DENIS J GLENN

Summary

Affiliation: University of California
Country: USA

Publications

  1. doi Cardiac steatosis potentiates angiotensin II effects in the heart
    DENIS J GLENN
    Department of Medicine and the Diabetes Center, University of California, San Francisco, California
    Am J Physiol Heart Circ Physiol 308:H339-50. 2015
  2. pmc A murine model of isolated cardiac steatosis leads to cardiomyopathy
    DENIS J GLENN
    Department of Medicine and Diabetes Center, University of California at San Francisco, San Francisco, CA 94143 0540, USA
    Hypertension 57:216-22. 2011
  3. pmc Expression of the vitamin d receptor is increased in the hypertrophic heart
    Songcang Chen
    Diabetes Center, University of California at San Francisco, CA 94143 0540, USA
    Hypertension 52:1106-12. 2008
  4. pmc Atrial natriuretic peptide suppresses endothelin gene expression and proliferation in cardiac fibroblasts through a GATA4-dependent mechanism
    DENIS J GLENN
    Department of Medicine, University of California at San Francisco, San Francisco, CA, USA
    Cardiovasc Res 84:209-17. 2009
  5. pmc Vitamin D and the heart
    David G Gardner
    Diabetes Center, University of California at San Francisco, San Francisco, California and
    Am J Physiol Regul Integr Comp Physiol 305:R969-77. 2013
  6. ncbi Molecular biology of the natriuretic peptide system: implications for physiology and hypertension
    David G Gardner
    Diabetes Center, University of California at San Francisco, San Francisco, CA 94143 0540, USA
    Hypertension 49:419-26. 2007
  7. pmc Endothelin-stimulated human B-type natriuretic peptide gene expression is mediated by Yin Yang 1 in association with histone deacetylase 2
    DENIS J GLENN
    Department of Medicine and Diabetes Center, University of California at San Francisco, USA
    Hypertension 53:549-55. 2009

Detail Information

Publications7

  1. doi Cardiac steatosis potentiates angiotensin II effects in the heart
    DENIS J GLENN
    Department of Medicine and the Diabetes Center, University of California, San Francisco, California
    Am J Physiol Heart Circ Physiol 308:H339-50. 2015
    ..These results suggest that myocyte steatosis amplifies the fibrotic effects of ANG II through mechanisms that involve activation of TGF-β signaling and increased production of ROS. ..
  2. pmc A murine model of isolated cardiac steatosis leads to cardiomyopathy
    DENIS J GLENN
    Department of Medicine and Diabetes Center, University of California at San Francisco, San Francisco, CA 94143 0540, USA
    Hypertension 57:216-22. 2011
    ..These findings suggest that triglyceride accumulation, in the absence of systemic metabolic derangement, results in cardiac dysfunction and decreased mitochondrial biogenesis...
  3. pmc Expression of the vitamin d receptor is increased in the hypertrophic heart
    Songcang Chen
    Diabetes Center, University of California at San Francisco, CA 94143 0540, USA
    Hypertension 52:1106-12. 2008
    ....
  4. pmc Atrial natriuretic peptide suppresses endothelin gene expression and proliferation in cardiac fibroblasts through a GATA4-dependent mechanism
    DENIS J GLENN
    Department of Medicine, University of California at San Francisco, San Francisco, CA, USA
    Cardiovasc Res 84:209-17. 2009
    ..Atrial natriuretic peptide (ANP) is a hormone that has both antihypertrophic and antifibrotic properties in the heart. We hypothesized that myocyte-derived ANP inhibits endothelin (ET) gene expression in fibroblasts...
  5. pmc Vitamin D and the heart
    David G Gardner
    Diabetes Center, University of California at San Francisco, San Francisco, California and
    Am J Physiol Regul Integr Comp Physiol 305:R969-77. 2013
    ....
  6. ncbi Molecular biology of the natriuretic peptide system: implications for physiology and hypertension
    David G Gardner
    Diabetes Center, University of California at San Francisco, San Francisco, CA 94143 0540, USA
    Hypertension 49:419-26. 2007
  7. pmc Endothelin-stimulated human B-type natriuretic peptide gene expression is mediated by Yin Yang 1 in association with histone deacetylase 2
    DENIS J GLENN
    Department of Medicine and Diabetes Center, University of California at San Francisco, USA
    Hypertension 53:549-55. 2009
    ..These data suggest a model in which YY1 activates hBNP transcription through interaction with histone deacetylase 2...