Dimitry N Krementsov

Summary

Affiliation: University of Vermont
Country: USA

Publications

  1. pmc Tetraspanins regulate cell-to-cell transmission of HIV-1
    Dimitry N Krementsov
    Department of Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405, USA
    Retrovirology 6:64. 2009
  2. pmc Formation of syncytia is repressed by tetraspanins in human immunodeficiency virus type 1-producing cells
    Jia Weng
    Department of Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405, USA
    J Virol 83:7467-74. 2009
  3. ncbi HIV-1 assembly differentially alters dynamics and partitioning of tetraspanins and raft components
    Dimitry N Krementsov
    Graduate Program in Cell and Molecular Biology, University of Vermont, Burlington, VT 05405, USA
    Traffic 11:1401-14. 2010
  4. pmc Human immunodeficiency virus type 1 and influenza virus exit via different membrane microdomains
    Sandhya Khurana
    Department of Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405, USA
    J Virol 81:12630-40. 2007
  5. pmc Mapping of tetraspanin-enriched microdomains that can function as gateways for HIV-1
    Sascha Nydegger
    Graduate Program in Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405, USA
    J Cell Biol 173:795-807. 2006
  6. ncbi Sex-specific control of central nervous system autoimmunity by p38 mitogen-activated protein kinase signaling in myeloid cells
    Dimitry N Krementsov
    Department of Medicine, Immunobiology Program, University of Vermont, Burlington, VT
    Ann Neurol 75:50-66. 2014
  7. ncbi Histamine H2 receptor signaling x environment interactions determine susceptibility to experimental allergic encephalomyelitis
    Naresha Saligrama
    1Immunobiology Program, C331 Given Medical Building, University of Vermont, Burlington, VT 05405, USA
    FASEB J 28:1898-909. 2014

Collaborators

Detail Information

Publications7

  1. pmc Tetraspanins regulate cell-to-cell transmission of HIV-1
    Dimitry N Krementsov
    Department of Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405, USA
    Retrovirology 6:64. 2009
    ..Here we used overexpression and knockdown approaches to address this question...
  2. pmc Formation of syncytia is repressed by tetraspanins in human immunodeficiency virus type 1-producing cells
    Jia Weng
    Department of Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405, USA
    J Virol 83:7467-74. 2009
    ..Overall, these data establish tetraspanins as negative regulators of HIV-1-induced cell-cell fusion, and they start delineating the requirements for this regulation...
  3. ncbi HIV-1 assembly differentially alters dynamics and partitioning of tetraspanins and raft components
    Dimitry N Krementsov
    Graduate Program in Cell and Molecular Biology, University of Vermont, Burlington, VT 05405, USA
    Traffic 11:1401-14. 2010
    ..Importantly, our findings support the emerging concept that cellular and viral components, instead of clustering at preexisting microdomain platforms, direct the formation of distinct domains for the execution of specific functions...
  4. pmc Human immunodeficiency virus type 1 and influenza virus exit via different membrane microdomains
    Sandhya Khurana
    Department of Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405, USA
    J Virol 81:12630-40. 2007
    ..Altogether, these data strongly suggest that the bimodal egress of these two pathogenic viruses, like their entry into target cells, is guided by specific sets of host cell proteins...
  5. pmc Mapping of tetraspanin-enriched microdomains that can function as gateways for HIV-1
    Sascha Nydegger
    Graduate Program in Microbiology and Molecular Genetics, University of Vermont, Burlington, VT 05405, USA
    J Cell Biol 173:795-807. 2006
    ....
  6. ncbi Sex-specific control of central nervous system autoimmunity by p38 mitogen-activated protein kinase signaling in myeloid cells
    Dimitry N Krementsov
    Department of Medicine, Immunobiology Program, University of Vermont, Burlington, VT
    Ann Neurol 75:50-66. 2014
    ..Investigators have described p38 mitogen-activated protein kinase (MAPK) as a key regulator of inflammatory responses in autoimmunity, but its role in the sexual dimorphism in MS or MS models remains unexplored...
  7. ncbi Histamine H2 receptor signaling x environment interactions determine susceptibility to experimental allergic encephalomyelitis
    Naresha Saligrama
    1Immunobiology Program, C331 Given Medical Building, University of Vermont, Burlington, VT 05405, USA
    FASEB J 28:1898-909. 2014
    ..Saligrama, N., Case, L. K., Krementsov, D. N., Teuscher, C. Histamine H2 receptor signaling × environment interactions determine susceptibility to experimental allergic encephalomyelitis. ..