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Species | J L CummingsSummaryAffiliation: University of California Country: USA Publications
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Publications
Cholinesterase inhibitors: A new class of psychotropic compoundsJ L Cummings
Department of Neurology, UCLA School of Medicine, Los Angeles, CA 90095 1769, USA
Am J Psychiatry 157:4-15. 2000..This article reviews evidence indicating that acetylcholinesterase inhibitors have psychotropic properties...
Neuropsychiatric assessment of Alzheimer's disease and related dementiasJ L Cummings
Department of Neurology, UCLA School of Medicine, Los Angeles, California 90095 1769, USA
Aging (Milano) 13:240-6. 2001..Evaluation of neuropsychiatric symptoms is a critical aspect of dementia diagnosis and management...
Regression to the mean: implications for clinical trials of psychotropic agents in dementiaJeffrey L Cummings
Department of Neurology, UCLA School of Medicine, Los Angeles, CA 90095 1769, USA
Curr Alzheimer Res 1:323-8. 2004..Research design adjustments such as collection of both screening and baseline measures to determine eligibility may limit the effects of regression to the mean on trial outcomes and reduce this challenge to clinical trials...
Use of cholinesterase inhibitors in clinical practice: evidence-based recommendationsJeffrey L Cummings
Departments of Neurology and Psychiatry and Biobehavioral Sciences, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095 1769, USA
Am J Geriatr Psychiatry 11:131-45. 2003..Clinical-trial populations differ substantially from unselected populations of AD patients, and these selection biases demand that efficacy data from clinical trials be generalized with caution...
Alzheimer diseaseJeffrey L Cummings
Department of Neurology, University of California, Los Angeles, School of Medicine, Los Angeles, CA 90095, USA
JAMA 287:2335-8. 2002
Cognitive and behavioral heterogeneity in Alzheimer's disease: seeking the neurobiological basisJ L Cummings
Departments of Neurology and Psychiatry and Biobehavioral Sciences, UCLA School of Medicine, Reed Neurological Research Center, 710 Westwood Plaza, Los Angeles CA 90095 1769, USA
Neurobiol Aging 21:845-61. 2000..There are a plethora of opportunities for basic scientists to offer insights, develop strategies, and provide techniques and technologies relevant to understanding the clinical manifestations of AD...
Reduction of behavioral disturbances and caregiver distress by galantamine in patients with Alzheimer's diseaseJeffrey L Cummings
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles 90095 1769, USA
Am J Psychiatry 161:532-8. 2004....
Alzheimer's disease and its management in the year 2010J L Cummings
Department of Neurology, University of California, Los Angeles, School of Medicine, 90095 1769, USA
Psychiatr Serv 50:1173-7. 1999....
Behavioral differences between frontotemporal dementia and Alzheimer's disease: a comparison on the BEHAVE-AD rating scaleM F Mendez
Department of Neurology, University of California at Los Angeles, USA
Int Psychogeriatr 10:155-62. 1998..The assessment of neuropsychiatric symptoms with a standardized scale or inventory can help distinguish dementia patients with FTD and AD...
Frequency and characteristics of anxiety among patients with Alzheimer's disease and related dementiasVerna R Porter
Department of Neurology, University of California, Los Angeles, School of Medicine, USA
J Neuropsychiatry Clin Neurosci 15:180-6. 2003..These data suggest that anxiety is common among patients with diverse forms of dementia. In AD, anxiety is most common in those with more severe cognitive deterioration and an earlier age at onset...
Donepezil delays progression to AD in MCI subjects with depressive symptomsP H Lu
Departments of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA, USA
Neurology 72:2115-21. 2009..To determine whether the presence of depression predicts higher rate of progression to Alzheimer disease (AD) in patients with amnestic mild cognitive impairment (aMCI) and whether donepezil treatment beneficially affect this relationship...
Executive dysfunction in Alzheimer diseaseMargaret M Swanberg
Department of Neurology, David Geffen School of Medicine at UCLA, 90095-1769, USA
Arch Neurol 61:556-60. 2004..CONCLUSION: These findings support the assessment of executive function in persons with AD and the importance of frontal lobe dysfunction in AD...
ADCS Prevention Instrument Project: development of a brief verbal memory test for primary prevention clinical trialsDavid P Salmon
Department of Neurosciences, University of California, San Diego, La Jolla, California 92093 0948, USA
Alzheimer Dis Assoc Disord 20:S139-46. 2006....
Dementia with leukoaraiosis: clinical differentiation by temporoparietal hypometabolism on (18)FDG-PET imagingM F Mendez
Psychiatry, VA Greater Los Angeles Healthcare System, UCLA School of Medicine, Los Angeles, CA 90073, USA
Dement Geriatr Cogn Disord 10:518-25. 1999..Copyrightz1999S.KargerAG,Basel..
Cerebral correlates of psychotic symptoms in Alzheimer's diseaseM S Mega
Department of Neurology UCLA School of Medicine, UCLA School of Medicine, Los Angeles, California, USA
J Neurol Neurosurg Psychiatry 69:167-71. 2000..Psychotic symptoms are produced by distributed neuronal dysfunction. Abnormalities of reality testing and false inference implicate frontal lobe abnormalities...
Dementia with parkinsonism: what is the diagnosis?Mei S Chong
Department of Neurology, The David Geffen School of Medicine at UCLA, Los Angeles, CA, USA
Rev Neurol Dis 1:41-4. 2004..The 72-year-old patient whose case is reviewed here had abnormalities in all three domains...
Mild cognitive impairment is associated with characteristic neuropsychiatric symptomsTzung J Hwang
Department of Neurology, UCLA School of Medicine, Los Angeles, CA 90095-1769, USA
Alzheimer Dis Assoc Disord 18:17-21. 2004..Psychotic symptoms are significantly more common in the early stage of AD than in MCI. These results are derived from a limited clinical sample and require confirmation in longitudinal community-based investigations...
Dementia and neurodevelopmental predisposition: cognitive dysfunction in presymptomatic subjects precedes dementia by decades in frontotemporal dementiaD H Geschwind
Department of Neurology, University of California at Los Angeles School of Medicine, 90095 1769, USA
Ann Neurol 50:741-6. 2001....
Gender-related cognitive deficits in Alzheimer's diseaseS McPherson
Alzheimer s Disease Center, Department of Neurology, UCLA School of Medicine, Los Angeles, California 90095 1769, USA
Int Psychogeriatr 11:117-22. 1999..Results indicated the presence of gender-related cognitive deficits on tasks of confrontation naming, expressive word knowledge, and both episodic and semantic memory for women with AD, relative to findings in men...
Frontotemporal dementia versus vascular dementia: differential features on mental status examinationM M Cherrier
Department of Neurology, University of California, Los Angeles School of Medicine, USA
J Am Geriatr Soc 45:579-83. 1997..The purpose of this study was to elucidate distinct cognitive profiles of a large group of FTD and VaD patients on a brief, clinical mental status examination...
Progression of clinical deterioration and pathological changes in patients with Alzheimer disease evaluated at biopsy and autopsyP L Di Patre
Department of Pathology and Laboratory Medicine, University of California, Los Angeles, Medical Center, 90095 1732, USA
Arch Neurol 56:1254-61. 1999....
Efficacy of metrifonate in improving the psychiatric and behavioral disturbances of patients with Alzheimer's diseaseJ L Cummings
Reed Neurological Research Center, University of California at Los Angeles, 90095-1769, USA
J Geriatr Psychiatry Neurol 14:101-8. 2001..Metrifonate significantly reduced many of the psychiatric and behavioral symptoms of AD. The observations suggest that enhancement of cholinergic functions in AD has beneficial effects on behavior...
Neuropathologic correlates of activities of daily living in Alzheimer diseaseGad A Marshall
Department of Neurology, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA 90095-1769, USA
Alzheimer Dis Assoc Disord 20:56-9. 2006..Deteriorating activities of daily living in Alzheimer Disease subjects correlate with greater overall pathologic burden and possibly selectively with involvement of the medial temporal, occipital, and orbital frontal regions...
Early-onset Alzheimer's disease is associated with greater pathologic burdenGad A Marshall
Department of Neurology, David Geffen School of Medicine, University of California, Los Angeles, USA
J Geriatr Psychiatry Neurol 20:29-33. 2007..This suggests that late-onset AD patients have less cognitive reserve than early-onset patients and require fewer pathologic changes to exhibit cognitive deterioration...
Efficacy of olanzapine in the treatment of psychosis in dementia with lewy bodiesJeffrey L Cummings
Department of Neurology, UCLA School of Medicine, Los Angeles, Calif, USA
Dement Geriatr Cogn Disord 13:67-73. 2002..CONCLUSIONS: This preliminary analysis suggests that olanzapine (5 or 10 mg) reduces psychosis in patients with DLB without worsening parkinsonism...
Cognitive and metabolic responses to metrifonate therapy in Alzheimer diseaseM S Mega
Department of Neurology, University of California at Los Angeles School of Medicine, 90095-1769, USA
Neuropsychiatry Neuropsychol Behav Neurol 14:63-8. 2001..CONCLUSION: The clinical benefits observed in AD with cholinesterase inhibitor therapy are associated with a metabolic increase of heteromodal cognitive and medial temporal networks...
Functional correlates of musical and visual ability in frontotemporal dementiaB L Miller
Department of Neurology, University of California at San Francisco School of Medicine, USA
Br J Psychiatry 176:458-63. 2000..The emergence of new skills in the setting of dementia suggests that loss of function in one brain area can release new functions elsewhere...
Treatment of Alzheimer's disease: current and future therapeutic approachesJeffrey L Cummings
Department of Neurology, University of California, The David Geffen School of Medicine, Los Angeles, CA, USA
Rev Neurol Dis 1:60-9. 2004..Improved methodologies for clinical trial design and analysis and the development of biological markers may hasten the identification of effective treatments for AD...
Positron emission tomography metabolic correlates of apathy in Alzheimer diseaseGad A Marshall
Department of Neurology, University of California, Los Angeles, CA, USA
Arch Neurol 64:1015-20. 2007....
Guidelines for managing Alzheimer's disease: part I. AssessmentJeffrey L Cummings
University of California, Los Angeles, School of Medicine, USA
Am Fam Physician 65:2263-72. 2002..Suggestions for easily administered and scored assessment tools are provided, and practical tips are given for supporting primary caregivers, thereby increasing efficiency and quality of care for patients with Alzheimer's disease...
Utility of clinical criteria in differentiating frontotemporal lobar degeneration (FTLD) from ADHoward J Rosen
Department of Neurology, Memory and Aging Center, University of California at San Francisco, 350 Parnassus Avenue, Suite 800, San Francisco, CA 94143 1207, USA
Neurology 58:1608-15. 2002..To assess the ability of the current diagnostic criteria for frontotemporal lobar degeneration (FTLD) to differentiate FTLD from AD...
Cerebral metabolic and cognitive decline in persons at genetic risk for Alzheimer's diseaseG W Small
Center on Aging and Department of Psychiatry and Biobehavioral Sciences, Neuropsychiatric Institute, 760 Westwood Plaza, Los Angeles, CA 90024, USA
Proc Natl Acad Sci U S A 97:6037-42. 2000..These results indicate that the combination of cerebral metabolic rates and genetic risk factors provides a means for preclinical AD detection that will assist in response monitoring during experimental treatments...
Cortical change in Alzheimer's disease detected with a disease-specific population-based brain atlasP M Thompson
Laboratory of Neuro Imaging, Department of Neurology, Division of Brain Mapping and Alzheimer s Disease Center, UCLA School of Medicine, Los Angeles, CA 90095 1769, USA
Cereb Cortex 11:1-16. 2001..These deficits can therefore be (i) charted in a living population and (ii) compared across individuals and groups, facilitating longitudinal, genetic and interventional studies of dementia...
Treatment of Alzheimer's diseaseJ L Cummings
UCLA Alzheimer's Disease Center, UCLA School of Medicine, Los Angeles, California, USA
Clin Cornerstone 3:27-39. 2001..The 3Rs--repeat, reassure, and redirect--can help caregivers reduce behavioral disturbances in patients with AD and limit the need for pharmacologic management...
Positron emission tomography in evaluation of dementia: Regional brain metabolism and long-term outcomeD H Silverman
Ahmanson Biological Imaging Center, CHS AR 144, Department of Molecular and Medical Pharmacology, University of California, Los Angeles School of Medicine, Los Angeles, CA 90095 6942, USA
JAMA 286:2120-7. 2001..Deficits in cerebral glucose utilization have been identified in patients with cognitive dysfunction attributed to various disease processes, but their prognostic and diagnostic value remains to be defined...
Treatment of Alzheimer's disease: the role of symptomatic agents in an era of disease-modifying therapiesJeffrey L Cummings
Departmentsof Neurology and Psychiatry, David Geffen School of Medicine at UCLA, Los Angeles, CA, USA
Rev Neurol Dis 4:57-62. 2007..Therapeutic regimens for the treatment of AD will include both disease-modifying and symptomatic agents...
Association of GSK3B with Alzheimer disease and frontotemporal dementiaBarbara A J Schaffer
Department of Neurology, David Geffen School of Medicine, University of California, Los Angeles, 2506 Gonda, 695 Charles E Young Dr S, Los Angeles, CA 90095 1761, USA
Arch Neurol 65:1368-74. 2008..As a known tau kinase, GSK3B is a promising candidate gene in the remaining cases of FTD and in AD, for which tau mutations have not been found...
Guidelines for managing Alzheimer's disease: Part II. TreatmentJeffrey L Cummings
University of California, Los Angeles, School of Medicine, USA
Am Fam Physician 65:2525-34. 2002..Throughout the course of the disease, routine use of community resources allows care to be provided by a network of professionals, many of whom will be specialists in Alzheimer's disease...
Creativity and dementia: emerging diagnostic and treatment methods for Alzheimer's diseaseJeffrey L Cummings
UCLA Alzheimer s Disease Research Center, University of California, Los Angeles, CA, USA
CNS Spectr 13:1-20; quiz 22. 2008..Debra Cherry, PhD, discusses the advocacy needs of Alzheimer's disease patients and their caregivers. In addition, Patricia Utermohlen, MA, provides a testimonial of the impact of Alzheimer's disease on an accomplished artist...
The prevalence and correlates of neuropsychiatric symptoms in a population with Parkinson's disease in MexicoJohn M Ringman
Department of Neurology, UC Irvine School of Medicine, Irvine, California, USA
Neuropsychiatry Neuropsychol Behav Neurol 15:99-105. 2002..To study the incidence of behavioral abnormalities in patients with Parkinson's disease (PD) and extend them to a Mexican population...
Orbitofrontal and anterior cingulate cortex neurofibrillary tangle burden is associated with agitation in Alzheimer diseaseS Tekin
Department of Neurology, UCLA School of Medicine, Los Angeles, CA 90095, USA
Ann Neurol 49:355-61. 2001..01). These observations suggest that agitation and aberrant motor behavior are correlates of greater NFT pathology in the orbitofrontal cortex in AD, whereas increasing apathy may relate to greater NFT burden in the anterior cingulate...
Consensus statement on the upcoming crisis in geriatric mental health: research agenda for the next 2 decadesD V Jeste
University of California, San Diego, VA San Diego Healthcare System, 92161, USA
Arch Gen Psychiatry 56:848-53. 1999..New methods of clinical and research training involving specialists, primary care clinicians, and the lay public are warranted...
Executive dysfunction in Alzheimer's disease: association with neuropsychiatric symptoms and functional impairmentS T Chen
Department of Psychiatry and Biobehavioral Sciences, UCLA School of Medicine, USA
J Neuropsychiatry Clin Neurosci 10:426-32. 1998..Executive skills deficits, neuropsychiatric symptoms, and functional disability may emerge from shared neurobiological mechanisms...
The spectrum of behavioral responses to cholinesterase inhibitor therapy in Alzheimer diseaseM S Mega
Laboratory of Neuro Imaging, Alzheimer s Disease Research Center, University of California, Los Angeles, School of Medicine, 90095 1769, USA
Arch Neurol 56:1388-93. 1999..Characterizing the pretreatment profile of patients who are likely to respond to cholinergic therapy will aid the efficient use of clinical resources...
Activities of daily living in Alzheimer's disease: neuropsychiatric, cognitive, and medical illness influencesS Tekin
Department of Neurology, Alzheimer Disease Research Center, and Department of Psychiatry and Biobehavioral Sciences, UCLA School of Medicine, Los Angeles, CA, USA
Am J Geriatr Psychiatry 9:81-6. 2001..IADLs changed with cognition and neuropsychiatric disturbances in AD. Medical illness burden had little influence on functional status and a limited impact on neuropsychiatric symptoms...
MRI evaluation of brain iron in earlier- and later-onset Parkinson's disease and normal subjectsG Bartzokis
North Little Rock VA Medical Center, Department of Psychiatry, University of Arkansas for Medical Sciences, 72114, USA
Magn Reson Imaging 17:213-22. 1999..The FDRI results suggest that disregulation of iron metabolism occurs in PD and that this disregulation may differ in earlier- versus later-onset PD...
Multiple system atrophy presenting with language impairmentL G Apostolova
Department of Neurology, University of California Los Angeles, USA
Neurology 67:726-7. 2006
Biochemical markers in persons with preclinical familial Alzheimer diseaseJ M Ringman
UCLA Department of Neurology, Alzheimer Disease Research Center, 10911 Weyburn Ave, Suite 200, Los Angeles, CA 90095 7226, USA
Neurology 71:85-92. 2008..Persons at risk for familial Alzheimer disease (FAD) provide a model in which biomarkers can be studied in presymptomatic disease...
3D mapping of language networks in clinical and pre-clinical Alzheimer's diseaseLiana G Apostolova
Department of Neurology, David Geffen School of Medicine, UCLA, CA, USA
Brain Lang 104:33-41. 2008..Using a novel 3D mapping technique, we demonstrated that in AD language abilities are strongly influenced by the integrity of the perisylvian cortical regions...
3D mapping of mini-mental state examination performance in clinical and preclinical Alzheimer diseaseLiana G Apostolova
Department of Neurology, David Geffen School of Medicine, UCLA, Los Angeles, CA 90095, USA
Alzheimer Dis Assoc Disord 20:224-31. 2006..As a global cognitive measure, MMSE depends on the integrity of widely distributed cortical areas in both brain hemispheres with left-sided predominance...
ADCS Prevention Instrument Project: behavioral measures in primary prevention trialsJeffrey L Cummings
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095 1769, USA
Alzheimer Dis Assoc Disord 20:S147-51. 2006..Experimental behavioral measures were included in the protocol to examine this hypothesis...
Effects of donepezil on neuropsychiatric symptoms in patients with dementia and severe behavioral disordersJeffrey L Cummings
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, California, USA
Am J Geriatr Psychiatry 14:605-12. 2006..The authors report the effects of donepezil on behavioral disturbances in patients with relatively severe psychopathology at baseline...
3D comparison of hippocampal atrophy in amnestic mild cognitive impairment and Alzheimer's diseaseLiana G Apostolova
Department of Neurology, David Geffen School of Medicine UCLA, CA, USA
Brain 129:2867-73. 2006..Novel hippocampal analytic techniques that can track the spread of hippocampal pathology in 3D with such precision are a promising research tool...
Treatments for behavioural disorders in neurodegenerative diseases: drug development strategiesJeffrey L Cummings
Department of Neurology, 710 Westwood Plaza, David Geffen School of Medicine, UCLA, Los Angeles, California 90095, USA
Nat Rev Drug Discov 5:64-74. 2006....
Neuropsychiatric and behavioral symptoms in a community sample of Hispanics with Alzheimer's diseaseFreddy Ortiz
The Neuropsychiatry Research Memory Clinic, Olive View UCLA Medical Center, Sylmar, CA 91342, USA
Am J Alzheimers Dis Other Demen 21:263-73. 2006..These findings have implications for differential sociocultural presentations of Alzheimer's disease among ethnic/racial groups...
Behavioral effects of memantine in Alzheimer disease patients receiving donepezil treatmentJeffrey L Cummings
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095 1769, USA
Neurology 67:57-63. 2006..To investigate the behavioral effects of memantine in moderate to severe Alzheimer disease (AD)...
Clinical evaluation as a biomarker for Alzheimer's diseaseJeffrey L Cummings
Department of Neurology, Alzheimer s Disease Research Center, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095 1769, USA
J Alzheimers Dis 8:327-37. 2005..Biomarkers must closely reflect clinical outcomes to be useful as evaluations of disease progression or as outcomes in clinical trials...
Deficits in facial emotion processing in mild cognitive impairmentEdmond Teng
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095 7226, USA
Dement Geriatr Cogn Disord 23:271-9. 2007..Patients with Alzheimer disease consistently demonstrate impaired performance on tests of facial emotion processing. However, it remains unclear how early in the neurodegenerative process these deficits emerge...
Effective pharmacologic management of Alzheimer's diseaseMartin R Farlow
Department of Neurology, Indiana University School of Medicine, Indianapolis 46202 5111, USA
Am J Med 120:388-97. 2007....
Effects of rivastigmine transdermal patch and capsule on aspects of clinical global impression of change in Alzheimer's disease: a retrospective analysisJeffrey L Cummings
Mary S Easton Center for Alzheimer s Disease Research at UCLA, Los Angeles, CA 90095 7226, USA
Dement Geriatr Cogn Disord 29:406-12. 2010..This was a hypothesis-forming study, designed to provide a basis for possible future research...
Integrating ADNI results into Alzheimer's disease drug development programsJeffrey L Cummings
Mary S Easton Center for Alzheimer s Disease Research at UCLA, Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095 7226, United States
Neurobiol Aging 31:1481-92. 2010....
3D comparison of low, intermediate, and advanced hippocampal atrophy in MCILiana G Apostolova
Department of Neurology, David Geffen School of Medicine, UCLA, CA, USA
Hum Brain Mapp 31:786-97. 2010..Greater CA1 and subicular atrophy can be demonstrated early and is predictive of future conversion to AD, whereas CA2-3 involvement becomes more evident as the disease progresses...
Automated 3D mapping of baseline and 12-month associations between three verbal memory measures and hippocampal atrophy in 490 ADNI subjectsLiana G Apostolova
Department of Neurology, David Geffen School of Medicine, UCLA, Los Angeles, CA 90095, USA
Neuroimage 51:488-99. 2010..After controlling for baseline hippocampal atrophy, memory performance showed regionally specific associations with hippocampal radial distance in predominantly CA1 but also in subicular distribution...
Rivastigmine transdermal patch skin tolerability: results of a 1-year clinical trial in patients with mild-to-moderate Alzheimer's diseaseJeffrey L Cummings
Mary S Easton Center for Alzheimer s Disease Research at UCLA, 10911 Weyburn Ave, Suite 200, Los Angeles, CA 90095 7226, USA
Clin Drug Investig 30:41-9. 2010..The aim of the component of the trial reported here was to evaluate the skin tolerability of the rivastigmine transdermal patch in patients with mild-to-moderate AD...
Defining and labeling disease-modifying treatments for Alzheimer's diseaseJeffrey L Cummings
Mary S Easton Center for Alzheimer s Disease Research at UCLA, Departments of Neurology and Psychiatry and Biobehavioral Sciences, David Geffen School of Medicine at UCLA, Los Angeles, CA, USA
Alzheimers Dement 5:406-18. 2009..Prevention claims will depend heavily on biomarker outcomes...
Current concepts of mild cognitive impairment and their applicability to persons at-risk for familial Alzheimer's diseaseJohn M Ringman
Mary S Easton Center for Alzheimer s Disease Research at UCLA, Associate Clinical Professor, UCLA Department of Neurology, 10911 Weyburn Ave, 200, Los Angeles, CA 90095 7226, USA
Curr Alzheimer Res 6:341-6. 2009..The study of FAD provides an opportunity to test various criteria for early AD and these observations should be taken into consideration in future iterations of such diagnostic criteria...
Surface feature-guided mapping of cerebral metabolic changes in cognitively normal and mildly impaired elderlyLiana G Apostolova
Department of Neurology, David Geffen School of Medicine, University of California Los Angeles, 10911 Weyburn Avenue, Los Angeles, CA 90095, USA
Mol Imaging Biol 12:218-24. 2010..The aim of this study was to investigate the longitudinal positron emission tomography (PET) metabolic changes in the elderly...
Controversies in Alzheimer's disease drug developmentJeffrey L Cummings
Departments of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA, USA
Int Rev Psychiatry 20:389-95. 2008..The questions surrounding these issues must be resolved as disease-modifying therapies for AD are advanced. These controversies are framed and potential directions towards resolution described...
Behavioral effects of current Alzheimer's disease treatments: a descriptive reviewJeffrey L Cummings
UCLA Alzheimer Disease Center, UCLA, Los Angeles, CA, USA
Alzheimers Dement 4:49-60. 2008....
Diagnosing depression in Alzheimer disease with the national institute of mental health provisional criteriaEdmond Teng
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90073, USA
Am J Geriatr Psychiatry 16:469-77. 2008....
Mary S. Easton Center of Alzheimer's Disease Research at UCLA: advancing the therapeutic imperativeJeffrey L Cummings
Department of Neurology, The Mary S Easton Center for Alzheimer s Disease Research at UCLA, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA
J Alzheimers Dis 19:375-88. 2010..The Center supports excellent senior 3 investigators and serves as an incubator for new scientists, agents, models, technologies and concepts that will significantly influence the future of AD treatment and AD research...
Neuropsychiatric manifestations in mild cognitive impairment: a systematic review of the literatureLiana G Apostolova
Department of Neurology, David Geffen School of Medicine, UCLA, USA
Dement Geriatr Cogn Disord 25:115-26. 2008..MCI is an important risk state for dementia. Neuropsychiatric symptoms may be present in MCI...
Three-dimensional gray matter atrophy mapping in mild cognitive impairment and mild Alzheimer diseaseLiana G Apostolova
Department of Neurology, David Geffen School of Medicine, University of California at Los Angeles, 10911 Westwood Blvd, Second Floor, Los Angeles, CA 90095, USA
Arch Neurol 64:1489-95. 2007..Most patients with MCI harbor the pathologic changes of AD and demonstrate transition to dementia at a rate of 10% to 15% per year. Patients with AD and MCI experience progressive brain atrophy...
Neuropsychiatric symptoms are associated with progression from mild cognitive impairment to Alzheimer's diseaseEdmond Teng
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095 7226, USA
Dement Geriatr Cogn Disord 24:253-9. 2007..Depression and apathy may identify a subset of MCI subjects at higher risk of progression to Alzheimer's disease (AD). However, it remains uncertain whether a broader spectrum of psychopathology is associated with progression to AD...
3D PIB and CSF biomarker associations with hippocampal atrophy in ADNI subjectsLiana G Apostolova
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA, United States
Neurobiol Aging 31:1284-303. 2010..p-tau(181) showed stronger correlation in ApoE4 carriers, while t-tau showed stronger correlation in ApoE4 noncarriers. Of the 3 PIB measures the precuneal SUVR showed strongest associations with hippocampal atrophy...
Neuropathologic correlates of apathy in Alzheimer's diseaseGad A Marshall
Department of Neurology, University of California at Los Angeles, Reed Neurological Research Center, 2 238, 710 Westwood Plaza, CA 90095 1769, USA
Dement Geriatr Cogn Disord 21:144-7. 2006..01, and r = 0.438, p = 0.032). This analysis suggests that chronic apathy in AD correlates with a greater anterior cingulate NFT burden and that chronic behavioral changes are more reflective than acute changes of disease pathology...
Relational integration and executive function in Alzheimer's diseaseJames A Waltz
Departjment of Psychology, University of California, Los Angeles, CA 90095-1563, USA
Neuropsychology 18:296-305. 2004....
Fluctuations in cognitive function in dementia with Lewy bodiesJeffrey L Cummings
David Geffen School of Medicine, UCLA, Los Angeles, CA, USA
Lancet Neurol 3:266. 2004
Heterogeneous age-related breakdown of white matter structural integrity: implications for cortical "disconnection" in aging and Alzheimer's diseaseGeorge Bartzokis
Department of Neurology, Univeristy of California School of Medicine, Los Angeles, CA 90095, USA
Neurobiol Aging 25:843-51. 2004..In AD this process is globally exacerbated, consistent with an extracellular deleterious process such as amyloid beta-peptide toxicity. Non-invasive measures such as R2 may be useful in primary prevention studies of AD...
Alzheimer's diseaseJeffrey L Cummings
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095-1769, USA
N Engl J Med 351:56-67. 2004
Dementia with lewy bodies: molecular pathogenesis and implications for classificationJeffrey L Cummings
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA, USA
J Geriatr Psychiatry Neurol 17:112-9. 2004..Classification systems based on symptom complexes related to the presence of protein misfolding will assist therapeutic decisions...
Subcortical hyperintensities in Alzheimer's disease: associated clinical and metabolic findingsDavid L Sultzer
Departments of Psychiatry and Biobehavioral Sciences, University of California Los Angeles, USA
J Neuropsychiatry Clin Neurosci 14:262-9. 2002..These preliminary findings indicate that subcortical MRI hyperintensities may play a role in the pathophysiology and clinical expression of Alzheimer's disease...
Speech and language in progressive nonfluent aphasia compared with early Alzheimer's diseaseMario F Mendez
Department of Neurology, David Geffen School of Medicine, University of California at Los Angeles, USA
Neurology 61:1108-13. 2003..The differentiation of PA from early Alzheimer's disease (AD) is important, given the presence of early language changes in AD...
Prognostic value of regional cerebral metabolism in patients undergoing dementia evaluation: comparison to a quantifying parameter of subsequent cognitive performance and to prognostic assessment without PETDaniel H S Silverman
Department of Molecular and Medical Pharmacology, University of California, Los Angeles, School of Medicine, Los Angeles, CA, USA
Mol Genet Metab 80:350-5. 2003..4). These data indicate that evaluation of brain metabolism by PET in appropriately selected patients may improve the accuracy of clinical prognostic assessment...
A potential role of the curry spice curcumin in Alzheimer's diseaseJohn M Ringman
University of California, Los Angeles, Department of Neurology, Alzheimer s Disease Research Center, Los Angeles, CA 90095, USA
Curr Alzheimer Res 2:131-6. 2005..We are therefore performing a study of curcumin in patients with AD to gather this information in addition to data on the effect of curcumin on biomarkers of AD pathology...
Alzheimer's disease: from molecular biology to neuropsychiatryJeffrey L Cummings
Department of Neurology and Psychiatry, Reed Neurological Research Center, UCLA School of Medicine, Los Angeles, CA 90095 1769, USA
Semin Clin Neuropsychiatry 8:31-6. 2003..The steps of the amyloid cascade present multiple targets for drug development and identification of effective disease-modifying anti-Alzheimer's disease treatments...
Apolipoprotein E genotype and age-related myelin breakdown in healthy individuals: implications for cognitive decline and dementiaGeorge Bartzokis
Department of Neurology, The David Geffen School of Medicine at UCLA, USA
Arch Gen Psychiatry 63:63-72. 2006..This may result in a progressive "disconnection" of widely distributed neural networks that may underlie the age risk factor for AD...
Effects of testosterone on cognition and mood in male patients with mild Alzheimer disease and healthy elderly menPo H Lu
Department of Neurology, David Geffen School of Medicine, University of California, Los Angeles, USA
Arch Neurol 63:177-85. 2006..There is a compelling need for therapies that prevent, defer the onset, slow the progression, or improve the symptoms of Alzheimer disease (AD)...
Subtle deficits in instrumental activities of daily living in subtypes of mild cognitive impairmentEdmond Teng
Neurobehavior Unit, Veterans Affairs Greater Los Angeles Healthcare System, Los Angeles, CA 90073, USA
Dement Geriatr Cogn Disord 30:189-97. 2010..We explored the relationship between these factors by comparing instrumental activities of daily living (IADLs) among cognitive subtypes of MCI and examining associations between IADL and neuropsychological indices...
Behavioral and neuropsychiatric outcomes in Alzheimer's diseaseJeffrey L Cummings
Alzheimer s Disease Research Center, Department of Neurology, University of California Los Angeles, 710 Westwood Plaza Rm 2 238, Los Angeles, CA 90095, USA
CNS Spectr 10:22-5. 2005....
Apathy and executive function in Alzheimer's diseaseSusan McPherson
Alzheimer s Disease Center, Department of Neurology, UCLA School of Medicine, Los Angeles, California 90095 1769, USA
J Int Neuropsychol Soc 8:373-81. 2002..Neuropsychological measures not dependent on executive functions were unrelated to apathy. Apathy is associated with executive dysfunction and not with other neuropsychological deficits. Apathy is distinct from dysphoria...
Diffusion tensor imaging in preclinical and presymptomatic carriers of familial Alzheimer's disease mutationsJohn M Ringman
UCLA Department of Neurology, Alzheimer s Disease Research Center, 10911 Weyburn Ave, Suite 200, Los Angeles, CA 90095 7226, USA
Brain 130:1767-76. 2007..Decreased FA in of the columns of the fornix is particularly robust in early FAD and may provide a biomarker for early disease in sporadic Alzheimer's disease...
Increased prevalence of significant recurrent headache in preclinical familial Alzheimer's disease mutation carriersJohn M Ringman
Alzheimer s Disease Research Center, UCLA, Los Angeles, CA 90095 7226, USA
Dement Geriatr Cogn Disord 25:380-4. 2008..In our study we compared the prevalence of headaches between nondemented FAD mutation carriers (MCs) and non-mutation-carrying controls (NCs)...
Persistence of neuropsychological testing deficits in mild cognitive impairmentEdmond Teng
Department of Neurology, David Geffen School of Medicine, University of California Los Angeles, Los Angeles, Calif, USA
Dement Geriatr Cogn Disord 28:168-78. 2009....
Delusional thoughts and regional frontal/temporal cortex metabolism in Alzheimer's diseaseDavid L Sultzer
Department of Psychiatry and Biobehavioral Sciences, David Geffen School of Medicine at UCLA, USA
Am J Psychiatry 160:341-9. 2003..In this study the authors examined the relationship between delusional thoughts and regional cortical metabolism in patients with Alzheimer's disease...
Disease-modifying therapies for Alzheimer disease: challenges to early interventionJeffrey L Cummings
Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA, USA
Neurology 69:1622-34. 2007..The neurologic health of the growing aging population demands disease-modifying therapies and the development of methods to identify and test promising candidate agents...
Structural correlates of apathy in Alzheimer's diseaseLiana G Apostolova
Department of Neurology, Laboratory of Neuro Imaging, David Geffen School of Medicine, University of California at Los Angeles, Los Angeles, California, USA
Dement Geriatr Cogn Disord 24:91-7. 2007..Apathy is the most common noncognitive symptom in Alzheimer's disease (AD). The structural correlates of apathy in AD have not yet been described...
Optimizing phase II of drug development for disease-modifying compoundsJeffrey L Cummings
Department of Neurology and Psychiatry, David Geffen School of Medicine at UCLA, Los Angeles, CA, USA
Alzheimers Dement 4:S15-20. 2008..in a successful drug development program; all have some promise for reforming phase II and answering the central question of "how much information is sufficient to proceed to phase III without excessive risk for failure?"..
White matter structural integrity in healthy aging adults and patients with Alzheimer disease: a magnetic resonance imaging studyGeorge Bartzokis
Department of Neurology, Alzheimer s Disease Center, University of California, Los Angeles, 710 Westwood Plaza, Room 2 238, Los Angeles, CA 90095 1769, USA
Arch Neurol 60:393-8. 2003..6 years and then declines. Postmortem evidence indicates that the structural integrity of myelin sheaths deteriorates during normal aging, especially in late myelinating regions such as the frontal lobes...
Brain ferritin iron as a risk factor for age at onset in neurodegenerative diseasesGeorge Bartzokis
Department of Neurology, UCLA, Los Angeles, California, USA
Ann N Y Acad Sci 1012:224-36. 2004..In vivo MRI can measure and track brain ferritin iron levels and provides an opportunity to design therapeutic interventions that target high-risk populations early in the course of illness, possibly even before symptoms appear...
Current therapeutic targets for the treatment of Alzheimer's diseaseJoshua D Grill
Mary S Easton Center for Alzheimer s Disease Research, Deane F Johnson Center for Neurotherapeutics, Department of Neurology, David Geffen School of Medicine at UCLA, Los Angeles, CA, USA
Expert Rev Neurother 10:711-28. 2010..We provide a review of these targets for drug development...
