M J Gandal

Summary

Affiliation: University of Pennsylvania
Country: USA

Publications

  1. doi Mice with reduced NMDA receptor expression: more consistent with autism than schizophrenia?
    M J Gandal
    Translational Neuroscience Program, Department of Psychiatry, University of Pennsylvania, Philadelphia, PA 19104, USA
    Genes Brain Behav 11:740-50. 2012
  2. pmc GABAB-mediated rescue of altered excitatory-inhibitory balance, gamma synchrony and behavioral deficits following constitutive NMDAR-hypofunction
    M J Gandal
    Translational Neuroscience Program, Department of Psychiatry, University of Pennsylvania, Philadelphia, PA 19104, USA
    Transl Psychiatry 2:e142. 2012
  3. doi N-methyl-d-aspartic acid receptor antagonist-induced frequency oscillations in mice recreate pattern of electrophysiological deficits in schizophrenia
    R S Ehrlichman
    Stanley Center for Experimental Therapeutics in Psychiatry, Department of Psychiatry, University of Pennsylvania, Philadelphia, PA 19104, USA
    Neuroscience 158:705-12. 2009

Collaborators

Detail Information

Publications3

  1. doi Mice with reduced NMDA receptor expression: more consistent with autism than schizophrenia?
    M J Gandal
    Translational Neuroscience Program, Department of Psychiatry, University of Pennsylvania, Philadelphia, PA 19104, USA
    Genes Brain Behav 11:740-50. 2012
    ..Neural synchrony abnormalities suggest an imbalance of glutamatergic and GABAergic coupling and may provide a target, along with behavioral phenotypes, for preclinical screening of novel therapeutics...
  2. pmc GABAB-mediated rescue of altered excitatory-inhibitory balance, gamma synchrony and behavioral deficits following constitutive NMDAR-hypofunction
    M J Gandal
    Translational Neuroscience Program, Department of Psychiatry, University of Pennsylvania, Philadelphia, PA 19104, USA
    Transl Psychiatry 2:e142. 2012
    ....
  3. doi N-methyl-d-aspartic acid receptor antagonist-induced frequency oscillations in mice recreate pattern of electrophysiological deficits in schizophrenia
    R S Ehrlichman
    Stanley Center for Experimental Therapeutics in Psychiatry, Department of Psychiatry, University of Pennsylvania, Philadelphia, PA 19104, USA
    Neuroscience 158:705-12. 2009
    ..Our findings support the hypothesis that NMDAR hypofunction contributes to the deficits in schizophrenia and that the dopaminergic pathways alone may not account for these changes...