B Gallis

Summary

Affiliation: University of Washington
Country: USA

Publications

  1. pmc Comparison of Francisella tularensis genomes reveals evolutionary events associated with the emergence of human pathogenic strains
    Laurence Rohmer
    Department of Genome Sciences, University of Washington, Campus Box 357710, 1705 NE Pacific Street Seattle, Washington 98195, USA
    Genome Biol 8:R102. 2007
  2. ncbi request reprint Identification of flow-dependent endothelial nitric-oxide synthase phosphorylation sites by mass spectrometry and regulation of phosphorylation and nitric oxide production by the phosphatidylinositol 3-kinase inhibitor LY294002
    B Gallis
    Department of Medicine, University of Washington, Seattle, Washington 98195, USA
    J Biol Chem 274:30101-8. 1999
  3. ncbi request reprint TNF-alpha inhibits flow and insulin signaling leading to NO production in aortic endothelial cells
    F Kim
    Department of Medicine, Division of Cardiology, Harborview Medical Center, University of Washington, Seattle, Washington 98104, USA
    Am J Physiol Cell Physiol 280:C1057-65. 2001
  4. ncbi request reprint A structure-function analysis of serine/threonine phosphorylation of the thrombopoietin receptor, c-Mpl
    Y Miyakawa
    Divisions of Hematology and Cardiology, University of Washington School of Medicine, Seattle, Washington 98195, USA
    J Biol Chem 275:32214-9. 2000

Collaborators

  • B C Berk
  • J G Drachman
  • D Figeys
  • Laurence Rohmer
  • F Kim
  • Y Miyakawa
  • Donald Bovee
  • Danielle Buckley
  • Becky Drees
  • Matthew Radey
  • David R Goodlett
  • Scott A Shaffer
  • Eric Haugen
  • Samuel I Miller
  • Larry A Gallagher
  • Simone Abmayr
  • Jean Chang
  • Rajinder Kaul
  • Christine Fong
  • Don Guenthener
  • Robert K Ernst
  • Mitchell J Brittnacher
  • Kerstin Svensson
  • Hillary S Hayden
  • Will Gillett
  • Yang Zhou
  • Maynard V Olson
  • Michael Wasnick
  • Theodore J Larson Freeman
  • Colin Manoil
  • Ruth Levy
  • Michael Jacobs
  • Jinzhi Chen
  • Regina Lim
  • Greg Taylor
  • Mats Forsman
  • David A D'Argenio
  • Allison Kang
  • Tina Guina
  • M A Corson
  • A Kaushansky
  • K Kaushansky

Detail Information

Publications4

  1. pmc Comparison of Francisella tularensis genomes reveals evolutionary events associated with the emergence of human pathogenic strains
    Laurence Rohmer
    Department of Genome Sciences, University of Washington, Campus Box 357710, 1705 NE Pacific Street Seattle, Washington 98195, USA
    Genome Biol 8:R102. 2007
    ....
  2. ncbi request reprint Identification of flow-dependent endothelial nitric-oxide synthase phosphorylation sites by mass spectrometry and regulation of phosphorylation and nitric oxide production by the phosphatidylinositol 3-kinase inhibitor LY294002
    B Gallis
    Department of Medicine, University of Washington, Seattle, Washington 98195, USA
    J Biol Chem 274:30101-8. 1999
    ..Finally, PKB phosphorylated eNOS in vitro at the same site phosphorylated in the cell and increased eNOS enzymatic activity by 15-20-fold...
  3. ncbi request reprint TNF-alpha inhibits flow and insulin signaling leading to NO production in aortic endothelial cells
    F Kim
    Department of Medicine, Division of Cardiology, Harborview Medical Center, University of Washington, Seattle, Washington 98104, USA
    Am J Physiol Cell Physiol 280:C1057-65. 2001
    ..This study suggests one potential mechanism for the development of endothelial dysfunction in disease states with alterations in insulin regulation and increased TNF-alpha levels...
  4. ncbi request reprint A structure-function analysis of serine/threonine phosphorylation of the thrombopoietin receptor, c-Mpl
    Y Miyakawa
    Divisions of Hematology and Cardiology, University of Washington School of Medicine, Seattle, Washington 98195, USA
    J Biol Chem 275:32214-9. 2000
    ..However, the mutation of Ser-18 to Ala nearly abrogates TPO-induced proliferation and co-precipitation of JAK2 with Mpl. This study provides the first systematic analysis of the role of Ser residues in c-Mpl signaling...