Zhengwei Cai

Summary

Affiliation: University of Mississippi Medical Center
Country: USA

Publications

  1. ncbi request reprint Intrauterine hypoxia-ischemia alters expression of the NMDA receptor in the young rat brain
    Z Cai
    Department of Pediatrics Newborn Medicine, University of Mississippi Medical Center, Jackson 39216, USA
    Neurochem Res 26:487-95. 2001
  2. ncbi request reprint Disturbance of oligodendrocyte development, hypomyelination and white matter injury in the neonatal rat brain after intracerebral injection of lipopolysaccharide
    Yi Pang
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, 2500 N State Street, Jackson, MS 39216, USA
    Brain Res Dev Brain Res 140:205-14. 2003
  3. ncbi request reprint Evaluation of the mechanism of vascular endothelial growth factor improvement of ischemic flap survival in rats
    Yi Pang
    Department of Pediatrics, Division of Plasic Surgery, University of Mississippoi Medical Center, Jackson, 39216, USA
    Plast Reconstr Surg 112:556-64. 2003
  4. ncbi request reprint Minocycline alleviates hypoxic-ischemic injury to developing oligodendrocytes in the neonatal rat brain
    Z Cai
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, 39216 4505, USA
    Neuroscience 137:425-35. 2006
  5. ncbi request reprint Role of interleukin-6 in lipopolysaccharide-induced brain injury and behavioral dysfunction in neonatal rats
    Y Pang
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, 2500 North State Street, Jackson, MS 39216, USA
    Neuroscience 141:745-55. 2006
  6. ncbi request reprint Neuroprotective effects of N-acetylaspartylglutamate in a neonatal rat model of hypoxia-ischemia
    Zhengwei Cai
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, 2500 North State Street, Jackson, MS 39216 4505, USA
    Eur J Pharmacol 437:139-45. 2002
  7. ncbi request reprint Differential roles of tumor necrosis factor-alpha and interleukin-1 beta in lipopolysaccharide-induced brain injury in the neonatal rat
    Zhengwei Cai
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Brain Res 975:37-47. 2003
  8. ncbi request reprint Brain injury induced by intracerebral injection of interleukin-1beta and tumor necrosis factor-alpha in the neonatal rat
    Zhengwei Cai
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Pediatr Res 56:377-84. 2004
  9. ncbi request reprint Prenatal hypoxia-ischemia alters expression and activity of nitric oxide synthase in the young rat brain and causes learning deficits
    Z Cai
    Department of Pediatrics, University of Mississippi Medical Center, Jackson 39216 4505, USA
    Brain Res Bull 49:359-65. 1999
  10. ncbi request reprint Intrauterine hypoxia-ischemia alters nitric oxide synthase expression and activity in fetal and neonatal rat brains
    Z Cai
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, 2500 N State Street, Jackson, MS 39216 4505, USA
    Brain Res Dev Brain Res 109:265-9. 1998

Detail Information

Publications47

  1. ncbi request reprint Intrauterine hypoxia-ischemia alters expression of the NMDA receptor in the young rat brain
    Z Cai
    Department of Pediatrics Newborn Medicine, University of Mississippi Medical Center, Jackson 39216, USA
    Neurochem Res 26:487-95. 2001
    ....
  2. ncbi request reprint Disturbance of oligodendrocyte development, hypomyelination and white matter injury in the neonatal rat brain after intracerebral injection of lipopolysaccharide
    Yi Pang
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, 2500 N State Street, Jackson, MS 39216, USA
    Brain Res Dev Brain Res 140:205-14. 2003
    ..These results indicate that developing OLs may be the target cells for LPS-induced brain injury and inflammatory cytokines are possible mediators of LPS-induced brain injury...
  3. ncbi request reprint Evaluation of the mechanism of vascular endothelial growth factor improvement of ischemic flap survival in rats
    Yi Pang
    Department of Pediatrics, Division of Plasic Surgery, University of Mississippoi Medical Center, Jackson, 39216, USA
    Plast Reconstr Surg 112:556-64. 2003
    ..It was concluded that administration of exogenous VEGF could protect flaps from ischemia-reperfusion injury through the regulation of proinflammatory cytokines and the inhibition of cytotoxic nitric oxide production...
  4. ncbi request reprint Minocycline alleviates hypoxic-ischemic injury to developing oligodendrocytes in the neonatal rat brain
    Z Cai
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, 39216 4505, USA
    Neuroscience 137:425-35. 2006
    ..The overall results suggest that reduction in microglial activation may protect developing oligodendrocytes in the neonatal brain from hypoxia-ischemia injury...
  5. ncbi request reprint Role of interleukin-6 in lipopolysaccharide-induced brain injury and behavioral dysfunction in neonatal rats
    Y Pang
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, 2500 North State Street, Jackson, MS 39216, USA
    Neuroscience 141:745-55. 2006
    ..The present study indicates that the contribution to brain injury by interleukin-6 depends on its interaction with other lipopolysaccharide-induced agents and not on interleukin-6 alone...
  6. ncbi request reprint Neuroprotective effects of N-acetylaspartylglutamate in a neonatal rat model of hypoxia-ischemia
    Zhengwei Cai
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, 2500 North State Street, Jackson, MS 39216 4505, USA
    Eur J Pharmacol 437:139-45. 2002
    ..The overall results suggest that the neuroprotective effect of NAAG is largely associated with activation of mGlu2/3 receptor...
  7. ncbi request reprint Differential roles of tumor necrosis factor-alpha and interleukin-1 beta in lipopolysaccharide-induced brain injury in the neonatal rat
    Zhengwei Cai
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Brain Res 975:37-47. 2003
    ..These data suggest that IL-1beta may play an important role in mediating LPS-induced brain injury and TNFalpha may have complicated, probably dual, effects in LPS-induced brain injury...
  8. ncbi request reprint Brain injury induced by intracerebral injection of interleukin-1beta and tumor necrosis factor-alpha in the neonatal rat
    Zhengwei Cai
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Pediatr Res 56:377-84. 2004
    ..The overall results indicate that IL-1beta may directly cause injuries to developing OLs and impair myelination in the neonatal rat brain and TNFalpha may have different roles in mediating brain injury...
  9. ncbi request reprint Prenatal hypoxia-ischemia alters expression and activity of nitric oxide synthase in the young rat brain and causes learning deficits
    Z Cai
    Department of Pediatrics, University of Mississippi Medical Center, Jackson 39216 4505, USA
    Brain Res Bull 49:359-65. 1999
    ..The learning deficit in offspring is possibly associated with the reduction in expression of NOS mRNA and NOS activity in the hippocampus of these animals...
  10. ncbi request reprint Intrauterine hypoxia-ischemia alters nitric oxide synthase expression and activity in fetal and neonatal rat brains
    Z Cai
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, 2500 N State Street, Jackson, MS 39216 4505, USA
    Brain Res Dev Brain Res 109:265-9. 1998
    ....
  11. pmc Interleukin-1beta-induced brain injury in the neonatal rat can be ameliorated by alpha-phenyl-n-tert-butyl-nitrone
    Lir Wan Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216 4504, USA
    Exp Neurol 220:143-53. 2009
    ....
  12. ncbi request reprint Suppression of glial activation is involved in the protection of IL-10 on maternal E. coli induced neonatal white matter injury
    Yi Pang
    Department of Pediatrics, University of Mississippi Med Ctr, Jackson, MS 39216 4505, USA
    Brain Res Dev Brain Res 157:141-9. 2005
    ..These results indicate that the protective effect of IL-10 against brain WMD is linked with suppression of microglial activation/macrophage infiltration, as shown by significantly reduced ED-1+ cells in the white matter...
  13. pmc IGF-1 can either protect against or increase LPS-induced damage in the developing rat brain
    Yi Pang
    Department of Pediatrics, University of Mississippi Medical Center, Jackson, Mississippi 39216, USA
    Pediatr Res 67:579-84. 2010
    ..However, these adverse effects were not found in rats treated with IGF-1 alone. This study provides the alarming evidence that in an acute inflammatory condition, IGF-1 may have severe, harmful effects on the developing brain...
  14. ncbi request reprint Minocycline attenuates hypoxia-ischemia-induced neurological dysfunction and brain injury in the juvenile rat
    Lir Wan Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, 39216, USA
    Eur J Neurosci 24:341-50. 2006
    ..The present results show that minocycline has long-lasting protective effects in the neonatal rat brain in terms of both hypoxia-ischemia-induced brain injury and the associated neurological dysfunction...
  15. ncbi request reprint Hypoxia-ischemia induced neurological dysfunction and brain injury in the neonatal rat
    Lir Wan Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Behav Brain Res 165:80-90. 2005
    ..The present data also indicate that the degree of brain injury and the deficits of neurobehavioral performance in the rat are dependent on the hypoxic-ischemic condition, i.e., the exposure time to hypoxia...
  16. pmc alpha-Phenyl-n-tert-butyl-nitrone attenuates lipopolysaccharide-induced brain injury and improves neurological reflexes and early sensorimotor behavioral performance in juvenile rats
    Lir Wan Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216 4505, USA
    J Neurosci Res 86:3536-47. 2008
    ....
  17. ncbi request reprint alpha-Phenyl-n-tert-butyl-nitrone reduces lipopolysaccharide-induced white matter injury in the neonatal rat brain
    Lir Wan Fan
    Department of Pediatrics, University of Mississippi Medical Center, Jackson, Mississippi 39216, USA
    Dev Neurobiol 68:365-78. 2008
    ..The overall results suggest that antioxidant PBN, more than a straightforward free radical scavenger, may also involve anti-inflammatory and anti-apoptotic properties in protection of the neonatal rat brain from LPS-induced injury...
  18. pmc Neonatal exposure to lipopolysaccharide enhances vulnerability of nigrostriatal dopaminergic neurons to rotenone neurotoxicity in later life
    Lir Wan Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Neurobiol Dis 44:304-16. 2011
    ..Our model may be useful for studying mechanisms involved in the pathogenesis of nonfamilial Parkinson's disease and the development of potential therapeutic treatments...
  19. ncbi request reprint Minocycline reduces lipopolysaccharide-induced neurological dysfunction and brain injury in the neonatal rat
    Lir Wan Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, Mississippi 39216 4504, USA
    J Neurosci Res 82:71-82. 2005
    ..These results suggest that inhibition of microglial activation by minocycline may have long-term protective effects in the neonatal brain on infection-induced brain injury and associated neurologic dysfunction in the rat...
  20. pmc Neonatal systemic exposure to lipopolysaccharide enhances susceptibility of nigrostriatal dopaminergic neurons to rotenone neurotoxicity in later life
    Zhengwei Cai
    Division of Newborn Medicine, Department of Pediatrics, University of Mississippi Medical Center, Jackson, MS 39216 4504, USA
    Dev Neurosci 35:155-71. 2013
    ....
  21. pmc Dopaminergic neuronal injury in the adult rat brain following neonatal exposure to lipopolysaccharide and the silent neurotoxicity
    Lir Wan Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216 4504, USA
    Brain Behav Immun 25:286-97. 2011
    ..Our data further suggest that the compromised dendritic mitochondrial function might contribute, at least partially, to the silent neurotoxicity...
  22. doi request reprint Alpha-phenyl-n-tert-butyl-nitrone ameliorates hippocampal injury and improves learning and memory in juvenile rats following neonatal exposure to lipopolysaccharide
    Lir Wan Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Eur J Neurosci 27:1475-84. 2008
    ..Treatment with PBN provided long-lasting protection against the LPS-induced axonal injury and neuronal loss, and improved the associated neurological dysfunctions in juvenile rats...
  23. ncbi request reprint IGF-1 protects oligodendrocyte progenitor cells and improves neurological functions following cerebral hypoxia-ischemia in the neonatal rat
    Shuying Lin
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Brain Res 1063:15-26. 2005
    ..The neuroprotection of IGF-1 was associated with its antiapoptotic and mitogenic effects...
  24. ncbi request reprint IGF-1 protects oligodendrocyte progenitors against TNFalpha-induced damage by activation of PI3K/Akt and interruption of the mitochondrial apoptotic pathway
    Yi Pang
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, Mississippi 39216, USA
    Glia 55:1099-107. 2007
    ..These observations indicate that the protection of OPCs by IGF-1 is mediated, at least partially, by interruption of the mitochondrial apoptotic pathway via activation of PI3K/Akt...
  25. pmc Intranasal administration of IGF-1 attenuates hypoxic-ischemic brain injury in neonatal rats
    Shuying Lin
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Exp Neurol 217:361-70. 2009
    ..rhIGF-1 enhanced proliferation of neuronal and oligodendroglial progenitors after cerebral HI as well. These data suggest that iN administration of IGF-1 has the potential to be used for clinical treatment...
  26. ncbi request reprint Neuroprotection of alpha-phenyl-n-tert-butyl-nitrone on the neonatal white matter is associated with anti-inflammation
    Shuying Lin
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, United States
    Neurosci Lett 405:52-6. 2006
    ..The overall results indicate that besides free radical scavenging, anti-inflammation might partly contribute to the neuroprotection afforded by PBN on neonatal white matter following cerebral HI...
  27. pmc Celecoxib reduces brain dopaminergic neuronaldysfunction, and improves sensorimotor behavioral performance in neonatal rats exposed to systemic lipopolysaccharide
    Asuka Kaizaki
    Division of Newborn Medicine, Department of Pediatrics, University of Mississippi Medical Center, Jackson, MS 39216, USA
    J Neuroinflammation 10:45. 2013
    ....
  28. pmc Dexamethasone and betamethasone protect against lipopolysaccharide-induced brain damage in neonatal rats
    Yi Pang
    Department of Pediatrics, University of Mississippi Medical Center, Jackson, Mississippi, USA
    Pediatr Res 71:552-8. 2012
    ..The aim of this study was to test whether dexamethasone (Dex) and betamethasone (Beta), two of the most commonly used corticosteroids, protect against lipopolysaccharide (LPS)-induced white matter damage and neurobehavioral dysfunction...
  29. ncbi request reprint alpha-Phenyl-n-tert-butyl-nitrone attenuates hypoxic-ischemic white matter injury in the neonatal rat brain
    Shuying Lin
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Brain Res 1007:132-41. 2004
    ..FR scavenging appears to be the primary mechanism underlying its neuroprotective effect...
  30. ncbi request reprint Effect of tumor necrosis factor-alpha on developing optic nerve oligodendrocytes in culture
    Yi Pang
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, Mississippi 39216, USA
    J Neurosci Res 80:226-34. 2005
    ..Consistent with reports that progenitor cells are preferentially injured in PVL, our results may further support the role of TNF-alpha as a potential mediator of PVL...
  31. pmc Control of blood pressure, appetite, and glucose by leptin in mice lacking leptin receptors in proopiomelanocortin neurons
    Jussara M do Carmo
    Department of Physiology and Biophysics, University of Mississippi Medical Center, 2500 North State St, Jackson, MS 39216, USA
    Hypertension 57:918-26. 2011
    ....
  32. ncbi request reprint Small intestinal submucosa in abdominal wall repair after TRAM flap harvesting in a rat model
    Feng Zhang
    Department of Pediatrics, Division of Plastic Surgery, University of Mississippi Medical Center, Jackson, 39216, USA
    Plast Reconstr Surg 112:565-70. 2003
    ..The use of small intestinal submucosa placement in combination with fascial repair can significantly improve the strength of the repaired abdominal wall after transverse rectus abdominis myocutaneous flap harvesting...
  33. pmc Interleukin-1beta-induced brain injury and neurobehavioral dysfunctions in juvenile rats can be attenuated by alpha-phenyl-n-tert-butyl-nitrone
    L W Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, 2500 North State Street, Jackson, MS 39216, USA
    Neuroscience 168:240-52. 2010
    ....
  34. ncbi request reprint Minocycline attenuates lipopolysaccharide-induced white matter injury in the neonatal rat brain
    L W Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216 4505, USA
    Neuroscience 133:159-68. 2005
    ....
  35. ncbi request reprint Acute, chronic and differential effects of several anesthetic barbiturates on glutamate receptor activation in neuronal culture
    Z Cai
    Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson 39216 4505
    Brain Res 611:181-6. 1993
    ..A similar hyperresponsiveness was not seen after the chronic treatment with phenobarbital.(ABSTRACT TRUNCATED AT 250 WORDS)..
  36. doi request reprint Lipopolysaccharide-activated microglia induce death of oligodendrocyte progenitor cells and impede their development
    Y Pang
    Department of Pediatrics, University of Mississippi Medical Center, 2500 North State Street, Jackson, MS 39216, USA
    Neuroscience 166:464-75. 2010
    ..Our finding that LPS-activated microglia not only induce two waves of cell death but also greatly impair OL development may shed some light on the mechanisms underlying selective white matter damage and hypomyelination in PVL...
  37. pmc Whole body hypothermia broadens the therapeutic window of intranasally administered IGF-1 in a neonatal rat model of cerebral hypoxia-ischemia
    Shuying Lin
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Brain Res 1385:246-56. 2011
    ....
  38. pmc Perinatal antidepressant exposure alters cortical network function in rodents
    Kimberly L Simpson
    Department of Neurobiology and Anatomical Sciences, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Proc Natl Acad Sci U S A 108:18465-70. 2011
    ..These findings indicate that 5-HT homeostasis is required for proper brain maturation and that fetal/infant exposure to SSRIs should be examined in humans, particularly those with developmental dysfunction, such as autism...
  39. ncbi request reprint Chronic ischemia preferentially causes white matter injury in the neonatal rat brain
    Z Cai
    Department of Pediatrics Newborn Medicine, University of Mississippi Medical Center, 2500 North State Street, Jackson, MS 39216 4505
    Brain Res 898:126-35. 2001
    ..The overall results suggest that pre-oligodendrocytes and immature oligodendrocytes might be major targets for chronic ischemic insults and activated microglia/macrophages are possibly involved in the process of white matter injury...
  40. ncbi request reprint Intrauterine hypoxia-ischemia reduces phosphoinositide hydrolysis stimulated by metabotropic glutamate receptor agonists in cultured rat cerebellar granule cells
    P G Rhodes
    Department of Pediatrics, University of Mississippi Medical Center, Jackson 39216 4505, USA
    Brain Res Dev Brain Res 93:129-35. 1996
    ....
  41. ncbi request reprint Prenatal ethanol exposure enhances glutamate release stimulated by quisqualate in rat cerebellar granule cell cultures
    P G Rhodes
    Department of Pediatrics, University of Mississippi Medical Center, Jackson 39216 4505, USA
    Mol Chem Neuropathol 33:99-111. 1998
    ....
  42. ncbi request reprint Maternal infection-induced white matter injury is reduced by treatment with interleukin-10
    Sheryl Rodts-Palenik
    Department of Obstetrics and Gynecology, University of Mississippi Medical Center, Jackson, MS, USA
    Am J Obstet Gynecol 191:1387-92. 2004
    ..The purpose of this study was to test the hypothesis that interleukin-10 can prevent white matter injury in neonatal rats that are born to infected dams...
  43. pmc Alpha-Phenyl-n-tert-butyl-nitrone attenuates lipopolysaccharide-induced neuronal injury in the neonatal rat brain
    L W Fan
    Department of Pediatrics, Division of Newborn Medicine, University of Mississippi Medical Center, 2500 North State Street, Jackson, MS 39216 4505, USA
    Neuroscience 151:737-44. 2008
    ..The protection of PBN may indicate the potential usefulness of antioxidants for treatment of these neuronal dysfunctions...
  44. ncbi request reprint Developmental expression of endothelial nitric oxide synthase (eNOS) in the rat liver
    Michael J Nowicki
    Division of Pediatric Gastroenterology, University of Mississippi Medical Center, Jackson, MS 39216, USA
    Pediatr Res 54:732-8. 2003
    ..eNOS expression and activity are developmentally regulated, increasing after birth coincident with an initial relative hypoxia and an increase in shear forces upon closure of the ductus venosus...
  45. ncbi request reprint A quantitative histochemistry technique for measuring regional distribution of acetylcholinesterase in the brain using digital scanning densitometry
    T Ma
    Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson, Mississippi 39216, USA
    Anal Biochem 296:18-28. 2001
    ..Because the digital scanning densitometers provide high quality and sensitive imaging, wide dynamic ranges, and convenient image analysis software, they are very useful tools in quantitative histochemistry...
  46. ncbi request reprint Hepatotoxicity and lethality of halomethanes in Mongolian gerbils pretreated with chlordecone, phenobarbital or mirex
    Z Cai
    Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson 39216 4505
    Arch Toxicol 65:204-12. 1991
    ..Evidence from histomorphometric studies was consistent with serum enzyme and 3H-T incorporation data.(ABSTRACT TRUNCATED AT 250 WORDS)..
  47. ncbi request reprint Analysis of genes differentially expressed in astrocytes stimulated with lipopolysaccharide using cDNA arrays
    Y Pang
    Division of Newborn Medicine, Department of Pediatrics, University of Mississippi Medical Center, 2500 N State Street, Jackson, MS 39216, USA
    Brain Res 914:15-22. 2001
    ..These results indicate that both up-regulation of inflammatory cytokine expression and down-regulation of growth factor expression are probably involved in the response of astrocytes upon exposure to LPS...

Research Grants9

  1. Perinatal hypoxia-ischemia, glutamate receptors, and Nitric Oxide Synthase
    Zhengwei Cai; Fiscal Year: 2007
    ..Direct effects of inflammatory cytokines and IGF-1 on survival and differentiation of OLs will be examined in cultures of OLs at various maturation stages. ..
  2. Perinatal hypoxia-ischemia, glutamate receptors, and NOS
    Zhengwei Cai; Fiscal Year: 2003
    ..Direct effects of inflammatory cytokines and IGF-1 on survival and differentiation of OLs will be examined in cultures of OLs at various maturation stages. ..
  3. Perinatal hypoxia-ischemia, glutamate receptors, and NOS
    Zhengwei Cai; Fiscal Year: 2004
    ..Direct effects of inflammatory cytokines and IGF-1 on survival and differentiation of OLs will be examined in cultures of OLs at various maturation stages. ..
  4. Perinatal hypoxia-ischemia, glutamate receptors, and NOS
    Zhengwei Cai; Fiscal Year: 2005
    ..Direct effects of inflammatory cytokines and IGF-1 on survival and differentiation of OLs will be examined in cultures of OLs at various maturation stages. ..
  5. Perinatal infection, cytokines and brain injury
    Zhengwei Cai; Fiscal Year: 2005
    ....
  6. Perinatal hypoxia-ischemia, glutamate receptors, and NOS
    Zhengwei Cai; Fiscal Year: 2006
    ..Direct effects of inflammatory cytokines and IGF-1 on survival and differentiation of OLs will be examined in cultures of OLs at various maturation stages. ..
  7. Perinatal infection, cytokines and brain injury
    Zhengwei Cai; Fiscal Year: 2006
    ....
  8. Perinatal infection, cytokines and brain injury
    Zhengwei Cai; Fiscal Year: 2007
    ....