Affiliation: University of North Carolina
- Mucosal healing and fibrosis after acute or chronic inflammation in wild type FVB-N mice and C57BL6 procollagen α1(I)-promoter-GFP reporter miceShengli Ding
Department of Cell and Molecular Physiology, The University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, United States of America
PLoS ONE 7:e42568. 2012..Intestinal fibrosis is a major complication of Crohn's disease. The cellular and molecular basis of mucosal healing and intestinal fibrosis are not well defined and better understanding requires well characterized mouse models...
- High-fat diet: bacteria interactions promote intestinal inflammation which precedes and correlates with obesity and insulin resistance in mouseShengli Ding
Department of Cell and Molecular Physiology, University of North Carolina at Chapel Hill, North Carolina, United States of America
PLoS ONE 5:e12191. 2010..This study tested the hypothesis that HF western diet and gut bacteria interact to promote intestinal inflammation, which contributes to the progression of obesity and insulin resistance...
- Role of intestinal inflammation as an early event in obesity and insulin resistanceShengli Ding
Department of Cell and Molecular Physiology, Center for Gastrointestinal Biology and Disease University of North Carolina at Chapel Hill, Chapel Hill, North Carolina 27599 7574, USA
Curr Opin Clin Nutr Metab Care 14:328-33. 2011..To highlight recent evidence supporting a concept that intestinal inflammation is a mediator or contributor to development of obesity and insulin resistance...