R Moslehi

Summary

Affiliation: University at Albany
Country: USA

Publications

  1. pmc Integrative transcriptome analysis reveals dysregulation of canonical cancer molecular pathways in placenta leading to preeclampsia
    Roxana Moslehi
    Department of Epidemiology and Biostatistics, School of Public Health, University at Albany, NY 12144, USA
    Sci Rep 3:2407. 2013
  2. pmc Phenotype-specific adverse effects of XPD mutations on human prenatal development implicate impairment of TFIIH-mediated functions in placenta
    Roxana Moslehi
    Department of Epidemiology and Biostatistics, School of Public Health, University at Albany, Albany, NY 12144, USA
    Eur J Hum Genet 20:626-31. 2012
  3. pmc Impact of BRCA mutations on female fertility and offspring sex ratio
    Roxana Moslehi
    Department of Epidemiology and Biostatistics, School of Public Health, State University of New York at Albany, 1 Discovery Drive, Rensselaer, NY 12144, USA
    Am J Hum Biol 22:201-5. 2010
  4. pmc Adverse effects of trichothiodystrophy DNA repair and transcription gene disorder on human fetal development
    R Moslehi
    Biostatistics Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD, USA
    Clin Genet 77:365-73. 2010
  5. ncbi request reprint Cigarette smoking, N-acetyltransferase genes and the risk of advanced colorectal adenoma
    Roxana Moslehi
    Division of Cancer Epidemiology and Genetics, National Cancer Institute, NIH, 6120 Executive Blvd, EPS 8047, Rockville, MD 20852 USA
    Pharmacogenomics 7:819-29. 2006
  6. pmc A web server for inferring the human N-acetyltransferase-2 (NAT2) enzymatic phenotype from NAT2 genotype
    Igor B Kuznetsov
    Gen NY Sis Center for Excellence in Cancer Genomics, Department of Epidemiology and Biostatistics, University at Albany, One Discovery Drive, Rensselaer, NY 12144, USA
    Bioinformatics 25:1185-6. 2009
  7. pmc Powerful multilocus tests of genetic association in the presence of gene-gene and gene-environment interactions
    Nilanjan Chatterjee
    Division of Cancer Epidemiology and Genetics, National Cancer Institute, Bethesda, MD, USA
    Am J Hum Genet 79:1002-16. 2006
  8. ncbi request reprint Analysis of metabolic syndrome phenotypes in Framingham Heart Study families from Genetic Analysis Workshop 13
    Lynn R Goldin
    Genetic Epidemiology Branch, DCEG, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892 7236, USA
    Genet Epidemiol 25:S78-89. 2003

Detail Information

Publications8

  1. pmc Integrative transcriptome analysis reveals dysregulation of canonical cancer molecular pathways in placenta leading to preeclampsia
    Roxana Moslehi
    Department of Epidemiology and Biostatistics, School of Public Health, University at Albany, NY 12144, USA
    Sci Rep 3:2407. 2013
    ..We propose that dysregulation of these canonical cancer molecular pathways occurs in preeclampsia and delineate the relevance of TFIIH, providing etiologic clues which could eventually translate into a therapeutic approach. ..
  2. pmc Phenotype-specific adverse effects of XPD mutations on human prenatal development implicate impairment of TFIIH-mediated functions in placenta
    Roxana Moslehi
    Department of Epidemiology and Biostatistics, School of Public Health, University at Albany, Albany, NY 12144, USA
    Eur J Hum Genet 20:626-31. 2012
    ..Therefore, future studies of genetic associations of preeclampsia and other placental vascular complications may benefit from focusing on genetic variants within the fetal DNA...
  3. pmc Impact of BRCA mutations on female fertility and offspring sex ratio
    Roxana Moslehi
    Department of Epidemiology and Biostatistics, School of Public Health, State University of New York at Albany, 1 Discovery Drive, Rensselaer, NY 12144, USA
    Am J Hum Biol 22:201-5. 2010
    ..Potential contribution of observed sex ratio distortions to positive selection for BRCA mutations may warrant further investigation...
  4. pmc Adverse effects of trichothiodystrophy DNA repair and transcription gene disorder on human fetal development
    R Moslehi
    Biostatistics Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD, USA
    Clin Genet 77:365-73. 2010
    ..Thus, we hypothesize that TTD DNA repair and transcription genes play an important role in normal human placental development...
  5. ncbi request reprint Cigarette smoking, N-acetyltransferase genes and the risk of advanced colorectal adenoma
    Roxana Moslehi
    Division of Cancer Epidemiology and Genetics, National Cancer Institute, NIH, 6120 Executive Blvd, EPS 8047, Rockville, MD 20852 USA
    Pharmacogenomics 7:819-29. 2006
    ..Our interest is in the polymorphisms within the NAT1 and NAT2 genes that influence the tobacco-colorectal tumor relationship by impacting on the metabolic activation and detoxification of tobacco smoke-derived carcinogens...
  6. pmc A web server for inferring the human N-acetyltransferase-2 (NAT2) enzymatic phenotype from NAT2 genotype
    Igor B Kuznetsov
    Gen NY Sis Center for Excellence in Cancer Genomics, Department of Epidemiology and Biostatistics, University at Albany, One Discovery Drive, Rensselaer, NY 12144, USA
    Bioinformatics 25:1185-6. 2009
    ..The web server can be used for a fast determination of NAT2 phenotypes in genetic screens...
  7. pmc Powerful multilocus tests of genetic association in the presence of gene-gene and gene-environment interactions
    Nilanjan Chatterjee
    Division of Cancer Epidemiology and Genetics, National Cancer Institute, Bethesda, MD, USA
    Am J Hum Genet 79:1002-16. 2006
    ....
  8. ncbi request reprint Analysis of metabolic syndrome phenotypes in Framingham Heart Study families from Genetic Analysis Workshop 13
    Lynn R Goldin
    Genetic Epidemiology Branch, DCEG, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892 7236, USA
    Genet Epidemiol 25:S78-89. 2003
    ..Over 40 genome-wide linkage analyses were conducted. Despite the broad range of approaches, several regions of the genome were repeatedly identified across multiple analyses...