P Krogstad

Summary

Affiliation: University of California
Country: USA

Publications

  1. ncbi Treatment of human immunodeficiency virus 1-infected infants and children with the protease inhibitor nelfinavir mesylate
    P Krogstad
    Department of Pediatrics, UCLA, Los Angeles, California, 90095 3075, USA
    Clin Infect Dis 28:1109-18. 1999
  2. ncbi Genetic evaluation of suspected cases of transient HIV-1 infection of infants
    L M Frenkel
    Department of Pediatrics, University of Rochester, Rochester, NY 14642, USA
    Science 280:1073-7. 1998
  3. ncbi HIV-1 Gag protein associates with F-actin present in microfilaments
    O Rey
    Department of Pediatrics, School of Medicine, University of California at Los Angeles 90095, USA
    Virology 220:530-4. 1996
  4. ncbi Cryoelectron microscopic examination of human immunodeficiency virus type 1 virions with mutations in the cyclophilin A binding loop
    L B Kong
    Department of Molecular and Medical Pharmacology and Crump Institute for Biological Imaging, UCLA School of Medicine, Los Angeles, California 90095, USA
    J Virol 72:4403-7. 1998
  5. ncbi HIV type 1 Gag and nucleocapsid proteins: cytoskeletal localization and effects on cell motility
    F J Ibarrondo
    Department of Pediatrics, UCLA School of Medicine, Los Angeles, California 90095, USA
    AIDS Res Hum Retroviruses 17:1489-500. 2001
  6. ncbi Cells with high cyclophilin A content support replication of human immunodeficiency virus type 1 Gag mutants with decreased ability to incorporate cyclophilin A
    B Ackerson
    Department of Pediatrics, University of California, Los Angeles 90095, USA
    J Virol 72:303-8. 1998
  7. ncbi Performance of the applied biosystems ViroSeq human immunodeficiency virus type 1 (HIV-1) genotyping system for sequence-based analysis of HIV-1 in pediatric plasma samples
    S Cunningham
    Department of Pathology, Johns Hopkins Medical Institutions, Baltimore, Maryland 21205, USA
    J Clin Microbiol 39:1254-7. 2001

Collaborators

Detail Information

Publications7

  1. ncbi Treatment of human immunodeficiency virus 1-infected infants and children with the protease inhibitor nelfinavir mesylate
    P Krogstad
    Department of Pediatrics, UCLA, Los Angeles, California, 90095 3075, USA
    Clin Infect Dis 28:1109-18. 1999
    ..Suppression of viremia was achieved in children as young as 3 months of age...
  2. ncbi Genetic evaluation of suspected cases of transient HIV-1 infection of infants
    L M Frenkel
    Department of Pediatrics, University of Rochester, Rochester, NY 14642, USA
    Science 280:1073-7. 1998
    ..These findings argue that transient HIV-1 infection, if it exists, will only rarely be satisfactorily documented...
  3. ncbi HIV-1 Gag protein associates with F-actin present in microfilaments
    O Rey
    Department of Pediatrics, School of Medicine, University of California at Los Angeles 90095, USA
    Virology 220:530-4. 1996
    ..In vivo and in vitro analyses of this interaction indicated that the unprocessed Gag polyprotein is capable of association with polymerized actin (F-actin). Binding of Gag to F-actin may be involved in the assembly or budding of HIV-1...
  4. ncbi Cryoelectron microscopic examination of human immunodeficiency virus type 1 virions with mutations in the cyclophilin A binding loop
    L B Kong
    Department of Molecular and Medical Pharmacology and Crump Institute for Biological Imaging, UCLA School of Medicine, Los Angeles, California 90095, USA
    J Virol 72:4403-7. 1998
    ..Although viral RNA incorporation and protease cleavage of the Gag precursor were not affected by these mutations, cryoelectron microscopy revealed a loss of virion maturation in P231A particles...
  5. ncbi HIV type 1 Gag and nucleocapsid proteins: cytoskeletal localization and effects on cell motility
    F J Ibarrondo
    Department of Pediatrics, UCLA School of Medicine, Los Angeles, California 90095, USA
    AIDS Res Hum Retroviruses 17:1489-500. 2001
    ..These data suggest that interactions between HIV-1 Gag and actin in infected cells enhance cell motility. Ultimately this enhanced motility of infected cells could promote the dissemination of virus into the brain and other tissues...
  6. ncbi Cells with high cyclophilin A content support replication of human immunodeficiency virus type 1 Gag mutants with decreased ability to incorporate cyclophilin A
    B Ackerson
    Department of Pediatrics, University of California, Los Angeles 90095, USA
    J Virol 72:303-8. 1998
    ..These experiments demonstrated that a higher cellular CyPA content of some cells was able to compensate for the decreased binding affinity of P222A mutant Gag for CyPA, allowing virus replication to occur...
  7. ncbi Performance of the applied biosystems ViroSeq human immunodeficiency virus type 1 (HIV-1) genotyping system for sequence-based analysis of HIV-1 in pediatric plasma samples
    S Cunningham
    Department of Pathology, Johns Hopkins Medical Institutions, Baltimore, Maryland 21205, USA
    J Clin Microbiol 39:1254-7. 2001
    ..This genotyping system performs well for analysis of HIV-1 in pediatric plasma samples, including those with low volume and low viral load. The availability of this system should facilitate studies of HIV-1 drug resistance...