Matthew J DiMagno

Summary

Affiliation: University of Michigan
Country: USA

Publications

  1. ncbi New advances in acute pancreatitis
    Matthew J DiMagno
    University of Michigan Medical School, Division of Gastroenterology and Hepatology, Department of Internal Medicine, Ann Arbor, Michigan, USA
    Curr Opin Gastroenterol 23:494-501. 2007
  2. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan 48109, USA
    Curr Opin Gastroenterol 25:454-9. 2009
  3. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan 48109 0682, USA
    Curr Opin Gastroenterol 28:523-31. 2012
  4. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan 48109 0682, USA
    Curr Opin Gastroenterol 27:452-9. 2011
  5. ncbi Pancreas divisum
    Matthew J DiMagno
    Department of Internal Medicine, Division of Gastroenterology and Hepatology, University of Michigan Medical School, 1150 West Medical Center Drive, Room 6520 MSRB I, Ann Arbor, MI 48109 0682, USA
    Curr Gastroenterol Rep 13:150-6. 2011
  6. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Department of Internal Medicine, Division of Gastroenterology and Hepatology, University of Michigan Medical School, Ann Arbor, Michigan 48109 0682, USA
    Curr Opin Gastroenterol 26:490-8. 2010
  7. ncbi Inhibition of acinar apoptosis occurs during acute pancreatitis in the human homologue DeltaF508 cystic fibrosis mouse
    Matthew J DiMagno
    Dept of Internal Medicine, Univ of Michigan Medical School, Ann Arbor, 48109, USA
    Am J Physiol Gastrointest Liver Physiol 299:G400-12. 2010
  8. ncbi Nitric oxide pathways and evidence-based perturbations in acute pancreatitis
    Matthew J DiMagno
    Department of Internal Medicine, Division of Gastroenterology and Hepatology, University of Michigan School of Medicine, Ann Arbor, MI 48109, USA
    Pancreatology 7:403-8. 2007
  9. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, MI 48109 0362, USA
    Curr Opin Gastroenterol 21:544-54. 2005
  10. ncbi Secretagogue-stimulated pancreatic secretion is differentially regulated by constitutive NOS isoforms in mice
    Matthew J DiMagno
    Department of Internal Medicine, The University of Michigan Medical School, 1500 E Medical Center Dr, 3912 Taubman Center, Ann Arbor, MI 48109 0362, USA
    Am J Physiol Gastrointest Liver Physiol 286:G428-36. 2004

Research Grants

  1. Inflammatory response to pancreatitis in CF mice: sodium & impaired apoptosis
    MATTHEW DIMAGNO; Fiscal Year: 2007

Collaborators

Detail Information

Publications23

  1. ncbi New advances in acute pancreatitis
    Matthew J DiMagno
    University of Michigan Medical School, Division of Gastroenterology and Hepatology, Department of Internal Medicine, Ann Arbor, Michigan, USA
    Curr Opin Gastroenterol 23:494-501. 2007
    ..We focused on clinical aspects of acute pancreatitis, which contained new observations or insights into new or old concepts. For experimental acute pancreatitis we refer readers to a recent comprehensive review...
  2. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan 48109, USA
    Curr Opin Gastroenterol 25:454-9. 2009
    ..We review important new clinical observations in chronic pancreatitis made in the last year...
  3. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan 48109 0682, USA
    Curr Opin Gastroenterol 28:523-31. 2012
    ..We review important new clinical observations in chronic pancreatitis reported in 2011...
  4. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan 48109 0682, USA
    Curr Opin Gastroenterol 27:452-9. 2011
    ..We review important new clinical observations in chronic pancreatitis made in the past year...
  5. ncbi Pancreas divisum
    Matthew J DiMagno
    Department of Internal Medicine, Division of Gastroenterology and Hepatology, University of Michigan Medical School, 1150 West Medical Center Drive, Room 6520 MSRB I, Ann Arbor, MI 48109 0682, USA
    Curr Gastroenterol Rep 13:150-6. 2011
    ....
  6. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Department of Internal Medicine, Division of Gastroenterology and Hepatology, University of Michigan Medical School, Ann Arbor, Michigan 48109 0682, USA
    Curr Opin Gastroenterol 26:490-8. 2010
    ..We review important new clinical observations in chronic pancreatitis made in the past year...
  7. ncbi Inhibition of acinar apoptosis occurs during acute pancreatitis in the human homologue DeltaF508 cystic fibrosis mouse
    Matthew J DiMagno
    Dept of Internal Medicine, Univ of Michigan Medical School, Ann Arbor, 48109, USA
    Am J Physiol Gastrointest Liver Physiol 299:G400-12. 2010
    ..Impaired transduction of increased apoptosis signaling in DeltaF508-CF acini may be biologically relevant to the pathogenesis of AP associated with CFTR mutations...
  8. ncbi Nitric oxide pathways and evidence-based perturbations in acute pancreatitis
    Matthew J DiMagno
    Department of Internal Medicine, Division of Gastroenterology and Hepatology, University of Michigan School of Medicine, Ann Arbor, MI 48109, USA
    Pancreatology 7:403-8. 2007
    ..The aims of this minireview are to describe normal intrapancreatic NO pathways, perturbations during experimental AP and due to epidemiological factors associated with pancreatic pathology, and the clinical implications of NO on AP...
  9. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, MI 48109 0362, USA
    Curr Opin Gastroenterol 21:544-54. 2005
    ..We included articles, including review articles, only if they contained new observations or readdressed old questions and provided new insights into old and new concepts...
  10. ncbi Secretagogue-stimulated pancreatic secretion is differentially regulated by constitutive NOS isoforms in mice
    Matthew J DiMagno
    Department of Internal Medicine, The University of Michigan Medical School, 1500 E Medical Center Dr, 3912 Taubman Center, Ann Arbor, MI 48109 0362, USA
    Am J Physiol Gastrointest Liver Physiol 286:G428-36. 2004
    ..eNOS likely plays a dominant role, because eNOS deletion mimics NOS blockade by inhibiting CCK-8 and carbachol-stimulated secretion, whereas nNOS deletion augments CCK-8 but not carbachol-stimulated secretion...
  11. ncbi Chronic pancreatitis
    Matthew J DiMagno
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan, USA
    Curr Opin Gastroenterol 22:487-97. 2006
    ..Topics recently reviewed were truncated to accommodate a surge in publications on clinical aspects of chronic pancreatitis, which contained new observations or insights into new or old concepts...
  12. ncbi Endothelial nitric oxide synthase is protective in the initiation of caerulein-induced acute pancreatitis in mice
    Matthew J DiMagno
    Department of Internal Medicine, Division of Gastroenterology, The University of Michigan Medical School, 1500 E Medical Center Drive, 3912 Taubman Center, Ann Arbor, MI 48109 0362, USA
    Am J Physiol Gastrointest Liver Physiol 287:G80-7. 2004
    ..Thus eNOS is the main isoform influencing the initiation of caerulein-induced AP. eNOS-derived NO exerts a protective effect through actions on nonacinar cell types, most likely endothelial cells, to produce greater PMBF...
  13. ncbi A proinflammatory, antiapoptotic phenotype underlies the susceptibility to acute pancreatitis in cystic fibrosis transmembrane regulator (-/-) mice
    Matthew J DiMagno
    Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, 48109, USA
    Gastroenterology 129:665-81. 2005
    ..We hypothesized that cftr(m1UNC) (-/-) mice, which have no evidence of chronic pancreatitis, are susceptible to developing acute pancreatitis...
  14. ncbi Pioglitazone reverses insulin resistance and impaired CCK-stimulated pancreatic secretion in eNOS(-/-) mice: therapy for exocrine pancreatic disorders?
    Raju C Reddy
    Department of Internal Medicine, Division of Gastroenterology and Hepatology, University of Michigan School of Medicine, 1150 W Medical Center Drive, 6520 MSRB 1, Ann Arbor, MI 48109, USA
    Am J Physiol Gastrointest Liver Physiol 293:G112-20. 2007
    ..Insulin-sensitizing PPAR-gamma agonists such as pioglitazone may thus simultaneously correct endocrine and exocrine pancreatic disorders...
  15. ncbi Alcohol use and cigarette smoking as risk factors for post-endoscopic retrograde cholangiopancreatography pancreatitis
    Anthony T DeBenedet
    Department of Internal Medicine, University of Michigan School of Medicine, Ann Arbor, Michigan 48109, USA
    Clin Gastroenterol Hepatol 7:353-8e4. 2009
    ..We performed a retrospective case-control study to determine if these activities increase the risk of PEP...
  16. ncbi Caerulein-induced acute pancreatitis inhibits protein synthesis through effects on eIF2B and eIF4F
    Maria Dolors Sans
    Department of Molecular and Integrative Physiology, The University of Michigan Medical School, Ann Arbor, MI 48109 0622, USA
    Am J Physiol Gastrointest Liver Physiol 285:G517-28. 2003
    ....
  17. ncbi Abdominal wall pain and crepitus in Marfan's syndrome caused by subcutaneous air
    Frederick K Shieh
    Department of Internal Medicine, Division of Gastroenterology and Hepatology, University of Michigan School of Medicine, Ann Arbor, Michigan, USA
    Clin Gastroenterol Hepatol 6:A24. 2008
  18. ncbi Listeria monocytogenes exploits cystic fibrosis transmembrane conductance regulator (CFTR) to escape the phagosome
    Andrea L Radtke
    Department of Microbiology and Immunology, University of Michigan Medical School, Ann Arbor, MI 48109, USA
    Proc Natl Acad Sci U S A 108:1633-8. 2011
    ..Our studies suggest that Listeria exploits mechanisms of cellular ion homeostasis to escape the phagosome and emphasize host ion-channel function as a key parameter of bacterial virulence...
  19. ncbi Massive lower gastrointestinal bleeding after rejection of pancreatic transplants
    Peter D R Higgins
    Division of Gastroenterology, University of Michigan Medical Center, Ann Arbor, MI 48109, USA
    Nat Clin Pract Gastroenterol Hepatol 2:240-4; quiz 1 p following 244. 2005
    ..He presented in late 2003 with a single episode of coffee-ground emesis and two episodes of brisk hematochezia...
  20. ncbi Cirrhosis, spontaneous venous thromboses, and splenic gas gangrene
    Matthew J DiMagno
    University of Michigan School of Medicine, Ann Arbor, Michigan, USA
    Clin Gastroenterol Hepatol 5:e14. 2007
  21. ncbi Editorial: Pancreas Divisum Does Not Cause Pancreatitis, But Associates With CFTR Mutations
    Matthew J DiMagno
    Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Michigan School of Medicine, Ann Arbor, Michigan, USA
    Am J Gastroenterol 107:318-20. 2012
    ..In lieu of CFTR mutations undertake no endoscopic/surgical procedure; if CFTR mutations are found, then refer patients for genetic counseling and withhold endoscopic/surgical therapy unless randomized studies show benefit...
  22. ncbi Overexpression of heat shock protein Hsp27 protects against cerulein-induced pancreatitis
    Constanze Kubisch
    Department of Internal Medicine II, Klinikum Grosshadern, Ludwig Maximilians University Munich, Germany
    Gastroenterology 127:275-86. 2004
    ....
  23. ncbi Does endoscopic therapy favorably affect the outcome of patients who have recurrent acute pancreatitis and pancreas divisum?
    Evan L Fogel
    Division of Gastroenterology Hepatology, Indiana University Medical Center, Indiana, IN, USA
    Pancreas 34:21-45. 2007

Research Grants4

  1. Inflammatory response to pancreatitis in CF mice: sodium & impaired apoptosis
    MATTHEW DIMAGNO; Fiscal Year: 2007
    ..Understanding of the pathophysiology of AP is incomplete & therapies are lacking. The proposed study may provide insight into the pathogenesis of AP & CF & contribute to generation of novel therapies. ..