Joseph F Gera

Summary

Affiliation: University of California
Country: USA

Publications

  1. ncbi request reprint AKT activity determines sensitivity to mammalian target of rapamycin (mTOR) inhibitors by regulating cyclin D1 and c-myc expression
    Joseph F Gera
    Department of Medicine, West Los Angeles Veteran s Administration UCLA Medical Center, University of California School of Medicine, Los Angeles, California 90073, USA
    J Biol Chem 279:2737-46. 2004
  2. pmc IL-6-induced stimulation of c-myc translation in multiple myeloma cells is mediated by myc internal ribosome entry site function and the RNA-binding protein, hnRNP A1
    Yijiang Shi
    Department of Medicine, Greater Los Angeles VA Healthcare System and University of California at Los Angeles Medical School, Los Angeles, California, USA
    Cancer Res 68:10215-22. 2008

Collaborators

Detail Information

Publications2

  1. ncbi request reprint AKT activity determines sensitivity to mammalian target of rapamycin (mTOR) inhibitors by regulating cyclin D1 and c-myc expression
    Joseph F Gera
    Department of Medicine, West Los Angeles Veteran s Administration UCLA Medical Center, University of California School of Medicine, Los Angeles, California 90073, USA
    J Biol Chem 279:2737-46. 2004
    ..These results identify two critical downstream molecular targets whose expression is regulated by AKT activity and whose down-regulation is required for rapamycin/CCI-779 sensitivity...
  2. pmc IL-6-induced stimulation of c-myc translation in multiple myeloma cells is mediated by myc internal ribosome entry site function and the RNA-binding protein, hnRNP A1
    Yijiang Shi
    Department of Medicine, Greater Los Angeles VA Healthcare System and University of California at Los Angeles Medical School, Los Angeles, California, USA
    Cancer Res 68:10215-22. 2008
    ..These data point to hnRNP A1 as a critical regulator of c-myc translation and a potential therapeutic target in multiple myeloma...