Jeffrey M Gelfand

Summary

Affiliation: University of California
Country: USA

Publications

  1. ncbi request reprint Multiple sclerosis: diagnosis, differential diagnosis, and clinical presentation
    Jeffrey M Gelfand
    Department of Neurology, University of California, San Francisco, USA Electronic address
    Handb Clin Neurol 122:269-90. 2014
  2. doi request reprint Microcystic inner nuclear layer abnormalities and neuromyelitis optica
    Jeffrey M Gelfand
    Multiple Sclerosis Center, Department of Neurology, University of California, San Francisco, San Francisco, CA 94158, USA
    JAMA Neurol 70:629-33. 2013
  3. pmc Retinal axonal loss begins early in the course of multiple sclerosis and is similar between progressive phenotypes
    Jeffrey M Gelfand
    University of California, San Francisco Department of Neurology, Multiple Sclerosis Center, University of California San Francisco, San Francisco, California, United States of America
    PLoS ONE 7:e36847. 2012
  4. pmc Fingolimod treatment in multiple sclerosis leads to increased macular volume
    Rachel Nolan
    Multiple Sclerosis Center, UCSF Department of Neurology, San Francisco, CA, USA
    Neurology 80:139-44. 2013
  5. pmc Microcystic macular oedema in multiple sclerosis is associated with disease severity
    Jeffrey M Gelfand
    Multiple Sclerosis Centre, University of California, San Francisco, Department of Neurology, 400 Parnassus Ave, San Francisco, CA 94143 0114, USA
    Brain 135:1786-93. 2012
  6. pmc Microcystic macular oedema, thickness of the inner nuclear layer of the retina, and disease characteristics in multiple sclerosis: a retrospective study
    Shiv Saidha
    Department of Neurology, Johns Hopkins University School of Medicine, Baltimore, MD, USA
    Lancet Neurol 11:963-72. 2012

Detail Information

Publications6

  1. ncbi request reprint Multiple sclerosis: diagnosis, differential diagnosis, and clinical presentation
    Jeffrey M Gelfand
    Department of Neurology, University of California, San Francisco, USA Electronic address
    Handb Clin Neurol 122:269-90. 2014
    ....
  2. doi request reprint Microcystic inner nuclear layer abnormalities and neuromyelitis optica
    Jeffrey M Gelfand
    Multiple Sclerosis Center, Department of Neurology, University of California, San Francisco, San Francisco, CA 94158, USA
    JAMA Neurol 70:629-33. 2013
    ..Acute optic neuritis is a cardinal manifestation of neuromyelitis optica (NMO). To our knowledge, microcystic inner nuclear layer abnormalities have not been investigated in NMO...
  3. pmc Retinal axonal loss begins early in the course of multiple sclerosis and is similar between progressive phenotypes
    Jeffrey M Gelfand
    University of California, San Francisco Department of Neurology, Multiple Sclerosis Center, University of California San Francisco, San Francisco, California, United States of America
    PLoS ONE 7:e36847. 2012
    ....
  4. pmc Fingolimod treatment in multiple sclerosis leads to increased macular volume
    Rachel Nolan
    Multiple Sclerosis Center, UCSF Department of Neurology, San Francisco, CA, USA
    Neurology 80:139-44. 2013
    ..To determine whether fingolimod, an oral sphingosine-1-phosphate receptor modulator approved for treatment of multiple sclerosis (MS), generally leads to increased retinal tissue volume...
  5. pmc Microcystic macular oedema in multiple sclerosis is associated with disease severity
    Jeffrey M Gelfand
    Multiple Sclerosis Centre, University of California, San Francisco, Department of Neurology, 400 Parnassus Ave, San Francisco, CA 94143 0114, USA
    Brain 135:1786-93. 2012
    ..These findings also have implications for clinical monitoring in patients with multiple sclerosis on sphingosine 1-phosphate receptor modulating agents...
  6. pmc Microcystic macular oedema, thickness of the inner nuclear layer of the retina, and disease characteristics in multiple sclerosis: a retrospective study
    Shiv Saidha
    Department of Neurology, Johns Hopkins University School of Medicine, Baltimore, MD, USA
    Lancet Neurol 11:963-72. 2012
    ..We aimed to determine whether MMO of the INL, and increased thickness of the INL are associated with disease activity or disability progression...