P Kofuji

Summary

Affiliation: University of Minnesota
Country: USA

Publications

  1. ncbi Genetic inactivation of an inwardly rectifying potassium channel (Kir4.1 subunit) in mice: phenotypic impact in retina
    P Kofuji
    Departments of Neuroscience and Physiology, University of Minnesota, Minneapolis 55455, USA
    J Neurosci 20:5733-40. 2000
  2. ncbi Inwardly rectifying potassium channel Kir4.1 is responsible for the native inward potassium conductance of satellite glial cells in sensory ganglia
    X Tang
    Department of Neuroscience, University of Minnesota, 6 145 Jackson Hall, 321 Church Street SE, Minneapolis, MN 55455, USA
    Neuroscience 166:397-407. 2010
  3. ncbi Stoichiometry of N-methyl-D-aspartate receptors within the suprachiasmatic nucleus
    J P Clark
    Department of Neuroscience, University of Minnesota, 6 145 Jackson Hall, 321 Church St SE, Minneapolis, MN 55455, USA
    J Neurophysiol 103:3448-64. 2010
  4. ncbi Potassium buffering in the central nervous system
    P Kofuji
    Department of Neuroscience, University of Minnesota, 6 145 Jackson Hall, 321 Church Street SE, Minneapolis, MN 55455, USA
    Neuroscience 129:1045-56. 2004
  5. ncbi Kir4.1 potassium channel subunit is crucial for oligodendrocyte development and in vivo myelination
    C Neusch
    Division of Biology, California Institute of Technology, Pasadena, California 91125, USA
    J Neurosci 21:5429-38. 2001
  6. ncbi Functional analysis of the weaver mutant GIRK2 K+ channel and rescue of weaver granule cells
    P Kofuji
    Division of Biology, California Institute of Technology, Pasadena, California 91125, USA
    Neuron 16:941-52. 1996
  7. ncbi A regenerative link in the ionic fluxes through the weaver potassium channel underlies the pathophysiology of the mutation
    S K Silverman
    Division of Chemistry and Chemical Engineering, California Institute of Technology, Pasadena 91125, USA
    Proc Natl Acad Sci U S A 93:15429-34. 1996
  8. ncbi The GABA(A) receptor gamma1-subunit in seizure prone (DBA/2) and resistant (C57BL/6) mice
    J B Wang
    Department of Pharmacology, University of Maryland School of Medicine, Baltimore 21201 1559, USA
    Brain Res Bull 45:421-5. 1998
  9. ncbi Na+/Ca2+ exchanger in Drosophila: cloning, expression, and transport differences
    A Ruknudin
    Department of Microbiology and Immunology, University of Maryland School of Medicine, Baltimore, Maryland, USA
    Am J Physiol 273:C257-65. 1997
  10. ncbi GABAA receptor beta 1, beta 2, and beta 3 subunits: comparisons in DBA/2J and C57BL/6J mice
    G L Kamatchi
    Department of Pharmacology and Experimental Therapeutics, University of Maryland School of Medicine, Baltimore 21201 1559
    Biochim Biophys Acta 1261:134-42. 1995

Collaborators

Detail Information

Publications13

  1. ncbi Genetic inactivation of an inwardly rectifying potassium channel (Kir4.1 subunit) in mice: phenotypic impact in retina
    P Kofuji
    Departments of Neuroscience and Physiology, University of Minnesota, Minneapolis 55455, USA
    J Neurosci 20:5733-40. 2000
    ..The highly regulated localization and the functional properties of Kir4.1 in Müller cells suggest the involvement of this channel in the regulation of extracellular K(+) in the mouse retina...
  2. ncbi Inwardly rectifying potassium channel Kir4.1 is responsible for the native inward potassium conductance of satellite glial cells in sensory ganglia
    X Tang
    Department of Neuroscience, University of Minnesota, 6 145 Jackson Hall, 321 Church Street SE, Minneapolis, MN 55455, USA
    Neuroscience 166:397-407. 2010
    ..1 is the principal Kir channel type in SGCs. Therefore Kir4.1 emerges as a key regulator of SGC function and possibly neuronal excitability in sensory ganglia...
  3. ncbi Stoichiometry of N-methyl-D-aspartate receptors within the suprachiasmatic nucleus
    J P Clark
    Department of Neuroscience, University of Minnesota, 6 145 Jackson Hall, 321 Church St SE, Minneapolis, MN 55455, USA
    J Neurophysiol 103:3448-64. 2010
    ....
  4. ncbi Potassium buffering in the central nervous system
    P Kofuji
    Department of Neuroscience, University of Minnesota, 6 145 Jackson Hall, 321 Church Street SE, Minneapolis, MN 55455, USA
    Neuroscience 129:1045-56. 2004
    ..We also discuss intriguing new data that suggest a close physical and functional relationship between Kir and water channels in glial cells...
  5. ncbi Kir4.1 potassium channel subunit is crucial for oligodendrocyte development and in vivo myelination
    C Neusch
    Division of Biology, California Institute of Technology, Pasadena, California 91125, USA
    J Neurosci 21:5429-38. 2001
    ..Therefore, this mouse shows how an ion channel mutation could contribute to the polygenic demyelinating diseases...
  6. ncbi Functional analysis of the weaver mutant GIRK2 K+ channel and rescue of weaver granule cells
    P Kofuji
    Division of Biology, California Institute of Technology, Pasadena, California 91125, USA
    Neuron 16:941-52. 1996
    ..Pharmacological blockade of the Na+ influx in weaver granule cells restores their ability to differentiate normally. Thus, Na+ flux through the weaver GIRK2 channel underlies the failure of granule cell development in situ...
  7. ncbi A regenerative link in the ionic fluxes through the weaver potassium channel underlies the pathophysiology of the mutation
    S K Silverman
    Division of Chemistry and Chemical Engineering, California Institute of Technology, Pasadena 91125, USA
    Proc Natl Acad Sci U S A 93:15429-34. 1996
    ....
  8. ncbi The GABA(A) receptor gamma1-subunit in seizure prone (DBA/2) and resistant (C57BL/6) mice
    J B Wang
    Department of Pharmacology, University of Maryland School of Medicine, Baltimore 21201 1559, USA
    Brain Res Bull 45:421-5. 1998
    ..No differences have been found for the subunit between DBA/2 and C57BL/6 mice either for nucleotide sequence or for level of expression of the subunit's mRNA in whole brain by Northern blots at 3 weeks of age...
  9. ncbi Na+/Ca2+ exchanger in Drosophila: cloning, expression, and transport differences
    A Ruknudin
    Department of Microbiology and Immunology, University of Maryland School of Medicine, Baltimore, Maryland, USA
    Am J Physiol 273:C257-65. 1997
    ....
  10. ncbi GABAA receptor beta 1, beta 2, and beta 3 subunits: comparisons in DBA/2J and C57BL/6J mice
    G L Kamatchi
    Department of Pharmacology and Experimental Therapeutics, University of Maryland School of Medicine, Baltimore 21201 1559
    Biochim Biophys Acta 1261:134-42. 1995
    ..We have also tested mouse beta 1 and beta 2 subunits for internal alternative splicing, detecting none...
  11. ncbi Generation of two forms of the gamma-aminobutyric acidA receptor gamma 2-subunit in mice by alternative splicing
    P Kofuji
    Department of Pharmacology, University of Maryland School of Medicine, Baltimore 21201
    J Neurochem 56:713-5. 1991
    ..The eight-amino-acid insertion encodes a potential consensus serine phosphorylation site for protein kinase C. These results suggest a novel mechanism for the regulation of the GABAA receptor by protein phosphorylation...
  12. ncbi The alpha 1, alpha 2, and alpha 3 subunits of GABAA receptors: comparison in seizure-prone and -resistant mice and during development
    J B Wang
    Department of Pharmacology, University of Maryland School of Medicine, Baltimore 21201
    J Mol Neurosci 3:177-84. 1992
    ....
  13. ncbi Strain comparisons and developmental profile of the delta subunit of the murine GABAA receptor
    J B Wang
    Department of Pharmacology, University of Maryland School of Medicine, Baltimore 21201
    Brain Res Bull 29:119-23. 1992
    ....