J L Hall
Affiliation: University of Minnesota
- Overexpression of Ref-1 inhibits hypoxia and tumor necrosis factor-induced endothelial cell apoptosis through nuclear factor-kappab-independent and -dependent pathwaysJ L Hall
Division of Cardiovascular Medicine, Brigham and Women s Hospital, Harvard Medical School, Boston, Mass, USA
Circ Res 88:1247-53. 2001..Moreover, it seems that Ref-1 may act as a critical cofactor, mediating the TNF-induced NF-kappaB response in the vascular endothelium...
- Determinants of vascular smooth muscle cell apoptosis after balloon angioplasty injury. Influence of redox state and cell phenotypeM J Pollman
Molecular Vascular Cell Biology Research Laboratory, Brigham and Women s Hospital, Harvard Medical School, Boston, MA, USA
Circ Res 84:113-21. 1999..Angioplasty-induced vascular cell apoptosis may be an important determinant of vascular remodeling and restenosis...
- Upregulation of glucose metabolism during intimal lesion formation is coupled to the inhibition of vascular smooth muscle cell apoptosis. Role of GSK3betaJ L Hall
Division of Cardiovascular Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts, USA
Diabetes 50:1171-9. 2001..Taken together, upregulation of glucose metabolism during intimal lesion formation promotes an antiapoptotic signaling pathway that is linked to the inactivation of GSK3beta...