Antonei B Csoka

Summary

Affiliation: University of Pittsburgh
Country: USA

Publications

  1. ncbi Genome-scale expression profiling of Hutchinson-Gilford progeria syndrome reveals widespread transcriptional misregulation leading to mesodermal/mesenchymal defects and accelerated atherosclerosis
    Antonei B Csoka
    Department of Molecular Biology, Cell Biology and Biochemistry, Brown University, Providence, RI 02912, USA
    Aging Cell 3:235-43. 2004
  2. ncbi Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome
    Maria Eriksson
    Department of Human Genetics, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, New York 10314, USA
    Nature 423:293-8. 2003
  3. ncbi Lamin A/C expression is a marker of mouse and human embryonic stem cell differentiation
    Dan Constantinescu
    Department of Cell Biology and Physiology, University of Pittsburgh School of Medicine, Pittsburgh Development Center of Magee Womens Research Institute, 204 Craft Ave, Pittsburgh, Pennsylvania 15213, USA
    Stem Cells 24:177-85. 2006

Collaborators

  • David G Ginzinger
  • Sangeeta B English
  • Atul Butte
  • Leslie B Gordon
  • Dan Constantinescu
  • Maria Eriksson
  • Gerald P Schatten
  • Paul J Sammak
  • Heather L Gray
  • Tracy Y Moses
  • Francis S Collins
  • Amalia Dutra
  • Christiane M Robbins
  • Laura Scott
  • Sandra Durkin
  • Michael Boehnke
  • Evgenia Pak
  • W Ted Brown
  • Thomas W Glover
  • Peter Berglund
  • Michael W Glynn
  • Michael R Erdos
  • Joel Singer

Detail Information

Publications3

  1. ncbi Genome-scale expression profiling of Hutchinson-Gilford progeria syndrome reveals widespread transcriptional misregulation leading to mesodermal/mesenchymal defects and accelerated atherosclerosis
    Antonei B Csoka
    Department of Molecular Biology, Cell Biology and Biochemistry, Brown University, Providence, RI 02912, USA
    Aging Cell 3:235-43. 2004
    ..The identification of a large number of genes implicated in atherosclerosis is especially valuable, because it provides clues to pathological processes that can now be investigated in HGPS patients or animal models...
  2. ncbi Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome
    Maria Eriksson
    Department of Human Genetics, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, New York 10314, USA
    Nature 423:293-8. 2003
    ..The discovery of the molecular basis of this disease may shed light on the general phenomenon of human ageing...
  3. ncbi Lamin A/C expression is a marker of mouse and human embryonic stem cell differentiation
    Dan Constantinescu
    Department of Cell Biology and Physiology, University of Pittsburgh School of Medicine, Pittsburgh Development Center of Magee Womens Research Institute, 204 Craft Ave, Pittsburgh, Pennsylvania 15213, USA
    Stem Cells 24:177-85. 2006
    ..Our results identify the absence of A-type lamin expression as a novel marker for undifferentiated ES cells and further support a role for nuclear lamins in cell maintenance and differentiation...