Gregg Hadley

Summary

Affiliation: University of Maryland
Country: USA

Publications

  1. pmc CD103 expression is required for destruction of pancreatic islet allografts by CD8(+) T cells
    Ye Feng
    Division of Transplantation, Department of Surgery, The University of Maryland Medical Center, Baltimore 21201, USA
    J Exp Med 196:877-86. 2002
  2. pmc TGF-{beta}-dependent CD103 expression by CD8(+) T cells promotes selective destruction of the host intestinal epithelium during graft-versus-host disease
    Riham El-Asady
    Department of Surgery, University of Maryland Medical School, Baltimore, MD 21201, USA
    J Exp Med 201:1647-57. 2005
  3. ncbi request reprint Role of integrin CD103 in promoting destruction of renal allografts by CD8 T cells
    Gregg Hadley
    University of Maryland Medical School, Surgery, Baltimore, MD, USA
    Am J Transplant 4:1026-32. 2004
  4. ncbi request reprint Steroid-free maintenance of islet allografts using mycophenolate mofetil and cyclosporine in the non-human primate
    Jill Buss
    Department of Surgery, Division of Transplantation, The Ohio State University, Columbus, USA
    Ann Transplant 16:88-97. 2011
  5. pmc An experimental model of acute humoral rejection of renal allografts associated with concomitant cellular rejection
    Alice Bickerstaff
    Department of Pathology, The Ohio State University College of Medicine and Public Health, Columbus, OH 43210, USA
    Am J Pathol 173:347-57. 2008

Collaborators

  • Philip J Lucas
  • Amer Rajab
  • Jill Buss
  • Alice Bickerstaff
  • Riham El-Asady
  • Donghua Wang
  • Rongwen Yuan
  • Ye Feng
  • Elizabeth Essig
  • Kwame Osei
  • Sergey Brodsky
  • Nicholas DiPaola
  • Jiao Jing Wang
  • Gyongyi Nadasdy
  • Anjali Satoskar
  • Tibor Nadasdy
  • Ronald Pelletier
  • Charles Orosz
  • Ronald E Gress
  • Cinthia B Drachenberg
  • Kechang Liu
  • Donna L Farber
  • Christina M Parker

Detail Information

Publications5

  1. pmc CD103 expression is required for destruction of pancreatic islet allografts by CD8(+) T cells
    Ye Feng
    Division of Transplantation, Department of Surgery, The University of Maryland Medical Center, Baltimore 21201, USA
    J Exp Med 196:877-86. 2002
    ....
  2. pmc TGF-{beta}-dependent CD103 expression by CD8(+) T cells promotes selective destruction of the host intestinal epithelium during graft-versus-host disease
    Riham El-Asady
    Department of Surgery, University of Maryland Medical School, Baltimore, MD 21201, USA
    J Exp Med 201:1647-57. 2005
    ..Collectively, these data document a pivotal role for TGF-beta-dependent CD103 expression in dictating the gut tropism, and hence the destructive potential, of CD8(+) T cells during GVHD pathogenesis...
  3. ncbi request reprint Role of integrin CD103 in promoting destruction of renal allografts by CD8 T cells
    Gregg Hadley
    University of Maryland Medical School, Surgery, Baltimore, MD, USA
    Am J Transplant 4:1026-32. 2004
    ..The purpose of this communication is to review the known properties of the CD103 molecule and its postulated role in the efferent phase of renal allograft rejection...
  4. ncbi request reprint Steroid-free maintenance of islet allografts using mycophenolate mofetil and cyclosporine in the non-human primate
    Jill Buss
    Department of Surgery, Division of Transplantation, The Ohio State University, Columbus, USA
    Ann Transplant 16:88-97. 2011
    ..The goal of this study is to evaluate the short-term follow-up (90 day) of a steroid-free maintenance immunosuppression protocol of mycophenolate mofetil (MMF) and cyclosporine (CSA) in non-human primate islet allotransplantation...
  5. pmc An experimental model of acute humoral rejection of renal allografts associated with concomitant cellular rejection
    Alice Bickerstaff
    Department of Pathology, The Ohio State University College of Medicine and Public Health, Columbus, OH 43210, USA
    Am J Pathol 173:347-57. 2008
    ..This animal model provides a valuable tool to study the pathogenesis of AHR, its relationship to cellular alloimmunity, its contribution to graft injury, and the effects of various potential therapeutic interventions...