. ncbi request reprint Corticotropin-releasing hormone (CRH) expression and protein kinase A mediated CRH receptor signalling in an immortalized hypothalamic cell line
    J Kasckow
    Cincinnati VAMC, Psychiatry Service 116A, and School of Medicine, Department of Psychiatry, and Neurosciences Program, University of Cincinnati, OH 45267 0559, USA
    J Neuroendocrinol 15:521-9. 2003
    ..The characteristics of IVB cells, such as CRH and VP coexpression, GR expression and a biologically active CRH-R1-mediated signalling pathway, suggest that this neuronal cell line may serve as model of parvocellular CRH neurones...
  11. ncbi request reprint Quantification and localization of kainic acid-induced neurotoxicity employing a new biomarker of cell death: cleaved microtubule-associated protein-tau (C-tau)
    F P Zemlan
    Department of Psychiatry, College of Medicine, 231 Sabin Avenue, University of Cincinnati, Cincinnati, OH 45267, USA
    Neuroscience 121:399-409. 2003
    ..These data suggest that C-tau proteolysis may prove a reliable species independent biomarker of neuronal degeneration regardless of source of injury...