B Lin

Summary

Affiliation: University of Miami
Country: USA

Publications

  1. pmc Forebrain ischemia triggers GABAergic system degeneration in substantia nigra at chronic stages in rats
    B Lin
    Department of Neurology D4 5, Cerebral Vascular Disease Research Center, University of Miami Miller School of Medicine, P O Box 016960, Miami, FL 33101, USA
    Cardiovasc Psychiatry Neurol 2010:506952. 2010
  2. ncbi request reprint Encephalopathy: a vicious cascade following forebrain ischemia and hypoxia
    Baowan Lin
    Department of Neurology, University of Miami Miller School of Medicine, PO Box 016960, Miami, FL 33101, USA
    Cent Nerv Syst Agents Med Chem 13:57-70. 2013
  3. ncbi request reprint Polyphenols and neuroprotection against ischemia and neurodegeneration
    B Lin
    Department of Neurology, University of Miami, Miller School of Medicine, Miami, Florida, USA
    Mini Rev Med Chem 11:1222-38. 2011
  4. ncbi request reprint Quantitative analysis of microvascular alterations in traumatic brain injury by endothelial barrier antigen immunohistochemistry
    B Lin
    Cerebral Vascular Disease Research Center, Department of Neurology, University of Miami School of Medicine, Florida 33101, USA
    J Neurotrauma 18:389-97. 2001
  5. ncbi request reprint Transient changes of brain-derived neurotrophic factor (BDNF) mRNA expression in hippocampus during moderate ischemia induced by chronic bilateral common carotid artery occlusions in the rat
    R Schmidt-Kastner
    Cerebral Vascular Disease Research Center, Department of Neurology, University of Miami School of Medicine, PO Box 016960, Miami, FL 33101, USA
    Brain Res Mol Brain Res 92:157-66. 2001

Collaborators

Detail Information

Publications5

  1. pmc Forebrain ischemia triggers GABAergic system degeneration in substantia nigra at chronic stages in rats
    B Lin
    Department of Neurology D4 5, Cerebral Vascular Disease Research Center, University of Miami Miller School of Medicine, P O Box 016960, Miami, FL 33101, USA
    Cardiovasc Psychiatry Neurol 2010:506952. 2010
    ..2 sec versus 11.8 ± 5.4 sec; sham versus ischemia, p < .05, n = 5, and 7, resp.). Our results demonstrate delayed impairment of the GABAergic system components in SN and associated with movement deficits after global ischemia...
  2. ncbi request reprint Encephalopathy: a vicious cascade following forebrain ischemia and hypoxia
    Baowan Lin
    Department of Neurology, University of Miami Miller School of Medicine, PO Box 016960, Miami, FL 33101, USA
    Cent Nerv Syst Agents Med Chem 13:57-70. 2013
    ..A combination of protecting endothelia, inhibiting platelet activity and improving cerebral circulation is a fundamental strategy to block this vicious cascade, thereby ameliorating or preventing the encephalopathy...
  3. ncbi request reprint Polyphenols and neuroprotection against ischemia and neurodegeneration
    B Lin
    Department of Neurology, University of Miami, Miller School of Medicine, Miami, Florida, USA
    Mini Rev Med Chem 11:1222-38. 2011
    ..Pharmacology of the 7 herbs and their major relative polyphenols is depicted in the article...
  4. ncbi request reprint Quantitative analysis of microvascular alterations in traumatic brain injury by endothelial barrier antigen immunohistochemistry
    B Lin
    Cerebral Vascular Disease Research Center, Department of Neurology, University of Miami School of Medicine, Florida 33101, USA
    J Neurotrauma 18:389-97. 2001
    ....
  5. ncbi request reprint Transient changes of brain-derived neurotrophic factor (BDNF) mRNA expression in hippocampus during moderate ischemia induced by chronic bilateral common carotid artery occlusions in the rat
    R Schmidt-Kastner
    Cerebral Vascular Disease Research Center, Department of Neurology, University of Miami School of Medicine, PO Box 016960, Miami, FL 33101, USA
    Brain Res Mol Brain Res 92:157-66. 2001
    ..Prolonged moderate ischemia (oligemia) is a sufficient stimulus for BDNF gene expression in the hippocampus. These molecular studies provide direct evidence for an involvement of the hippocampus in the BCCAO model...