Research Topics
Species | Eric L LogigianSummaryAffiliation: University of Rochester Country: USA Publications
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Publications
Quantitative analysis of the "warm-up" phenomenon in myotonic dystrophy type 1E L Logigian
Department of Neurology, University of Rochester Medical Center, Box 673, 601 Elmwood Avenue, Rochester, New York 14642, USA
Muscle Nerve 32:35-42. 2005....
Mexiletine is an effective antimyotonia treatment in myotonic dystrophy type 1E L Logigian
Department of Neurology, University of Rochester, Rochester, NY, USA
Neurology 74:1441-8. 2010..To determine if mexiletine is safe and effective in reducing myotonia in myotonic dystrophy type 1 (DM1)...- Gentle dorsal root retraction and dissection can cause areflexia: implications for intraoperative monitoring during "selective" partial dorsal rhizotomyE L Logigian
Department of Neurology, Strong Memorial Hospital, University of Rochester School of Medicine and Dentistry, 601 Elmwood Avenue, Rochester, New York 14642, USA
Muscle Nerve 24:1352-8. 2001..High-intensity dorsal rootlet stimulation distal to the site of conduction block may then evoke not reflex responses, but rather nonreflex motor responses, due to the costimulation of adjacent ventral roots... - Leukocyte CTG repeat length correlates with severity of myotonia in myotonic dystrophy type 1E L Logigian
Department of Neurology, University of Rochester, NY, USA
Neurology 62:1081-9. 2004..To quantitate hand muscle myotonia and to assess the relationship between CTG repeat length and myotonia in myotonic dystrophy type 1 (DM1)... - Severity, type, and distribution of myotonic discharges are different in type 1 and type 2 myotonic dystrophyEric L Logigian
Neuromuscular Division, Department of Neurology, Box 673, 601 Elmwood Avenue, University of Rochester Medical Center, Rochester, New York 14642, USA
Muscle Nerve 35:479-85. 2007..It tends to be waxing-waning in DM1 but waning in DM2, thus making electrodiagnosis of DM2 more challenging. Its severity correlates with muscle weakness and the presence of waxing-waning discharges in DM1 but not DM2... 
Evoked myotonia can be "dialed-up" by increasing stimulus train length in myotonic dystrophy type 1Eric L Logigian
Department of Neurology, University of Rochester Medical Center, Box 673, 601 Elmwood Avenue, Rochester, New York, USA
Muscle Nerve 41:191-6. 2010..Evoked myotonia testing utilizing a stimulus paradigm of at least 20 stimuli at 30-50 HZ may be useful in antimyotonic drug trials, particularly when grip RT is normal or equivocal...- Computerized hand grip myometry reliably measures myotonia and muscle strength in myotonic dystrophy (DM1)Richard T Moxley
Department of Neurology, University of Rochester Medical Center, Box 673, 601 Elmwood Avenue, Rochester, New York 14642, USA
Muscle Nerve 36:320-8. 2007..We conclude that computerized handgrip myometry provides a sensitive, reliable measure of myotonia and strength in DM1 and offers a method to assess natural history and response to treatment... - Acute inflammatory demyelinating polyneuropathy: contribution of a dispersed distal compound muscle action potential to electrodiagnosisJames C Cleland
Department of Neurology, University of Rochester Medical Center, 601 Elmwood Avenue, Box 673, Rochester, New York 14642, USA
Muscle Nerve 33:771-7. 2006.... - Dispersion of the distal compound muscle action potential as a diagnostic criterion for chronic inflammatory demyelinating polyneuropathyPariwat Thaisetthawatkul
Departments of Neurology, University of Rochester Medical Center, NY 14642, USA
Neurology 59:1526-32. 2002..CONCLUSION: Quantitation of DCMAP dispersion shows promise as a sensitive and specific adjunctive electrodiagnostic criterion for CIDP... - Yield of the sural/radial ratio versus the medial plantar nerve in sensory neuropathies with a normal sural responseJohn P Sullivan
Department of Neurology, University of Rochester Medical Center, Rochester, NY 14642, USA
J Clin Neurophysiol 25:111-4. 2008..The yield of the SRAR and plantar NAP amplitude is poor when clinical signs of large-fiber sensory dysfunction are lacking... - Dispersion of compound muscle action potential in hereditary neuropathies and chronic inflammatory demyelinating polyneuropathyMichael Stanton
Department of Neurology, University of Rochester Medical Center, Rochester, New York 14642, USA
Muscle Nerve 34:417-22. 2006.... - Evaluation of patients with recurrent symptoms after ulnar nerve transpositionCristina I Matei
Department of Neurology, Upstate Medical University, 750 East Adams Street, Syracuse, New York 13210, USA
Muscle Nerve 30:493-6. 2004..The prevalence of this medial location of the ulnar nerve in asymptomatic postsurgical patients is unknown... - Electrodiagnosis of ulnar neuropathy at the wrist: conduction block versus traditional testsSusan R Cowdery
Department of Neurology, University of Rochester Medical Center, 601 Elmwood Avenue, Rochester, NY 14642, USA
Neurology 59:420-7. 2002..CONCLUSIONS: In UNW, an additional palmar stimulation site improves electrodiagnostic yield, and demonstrates that CB is an important cause of muscle weakness... - Late-onset Tay-Sachs disease: the spectrum of peripheral neuropathy in 30 affected patientsBarbara E Shapiro
Neuromuscular Division, Department of Neurology, Neurological Institute, University Hospitals Case Medical Center, Cleveland, Ohio, USA
Muscle Nerve 38:1012-5. 2008.... - Dispersion of the distal compound muscle action potential in chronic inflammatory demyelinating polyneuropathy and carpal tunnel syndromeJames C Cleland
Department of Neurology, University of Rochester, Box 673, 601 Elmwood Avenue, Rochester, New York 14642, USA
Muscle Nerve 28:189-93. 2003.... - Unexpected neuroimaging abnormalities in patients with apparent C8 radiculopathy: broadening the clinical spectrumMichael K Hehir
University of Vermont, Department of Neurology, Burlington, Vermont, USA
Muscle Nerve 45:859-65. 2012..We hypothesized that C7 or T1 root lesions (with a pre- or postfixed plexus) or cervical myelopathy might explain some "C8 radiculopathies" without C8 root compression... - Clinical evaluation of membrane excitability in muscle channel disorders: potential applications in clinical trialsJames C Cleland
University of Rochester School of Medicine and Dentistry, Rochester, New York, USA
Neurotherapeutics 4:205-15. 2007..Even in genetically defined cases, minimal clinical expressivity due to incomplete penetrance poses a significant challenge to currently available nonmolecular testing... - Plantar nerve AP and skin biopsy in sensory neuropathies with normal routine conduction studiesD N Herrmann
Department of Neurology, University of Rochester, NY, USA
Neurology 63:879-85. 2004..To assess the medial plantar nerve action potential (NAP) and skin biopsy in the evaluation of suspected distal sensory neuropathies (SN) with normal routine nerve conduction studies (NCS)... - Localization of ulnar neuropathy with conduction block across the elbowD N Herrmann
Department of Neurology, University of Rochester, SMH 601 Elmwood Avenue, Box 673, Rochester, New York 14642, USA
Muscle Nerve 24:698-700. 2001..The ulnar nerve may be more prone to external compression above the elbow than previously recognized. Short segment incremental studies are useful to identify conduction block above the elbow in such patients... - Successful treatment of Lyme encephalopathy with intravenous ceftriaxoneE L Logigian
Department of Neurology, University of Rochester Medical Center, Rochester, NY 14642, USA
J Infect Dis 180:377-83. 1999..01). Twelve to 24 months after treatment, all 18 patients rated themselves as back to normal or improved. We conclude that Lyme encephalopathy can be treated successfully with ceftriaxone... - Open-label trial of recombinant human insulin-like growth factor 1/recombinant human insulin-like growth factor binding protein 3 in myotonic dystrophy type 1Chad R Heatwole
University of Rochester Medical Center, Rochester, NY 14642, USA
Arch Neurol 68:37-44. 2011..To evaluate the safety and tolerability of recombinant human insulin-like growth factor 1 (rhIGF-1) complexed with IGF binding protein 3 (rhIGF-1/rhIGFBP-3) in patients with myotonic dystrophy type 1 (DM1)... - Conduction slowing in diabetic distal polyneuropathyDavid N Herrmann
Department of Neurology, University of Rochester, SMH 601 Elmwood Avenue, Box 673, Rochester, New York 14642, USA
Muscle Nerve 26:232-7. 2002..CMAP amplitude vs. CV and DL regression analyses using ALS as a control group for relatively pure axon loss may provide pathophysiologic information about motor nerves in other neuropathic disorders... - Class of nerve fiber involvement in sensory neuropathies: clinical characterization and utility of the plantar nerve action potentialHiroyuki Nodera
Department of Neurology, University of Rochester Medical Center, Box 673, 601 Elmwood Avenue, Rochester, New York 14642, USA
Muscle Nerve 26:212-7. 2002.... - Electrodiagnostic approach to the patient with suspected mononeuropathy of the upper extremityDavid N Herrmann
Department of Neurology, University of Rochester, SMH 601 Elmwood Ave, Box 673, Rochester, NY 14642, USA
Neurol Clin 20:451-78, vii. 2002..Insights gleaned from the electrodiagnostic report guide clinicians in the optimal management of mononeuropathies of the upper extremity... - Painful small-fiber neuropathy in Sjogren syndromeJ Chai
Department of Neurology, University of Rochester, Rochester, NY, USA
Neurology 65:925-7. 2005..ENF loss was frequently non length dependent, suggesting that patients with this disorder commonly have a small-fiber sensory neuronopathy rather than a "dying-back" axonopathy... - A Bayesian argument against rigid cut-offs in electrodiagnosis of median neuropathy at the wristH Nodera
Department of Neurology, University of Rochester School of Medicine and Dentistry, Rochester, NY, USA
Neurology 60:458-64. 2003..Use of rigid cut-off values to confirm MNW is problematic, because more conservative cut-offs are required for low pretest probability. Conversely, NC tests with sensitivity <95% cannot exclude MNW when pretest probability is high... - The diagnosis and treatment of myotonic disordersChad R Heatwole
Department of Neurology, Box 673, University of Rochester Medical Center, 601 Elmwood Avenue, Rochester, New York, New York 14642, USA
Muscle Nerve 47:632-48. 2013..Muscle Nerve 47: 632-648, 2013... - Electrodiagnosis of myotonic disordersMichael K Hehir
Department of Neurology, University of Vermont, Burlington, VT 05401, USA
Phys Med Rehabil Clin N Am 24:209-20. 2013..This article reviews myotonia and its differential diagnosis. The use of electrodiagnostic testing to evaluate the primary myotonic disorders (myotonic dystrophy and the nondystrophic myotonias) is also discussed... - Evidence in support of a feedback-sensitive central timekeeper for an over-learned repetitive motor behavior (pencil shading)G M Plotkin
Department of Neurology, Strong Memorial Hospital, University of Rochester School of Medicine and Dentistry, Rochester, NY, USA
Electromyogr Clin Neurophysiol 42:243-51. 2002..CONCLUSIONS: These data support the hypothesis that certain repetitive voluntary movements, such as pencil shading, are paced by central timekeepers that are influenced by changes in sensory feedback... - Genotype-phenotype correlation in a family with late onset CMT and an MPZ lys236del mutationJ E Sowden
Department of Neurology, University of Rochester Medical Center, 601 Elmwood Ave, Rochester, NY 14642, USA
J Neurol Neurosurg Psychiatry 76:442-4. 2005.... - HNPP due to a novel missense mutation of the PMP22 geneHiroyuki Nodera
Department of Clinical Neuroscience, University of Tokushima, Japan. tokushima-u.ac.jp
Neurology 60:1863-4. 2003