Sally A Huber

Summary

Affiliation: University of Vermont
Country: USA

Publications

  1. pmc Sudden unexpected death related to enterovirus myocarditis: histopathology, immunohistochemistry and molecular pathology diagnosis at post-mortem
    Imed Gaaloul
    Laboratory of Transmissible Diseases LR99 ES27, Faculty of Pharmacy, Avenue Avicenne, 5000, Monastir, Tunisia
    BMC Infect Dis 12:212. 2012
  2. pmc Sex-specific signaling through Toll-Like Receptors 2 and 4 contributes to survival outcome of Coxsackievirus B3 infection in C57Bl/6 mice
    Brian J Roberts
    Department of Pathology, Center for Immunology and Infectious Disease, University of Vermont, Burlington, VT, USA
    Biol Sex Differ 3:25. 2012
  3. pmc Slam haplotype 2 promotes NKT but suppresses Vγ4+ T-cell activation in coxsackievirus B3 infection leading to increased liver damage but reduced myocarditis
    Sally Ann Huber
    Department of Pathology, University of Vermont, Burlington, Vermont 05446, USA
    Am J Pathol 182:401-9. 2013
  4. pmc Cross-talk between cd1d-restricted nkt cells and γδ cells in t regulatory cell response
    Wei Liu
    The First Affiliated Hospital of Harbin Medical University, Harbin 150001, PR China
    Virol J 8:32. 2011
  5. pmc Sex chromosome complement contributes to sex differences in coxsackievirus B3 but not influenza A virus pathogenesis
    Dionne P Robinson
    The W Harry Feinstone Department of Molecular Microbiology and Immunology, The Johns Hopkins Bloomberg School of Public Health, Baltimore, MD 21205, USA
    Biol Sex Differ 2:8. 2011
  6. ncbi request reprint T cells in coxsackievirus-induced myocarditis
    Sally Huber
    Department of Pathology, University of Vermont, Bington, Vermont 05405, USA
    Viral Immunol 17:152-64. 2004
  7. ncbi request reprint CD1d expression on hemopoietic cells promotes CD4+ Th1 response in coxsackievirus B3 induced myocarditis
    S A Huber
    University of Vermont, Department of Pathology, 208 South Park Drive, Suite 2, Burlington, VT 05446, USA
    Virology 352:226-36. 2006
  8. ncbi request reprint Autoimmunity in coxsackievirus B3 induced myocarditis
    Sally A Huber
    University of Vermont, Department of Pathology, Colchester, VT 05446, USA
    Autoimmunity 39:55-61. 2006
  9. ncbi request reprint Decay-accelerating factor (CD55) promotes CD1d expression and Vgamma4+ T-cell activation in coxsackievirus B3-induced myocarditis
    Sally Huber
    Department of Pathology, University of Vermont, Colchester, Vermont 05446, USA
    Viral Immunol 19:156-66. 2006
  10. ncbi request reprint Cellular autoimmunity in myocarditis
    Sally Huber
    University of Vermont, Colchester, VT 05446, USA
    Heart Fail Clin 1:321-31. 2005

Research Grants

  1. CDI and Gamma Delta+ in Viral Myocarditis
    Sally Huber; Fiscal Year: 2005
  2. Sex in Myocarditis
    Sally Huber; Fiscal Year: 2005
  3. Sex in Myocarditis
    Sally Huber; Fiscal Year: 2006
  4. T reg Cells in Myocarditis
    Sally Huber; Fiscal Year: 2007
  5. Sex in Myocarditis
    Sally Huber; Fiscal Year: 2007
  6. T reg Cells in Myocarditis
    Sally Huber; Fiscal Year: 2009
  7. CDI and Gamma Delta+ in Viral Myocarditis
    Sally Huber; Fiscal Year: 2004
  8. Sex in Viral Myocarditis
    Sally Huber; Fiscal Year: 2004
  9. CDI and Gamma Delta+ in Viral Myocarditis
    Sally Huber; Fiscal Year: 2003
  10. TH1 AND TH2 RESPONSES IN PATHOGENIC CVB3 INFECTIONS
    Sally Huber; Fiscal Year: 1999

Collaborators

Detail Information

Publications41

  1. pmc Sudden unexpected death related to enterovirus myocarditis: histopathology, immunohistochemistry and molecular pathology diagnosis at post-mortem
    Imed Gaaloul
    Laboratory of Transmissible Diseases LR99 ES27, Faculty of Pharmacy, Avenue Avicenne, 5000, Monastir, Tunisia
    BMC Infect Dis 12:212. 2012
    ..When supplemented with immunohistochemistry and molecular pathology, conventional histopathology may provide important clues regarding myocarditis underlying etiology...
  2. pmc Sex-specific signaling through Toll-Like Receptors 2 and 4 contributes to survival outcome of Coxsackievirus B3 infection in C57Bl/6 mice
    Brian J Roberts
    Department of Pathology, Center for Immunology and Infectious Disease, University of Vermont, Burlington, VT, USA
    Biol Sex Differ 3:25. 2012
    ..The goal of this study is to determine which TLRs show differential expression in CVB3 infected mice corresponding to male susceptibility and female resistance in this disease...
  3. pmc Slam haplotype 2 promotes NKT but suppresses Vγ4+ T-cell activation in coxsackievirus B3 infection leading to increased liver damage but reduced myocarditis
    Sally Ann Huber
    Department of Pathology, University of Vermont, Burlington, Vermont 05446, USA
    Am J Pathol 182:401-9. 2013
    ..Liver injuries, as determined by alanine aminotransferase levels in plasma, were increased significantly, confirming that NKT cells are protective for myocarditis but pathogenic in the liver...
  4. pmc Cross-talk between cd1d-restricted nkt cells and γδ cells in t regulatory cell response
    Wei Liu
    The First Affiliated Hospital of Harbin Medical University, Harbin 150001, PR China
    Virol J 8:32. 2011
    ..CD1d-restricted immunity can therefore lead to either immunosuppression or autoimmunity depending upon the type of innate effector dominating during the infection...
  5. pmc Sex chromosome complement contributes to sex differences in coxsackievirus B3 but not influenza A virus pathogenesis
    Dionne P Robinson
    The W Harry Feinstone Department of Molecular Microbiology and Immunology, The Johns Hopkins Bloomberg School of Public Health, Baltimore, MD 21205, USA
    Biol Sex Differ 2:8. 2011
    ..abstract:..
  6. ncbi request reprint T cells in coxsackievirus-induced myocarditis
    Sally Huber
    Department of Pathology, University of Vermont, Bington, Vermont 05405, USA
    Viral Immunol 17:152-64. 2004
    ..The ligand for Vgamma1+ cells is unknown. Only infected myocytes up-regulate CD1d. Signaling through both infection (double stranded RNA) and TNFalpha is required for CD1d up-regulation...
  7. ncbi request reprint CD1d expression on hemopoietic cells promotes CD4+ Th1 response in coxsackievirus B3 induced myocarditis
    S A Huber
    University of Vermont, Department of Pathology, 208 South Park Drive, Suite 2, Burlington, VT 05446, USA
    Virology 352:226-36. 2006
    ..These results show that Vgamma4+ cells modulate developing adaptive immunity through recognition of CD1d on CD4+ T cells, and that this interaction, more than Vgamma4+ cell interaction with infected cardiocytes, determines pathogenicity...
  8. ncbi request reprint Autoimmunity in coxsackievirus B3 induced myocarditis
    Sally A Huber
    University of Vermont, Department of Pathology, Colchester, VT 05446, USA
    Autoimmunity 39:55-61. 2006
    ..Evidence for these mechanisms in coxsackievirus B3 induced myocarditis is discussed...
  9. ncbi request reprint Decay-accelerating factor (CD55) promotes CD1d expression and Vgamma4+ T-cell activation in coxsackievirus B3-induced myocarditis
    Sally Huber
    Department of Pathology, University of Vermont, Colchester, Vermont 05446, USA
    Viral Immunol 19:156-66. 2006
    ..Anti-CD8 depletion of CD8+ T cells protects infected BALB/c mice but aggravates disease in infected DAF-/- animals, indicating that the immunopathogenicity of viral myocarditis differs in the absence of the DAF virus receptor...
  10. ncbi request reprint Cellular autoimmunity in myocarditis
    Sally Huber
    University of Vermont, Colchester, VT 05446, USA
    Heart Fail Clin 1:321-31. 2005
  11. ncbi request reprint Host immune responses to coxsackievirus B3
    S Huber
    University of Vermont, Burlington, VT 05405, USA
    Curr Top Microbiol Immunol 323:199-221. 2008
    ....
  12. pmc Coxsackievirus B3-induced myocarditis: infection of females during the estrus phase of the ovarian cycle leads to activation of T regulatory cells
    S A Huber
    University of Vermont, Department of Pathology, 208 South Park Drive, Suite 2, Colchester, Vermont 05446, USA
    Virology 378:292-8. 2008
    ..These results demonstrate that hormonal fluctuations occurring in normally cycling females can determine T regulatory cell response and control virus-induced pathogenesis...
  13. pmc Coxsackievirus B3 induction of NFAT: requirement for myocarditis susceptibility
    S A Huber
    Department of Pathology, University of Vermont, Colchester, VT 05446, USA
    Virology 381:155-60. 2008
    ..FasL expression by V gamma 4+ cells was also suppressed. Inhibition of FasL expression by V gamma 4+ cells is consistent with myocarditis protection in dnNFAT mice...
  14. pmc Tumor necrosis factor-alpha promotes myocarditis in female mice infected with coxsackievirus B3 through upregulation of CD1d on hematopoietic cells
    Sally Huber
    Department of Pathology, University of Vermont, Colchester, Vermont 05446, USA
    Viral Immunol 23:79-86. 2010
    ....
  15. pmc γδ T lymphocytes kill T regulatory cells through CD1d
    Sally A Huber
    Department of Pathology, University of Vermont, Colchester, VT 05446, USA
    Immunology 131:202-9. 2010
    ..These results demonstrate that Vγ4+ cells selectively abrogate T regulatory cells through recognition of CD1d expressed on the regulatory cells and caspase-dependent apoptosis...
  16. ncbi request reprint T cells expressing the Vgamma1 T-cell receptor enhance virus-neutralizing antibody response during coxsackievirus B3 infection of BALB/c mice: differences in male and female mice
    Sally Huber
    Department of Pathology, University of Vermont, Colchester, Vermont 05446, USA
    Viral Immunol 18:730-9. 2005
    ..These results demonstrate that two gammadelta+ T cell populations, Vgamma1+ and Vgamma4+, have different functions during myocarditis, in that Vgamma1+ cells promote humoral immunity and protection whereas Vgamma4+ cells are pathogenic...
  17. ncbi request reprint Dual functions of murine gammadelta cells in inflammation and autoimmunity in coxsackievirus B3-induced myocarditis: role of Vgamma1+ and Vgamma4+ cells
    S A Huber
    Department of Pathology, University of Vermont, Burlington, VT, USA
    Microbes Infect 7:537-43. 2005
    ..The CD4+ Th1 response is necessary for activation of the autoimmune CD8+ T cells, which kill uninfected cardiocytes through perforin-dependent mechanisms...
  18. ncbi request reprint Coxsackievirus B3 induces T regulatory cells, which inhibit cardiomyopathy in tumor necrosis factor-alpha transgenic mice
    Sally A Huber
    Department of Pathology, University of Vermont, Colchester 05446, USA
    Circ Res 99:1109-16. 2006
    ..H3 virus infection induces high levels of systemic TNF-alpha, whereas H310A1 virus does not. The low TNF-alpha response during H310A1 infections is likely responsible for the T regulatory cell response in these animals...
  19. pmc Gammadelta T cells promote a Th1 response during coxsackievirus B3 infection in vivo: role of Fas and Fas ligand
    Sally Huber
    Department of Pathology, College of Medicine, The University of Vermont, Burlington, VT 05405, USA
    J Virol 76:6487-94. 2002
    ..These results demonstrate that Fas/FasL interactions conferred by gammadelta T cells on lymphocyte subpopulations may regulate the cytokine response to CVB3 infection and pathogenicity...
  20. pmc Vgamma4(+) T cells promote autoimmune CD8(+) cytolytic T-lymphocyte activation in coxsackievirus B3-induced myocarditis in mice: role for CD4(+) Th1 cells
    S A Huber
    Department of Pathology, University of Vermont, Burlington, Vermont, USA
    J Virol 76:10785-90. 2002
    ..Vgamma4(+) cells enhance CD4(+) Th1 (IFN-gamma(+)) cell activation through IFN-gamma- and CD1-dependent mechanisms. CD4(+) Th1 cells promote activation of the autoimmune CD8(+) alphabeta TCR(+) effectors...
  21. ncbi request reprint Role of CD1d in coxsackievirus B3-induced myocarditis
    Sally Huber
    Department of Pathology, University of Vermont, Burlington, VT 05446, USA
    J Immunol 170:3147-53. 2003
    ....
  22. ncbi request reprint Increased susceptibility of male BALB/c mice to coxsackievirus B3-induced myocarditis: role for CD1d
    Sally A Huber
    University of Vermont, Department of Pathology, 208 South Park Drive Suite 2, Colchester, VT 05446, USA
    Med Microbiol Immunol 194:121-7. 2005
    ..These results demonstrate that CVB3 infections produce gender-specific differences in both innate and adaptive immunity, which may explain the difference in disease susceptibility...
  23. pmc Roles of tumor necrosis factor alpha (TNF-alpha) and the p55 TNF receptor in CD1d induction and coxsackievirus B3-induced myocarditis
    S A Huber
    University of Vermont, Department of Pathology, 208 South Park Dr, Suite 2, Colchester, VT 05446, USA
    J Virol 79:2659-65. 2005
    ..TNF-alpha treatment of uninfected endothelial cells had only a modest effect on CD1d expression, suggesting that optimal CD1d upregulation requires both infection and TNF-alpha signaling...
  24. pmc The Y chromosome as a regulatory element shaping immune cell transcriptomes and susceptibility to autoimmune disease
    Laure K Case
    Department of Medicine, University of Vermont, Burlington, Vermont 05405, USA
    Genome Res 23:1474-85. 2013
    ..Thus, as in Drosophila, these data establish the mammalian ChrY as a member of the regulatory genome due to its ability to epigenetically regulate genome-wide gene expression in immune cells. ..
  25. pmc Sex differences in TLR2 and TLR4 expression and their effect on coxsackievirus-induced autoimmune myocarditis
    Brian J Roberts
    Department of Pathology, Center for Immunology and Infectious Disease, University of Vermont, Burlington, VT 05446, United States
    Exp Mol Pathol 94:58-64. 2013
    ..The suppression of T regulatory cells by TLR signaling in males but not females correlates with the increased myocarditis susceptibility of the males...
  26. ncbi request reprint T helper-cell phenotype regulates atherosclerosis in mice under conditions of mild hypercholesterolemia
    S A Huber
    Department of Pathology, University of Vermont, Burlington, USA
    Circulation 103:2610-6. 2001
    ..T cells are implicated in atherosclerosis, but little is known about the genetic control or molecular pathways, especially under conditions of mild hypercholesterolemia...
  27. pmc T helper cell polarization in healthy people: implications for cardiovascular disease
    Nels C Olson
    Department of Pathology, University of Vermont College of Medicine, Burlington, VT, USA
    J Cardiovasc Transl Res 6:772-86. 2013
    ..We also present data from our laboratory demonstrating inherent Th1/Th2 phenotypes in apparently healthy human volunteers that are stable over time and discuss the potential implications for cardiovascular disease. ..
  28. pmc Decreased naive and increased memory CD4(+) T cells are associated with subclinical atherosclerosis: the multi-ethnic study of atherosclerosis
    Nels C Olson
    Department of Pathology, University of Vermont College of Medicine, Burlington, Vermont, USA
    PLoS ONE 8:e71498. 2013
    ..We hypothesized that activation of adaptive immune responses, as reflected by higher proportions of circulating CD4(+) memory cells and lower proportions of naive cells, would be associated with subclinical atherosclerosis...
  29. pmc Histamine H(1) receptor signaling regulates effector T cell responses and susceptibility to coxsackievirus B3-induced myocarditis
    Laure K Case
    Department of Medicine, University of Vermont, Burlington, VT 05405, USA
    Cell Immunol 272:269-74. 2012
    ..These data establish a critical role for histamine and H(1)R signaling in regulating T cell responses and susceptibility to CVB3-induced myocarditis in B6 mice...
  30. ncbi request reprint c-FLIP-Short Reduces Type I Interferon Production and Increases Viremia with Coxsackievirus B3
    Iwona A Buskiewicz
    Department of Pathology, Vermont Center for Immunology and Infectious Diseases, University of Vermont, Burlington, Vermont, United States of America
    PLoS ONE 9:e96156. 2014
    ....
  31. pmc Depletion of gammadelta+ T cells increases CD4+ FoxP3 (T regulatory) cell response in coxsackievirus B3-induced myocarditis
    Sally A Huber
    Department of Pathology, University of Vermont, Colchester, VT 05446, USA
    Immunology 127:567-76. 2009
    ..This study shows that gammadelta(+) cells promote CD4(+) IFN-gamma(+) acute and memory responses by limiting FoxP3(+) T regulatory cell activation...
  32. pmc gamma delta+ T cells regulate major histocompatibility complex class II(IA and IE)-dependent susceptibility to coxsackievirus B3-induced autoimmune myocarditis
    S A Huber
    Department of Pathology, Department of Medicine, University of Vermont College of Medicine, Burlington, Vermont 05405, USA
    J Virol 73:5630-6. 1999
    ....
  33. ncbi request reprint Interleukin-6 exacerbates early atherosclerosis in mice
    S A Huber
    Department of Pathology, University of Vermont, Burlington, VT 05405, USA
    Arterioscler Thromb Vasc Biol 19:2364-7. 1999
    ....
  34. ncbi request reprint T cells expressing the gamma delta T cell receptor induce apoptosis in cardiac myocytes
    S A Huber
    Department of Pathology, University of Vermont, Colchester 05446, USA
    Cardiovasc Res 45:579-87. 2000
    ..The objective of the present studies is to demonstrate that gamma delta T cells directly induce greater Fas-dependent apoptosis of cultured myocytes than T cells expressing the alpha beta TcR...
  35. pmc Hormonal regulation of CD4(+) T-cell responses in coxsackievirus B3-induced myocarditis in mice
    S A Huber
    Departments of Pathology and Medicine, University of Vermont, Burlington, Vermont 05405, USA
    J Virol 73:4689-95. 1999
    ..Taken together, our results indicate that testosterone promotes a CD4(+) Th1 cell response and myocarditis by promoting increased gammadelta+ cell activation...
  36. ncbi request reprint V gamma 1+ T cells suppress and V gamma 4+ T cells promote susceptibility to coxsackievirus B3-induced myocarditis in mice
    S A Huber
    Department of Pathology, University of Vermont, Burlington, VT 05446, USA
    J Immunol 165:4174-81. 2000
    ..Th subset analysis suggests that Vgamma1(+) cells biased the CD4(+) T cells to a dominant Th2 cell response, whereas Vgamma4(+) cells biased CD4(+) T cells toward a dominant Th1 cell response...
  37. pmc Cytokine production by Vgamma(+)-T-cell subsets is an important factor determining CD4(+)-Th-cell phenotype and susceptibility of BALB/c mice to coxsackievirus B3-induced myocarditis
    S A Huber
    Department of Pathology, University of Vermont, Burlington, Vermont, USA
    J Virol 75:5860-9. 2001
    ..The cytokines produced by the Vgamma subpopulation have a significant influence on the CD4(+)-Th-cell phenotype...
  38. ncbi request reprint The role of sex differences in autophagy in the heart during coxsackievirus B3-induced myocarditis
    Andreas Koenig
    Department of Medicine, Vermont Center for Immunology and Infectious Diseases, University of Vermont, Burlington, VT, 05405, USA
    J Cardiovasc Transl Res 7:182-91. 2014
    ..This review discusses the aspects of sex bias in autophagy induction in cardiomyocytes. ..
  39. pmc T-helper type 1 bias in healthy people is associated with cytomegalovirus serology and atherosclerosis: the Multi-Ethnic Study of Atherosclerosis
    Russell P Tracy
    Department of Pathology, University of Vermont, Burlington, VT 05446, USA
    J Am Heart Assoc 2:e000117. 2013
    ..Although T-helper type 1 (Th1) cells are considered important in atherosclerosis, the relationships between Th1 and Th2 cells and atherosclerosis have not been examined in population-based studies...
  40. ncbi request reprint Different potentials of gamma delta T cell subsets in regulating airway responsiveness: V gamma 1+ cells, but not V gamma 4+ cells, promote airway hyperreactivity, Th2 cytokines, and airway inflammation
    Youn Soo Hahn
    Department of Immunology, National Jewish Medical and Research Center, Denver, CO 80206, USA
    J Immunol 172:2894-902. 2004
    ..These findings show how gammadelta T cells can be both inhibitors and enhancers of AHR and airway inflammation, and they provide further support for the hypothesis that TCR expression and function cosegregate in gammadelta T cells...
  41. ncbi request reprint Gene expression profiling during the transition to failure in TNF-alpha over-expressing mice demonstrates the development of autoimmune myocarditis
    Zhonghua Tang
    Department of Medicine, Center for Translational Medicine, Thomas Jefferson University, Suite 813 College Building, 1025 Walnut Street, Philadelphia, PA 19107, USA
    J Mol Cell Cardiol 36:515-30. 2004
    ..The autoimmune response occurs concomitantly with cardiac decompensation and may participate in triggering the transition to failure in TNF1.6 mice...

Research Grants21

  1. CDI and Gamma Delta+ in Viral Myocarditis
    Sally Huber; Fiscal Year: 2005
    ..abstract_text> ..
  2. Sex in Myocarditis
    Sally Huber; Fiscal Year: 2005
    ..By understanding the factors controlling virus infection and induction of innate immunity, better mechanism for controlling viral disease should be possible. ..
  3. Sex in Myocarditis
    Sally Huber; Fiscal Year: 2006
    ..By understanding the factors controlling virus infection and induction of innate immunity, better mechanism for controlling viral disease should be possible. ..
  4. T reg Cells in Myocarditis
    Sally Huber; Fiscal Year: 2007
    ..Determine whether the T reg cells are antigen specific (virus), cardiovascular specific (autoimmunity), or non-antigen specific. ..
  5. Sex in Myocarditis
    Sally Huber; Fiscal Year: 2007
    ..By understanding the factors controlling virus infection and induction of innate immunity, better mechanism for controlling viral disease should be possible. ..
  6. T reg Cells in Myocarditis
    Sally Huber; Fiscal Year: 2009
    ..Determine whether the T reg cells are antigen specific (virus), cardiovascular specific (autoimmunity), or non-antigen specific. ..
  7. CDI and Gamma Delta+ in Viral Myocarditis
    Sally Huber; Fiscal Year: 2004
    ..abstract_text> ..
  8. Sex in Viral Myocarditis
    Sally Huber; Fiscal Year: 2004
    ..These studies may provide new insights as to how viruses affect developing host defense responses and how hormones can modulate this initial response. ..
  9. CDI and Gamma Delta+ in Viral Myocarditis
    Sally Huber; Fiscal Year: 2003
    ..abstract_text> ..
  10. TH1 AND TH2 RESPONSES IN PATHOGENIC CVB3 INFECTIONS
    Sally Huber; Fiscal Year: 1999
    ....
  11. CD4+ T CELLS PROMOTE EARLY ATHEROSCLEROSIS
    Sally Huber; Fiscal Year: 1999
    ..Specific Aim 3 will investigate the role of CD4+ T cells in atherogenesis and evaluate whether these cells promote Th1 cell dominance. ..
  12. TH1 AND TH2 RESPONSES IN PATHOGENIC CVB3 INFECTIONS
    Sally Huber; Fiscal Year: 2000
    ....
  13. CD4+ T CELLS PROMOTE EARLY ATHEROSCLEROSIS
    Sally Huber; Fiscal Year: 2000
    ..Specific Aim 3 will investigate the role of CD4+ T cells in atherogenesis and evaluate whether these cells promote Th1 cell dominance. ..
  14. TH1 AND TH2 RESPONSES IN PATHOGENIC CVB3 INFECTIONS
    Sally Huber; Fiscal Year: 2001
    ....
  15. CD4+ T CELLS PROMOTE EARLY ATHEROSCLEROSIS
    Sally Huber; Fiscal Year: 2001
    ..Specific Aim 3 will investigate the role of CD4+ T cells in atherogenesis and evaluate whether these cells promote Th1 cell dominance. ..
  16. CD4+ T CELLS PROMOTE EARLY ATHEROSCLEROSIS
    Sally Huber; Fiscal Year: 2002
    ..Specific Aim 3 will investigate the role of CD4+ T cells in atherogenesis and evaluate whether these cells promote Th1 cell dominance. ..
  17. CDI and Gamma Delta+ in Viral Myocarditis
    Sally Huber; Fiscal Year: 2002
    ..abstract_text> ..
  18. T reg Cells in Myocarditis
    Sally A Huber; Fiscal Year: 2010
    ..Determine whether the T reg cells are antigen specific (virus), cardiovascular specific (autoimmunity), or non-antigen specific. ..