Risa Chaisuparat

Summary

Affiliation: University of Maryland
Country: USA

Publications

  1. ncbi request reprint Primary intraosseous odontogenic carcinoma arising in an odontogenic cyst or de novo: a clinicopathologic study of six new cases
    Risa Chaisuparat
    Department of Diagnostic Sciences and Pathology, University of Maryland, Baltimore, MD, USA
    Oral Surg Oral Med Oral Pathol Oral Radiol Endod 101:194-200. 2006
  2. pmc Amplification of the angiogenic signal through the activation of the TSC/mTOR/HIF axis by the KSHV vGPCR in Kaposi's sarcoma
    Bruno C Jham
    Department of Oncology and Diagnostic Sciences, School of Dentistry, University of Maryland, Baltimore, Maryland, United States of America
    PLoS ONE 6:e19103. 2011
  3. doi request reprint Expression of midkine in ameloblastomas and its correlation with clinicopathologic parameters
    Mark A Scheper
    Department of Oncology and Diagnostic Sciences, School of Dentistry, University of Maryland, Maryland, USA
    Oral Surg Oral Med Oral Pathol Oral Radiol 114:497-502. 2012
  4. pmc Dual inhibition of PI3Kalpha and mTOR as an alternative treatment for Kaposi's sarcoma
    Risa Chaisuparat
    Department of Oncology and Diagnostic Sciences and Greenebaum Cancer Center, University of Maryland, Baltimore, Maryland 21201, USA
    Cancer Res 68:8361-8. 2008
  5. pmc Effect of zoledronic acid on oral fibroblasts and epithelial cells: a potential mechanism of bisphosphonate-associated osteonecrosis
    Mark A Scheper
    Department of Oncology and Diagnostic Sciences, Dental School, University of Maryland Medical Center, Baltimore, MD 21201, USA
    Br J Haematol 144:667-76. 2009
  6. pmc Zoledronic acid directly suppresses cell proliferation and induces apoptosis in highly tumorigenic prostate and breast cancers
    Hussain Almubarak
    University of Maryland and the Marlene and Stewart Greenebaum Cancer Center, 650 W Baltimore St, Baltimore, MD 21201, USA
    J Carcinog 10:2. 2011
  7. doi request reprint A clinicopathological study of malignant odontogenic tumours
    Risa Chaisuparat
    Department of Oral Pathology, Faculty of Dentistry, Chulalongkorn University, Bangkok, Thailand
    Histopathology 61:107-12. 2012
  8. pmc Sulindac induces apoptosis and inhibits tumor growth in vivo in head and neck squamous cell carcinoma
    Mark A Scheper
    Department of Diagnostic Sciences and Pathology, University of Maryland, Baltimore, MD 21201, USA
    Neoplasia 9:192-9. 2007
  9. ncbi request reprint Mesenchymal chondrosarcoma of the maxilla: case report and literature review
    Niven Tien
    Department of Oral and Maxillofacial Surgery, New York University College of Dentistry, New York, NY, USA
    J Oral Maxillofac Surg 65:1260-6. 2007
  10. ncbi request reprint The TSC2/mTOR pathway drives endothelial cell transformation induced by the Kaposi's sarcoma-associated herpesvirus G protein-coupled receptor
    Akrit Sodhi
    Oral and Pharyngeal Cancer Branch, National Institute of Dental and Craniofacial Research, National Institutes of Health, Bethesda, Maryland 20892, USA
    Cancer Cell 10:133-43. 2006

Collaborators

Detail Information

Publications11

  1. ncbi request reprint Primary intraosseous odontogenic carcinoma arising in an odontogenic cyst or de novo: a clinicopathologic study of six new cases
    Risa Chaisuparat
    Department of Diagnostic Sciences and Pathology, University of Maryland, Baltimore, MD, USA
    Oral Surg Oral Med Oral Pathol Oral Radiol Endod 101:194-200. 2006
    ..Knowledge of the clinical, radiographic, and histopathologic features of PIOC allows accurate diagnosis and appropriate treatment of this rare malignancy...
  2. pmc Amplification of the angiogenic signal through the activation of the TSC/mTOR/HIF axis by the KSHV vGPCR in Kaposi's sarcoma
    Bruno C Jham
    Department of Oncology and Diagnostic Sciences, School of Dentistry, University of Maryland, Baltimore, Maryland, United States of America
    PLoS ONE 6:e19103. 2011
    ..The driving force of the KS lesion, the KSHV-infected spindle cell, secretes elevated levels of vascular endothelial growth factor (VEGF), essential for KS development. However, the origin of VEGF in this tumor remains unclear...
  3. doi request reprint Expression of midkine in ameloblastomas and its correlation with clinicopathologic parameters
    Mark A Scheper
    Department of Oncology and Diagnostic Sciences, School of Dentistry, University of Maryland, Maryland, USA
    Oral Surg Oral Med Oral Pathol Oral Radiol 114:497-502. 2012
    ..Midkine (MK) is a heparin-binding growth factor that is overexpressed in various human cancers. The aim of this study was to investigate the expression of MK in ameloblastomas and correlate the results with clinicopathologic parameters...
  4. pmc Dual inhibition of PI3Kalpha and mTOR as an alternative treatment for Kaposi's sarcoma
    Risa Chaisuparat
    Department of Oncology and Diagnostic Sciences and Greenebaum Cancer Center, University of Maryland, Baltimore, Maryland 21201, USA
    Cancer Res 68:8361-8. 2008
    ..Moreover, as KS may serve as a model for pathologic angiogenesis, our results further provide the basis for the early assessment of PI-103 as an antiangiogenic chemotherapeutic...
  5. pmc Effect of zoledronic acid on oral fibroblasts and epithelial cells: a potential mechanism of bisphosphonate-associated osteonecrosis
    Mark A Scheper
    Department of Oncology and Diagnostic Sciences, Dental School, University of Maryland Medical Center, Baltimore, MD 21201, USA
    Br J Haematol 144:667-76. 2009
    ..These findings support the potential for soft tissue injury as an initiating/potentiating event for osteonecrosis...
  6. pmc Zoledronic acid directly suppresses cell proliferation and induces apoptosis in highly tumorigenic prostate and breast cancers
    Hussain Almubarak
    University of Maryland and the Marlene and Stewart Greenebaum Cancer Center, 650 W Baltimore St, Baltimore, MD 21201, USA
    J Carcinog 10:2. 2011
    ..The purpose of this study was to show that zoledronic acid (ZA) directly eradicates highly tumorigenic and potentially metastatic cancer cells...
  7. doi request reprint A clinicopathological study of malignant odontogenic tumours
    Risa Chaisuparat
    Department of Oral Pathology, Faculty of Dentistry, Chulalongkorn University, Bangkok, Thailand
    Histopathology 61:107-12. 2012
    ..Malignant odontogenic tumours (MOTs) are rare neoplasms occurring primarily within the jaw. The objective of this study was to determine the incidence, demographics and clinicopathological features of the MOTs from two institutions...
  8. pmc Sulindac induces apoptosis and inhibits tumor growth in vivo in head and neck squamous cell carcinoma
    Mark A Scheper
    Department of Diagnostic Sciences and Pathology, University of Maryland, Baltimore, MD 21201, USA
    Neoplasia 9:192-9. 2007
    ....
  9. ncbi request reprint Mesenchymal chondrosarcoma of the maxilla: case report and literature review
    Niven Tien
    Department of Oral and Maxillofacial Surgery, New York University College of Dentistry, New York, NY, USA
    J Oral Maxillofac Surg 65:1260-6. 2007
  10. ncbi request reprint The TSC2/mTOR pathway drives endothelial cell transformation induced by the Kaposi's sarcoma-associated herpesvirus G protein-coupled receptor
    Akrit Sodhi
    Oral and Pharyngeal Cancer Branch, National Institute of Dental and Craniofacial Research, National Institutes of Health, Bethesda, Maryland 20892, USA
    Cancer Cell 10:133-43. 2006
    ..Collectively, these results implicate mTOR in KS initiation and suggest that the sarcomagenic potential of KSHV may be a direct consequence of the profound sensitivity of endothelial cells to vGPCR dysregulation of the TSC2/mTOR pathway...
  11. ncbi request reprint Simvastatin, an HMG-CoA reductase inhibitor, reduced the expression of matrix metalloproteinase-9 (Gelatinase B) in osteoblastic cells and HT1080 fibrosarcoma cells
    Pasutha D Thunyakitpisal
    Department of Anatomy, Faculty of Dentistry, Chulalongkorn University, Bangkok, Thailand
    J Pharmacol Sci 94:403-9. 2004
    ..5 - 10 microM in primary calvaria, U2-OS, and HT1080 cells, respectively. Collectively, these results suggest that simvastatin is a potent drug for inhibition of MMP-9 expression in osteoblastic cells and HT1080 fibrosarcoma cells...