M Katherine Kelm

Summary

Affiliation: University of North Carolina
Country: USA

Publications

  1. ncbi Calcium release from presynaptic internal stores is required for ethanol to increase spontaneous gamma-aminobutyric acid release onto cerebellum Purkinje neurons
    M Katherine Kelm
    Bowles Center for Alcohol Studies, CB 7178, Thurston Bowles Building, Chapel Hill, NC 27599 7178, USA
    J Pharmacol Exp Ther 323:356-64. 2007
  2. ncbi The role of protein kinase A in the ethanol-induced increase in spontaneous GABA release onto cerebellar Purkinje neurons
    M Katherine Kelm
    Department of Pharmacology, University of North Carolina at Chapel Hill, Chapel Hill, NC 27599 7178, USA
    J Neurophysiol 100:3417-28. 2008
  3. ncbi The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release
    M Katherine Kelm
    Department of Pharmacology, School of Medicine, University of North Carolina, Chapel Hill, NC 27599 7365, USA
    Neuropharmacology 58:1179-86. 2010
  4. ncbi Brain regional differences in the effect of ethanol on GABA release from presynaptic terminals
    Hugh E Criswell
    Bowles Center for Alcohol Studies, University of North Carolina, Chapel Hill, NC 27599, USA
    J Pharmacol Exp Ther 326:596-603. 2008
  5. ncbi Ethanol-enhanced GABA release: a focus on G protein-coupled receptors
    M Katherine Kelm
    Department of Pharmacology, School of Medicine, University of North Carolina at Chapel Hill, NC 27599 7178, USA
    Brain Res Rev 65:113-23. 2011

Detail Information

Publications5

  1. ncbi Calcium release from presynaptic internal stores is required for ethanol to increase spontaneous gamma-aminobutyric acid release onto cerebellum Purkinje neurons
    M Katherine Kelm
    Bowles Center for Alcohol Studies, CB 7178, Thurston Bowles Building, Chapel Hill, NC 27599 7178, USA
    J Pharmacol Exp Ther 323:356-64. 2007
    ..Overall, these data suggest that calcium release from presynaptic internal stores plays a vital role in the mechanism of ethanol-enhanced spontaneous GABA release at the interneuron-Purkinje cell synapse...
  2. ncbi The role of protein kinase A in the ethanol-induced increase in spontaneous GABA release onto cerebellar Purkinje neurons
    M Katherine Kelm
    Department of Pharmacology, University of North Carolina at Chapel Hill, Chapel Hill, NC 27599 7178, USA
    J Neurophysiol 100:3417-28. 2008
    ..Overall, these results suggest that PKA activation is necessary for ethanol to increase spontaneous GABA release...
  3. ncbi The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release
    M Katherine Kelm
    Department of Pharmacology, School of Medicine, University of North Carolina, Chapel Hill, NC 27599 7365, USA
    Neuropharmacology 58:1179-86. 2010
    ..Overall, these results suggest that activation of the PLC/IP(3)R/PKC pathway is necessary for ethanol to increase spontaneous GABA release from presynaptic terminals onto Purkinje cells...
  4. ncbi Brain regional differences in the effect of ethanol on GABA release from presynaptic terminals
    Hugh E Criswell
    Bowles Center for Alcohol Studies, University of North Carolina, Chapel Hill, NC 27599, USA
    J Pharmacol Exp Ther 326:596-603. 2008
    ..This regional difference is consistent with earlier in vivo studies in which ethanol affected neural activity and sensitivity to GABA in some, but not all, brain sites...
  5. ncbi Ethanol-enhanced GABA release: a focus on G protein-coupled receptors
    M Katherine Kelm
    Department of Pharmacology, School of Medicine, University of North Carolina at Chapel Hill, NC 27599 7178, USA
    Brain Res Rev 65:113-23. 2011
    ....