Dawn Belt Davis

Summary

Affiliation: University of Wisconsin
Country: USA

Publications

  1. pmc FoxM1 is up-regulated by obesity and stimulates beta-cell proliferation
    Dawn Belt Davis
    Department of Medicine, University of Wisconsin, Madison, WI, USA
    Mol Endocrinol 24:1822-34. 2010
  2. pmc Cholecystokinin is up-regulated in obese mouse islets and expands beta-cell mass by increasing beta-cell survival
    Jeremy A Lavine
    Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706, USA
    Endocrinology 151:3577-88. 2010
  3. pmc A gene expression network model of type 2 diabetes links cell cycle regulation in islets with diabetes susceptibility
    Mark P Keller
    Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53076, USA
    Genome Res 18:706-16. 2008
  4. pmc Tcf19 is a novel islet factor necessary for proliferation and survival in the INS-1 β-cell line
    Kimberly A Krautkramer
    Department of Medicine, Division of Endocrinology, Diabetes, and Metabolism, University of Wisconsin, Madison, Wisconsin
    Am J Physiol Endocrinol Metab 305:E600-10. 2013
  5. pmc Overexpression of pre-pro-cholecystokinin stimulates beta-cell proliferation in mouse and human islets with retention of islet function
    Jeremy A Lavine
    Department of Biochemistry, University of Wisconsin Madison, Madison, Wisconsin 53706, USA
    Mol Endocrinol 22:2716-28. 2008

Collaborators

  • Alan D Attie
  • Brian S Yandell
  • Eric E Schadt
  • Ping Wang
  • Christopher B Newgard
  • Luis A Fernandez
  • Jeremy A Lavine
  • Mark P Keller
  • Mary E Rabaglia
  • Kimberly A Krautkramer
  • Marc K Hellerstein
  • Donald S Stapleton
  • Philipp W Raess
  • Margery C Beinfeld
  • Nathan A Truchan
  • Ondine Cleaver
  • Michelle E Kimple
  • Ted W Harris
  • Amy L Whillock
  • Danielle A Fontaine
  • Gregory J Schleis
  • Leilani Marty-Santos
  • Amelia K Linnemann
  • John A Dawson
  • Linda C Samuelson
  • James E Koltes
  • Joshua I Suhonen
  • Kathryn L Schueler
  • H Adam Steinberg
  • Christina Kendziorski
  • Carmen Argmann
  • Alan S Kopin
  • Elias Chaibub Neto
  • Kathy L Schueler
  • Younjeong Choi
  • Robert Kleinhanz
  • Brent K Presley
  • Steve Edwards
  • Scott Turner
  • Angie T Oler

Detail Information

Publications5

  1. pmc FoxM1 is up-regulated by obesity and stimulates beta-cell proliferation
    Dawn Belt Davis
    Department of Medicine, University of Wisconsin, Madison, WI, USA
    Mol Endocrinol 24:1822-34. 2010
    ..Our data suggest that beta-cell proliferation occurs in adult obese humans in an attempt to expand beta-cell mass to compensate for insulin resistance, and that the FoxM1 transcriptional program plays a key role in this process...
  2. pmc Cholecystokinin is up-regulated in obese mouse islets and expands beta-cell mass by increasing beta-cell survival
    Jeremy A Lavine
    Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706, USA
    Endocrinology 151:3577-88. 2010
    ..In summary, CCK is up-regulated by islet cells during obesity and functions as a paracrine or autocrine factor to increase beta-cell survival and expand beta-cell mass to compensate for obesity-induced insulin resistance...
  3. pmc A gene expression network model of type 2 diabetes links cell cycle regulation in islets with diabetes susceptibility
    Mark P Keller
    Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53076, USA
    Genome Res 18:706-16. 2008
    ....
  4. pmc Tcf19 is a novel islet factor necessary for proliferation and survival in the INS-1 β-cell line
    Kimberly A Krautkramer
    Department of Medicine, Division of Endocrinology, Diabetes, and Metabolism, University of Wisconsin, Madison, Wisconsin
    Am J Physiol Endocrinol Metab 305:E600-10. 2013
    ..Therefore, Tcf19 is necessary for both proliferation and survival and is a novel regulator of these pathways...
  5. pmc Overexpression of pre-pro-cholecystokinin stimulates beta-cell proliferation in mouse and human islets with retention of islet function
    Jeremy A Lavine
    Department of Biochemistry, University of Wisconsin Madison, Madison, Wisconsin 53706, USA
    Mol Endocrinol 22:2716-28. 2008
    ..CCK triggers beta-cell proliferation without disrupting islet function, up-regulates a distinct set of cell cycle regulators in islets, and signals independently of the CCK receptors...