C L Heaps

Summary

Affiliation: University of Missouri
Country: USA

Publications

  1. ncbi Enhanced L-type Ca2+ channel current density in coronary smooth muscle of exercise-trained pigs is compensated to limit myoplasmic free Ca2+ accumulation
    C L Heaps
    Dalton Cardiovascular Research Center and Department of Physiology, University of Missouri, Columbia, MO 65211, USA
    J Physiol 528:435-45. 2000
  2. ncbi Sarcoplasmic reticulum Ca(2+) uptake is impaired in coronary smooth muscle distal to coronary occlusion
    C L Heaps
    Dalton Cardiovascular Research Center, University of Missouri, Columbia, Missouri 65211, USA
    Am J Physiol Heart Circ Physiol 281:H223-31. 2001
  3. ncbi Calcium buffering in coronary smooth muscle after chronic occlusion and exercise training
    J J Jones
    Vascular Biology Laboratory, Dalton Cardiovascular Research Center, and Department of Physiology, University of Missouri, Columbia, MO 65211, USA
    Cardiovasc Res 51:359-67. 2001
  4. ncbi Hypercholesterolemia abolishes voltage-dependent K+ channel contribution to adenosine-mediated relaxation in porcine coronary arterioles
    C L Heaps
    Department of Biomedical Sciences, College of Veterinary Medicine, University of Missouri, Columbia, Missouri 65211, USA
    Am J Physiol Heart Circ Physiol 288:H568-76. 2005
  5. ncbi Endogenous testosterone increases L-type Ca2+ channel expression in porcine coronary smooth muscle
    D K Bowles
    E102 Veterinary Medicine, Univ of Missouri, Columbia, MO 65211, USA
    Am J Physiol Heart Circ Physiol 287:H2091-8. 2004
  6. ncbi Hypercholesterolemia inhibits L-type calcium current in coronary macro-, not microcirculation
    D K Bowles
    Department of Biomedical Sciences, University of Missouri, Columbia, MO 65211, USA
    J Appl Physiol 96:2240-8. 2004

Collaborators

Detail Information

Publications6

  1. ncbi Enhanced L-type Ca2+ channel current density in coronary smooth muscle of exercise-trained pigs is compensated to limit myoplasmic free Ca2+ accumulation
    C L Heaps
    Dalton Cardiovascular Research Center and Department of Physiology, University of Missouri, Columbia, MO 65211, USA
    J Physiol 528:435-45. 2000
    ..Additional potential compensatory mechanisms include Ca2+ extrusion via plasma membrane Ca2+-ATPase, mitochondrial uptake, myoplasmic Ca2+-binding proteins and other sources of VGCC inactivation...
  2. ncbi Sarcoplasmic reticulum Ca(2+) uptake is impaired in coronary smooth muscle distal to coronary occlusion
    C L Heaps
    Dalton Cardiovascular Research Center, University of Missouri, Columbia, Missouri 65211, USA
    Am J Physiol Heart Circ Physiol 281:H223-31. 2001
    ..Impaired SR Ca(2+) uptake was independent of SERCA protein levels and was not prevented by exercise training...
  3. ncbi Calcium buffering in coronary smooth muscle after chronic occlusion and exercise training
    J J Jones
    Vascular Biology Laboratory, Dalton Cardiovascular Research Center, and Department of Physiology, University of Missouri, Columbia, MO 65211, USA
    Cardiovasc Res 51:359-67. 2001
    ....
  4. ncbi Hypercholesterolemia abolishes voltage-dependent K+ channel contribution to adenosine-mediated relaxation in porcine coronary arterioles
    C L Heaps
    Department of Biomedical Sciences, College of Veterinary Medicine, University of Missouri, Columbia, Missouri 65211, USA
    Am J Physiol Heart Circ Physiol 288:H568-76. 2005
    ....
  5. ncbi Endogenous testosterone increases L-type Ca2+ channel expression in porcine coronary smooth muscle
    D K Bowles
    E102 Veterinary Medicine, Univ of Missouri, Columbia, MO 65211, USA
    Am J Physiol Heart Circ Physiol 287:H2091-8. 2004
    ..2 expression. Furthermore, this effect was blocked by the androgen receptor antagonist cyproterone. We conclude that endogenous testosterone is a primary regulator of Cav1.2 expression and activity in coronary arteries of males...
  6. ncbi Hypercholesterolemia inhibits L-type calcium current in coronary macro-, not microcirculation
    D K Bowles
    Department of Biomedical Sciences, University of Missouri, Columbia, MO 65211, USA
    J Appl Physiol 96:2240-8. 2004
    ..The inhibition of calcium channel activity in CASM of large coronary arteries is likely due to increases in membrane free cholesterol...