JACQUELINE JONES COALSON

Summary

Affiliation: University of Texas Health Science Center
Country: USA

Publications

  1. ncbi request reprint Pathology of new bronchopulmonary dysplasia
    Jacqueline J Coalson
    University of Texas Health Science Center, 7703 Floyd Curl Drive, 78229, San Antonio, TX, USA
    Semin Neonatol 8:73-81. 2003
  2. pmc Coccidioides posadasii infection alters the expression of pulmonary surfactant proteins (SP)-A and SP-D
    Shanjana Awasthi
    Department of Pathology, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA
    Respir Res 5:28. 2004
  3. ncbi request reprint Neonatal chronic lung disease in extremely immature baboons
    J J Coalson
    Department of Pathology, University of Texas Health Science Center San Antonio, San Antonio, USA
    Am J Respir Crit Care Med 160:1333-46. 1999
  4. ncbi request reprint Pathology of bronchopulmonary dysplasia
    Jacqueline J Coalson
    University of Texas Health Science Center at San Antonio, San Antonio, TX 78229, USA
    Semin Perinatol 30:179-84. 2006
  5. ncbi request reprint Impaired urinary cortisol excretion and early cardiopulmonary dysfunction in immature baboons
    BradleyA Yoder
    Department of Pathology, University of Texas Health Science Center, San Antonio, TX 78284, USA
    Pediatr Res 51:426-32. 2002
  6. ncbi request reprint Effects of antenatal colonization with ureaplasma urealyticum on pulmonary disease in the immature baboon
    Bradley A Yoder
    Department of Pathology, University of Texas Health Science Center, San Antonio, TX 78284, USA
    Pediatr Res 54:797-807. 2003
  7. ncbi request reprint Efficacy of antigen 2/proline-rich antigen cDNA-transfected dendritic cells in immunization of mice against Coccidioides posadasii
    Shanjana Awasthi
    Department of Pathology, University of Texas Health Science Center, San Antonio, TX 78229, USA
    J Immunol 175:3900-6. 2005
  8. ncbi request reprint Inhaled NO improves early pulmonary function and modifies lung growth and elastin deposition in a baboon model of neonatal chronic lung disease
    Donald C McCurnin
    Department of Pediatrics, University of Texas Health Science Center, San Antonio, Texas, USA
    Am J Physiol Lung Cell Mol Physiol 288:L450-9. 2005
  9. ncbi request reprint Chlamydia trachomatis pulmonary infection induces greater inflammatory pathology in immunoglobulin A deficient mice
    Ashlesh K Murthy
    Department of Biology, University of Texas at San Antonio, San Antonio, TX 78249, USA
    Cell Immunol 230:56-64. 2004
  10. ncbi request reprint Resistant Candida parapsilosis associated with long term fluconazole prophylaxis in an animal model
    Bradley A Yoder
    Department of Medicine and Physiology, Southwest Foundation for Biomedical Research, University of Texas Health Science Center, San Antonio, Texas, USA
    Pediatr Infect Dis J 23:687-8. 2004

Research Grants

  1. COLLABORATIVE PROGRAM IN BPD
    Jacqueline Coalson; Fiscal Year: 2007

Detail Information

Publications11

  1. ncbi request reprint Pathology of new bronchopulmonary dysplasia
    Jacqueline J Coalson
    University of Texas Health Science Center, 7703 Floyd Curl Drive, 78229, San Antonio, TX, USA
    Semin Neonatol 8:73-81. 2003
    ..The alveolar and capillary hypoplasia of new BPD will require the development of specific therapies, but avoiding volutrauma, oxidant injury and inflammation/infection will improve lung morphology...
  2. pmc Coccidioides posadasii infection alters the expression of pulmonary surfactant proteins (SP)-A and SP-D
    Shanjana Awasthi
    Department of Pathology, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA
    Respir Res 5:28. 2004
    ..Pulmonary surfactant proteins (SP)-A and SP-D have been recognized to play an important role in binding and phagocytosis of various microorganisms, but their roles in Coccidioides infection are not known...
  3. ncbi request reprint Neonatal chronic lung disease in extremely immature baboons
    J J Coalson
    Department of Pathology, University of Texas Health Science Center San Antonio, San Antonio, USA
    Am J Respir Crit Care Med 160:1333-46. 1999
    ..This model demonstrates that impaired alveolization and capillary development occur in immature lungs, even in the absence of marked hyperoxia and high ventilation settings...
  4. ncbi request reprint Pathology of bronchopulmonary dysplasia
    Jacqueline J Coalson
    University of Texas Health Science Center at San Antonio, San Antonio, TX 78229, USA
    Semin Perinatol 30:179-84. 2006
    ....
  5. ncbi request reprint Impaired urinary cortisol excretion and early cardiopulmonary dysfunction in immature baboons
    BradleyA Yoder
    Department of Pathology, University of Texas Health Science Center, San Antonio, TX 78284, USA
    Pediatr Res 51:426-32. 2002
    ..Adrenal hypofunction in the immature baboon is similar to the very preterm human and could serve as a model for future postnatal investigations...
  6. ncbi request reprint Effects of antenatal colonization with ureaplasma urealyticum on pulmonary disease in the immature baboon
    Bradley A Yoder
    Department of Pathology, University of Texas Health Science Center, San Antonio, TX 78284, USA
    Pediatr Res 54:797-807. 2003
    ..Inherent immune system responses may determine outcome of perinatal Ureaplasma colonization...
  7. ncbi request reprint Efficacy of antigen 2/proline-rich antigen cDNA-transfected dendritic cells in immunization of mice against Coccidioides posadasii
    Shanjana Awasthi
    Department of Pathology, University of Texas Health Science Center, San Antonio, TX 78229, USA
    J Immunol 175:3900-6. 2005
    ..The results suggest that the DCs have a potent immunostimulatory activity, and immunization with DCs transfected with Ag2/proline-rich Ag-cDNA induces protective immunity against C. posadasii in C57BL6 mice...
  8. ncbi request reprint Inhaled NO improves early pulmonary function and modifies lung growth and elastin deposition in a baboon model of neonatal chronic lung disease
    Donald C McCurnin
    Department of Pediatrics, University of Texas Health Science Center, San Antonio, Texas, USA
    Am J Physiol Lung Cell Mol Physiol 288:L450-9. 2005
    ..Thus, in the baboon model of CLD, iNO improves early pulmonary function and alters lung growth and extracellular matrix deposition. As such, NO biosynthetic pathway dysfunction may contribute to the pathogenesis of CLD...
  9. ncbi request reprint Chlamydia trachomatis pulmonary infection induces greater inflammatory pathology in immunoglobulin A deficient mice
    Ashlesh K Murthy
    Department of Biology, University of Texas at San Antonio, San Antonio, TX 78249, USA
    Cell Immunol 230:56-64. 2004
    ..However, IgA(-/-) mice displayed exaggerated lung histopathology and altered cytokine production, indicating an important role for IgA in regulating C. trachomatis induced pulmonary inflammation and maintenance of mucosal homeostasis...
  10. ncbi request reprint Resistant Candida parapsilosis associated with long term fluconazole prophylaxis in an animal model
    Bradley A Yoder
    Department of Medicine and Physiology, Southwest Foundation for Biomedical Research, University of Texas Health Science Center, San Antonio, Texas, USA
    Pediatr Infect Dis J 23:687-8. 2004
    ..Where such programs are in place, evaluation of fungal isolates for drug resistance should be considered...
  11. ncbi request reprint Contribution of polymeric immunoglobulin receptor to regulation of intestinal inflammation in dextran sulfate sodium-induced colitis
    Ashlesh K Murthy
    Department of Biology, University of Texas at San Antonio, San Antonio, TX, USA
    J Gastroenterol Hepatol 21:1372-80. 2006
    ..Immunoglobulin A deficiency in humans has been associated with celiac disease and ulcerative colitis. However, the precise role of IgA in the pathogenesis of these disorders is yet to be fully understood...

Research Grants1

  1. COLLABORATIVE PROGRAM IN BPD
    Jacqueline Coalson; Fiscal Year: 2007
    ..It allows, at a national level, a continuing influx of outstanding scientists to address the major deficiencies in our knowledge concerning BPD. ..