Joseph O Humtsoe

Summary

Affiliation: University of California
Country: USA

Publications

  1. doi request reprint Transcriptional profiling identifies upregulated genes following induction of epithelial-mesenchymal transition in squamous carcinoma cells
    Joseph O Humtsoe
    Department of Cell and Tissue Biology, School of Dentistry, 521 Parnassus Avenue, University of California at San Francisco, San Francisco, CA 94143, USA
    Exp Cell Res 318:379-90. 2012
  2. pmc Lipid phosphate phosphatase-3 regulates tumor growth via β-catenin and CYCLIN-D1 signaling
    Ishita Chatterjee
    Dept of Pharmacology, University of Illinois, 835 S Wolcott, Room E403, Chicago, IL 60612, USA
    Mol Cancer 10:51. 2011
  3. doi request reprint Bcl-2 overexpression induces a partial epithelial to mesenchymal transition and promotes squamous carcinoma cell invasion and metastasis
    Jianhong Zuo
    Department of Cell and Tissue Biology, University of California San Francisco, 521 Parnassus Avenue, San Francisco, CA 94143 0640, USA
    Mol Cancer Res 8:170-82. 2010
  4. pmc STAT3 signaling is induced by intercellular adhesion in squamous cell carcinoma cells
    Akiko Onishi
    Department of Cell and Tissue Biology, University of California San Francisco, San Francisco, California 94143 0640, USA
    Exp Cell Res 314:377-86. 2008
  5. pmc Lipid phosphate phosphatase 3 stabilization of beta-catenin induces endothelial cell migration and formation of branching point structures
    Joseph O Humtsoe
    Department of Cell and Tissue Biology, University of California San Francisco, San Francisco, CA 94143, USA
    Mol Cell Biol 30:1593-606. 2010

Collaborators

  • Randall H Kramer
  • Kishore K Wary
  • Ishita Chatterjee
  • Jianhong Zuo
  • Akiko Onishi
  • Erin E Kohler
  • Claudio Sorio
  • Tohru Ishikawa
  • Zhiqiang Xiao
  • Shadi Boutros
  • Qianming Chen

Detail Information

Publications5

  1. doi request reprint Transcriptional profiling identifies upregulated genes following induction of epithelial-mesenchymal transition in squamous carcinoma cells
    Joseph O Humtsoe
    Department of Cell and Tissue Biology, School of Dentistry, 521 Parnassus Avenue, University of California at San Francisco, San Francisco, CA 94143, USA
    Exp Cell Res 318:379-90. 2012
    ..This set of newly identified genes that are modulated during EMT-like conversion may be important diagnostic biomarkers during the process of HNSCC progression...
  2. pmc Lipid phosphate phosphatase-3 regulates tumor growth via β-catenin and CYCLIN-D1 signaling
    Ishita Chatterjee
    Dept of Pharmacology, University of Illinois, 835 S Wolcott, Room E403, Chicago, IL 60612, USA
    Mol Cancer 10:51. 2011
    ..The goal of this study was to determine the role of LPP3 in tumor cells...
  3. doi request reprint Bcl-2 overexpression induces a partial epithelial to mesenchymal transition and promotes squamous carcinoma cell invasion and metastasis
    Jianhong Zuo
    Department of Cell and Tissue Biology, University of California San Francisco, 521 Parnassus Avenue, San Francisco, CA 94143 0640, USA
    Mol Cancer Res 8:170-82. 2010
    ....
  4. pmc STAT3 signaling is induced by intercellular adhesion in squamous cell carcinoma cells
    Akiko Onishi
    Department of Cell and Tissue Biology, University of California San Francisco, San Francisco, California 94143 0640, USA
    Exp Cell Res 314:377-86. 2008
    ..These results indicate that, in HNSCC cells, cadherin-mediated intercellular adhesion induces STAT signaling that may modulate cell survival and resistance to apoptosis during tumor progression...
  5. pmc Lipid phosphate phosphatase 3 stabilization of beta-catenin induces endothelial cell migration and formation of branching point structures
    Joseph O Humtsoe
    Department of Cell and Tissue Biology, University of California San Francisco, San Francisco, CA 94143, USA
    Mol Cell Biol 30:1593-606. 2010
    ..Thus, our study identified a key role for LPP3 in orchestrating PTEN-mediated beta-catenin/LEF-1 signaling in EC migration, cell-cell adhesion, and formation of branching point structures...