Marilie D Gammon

Summary

Affiliation: University of North Carolina
Country: USA

Publications

  1. pmc Imputation method for lifetime exposure assessment in air pollution epidemiologic studies
    Jan Beyea
    Consulting in the Public Interest CIPI, Lambertville, NJ, USA
    Environ Health 12:62. 2013
  2. ncbi request reprint Environmental toxins and breast cancer on Long Island. I. Polycyclic aromatic hydrocarbon DNA adducts
    Marilie D Gammon
    Department of Epidemiology, University of North Carolina, School of Public Health, Chapel Hill, North Carolina 27599 7435, USA
    Cancer Epidemiol Biomarkers Prev 11:677-85. 2002
  3. ncbi request reprint Environmental toxins and breast cancer on Long Island. II. Organochlorine compound levels in blood
    Marilie D Gammon
    Department of Epidemiology, University of North Carolina, School of Public Health, Chapel Hill, North Carolina 27599 7435, USA
    Cancer Epidemiol Biomarkers Prev 11:686-97. 2002
  4. ncbi request reprint The Long Island Breast Cancer Study Project: description of a multi-institutional collaboration to identify environmental risk factors for breast cancer
    Marilie D Gammon
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill 27599 7400, USA
    Breast Cancer Res Treat 74:235-54. 2002
  5. ncbi request reprint Alcohol dehydrogenase 3 and risk of esophageal and gastric adenocarcinomas
    Mary Beth Terry
    Mailman School of Public Health, Department of Epidemiology, Columbia University, 722 West 168th Street, Room 724A, New York, NY 10032, USA
    Cancer Causes Control 18:1039-46. 2007
  6. ncbi request reprint General and abdominal obesity and survival among young women with breast cancer
    Page E Abrahamson
    Department of Epidemiology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, USA
    Cancer Epidemiol Biomarkers Prev 15:1871-7. 2006
  7. ncbi request reprint Active and passive cigarette smoke and breast cancer survival
    Sharon K Sagiv
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC, USA
    Ann Epidemiol 17:385-93. 2007
  8. ncbi request reprint Polymorphisms in nucleotide excision repair genes, polycyclic aromatic hydrocarbon-DNA adducts, and breast cancer risk
    Katherine D Crew
    Department of Epidemiology, Mailman School of Public Health, Columbia University, 161 Fort Washington Avenue, New York, NY 10032, USA
    Cancer Epidemiol Biomarkers Prev 16:2033-41. 2007
  9. pmc Common genetic variations in the LEP and LEPR genes, obesity and breast cancer incidence and survival
    Rebecca J Cleveland
    Department of Nutrition, School of Public Health, University of North Carolina, Chapel Hill, NC 27599 7461, USA
    Breast Cancer Res Treat 120:745-52. 2010
  10. ncbi request reprint Polycyclic aromatic hydrocarbon-DNA adducts and breast cancer: a pooled analysis
    Marilie D Gammon
    Department of Epidemiology, University of North Carolina School of Public Health, Chapel Hill, North Carolina 27599 7435, USA
    Arch Environ Health 59:640-9. 2004

Collaborators

Detail Information

Publications96

  1. pmc Imputation method for lifetime exposure assessment in air pollution epidemiologic studies
    Jan Beyea
    Consulting in the Public Interest CIPI, Lambertville, NJ, USA
    Environ Health 12:62. 2013
    ..We also address challenges that arise, including choice of imputation method, potential bias in regression coefficients, and uncertainty in age-at-exposure sensitivities...
  2. ncbi request reprint Environmental toxins and breast cancer on Long Island. I. Polycyclic aromatic hydrocarbon DNA adducts
    Marilie D Gammon
    Department of Epidemiology, University of North Carolina, School of Public Health, Chapel Hill, North Carolina 27599 7435, USA
    Cancer Epidemiol Biomarkers Prev 11:677-85. 2002
    ..These data indicate that PAH-DNA adduct formation may influence breast cancer development, although the association does not appear to be dose dependent and may have a threshold effect...
  3. ncbi request reprint Environmental toxins and breast cancer on Long Island. II. Organochlorine compound levels in blood
    Marilie D Gammon
    Department of Epidemiology, University of North Carolina, School of Public Health, Chapel Hill, North Carolina 27599 7435, USA
    Cancer Epidemiol Biomarkers Prev 11:686-97. 2002
    ..These findings, based on the largest number of samples analyzed to date among primarily white women, do not support the hypothesis that organochlorines increase breast cancer risk among Long Island women...
  4. ncbi request reprint The Long Island Breast Cancer Study Project: description of a multi-institutional collaboration to identify environmental risk factors for breast cancer
    Marilie D Gammon
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill 27599 7400, USA
    Breast Cancer Res Treat 74:235-54. 2002
    ....
  5. ncbi request reprint Alcohol dehydrogenase 3 and risk of esophageal and gastric adenocarcinomas
    Mary Beth Terry
    Mailman School of Public Health, Department of Epidemiology, Columbia University, 722 West 168th Street, Room 724A, New York, NY 10032, USA
    Cancer Causes Control 18:1039-46. 2007
    ..The alcohol dehydrogenase gene has several polymorphisms which may lead to faster conversion of ethanol to acetaldehyde, which may increase cancer risk...
  6. ncbi request reprint General and abdominal obesity and survival among young women with breast cancer
    Page E Abrahamson
    Department of Epidemiology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, USA
    Cancer Epidemiol Biomarkers Prev 15:1871-7. 2006
    ..22; 95% CI, 1.45-3.40). This study provides evidence that breast cancer survival is reduced among younger women with general or abdominal obesity...
  7. ncbi request reprint Active and passive cigarette smoke and breast cancer survival
    Sharon K Sagiv
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC, USA
    Ann Epidemiol 17:385-93. 2007
    ..The association between active and passive cigarette smoking before breast cancer diagnosis and survival was investigated among a cohort of invasive breast cancer cases (n = 1273) participating in a population-based case-control study...
  8. ncbi request reprint Polymorphisms in nucleotide excision repair genes, polycyclic aromatic hydrocarbon-DNA adducts, and breast cancer risk
    Katherine D Crew
    Department of Epidemiology, Mailman School of Public Health, Columbia University, 161 Fort Washington Avenue, New York, NY 10032, USA
    Cancer Epidemiol Biomarkers Prev 16:2033-41. 2007
    ..Overall, this study suggests that the risk of breast cancer may be elevated among women with polymorphisms in NER pathway genes and detectable PAH-DNA adducts...
  9. pmc Common genetic variations in the LEP and LEPR genes, obesity and breast cancer incidence and survival
    Rebecca J Cleveland
    Department of Nutrition, School of Public Health, University of North Carolina, Chapel Hill, NC 27599 7461, USA
    Breast Cancer Res Treat 120:745-52. 2010
    ..In summary, our results show that a common variant in LEP may be associated with the risk of developing breast cancer supporting the hypothesis that leptin is involved in breast carcinogenesis...
  10. ncbi request reprint Polycyclic aromatic hydrocarbon-DNA adducts and breast cancer: a pooled analysis
    Marilie D Gammon
    Department of Epidemiology, University of North Carolina School of Public Health, Chapel Hill, North Carolina 27599 7435, USA
    Arch Environ Health 59:640-9. 2004
    ..These data provide only modest support for an association between PAH-DNA adducts and breast cancer development...
  11. ncbi request reprint Dietary flavonoid intake and breast cancer survival among women on Long Island
    Brian N Fink
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, North Carolina, USA
    Cancer Epidemiol Biomarkers Prev 16:2285-92. 2007
    ..However, the effects of flavonoids on survival are not known. In a population-based cohort of breast cancer patients, we investigated whether dietary flavonoid intake before diagnosis is associated with subsequent survival...
  12. ncbi request reprint Polymorphism in the DNA repair gene XPD, polycyclic aromatic hydrocarbon-DNA adducts, cigarette smoking, and breast cancer risk
    Mary Beth Terry
    Department of Epidemiology, Mailman School of Public Health, Columbia University, PH 18 102, 600 West 168th Street, New York, NY 10032, USA
    Cancer Epidemiol Biomarkers Prev 13:2053-8. 2004
    ..Overall, this study suggests that those individuals with this polymorphism in the XPD gene may face an increased risk of breast cancer from PAH-DNA adducts and cigarette smoking...
  13. ncbi request reprint Associations between breast cancer risk and the catalase genotype, fruit and vegetable consumption, and supplement use
    Jiyoung Ahn
    Department of Epidemiology, Roswell Park Cancer Institute, Buffalo, NY 14263, USA
    Am J Epidemiol 162:943-52. 2005
    ..CC genotypes were prevalent in approximately 64% of controls; thus, the preventive potential for fruit consumption has widespread implications...
  14. ncbi request reprint Genetic variation of TP53, polycyclic aromatic hydrocarbon-related exposures, and breast cancer risk among women on Long Island, New York
    Mia M Gaudet
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC, USA
    Breast Cancer Res Treat 108:93-9. 2008
    ..p53 participates in cell cycle control, programmed cell death/apoptosis, and DNA repair, all pathways involved in carcinogenesis. TP53 variants may influence p53 function...
  15. pmc Polycyclic aromatic hydrocarbon-DNA adducts and survival among women with breast cancer
    Sharon K Sagiv
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC, USA
    Environ Res 109:287-91. 2009
    ..Results from this large population-based study do not provide strong support for an association between detectable PAH-DNA adducts and survival among women with breast cancer, except perhaps among those receiving radiation treatment...
  16. ncbi request reprint MnSOD Val-9Ala genotype, pro- and anti-oxidant environmental modifiers, and breast cancer among women on Long Island, New York
    Mia M Gaudet
    Department of Epidemiology, School of Public Health, University of North Carolina, CB 7435, Chapel Hill, NC 27599 7435, USA
    Cancer Causes Control 16:1225-34. 2005
    ..This study provides little evidence that variation in Val-9Ala polymorphism of MnSOD alone or through substantial interaction with key exposures believed to be pro- or anti-oxidant properties influences breast cancer risk...
  17. ncbi request reprint Weight gain prior to diagnosis and survival from breast cancer
    Rebecca J Cleveland
    Department of Epidemiology, University of North Carolina, CB 7435 McGavran Greenberg Hall, Chapel Hill, NC 27599 7435, USA
    Cancer Epidemiol Biomarkers Prev 16:1803-11. 2007
    ....
  18. pmc Polymorphisms in DNA repair genes, recreational physical activity and breast cancer risk
    Lauren E McCullough
    Department of Epidemiology, Gillings School of Global Public Health, University of North Carolina, Chapel Hill, NC, USA
    Int J Cancer 134:654-63. 2014
    ..46; 95% CI, 0.30-0.71). Our data show that women with minor alleles in both MSH2 and MLH1 could be at increased breast cancer risk. RPA may be modified by genes in the DNA repair pathway, and merit further investigation. ..
  19. pmc Nutrient pathways and breast cancer risk: the Long Island Breast Cancer Study Project
    Patrick T Bradshaw
    Department of Nutrition, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina 27599, USA
    Nutr Cancer 65:345-54. 2013
    ..Our study emphasizes the importance of accounting for multiple nutrient pathways when examining associations between dietary intake and breast cancer...
  20. pmc Polymorphisms in oxidative stress genes, physical activity, and breast cancer risk
    Lauren E McCullough
    Department of Epidemiology, University of North Carolina, CB 7435, McGavran Greenberg Hall, Chapel Hill, NC 27599 7435, USA
    Cancer Causes Control 23:1949-58. 2012
    ..Given the paradoxical effects of physical activity, the oxidative stress pathway is of interest. Genetic variation in CAT or antioxidant-related polymorphisms may mediate the physical activity-breast cancer association...
  21. pmc Fat or fit: the joint effects of physical activity, weight gain, and body size on breast cancer risk
    Lauren E McCullough
    Department of Epidemiology, Gillings School of Global Public Health, University of North Carolina, Chapel Hill, North Carolina, USA
    Cancer 118:4860-8. 2012
    ..These include the minimum duration and intensity necessary for risk reduction and the optimal time period for occurrence, as well as subgroup effects, particularly with regard to tumor heterogeneity and body size...
  22. pmc PAH-DNA adducts, cigarette smoking, GST polymorphisms, and breast cancer risk
    Kathleen M McCarty
    Department of Epidemiology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, USA
    Environ Health Perspect 117:552-8. 2009
    ..Few breast cancer studies have investigated the joint effects of multiple GSTs and a PAH biomarker...
  23. ncbi request reprint Effects of glutathione S-transferase A1 (GSTA1) genotype and potential modifiers on breast cancer risk
    Jiyoung Ahn
    Department of Epidemiology, Roswell Park Cancer Institute, Buffalo, NY 14263, USA
    Carcinogenesis 27:1876-82. 2006
    ..These data indicate that GSTA1 genotypes related to reduced GSTA1 expression are associated with increased breast cancer primarily among women with lower consumption of cruciferous vegetables and among current smokers...
  24. pmc The association of diabetes with breast cancer incidence and mortality in the Long Island Breast Cancer Study Project
    Rebecca J Cleveland
    Department of Medicine, University of North Carolina, Chapel Hill, 27599 7280, USA
    Cancer Causes Control 23:1193-203. 2012
    ..Importantly, prior investigations have rarely considered the influence of factors associated with diabetes such as obesity, age at diabetes diagnosis, duration of diabetes, or diabetes treatments...
  25. ncbi request reprint Residential environmental exposures and other characteristics associated with detectable PAH-DNA adducts in peripheral mononuclear cells in a population-based sample of adult females
    Sumitra Shantakumar
    Department of Epidemiology, CB 7435 McGavran Greenberg Hall, University of North Carolina School of Public Health, Chapel Hill, North Carolina 27599 7435, USA
    J Expo Anal Environ Epidemiol 15:482-90. 2005
    ..These data suggest that PAH-DNA adducts detected in a population-based sample of adult women with ambient exposure levels reflect some key residential PAH exposure sources assessed in this study, such as cigarette smoking...
  26. ncbi request reprint Cooked meat and risk of breast cancer--lifetime versus recent dietary intake
    Susan E Steck
    Department of Nutrition, School of Public Health, University of North Carolina, Chapel Hill, NC, USA
    Epidemiology 18:373-82. 2007
    ..Polycyclic aromatic hydrocarbons (PAHs) and heterocyclic amines (HCAs) are carcinogens formed in or on the surface of well-done meat, cooked at high temperature...
  27. ncbi request reprint IGF1 CA repeat polymorphisms, lifestyle factors and breast cancer risk in the Long Island Breast Cancer Study Project
    Rebecca J Cleveland
    Department of Epidemiology, School of Public Health and Lineberger Comprehensive Cancer Center, University of North Carolina, Chapel Hill, NC 27599 7435, USA
    Carcinogenesis 27:758-65. 2006
    ..Our results demonstrate a role for alleles with fewer than (CA)19 repeats as a risk factor for breast cancer and also suggest that several traditional breast cancer risk factors modify the association of the IGF1 (CA)19 repeat genotype...
  28. ncbi request reprint Fruits, vegetables, and micronutrients in relation to breast cancer modified by menopause and hormone receptor status
    Mia M Gaudet
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, North Carolina 27599 7435, USA
    Cancer Epidemiol Biomarkers Prev 13:1485-94. 2004
    ..Our results support an inverse association for fruit and vegetable intake among postmenopausal but not premenopausal breast cancer, which may be more pronounced among women with ER+ tumors...
  29. ncbi request reprint A CYP19 (aromatase) polymorphism is associated with increased premenopausal breast cancer risk
    Kathryn E Talbott
    Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY, 10032, USA
    Breast Cancer Res Treat 111:481-7. 2008
    ..The finding supports the potential role of variation in estrogen biosynthesis genes in premenopausal breast cancer risk...
  30. pmc Common genetic variation in adiponectin, leptin, and leptin receptor and association with breast cancer subtypes
    Sarah J Nyante
    Department of Epidemiology, University of North Carolina, Chapel Hill, NC, USA
    Breast Cancer Res Treat 129:593-606. 2011
    ..Replication in additional studies where breast cancer subtypes have been defined is necessary to confirm these potential associations...
  31. pmc ADIPOQ, ADIPOR1, and ADIPOR2 polymorphisms in relation to serum adiponectin levels and BMI in black and white women
    Sarah S Cohen
    Department of Epidemiology, University of North Carolina, Chapel Hill, North Carolina, USA
    Obesity (Silver Spring) 19:2053-62. 2011
    ....
  32. pmc Postdiagnosis change in bodyweight and survival after breast cancer diagnosis
    Patrick T Bradshaw
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC 27599 7535, USA
    Epidemiology 23:320-7. 2012
    ..Weight gain after diagnosis is common among women with breast cancer, yet results have been inconsistent among the few studies examining its effects on survival...
  33. ncbi request reprint ADH3 genotype, alcohol intake and breast cancer risk
    Mary Beth Terry
    Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY 10032, USA, and Prosserman Centre for Health Research, Samuel Lunenfeld Research Institute, Mount Sinai Hospital, Toronto, Canada
    Carcinogenesis 27:840-7. 2006
    ..9, 95 % CI=1.2-7.1; postmenopausal women OR=1.8, 95% CI=0.9-3.8). These population-based data support the hypothesis that fast metabolizers of alcohol have a higher risk of breast cancer risk, from alcohol intake than slow metabolizers...
  34. ncbi request reprint Reproductive factors and breast cancer risk among older women
    Sumitra Shantakumar
    Department of Epidemiology, CB 7435 McGavran Greenberg Hall, University of North Carolina School of Public Health, Chapel Hill, NC 27599 7435, USA
    Breast Cancer Res Treat 102:365-74. 2007
    ..Moreover, breastfeeding, one of the few potentially modifiable risk factors for breast cancer, was an important factor in decreasing risk among older parous postmenopausal women...
  35. ncbi request reprint Dietary flavonoid intake and breast cancer risk among women on Long Island
    Brian N Fink
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC, USA
    Am J Epidemiol 165:514-23. 2007
    ..These results suggest that US women can consume sufficient levels of flavonoids to benefit from their potential chemopreventive effects...
  36. ncbi request reprint Catechol-O-methyltransferase haplotypes and breast cancer among women on Long Island, New York
    Mia M Gaudet
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hil, NC 27516, USA
    Breast Cancer Res Treat 99:235-40. 2006
    ....
  37. ncbi request reprint Demographic and lifestyle predictors of survival in patients with esophageal or gastric cancers
    Katrina F Trivers
    Department of Epidemiology, University of North Carolina at Chapel Hill, CB 7435, Chapel Hill, North Carolina 27599 7435, USA
    Clin Gastroenterol Hepatol 3:225-30. 2005
    ..Risk factors for subtypes of esophageal and gastric cancer recently have been identified, but their effect on survival is unknown...
  38. pmc Antioxidant intake and pancreatic cancer risk: the Vitamins and Lifestyle (VITAL) Study
    Xuesong Han
    Department of Nutrition, Gillings School of Global Public Health, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA
    Cancer 119:1314-20. 2013
    ..In this study, the authors examined the intake of antioxidants from diet and supplements in relation to pancreatic cancer risk among participants of the Vitamins and Lifestyle (VITAL) Study...
  39. pmc Influence of prediagnostic recreational physical activity on survival from breast cancer
    Rebecca J Cleveland
    Department of Medicine, University of North Carolina, Chapel Hill, North Carolina 27599 7280, USA
    Eur J Cancer Prev 21:46-54. 2012
    ..no RPA, the HR=0.61; 95% CI=0.39-0.94 and HR=1.00; 95% CI=0.65-1.54, respectively). This study provides support that RPA prior to breast cancer diagnosis improves survival...
  40. ncbi request reprint Age and menopausal effects of hormonal birth control and hormone replacement therapy in relation to breast cancer risk
    Sumitra Shantakumar
    Department of Epidemiology, University of North Carolina School of Public Health, Chapel Hill, NC, USA
    Am J Epidemiol 165:1187-98. 2007
    ..These results emphasize that timing of exogenous hormone use is important. Women who used these hormones before menopause had elevated risks, but the harmful effects began to decline with age after menopause...
  41. ncbi request reprint Association between reproductive factors and breast cancer survival in younger women
    Katrina F Trivers
    Department of Epidemiology, University of North Carolina at Chapel Hill, CB 7435, Chapel Hill, NC 27599 7435, USA
    Breast Cancer Res Treat 103:93-102. 2007
    ..These results may help elucidate breast cancer progression mechanisms and enable a better understanding of how reproductive characteristics influence breast cancer survival...
  42. ncbi request reprint No association between glutathione peroxidase Pro198Leu polymorphism and breast cancer risk
    Jiyoung Ahn
    Department of Epidemiology, Roswell Park Cancer Institute, Elm and Carlton Streets, Buffalo, NY 14263, USA
    Cancer Epidemiol Biomarkers Prev 14:2459-61. 2005
  43. ncbi request reprint Environmental tobacco smoke and breast cancer incidence
    Marilie D Gammon
    Department of Epidemiology, School of Public Health, University of North Carolina, CB 7435 McGavern Greenberg Hall, Chapel Hill, NC 27599 7435, USA
    Environ Res 96:176-85. 2004
    ..42 for ER+ PR+, 95% CI, 1.00, 2.00). Our data suggest that if there is an effect for ETS on breast cancer, that effect is restricted to selected subgroups of women, such as those with long-term exposure from a smoking spouse...
  44. ncbi request reprint Myeloperoxidase genotype, fruit and vegetable consumption, and breast cancer risk
    Jiyoung Ahn
    Department of Epidemiology, Roswell Park Cancer Institute, Buffalo, New York, USA
    Cancer Res 64:7634-9. 2004
    ....
  45. pmc Peroxisome proliferator-activated receptor-alpha (PPARA) genetic polymorphisms and breast cancer risk: a Long Island ancillary study
    Amanda K Golembesky
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC 27599, USA
    Carcinogenesis 29:1944-9. 2008
    ..Our results are the first to evaluate the relationship between PPARA and breast cancer incidence and suggest that replication in an independent cohort is warranted...
  46. ncbi request reprint Fruits, vegetables, and micronutrient intake in relation to breast cancer survival
    Brian N Fink
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC 27599 7435, USA
    Breast Cancer Res Treat 98:199-208. 2006
    ..To determine whether fruit, vegetable, and micronutrient intake 1 year prior to breast cancer diagnosis is associated with a reduction in the subsequent risk of all-cause or breast cancer-specific mortality...
  47. ncbi request reprint Val153Met polymorphism of catechol-O-methyltransferase and prevalence of uterine leiomyomata
    Kyna M Gooden
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC, USA
    Reprod Sci 14:117-20. 2007
    ..This study suggests that variation in this polymorphism alone does not affect fibroid prevalence. Additional research is needed to examine other variations and haplotypes within the COMT gene...
  48. pmc Prevalence and predictors of antioxidant supplement use during breast cancer treatment: the Long Island Breast Cancer Study Project
    Heather Greenlee
    Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY, USA
    Cancer 115:3271-82. 2009
    ..The authors of this report analyzed the prevalence of and factors associated with antioxidant supplement use during breast cancer (BC) treatment among women who participated in the Long Island Breast Cancer Study Project...
  49. pmc Exposure to fogger trucks and breast cancer incidence in the Long Island Breast Cancer Study Project: a case-control study
    Alexandra J White
    Department of Epidemiology, University of North Carolina, Chapel Hill, NC, USA
    Environ Health 12:24. 2013
    ..Self-reported acute exposure to a fogger truck, which sprayed DDT prior to 1972, was hypothesized to increase the risk of breast cancer, particularly among women exposed at a young age or diagnosed with ER+PR+ breast cancer...
  50. ncbi request reprint Preeclampsia, pregnancy-related hypertension, and breast cancer risk
    Mary Beth Terry
    Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY 10032, USA
    Am J Epidemiol 165:1007-14. 2007
    ..These data suggest that pregnancy conditions related to hypertension, particularly preeclampsia, play a role in reducing breast cancer risk. Possible biologic mechanisms underpinning these associations should be further explored...
  51. pmc A Bayesian proportional hazards regression model with non-ignorably missing time-varying covariates
    Patrick T Bradshaw
    Department of Epidemiology, University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA
    Stat Med 29:3017-29. 2010
    ..Our sensitivity analysis showed only slight differences between models with different assumptions on the missing data mechanism yet the complete-case analysis yielded markedly different results...
  52. doi request reprint Consumption of sweet foods and breast cancer risk: a case-control study of women on Long Island, New York
    Patrick T Bradshaw
    Department of Epidemiology, CB 7435 McGavran Greenberg Hall, School of Public Health, University of North Carolina, Chapel Hill, NC 27599 7435, USA
    Cancer Causes Control 20:1509-15. 2009
    ..These results are consistent with other studies that implicate insulin-related factors in breast carcinogenesis...
  53. ncbi request reprint Body size changes in relation to postmenopausal breast cancer among women on Long Island, New York
    Sybil M Eng
    Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY 10017, USA
    Am J Epidemiol 162:229-37. 2005
    ..55, 95% CI: 0.32, 0.96). These results add to the literature by focusing on the perimenopausal weight trajectory and support efforts urging women to avoid weight gain as they age...
  54. doi request reprint PAH, genetic susceptibility and breast cancer risk: an update from the Long Island Breast Cancer Study Project
    Marilie D Gammon
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC, USA
    Eur J Cancer 44:636-40. 2008
  55. ncbi request reprint Lifetime alcohol intake and breast cancer risk
    Mary Beth Terry
    Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY, USA
    Ann Epidemiol 16:230-40. 2006
    ....
  56. ncbi request reprint Association of frequency and duration of aspirin use and hormone receptor status with breast cancer risk
    Mary Beth Terry
    Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY 10032, USA
    JAMA 291:2433-40. 2004
    ..Given the importance of estrogen in the pathogenesis of breast cancer, the ability of aspirin and other NSAIDs to protect against breast cancer could vary according to hormone receptor status...
  57. ncbi request reprint Construction of a flavonoid database for assessing intake in a population-based sample of women on Long Island, New York
    Brian N Fink
    Department of Epidemiology, School of Public Health, University of North Carolina Chapel Hill, Chapel Hill, NC 27599, USA
    Nutr Cancer 56:57-66. 2006
    ..Each class of flavonoids and lignans exhibited a wide range of intake levels. This database is useful to quantify flavonoid and lignan intake for other observational studies conducted in the United States that utilize the Block FFQ...
  58. pmc Types of fish consumed and fish preparation methods in relation to pancreatic cancer incidence: the VITAL Cohort Study
    Ka He
    Department of Nutrition, Gillings School of Global Public Health, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, USA
    Am J Epidemiol 177:152-60. 2013
    ..LC-PUFAs, particularly docosahexaenoic acid, and nonfried fish, but not shellfish or fried fish, may be beneficial in the primary prevention of pancreatic cancer...
  59. ncbi request reprint Recreational physical activity and survival among young women with breast cancer
    Page E Abrahamson
    Department of Epidemiology, University of North Carolina, Chapel Hill, North Carolina, USA
    Cancer 107:1777-85. 2006
    ..Most epidemiologic studies report a reduced risk of developing breast cancer associated with higher levels of recreational physical activity, but little is known regarding its effect on prognosis...
  60. pmc Associations between polycyclic aromatic hydrocarbon-related exposures and p53 mutations in breast tumors
    Irina Mordukhovich
    Department of Epidemiology, University of North Carolina, Chapel Hill, North Carolina 27599 7435, USA
    Environ Health Perspect 118:511-8. 2010
    ..Objectives: We hypothesized that associations of PAH-related exposures with breast cancer would differ according to tumor p53 mutation status, effect, type, and number...
  61. pmc Serum adiponectin in relation to body mass index and other correlates in black and white women
    Sarah S Cohen
    Department of Epidemiology, University of North Carolina, Chapel Hill, USA
    Ann Epidemiol 21:86-94. 2011
    ..Adiponectin is a promising biomarker linking obesity and disease risk; however, limited data are available regarding adiponectin in black women among whom obesity is highly prevalent...
  62. doi request reprint Dichlorodiphenyldichloroethane and polychlorinated biphenyls: intraindividual changes, correlations, and predictors in healthy women from the southeastern United States
    Thao T Vo
    Department of Epidemiology, School of Public Health, University of North Carolina in Chapel Hill, Chapel Hill, North Carolina, USA
    Cancer Epidemiol Biomarkers Prev 17:2729-36. 2008
    ....
  63. ncbi request reprint Nonsteroidal anti-inflammatory drug use associated with reduced incidence of adenocarcinomas of the esophagus and gastric cardia that overexpress cyclin D1: a population-based study
    Marilie D Gammon
    University of North Carolina, School of Public Health, Department of Epidemiology, Chapel Hill, North Carolina, USA
    Cancer Epidemiol Biomarkers Prev 13:34-9. 2004
    ..These data suggest that the reduction in risk associated with NSAID use may be restricted to those esophageal and gastric cardia adenocarcinomas that overexpress cyclin D1...
  64. ncbi request reprint The epidemiology of gastric cancer
    Mary Beth Terry
    Columbia University Mailman School of Public Health, Department of Epidemiology, New York, NY 10032, USA
    Semin Radiat Oncol 12:111-27. 2002
    ..Future epidemiologic studies that include information on site of origin as well as molecular markers promise to yield more homogeneous classification of case groups, which will enhance identification of underlying disease processes...
  65. pmc Airborne emissions from 1961 to 2004 of benzo[a]pyrene from U.S. vehicles per km of travel based on tunnel studies
    Jan Beyea
    Consulting in the Public Interest CIPI, Lambertville, New Jersey, Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY 10032, USA
    Environ Sci Technol 42:7315-20. 2008
    ....
  66. ncbi request reprint The association between oral contraceptive use and lobular and ductal breast cancer in young women
    Sarah J Nyante
    Department of Epidemiology, University of North Carolina at Chapel Hill, Chapel Hill, NC 27599, USA
    Int J Cancer 122:936-41. 2008
    ..These results are similar to previous studies that report little difference in the effect of oral contraceptive use on breast cancer by histology...
  67. ncbi request reprint Obesity before age 30 years and risk of advanced prostate cancer
    Whitney R Robinson
    Department of Epidemiology, School of Public Health, University of North Carolina, Chapel Hill, NC 27599 7435, USA
    Am J Epidemiol 161:1107-14. 2005
    ....
  68. ncbi request reprint Oral contraceptive use and cyclin D1 overexpression in breast cancer among young women
    Mary Beth Terry
    Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, New York 10032, USA
    Cancer Epidemiol Biomarkers Prev 11:1100-3. 2002
    ..2-4.0) but not for breast cancer without cyclin D1 overexpression (OR, 1.1; 95% CI, 0.7-1.8). If replicated, these findings suggest that early OC use may be associated with the subset of mammary tumors that overexpress cyclin D1...
  69. ncbi request reprint Perceptions of participation in an observational epidemiologic study of cancer among African Americans
    Kyna M Gooden
    Department of Epidemiology, University of North Carolina at Chapel Hill, 2101 McGavran Greenberg Hall, CB 7435, Chapel Hill, NC 26599, USA
    Ethn Dis 15:68-75. 2005
    ..We assessed perceptions and attitudes of African Americans towards participation in an observational epidemiologic study of cancer, and attitudes toward the data collection process...
  70. pmc Choline metabolism and risk of breast cancer in a population-based study
    Xinran Xu
    Department of Community and Preventive Medicine, Mt Sinai School of Medicine, One Gustave L Levy Place, New York, NY 10029, USA
    FASEB J 22:2045-52. 2008
    ..We observed a significant interaction between dietary betaine intake and the PEMT rs7926 polymorphism (P(interaction)=0.04). Our findings suggest that choline metabolism may play an important role in breast cancer etiology...
  71. ncbi request reprint Polymorphisms in XRCC1 modify the association between polycyclic aromatic hydrocarbon-DNA adducts, cigarette smoking, dietary antioxidants, and breast cancer risk
    Jing Shen
    Department of Environmental Health Sciences, Mailman School of Public Health, Columbia University, New York, NY 10032, USA
    Cancer Epidemiol Biomarkers Prev 14:336-42. 2005
    ..Further understanding of the biological function of XRCC1 variants and their interactions with PAH-DNA adducts, antioxidants, and other genes in the pathway are needed...
  72. ncbi request reprint A functional polymorphism of toll-like receptor 4 gene increases risk of gastric carcinoma and its precursors
    Georgina L Hold
    Department of Medicine and Therapeutics, Aberdeen University, Aberdeen, Scotland
    Gastroenterology 132:905-12. 2007
    ..The TLR4+896A>G polymorphism linked with impaired reactivity to bacterial lipopolysaccharide may play a role in gastric carcinogenesis...
  73. ncbi request reprint A functional 19-base pair deletion polymorphism of dihydrofolate reductase (DHFR) and risk of breast cancer in multivitamin users
    Xinran Xu
    Departments of Community and Preventive Medicine and Epidemiology and Medicine, Columbia University, New York, NY, USA
    Am J Clin Nutr 85:1098-102. 2007
    ..Folic acid from multivitamins needs to be reduced by DHFR before it participates in cellular reactions...
  74. ncbi request reprint Population attributable risks of esophageal and gastric cancers
    Lawrence S Engel
    Division of Cancer Epidemiology and Genetics, National Cancer Institute, Bethesda, MD, USA
    J Natl Cancer Inst 95:1404-13. 2003
    ..Herein, we estimate the population attributable risks (PARs) for various risk factors observed in a multicenter population-based case-control study...
  75. ncbi request reprint Wire coding in the EMF and Breast Cancer on Long Island Study: relationship to magnetic fields
    Erin S O'Leary
    Department of Preventive Medicine, School of Medicine, Stony Brook University, Stony Brook, New York 11794 8036, USA
    J Expo Anal Environ Epidemiol 13:283-93. 2003
    ..Results also suggest that wire codes are a proxy measure, to some degree, for current in-home magnetic field measurements in this study...
  76. ncbi request reprint Breast cancers among very young premenopausal women (United States)
    Michelle D Althuis
    National Cancer Institute, Rockville, MD, USA
    Cancer Causes Control 14:151-60. 2003
    ..To assess risk factors for breast cancer among very young compared to older premenopausal women...
  77. pmc GST, NAT1, CYP1A1 polymorphisms and risk of esophageal and gastric adenocarcinomas
    Louise Wideroff
    Applied Research Program, Division of Cancer Control and Population Sciences, National Cancer Institute, EPN 4005, 6130 Executive Boulevard, MSC 7344, Bethesda, MD 20892 7344, USA
    Cancer Detect Prev 31:233-6. 2007
    ..In a population-based case-control study, we examined risks in relation to polymorphisms of the following genes: GSTP1; GSTM1; GSTT1; NAT1; and CYP1A1...
  78. ncbi request reprint Increased risk of noncardia gastric cancer associated with proinflammatory cytokine gene polymorphisms
    Emad M El-Omar
    Department of Medicine and Therapeutics, Aberdeen University, Foresterhill, Scotland
    Gastroenterology 124:1193-201. 2003
    ..The aim of this study was to evaluate the role of proinflammatory cytokine gene polymorphisms in gastric and esophageal cancers defined by anatomic subsite...
  79. ncbi request reprint Increased risk of early-stage breast cancer related to consumption of sweet foods among women less than age 45 in the United States
    Nancy Potischman
    Division of Cancer Control and Population Studies, Centers for Disease Conztrol annd Prevention, Atlanta, GA, USA
    Cancer Causes Control 13:937-46. 2002
    ..To evaluate the associations of dietary macronutrients, food groups, and eating patterns with risk of breast cancer in a population-based case-control study...
  80. ncbi request reprint Polymorphisms of one-carbon-metabolizing genes and risk of breast cancer in a population-based study
    Xinran Xu
    Department of Community and Preventive Medicine, Mount Sinai School of Medicine, New York, NY 10029, USA
    Carcinogenesis 28:1504-9. 2007
    ..Although the pathway is a network of interrelated enzymes, redundancy exists; evaluating the rate-limiting enzyme and its interaction with environment and other genes within the same pathway is critical in assessing breast cancer risk...
  81. ncbi request reprint Risk of breast cancer classified by joint estrogen receptor and progesterone receptor status among women 20-44 years of age
    Julie A Britton
    Division of Environmental Health Science, Mount Sinai School of Medicine, New York, NY 10029, USA
    Am J Epidemiol 156:507-16. 2002
    ..These findings only modestly support the hypothesis that hormonally related risk factors have differing relations with ER+PR+ versus ER-PR- tumors among younger women...
  82. pmc Genetic polymorphisms in the cyclooxygenase-2 gene, use of nonsteroidal anti-inflammatory drugs, and breast cancer risk
    Jing Shen
    Department of Environmental Health Sciences, Mailman School of Public Health, Columbia University, 630 West 168th Street, New York, New York 10032, USA
    Breast Cancer Res 8:R71. 2006
    ..926, COX-2 .5209, and COX-2 .8473) may reduce overall breast cancer risk or risk for subtypes of breast cancer by modulating the inflammatory response and may interact with aspirin or any NSAID use...
  83. ncbi request reprint OGG1 polymorphisms and breast cancer risk
    Pavel Rossner
    Department of Environmental Health Sciences, Mailman School of Public Health, Columbia University, New York, New York, USA
    Cancer Epidemiol Biomarkers Prev 15:811-5. 2006
    ..In addition, data on functionality of these polymorphisms are crucial to understand if these modest differences are important...
  84. ncbi request reprint One-carbon metabolism, MTHFR polymorphisms, and risk of breast cancer
    Jia Chen
    Department of Community and Preventive Medicine, Mount Sinai School of Medicine, New York, New York, USA
    Cancer Res 65:1606-14. 2005
    ..96) or total folate intake (OR, 1.71; 95% CI, 1.08-2.71). From a public heath perspective, it is important to identify risk factors, such as low B vitamin consumption, that may guide an effective prevention strategy against the disease...
  85. pmc Xeroderma pigmentosum complementation group C genotypes/diplotypes play no independent or interaction role with polycyclic aromatic hydrocarbons-DNA adducts for breast cancer risk
    Jing Shen
    Department of Environmental Health Sciences, Mailman School of Public Health, Columbia University, 630 West 168th Street, P and S 19 418, New York, NY 10032, USA
    Eur J Cancer 44:710-7. 2008
    ..69). These data suggest that XPCs have neither independent effects nor interactions with cigarette smoking and PAH-DNA adducts for breast cancer risk. Further studies with multiple genetic polymorphisms in NER pathway are warranted...
  86. ncbi request reprint IGHMBP2 Thr671Ala polymorphism might be a modifier for the effects of cigarette smoking and PAH-DNA adducts to breast cancer risk
    Jing Shen
    Department of Environmental Health Sciences, Mailman School of Public Health, Columbia University, New York, NY, USA
    Breast Cancer Res Treat 99:1-7. 2006
    ..Additional studies focused on the biological function of the variant-G allele and interactions with other genetic polymorphisms are necessary to confirm our findings...
  87. ncbi request reprint Carbonated soft drink consumption and risk of esophageal adenocarcinoma
    Susan T Mayne
    Department of Epidemiology and Public Health, Yale University School of Medicine and Cancer Center, New Haven, CT 06520 8034, USA
    J Natl Cancer Inst 98:72-5. 2006
    ..These findings indicate that CSD consumption (especially diet CSD) is inversely associated with risk of esophageal adenocarcinoma, and thus it is not likely to have contributed to the rising incidence rates...
  88. pmc Modification by N-acetyltransferase 1 genotype on the association between dietary heterocyclic amines and colon cancer in a multiethnic study
    Lesley M Butler
    University of California Davis, Department of Public Health Sciences, Division of Epidemiology, Davis, CA 95616, USA
    Mutat Res 638:162-74. 2008
    ....
  89. ncbi request reprint Shift work, light at night, and breast cancer on Long Island, New York
    Erin S O'Leary
    Department of Preventive Medicine, School of Medicine, Stony Brook University, Stony Brook, NY 11794 8036, USA
    Am J Epidemiol 164:358-66. 2006
    ..Furthermore, overall and evening shift work were not significant factors, and analyses of overnight shift workers yielded reduced risk estimates. The study thus provides mixed evidence for the light-at-night hypothesis...
  90. pmc Validation and calibration of a model used to reconstruct historical exposure to polycyclic aromatic hydrocarbons for use in epidemiologic studies
    Jan Beyea
    Consulting in the Public Interest, Lambertville, New Jersey, USA
    Environ Health Perspect 114:1053-8. 2006
    ..Here we report the results of four exercises to validate and calibrate the model...
  91. ncbi request reprint Improving organochlorine biomarker models for cancer research
    Mary S Wolff
    Department of Community and Preventive Medicine, Mount Sinai School of Medicine, New York, NY 10029, USA
    Cancer Epidemiol Biomarkers Prev 14:2224-36. 2005
    ..e., PCB) or long discontinued (i.e., DDE)...
  92. ncbi request reprint MGMT genotype modulates the associations between cigarette smoking, dietary antioxidants and breast cancer risk
    Jing Shen
    Department of Environmental Health Sciences, Columbia University, New York, NY 10032, USA
    Carcinogenesis 26:2131-7. 2005
    ....
  93. ncbi request reprint Genetic polymorphisms in the apoptosis-associated genes FAS and FASL and breast cancer risk
    Katherine D Crew
    Department of Medicine and the Herbert Irving Comprehensive Cancer Center, Columbia University, 161 Fort Washington Avenue, 10 1072, New York, NY 10032, USA
    Carcinogenesis 28:2548-51. 2007
    ..Overall, this study suggests that the risk of breast cancer may be elevated among women with polymorphisms in the FAS gene and detectable PAH-DNA adducts...
  94. ncbi request reprint Reported residential pesticide use and breast cancer risk on Long Island, New York
    Susan L Teitelbaum
    Department of Community Medicine, Mount Sinai School of Medicine, One Gustave Levy Place, New York, NY 10029, USA
    Am J Epidemiol 165:643-51. 2007
    ..This study is the first known to suggest that self-reported use of residential pesticides may increase breast cancer risk. Further investigation in other populations is necessary to confirm these findings...
  95. doi request reprint Estrogen-biosynthesis gene CYP17 and its interactions with reproductive, hormonal and lifestyle factors in breast cancer risk: results from the Long Island Breast Cancer Study Project
    Yu Chen
    Department of Environmental Medicine and New York University Cancer Institute, New York University School of Medicine, New York, NY 10016, USA
    Carcinogenesis 29:766-71. 2008
    ..In conclusion, the findings suggest that the CYP17 variant C allele may increase breast cancer risk in conjunction with long-term HRT use and high BMI in postmenopausal women...
  96. ncbi request reprint Occupation and risk of esophageal and gastric cardia adenocarcinoma
    Lawrence S Engel
    Occupational Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
    Am J Ind Med 42:11-22. 2002
    ..Adenocarcinomas of the esophagus and gastric cardia have risen dramatically in incidence over the past few decades, however, little research has been conducted on the occupational risk factors for these cancers...