C Giulivi

Summary

Affiliation: University of Southern California
Country: USA

Publications

  1. ncbi request reprint Production of nitric oxide by mitochondria
    C Giulivi
    Department of Molecular Pharmacology and Toxicology, University of Southern California, Los Angeles, California 90033, USA
    J Biol Chem 273:11038-43. 1998
  2. ncbi request reprint Iron primes hepatic macrophages for NF-kappaB activation in alcoholic liver injury
    H Tsukamoto
    Department of Medicine and Pathology, University of Southern California School of Medicine, Los Angeles 90033, California
    Am J Physiol 277:G1240-50. 1999
  3. pmc Functional implications of nitric oxide produced by mitochondria in mitochondrial metabolism
    C Giulivi
    Department of Molecular Pharmacology and Toxicology, University of Southern California, 1985 Zonal Ave, Los Angeles, CA 90033, USA
    Biochem J 332:673-9. 1998

Detail Information

Publications3

  1. ncbi request reprint Production of nitric oxide by mitochondria
    C Giulivi
    Department of Molecular Pharmacology and Toxicology, University of Southern California, Los Angeles, California 90033, USA
    J Biol Chem 273:11038-43. 1998
    ..This study demonstrates that mitochondria are a source of NO., the production of which may effect energy metabolism, O2 consumption, and O2 free radical formation...
  2. ncbi request reprint Iron primes hepatic macrophages for NF-kappaB activation in alcoholic liver injury
    H Tsukamoto
    Department of Medicine and Pathology, University of Southern California School of Medicine, Los Angeles 90033, California
    Am J Physiol 277:G1240-50. 1999
    ..These results support the pivotal role of heme-derived iron in priming HM for NF-kappaB activation and expression of proinflammatory genes in alcoholic liver injury...
  3. pmc Functional implications of nitric oxide produced by mitochondria in mitochondrial metabolism
    C Giulivi
    Department of Molecular Pharmacology and Toxicology, University of Southern California, 1985 Zonal Ave, Los Angeles, CA 90033, USA
    Biochem J 332:673-9. 1998
    ..This inhibition can be explained by the interaction of NO. with cytochrome oxidase at the binuclear centre. The role of NO. as a physiological modulator of cytochrome oxidase is discussed in terms of cellular metabolism...