Suil Kim

Summary

Affiliation: University of California
Country: USA

Publications

  1. pmc Fibrinogen binding to ICAM-1 promotes EGFR-dependent mucin production in human airway epithelial cells
    Suil Kim
    Department of Medicine, Cardiovascular Research Institute, University of California, San Francisco, California 94143 0130, USA
    Am J Physiol Lung Cell Mol Physiol 297:L174-83. 2009
  2. doi request reprint CCL20/CCR6 feedback exaggerates epidermal growth factor receptor-dependent MUC5AC mucin production in human airway epithelial (NCI-H292) cells
    Suil Kim
    Cardiovascular Research Institute, University of California San Francisco, San Francisco, CA 94143, USA
    J Immunol 186:3392-400. 2011
  3. doi request reprint Epidermal growth factor receptor reactivation induced by E-prostanoid-3 receptor- and tumor necrosis factor-alpha-converting enzyme-dependent feedback exaggerates interleukin-8 production in airway cancer (NCI-H292) cells
    Suil Kim
    Cardiovascular Research Institute, University of California San Francisco, San Francisco, CA 94143 0130, USA
    Exp Cell Res 317:2650-60. 2011
  4. ncbi request reprint Role of neutrophils in mucus hypersecretion in COPD and implications for therapy
    Suil Kim
    Cardiovascular Research Institute, Cancer Center, and Department of Medicine, University of California San Francisco, 94143 0130, USA
    Treat Respir Med 3:147-59. 2004
  5. ncbi request reprint E-cadherin promotes EGFR-mediated cell differentiation and MUC5AC mucin expression in cultured human airway epithelial cells
    Suil Kim
    Cardiovascular Research Institute, Box 0130, University of California San Francisco, San Francisco, CA 94143 0130, USA
    Am J Physiol Lung Cell Mol Physiol 289:L1049-60. 2005
  6. ncbi request reprint Pseudomonas lipopolysaccharide accelerates wound repair via activation of a novel epithelial cell signaling cascade
    Jonathan L Koff
    Cardiovascular Research Institute, Department of Medicine, University of California San Francisco, 505 Parnassus Avenue, San Francisco, CA 94143, USA
    J Immunol 177:8693-700. 2006
  7. ncbi request reprint IL-13-induced Clara cell secretory protein expression in airway epithelium: role of EGFR signaling pathway
    Suil Kim
    Cardiovascular Research Institute and Department of Medicine, University of California, San Francisco, California 94143 0130, USA
    Am J Physiol Lung Cell Mol Physiol 283:L67-75. 2002

Research Grants

Collaborators

Detail Information

Publications7

  1. pmc Fibrinogen binding to ICAM-1 promotes EGFR-dependent mucin production in human airway epithelial cells
    Suil Kim
    Department of Medicine, Cardiovascular Research Institute, University of California, San Francisco, California 94143 0130, USA
    Am J Physiol Lung Cell Mol Physiol 297:L174-83. 2009
    ....
  2. doi request reprint CCL20/CCR6 feedback exaggerates epidermal growth factor receptor-dependent MUC5AC mucin production in human airway epithelial (NCI-H292) cells
    Suil Kim
    Cardiovascular Research Institute, University of California San Francisco, San Francisco, CA 94143, USA
    J Immunol 186:3392-400. 2011
    ..These findings have potentially important therapeutic implications in obstructive airway diseases...
  3. doi request reprint Epidermal growth factor receptor reactivation induced by E-prostanoid-3 receptor- and tumor necrosis factor-alpha-converting enzyme-dependent feedback exaggerates interleukin-8 production in airway cancer (NCI-H292) cells
    Suil Kim
    Cardiovascular Research Institute, University of California San Francisco, San Francisco, CA 94143 0130, USA
    Exp Cell Res 317:2650-60. 2011
    ..Thus, we conclude that a positive feedback pathway involving COX-2/PGE2/EP3 receptor-dependent EGFR reactivation exaggerates IL-8 production in NCI-H292 cancer cells but not in NHBE (normal) cells...
  4. ncbi request reprint Role of neutrophils in mucus hypersecretion in COPD and implications for therapy
    Suil Kim
    Cardiovascular Research Institute, Cancer Center, and Department of Medicine, University of California San Francisco, 94143 0130, USA
    Treat Respir Med 3:147-59. 2004
    ..Interventions that target EGFR, neutrophil elastase, and reactive oxygen species exist and can be evaluated as treatments for neutrophil-dependent mucus hypersecretion...
  5. ncbi request reprint E-cadherin promotes EGFR-mediated cell differentiation and MUC5AC mucin expression in cultured human airway epithelial cells
    Suil Kim
    Cardiovascular Research Institute, Box 0130, University of California San Francisco, San Francisco, CA 94143 0130, USA
    Am J Physiol Lung Cell Mol Physiol 289:L1049-60. 2005
    ..Thus E-cadherin promotes EGFR-mediated cell differentiation and MUC5AC production, and our results suggest that this occurs via a pathway involving protein tyrosine phosphatase-dependent EGFR dephosphorylation...
  6. ncbi request reprint Pseudomonas lipopolysaccharide accelerates wound repair via activation of a novel epithelial cell signaling cascade
    Jonathan L Koff
    Cardiovascular Research Institute, Department of Medicine, University of California San Francisco, 505 Parnassus Avenue, San Francisco, CA 94143, USA
    J Immunol 177:8693-700. 2006
    ..Thus, LPS accelerates wound repair in airway epithelial cells via a novel TLR-4-->protein kinase C alphabeta-->dual oxidase 1-->reactive oxygen species-->TACE-->TGF-alpha-->EGFR phosphorylation pathway...
  7. ncbi request reprint IL-13-induced Clara cell secretory protein expression in airway epithelium: role of EGFR signaling pathway
    Suil Kim
    Cardiovascular Research Institute and Department of Medicine, University of California, San Francisco, California 94143 0130, USA
    Am J Physiol Lung Cell Mol Physiol 283:L67-75. 2002
    ..These findings indicate that IL-13 induces CCSP expression via an EGFR- and leukocyte-dependent pathway...

Research Grants2