Research Topics
| D Joshua LiaoSummaryAffiliation: University of Minnesota Country: USA Publications
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Detail Information
Publications
Gene expression profiles in primary pancreatic tumors and metastatic lesions of Ela-c-myc transgenic miceArchana Thakur
Department of Pathology, Karmanos Cancer Institute, Wayne State University School of Medicine, 110 E, Warren Ave, Detroit, Michigan 48201, USA
Mol Cancer 7:11. 2008..To further evaluate the relevance of some of the selected differentially expressed genes, we investigated their expression pattern in human pancreatic cancer cell lines with high and low metastatic potentials...
Perspectives on c-Myc, Cyclin D1, and their interaction in cancer formation, progression, and response to chemotherapyD Joshua Liao
Hormel Institute, University of Minnesota, 801 16th Ave NE, Austin, MN 55912, USA
Crit Rev Oncog 13:93-158. 2007....
Splicing of mouse p53 pre-mRNA does not always follow the "first come, first served" principle and may be influenced by cisplatin treatment and serum starvationMin Yang
Hormel Institute, University of Minnesota, 801 16th Avenue, NE, Austin, MN 55912, USA
Mol Biol Rep 39:9247-56. 2012..It remains possible that these p53-ICTs are splicing intermediates existing as a mechanism for the cell to respond more promptly to a demand for more p53 and that p53 protein may be required for a normal life of MEF...
CyclinD1 protein plays different roles in modulating chemoresponses in MCF7 and MDA-MB231 cellsYuan Sun
Department of Pathology, Guangxi Medical University, Nanning, Guangxi 530 021, P R China Hormel Institute, University of Minnesota, Austin, MN, USA
J Carcinog 11:12. 2012..The elevated cyclinD1 in some types of cancers is believed to be associated with tumor progression and response to systemic treatments...
Pseudogenes as weaknesses of ACTB (Actb) and GAPDH (Gapdh) used as reference genes in reverse transcription and polymerase chain reactionsYuan Sun
Hormel Institute, University of Minnesota, Austin, Minnesota, United States of America
PLoS ONE 7:e41659. 2012..We also propose a standard operation procedure in which design of primers for RT-PCR starts from avoiding mis-priming PGs and all primers need be tested for specificity with both cDNA and gDNA...
Expression profile of microRNAs in c-Myc induced mouse mammary tumorsYuan Sun
Hormel Institute, University of Minnesota, Austin, MN, 55912, USA
Breast Cancer Res Treat 118:185-96. 2009..These results suggest that certain mirRNAs and the chromosome 19 derived non-coding RNAs may mediate c-myc induced mammary carcinogenesis...
Effects of an Indolocarbazole-Derived CDK4 Inhibitor on Breast Cancer CellsYuan Sun
1 Hormel Institute, University of Minnesota, Austin, MN 55912, USA
J Cancer 2:36-51. 2011..Decreased phosphorylation of Rb by D1-CDK4/6 and decreased p27kip1 protein level may be part of the underlying mechanism...
Cyclin D1 inhibits whereas c-Myc enhances the cytotoxicity of cisplatin in mouse pancreatic cancer cells via regulation of several members of the NF-κB and Bcl-2 familiesAyman El-Kady
Department of the University of Minnesota, Hormel Institute, Austin, MN 55912, USA
J Carcinog 10:24. 2011..This study aims to determine whether and how cyclin D1 (D1) and c-Myc influence the response of pancreatic cancer cells to CDDP...
A quick, cost-free method of purification of DNA fragments from agarose gelYuan Sun
1 Hormel Institute, The University of Minnesota, Austin, MN 55912, USA
J Cancer 3:93-5. 2012....
Complex alternative splicing of the smarca2 gene suggests the importance of smarca2-B variantsMin Yang
1 Hormel Institute, The University of Minnesota, Austin, MN 55912, USA
J Cancer 2:386-400. 2011....
Synergistic effect of cyclin D1 and c-Myc leads to more aggressive and invasive mammary tumors in severe combined immunodeficient miceYong Wang
Department of Pathology, Barbara Ann Karmanos Cancer Institute, Hudson Webber Cancer Research Center, Wayne State University School of Medicine, Detroit, MI, USA
Cancer Res 67:3698-707. 2007..In summary, overexpression of cyclin D1 may commit mammary epithelia to a tumor-prone phenotype in which cooperation with other genes, such as synergy with c-myc, may lead to a more aggressive phenotype...
