Edwin G Bovill

Summary

Affiliation: University of Vermont
Country: USA

Publications

  1. doi request reprint Venous valvular stasis-associated hypoxia and thrombosis: what is the link?
    Edwin G Bovill
    Department of Pathology, University of Vermont College of Medicine, Burlington, 05405, USA
    Annu Rev Physiol 73:527-45. 2011
  2. ncbi request reprint Effect of tamoxifen on venous thrombosis risk factors in women without cancer: the Breast Cancer Prevention Trial
    Mary Cushman
    Department of Medicine, University of Vermont, Burlington, VT, USA
    Br J Haematol 120:109-16. 2003
  3. pmc Imaging aspects of cardiovascular disease at the cell and molecular level
    Douglas J Taatjes
    Department of Pathology, College of Medicine, University of Vermont, 89 Beaumont Avenue, Burlington, VT 05405, USA
    Histochem Cell Biol 130:235-45. 2008
  4. ncbi request reprint Evaluating chronic venous disease with a new venous severity scoring system
    Michael A Ricci
    Department of Surgery, Vermont College of Medicine, Burlington, 05405 0068, USA
    J Vasc Surg 38:909-15. 2003
  5. ncbi request reprint SELDI-TOF plasma profiles distinguish individuals in a protein C-deficient family with thrombotic episodes occurring before age 40
    Annika M Svensson
    Department of Pathology, University of Vermont College of Medicine, Burlington, Vermont, USA
    Thromb Haemost 96:725-30. 2006
  6. ncbi request reprint Normal retinal vasculature despite familial protein C deficiency
    Carla Y Vossen
    Department of Pathology, University of Vermont, 199 Main Street, Burlington, VT 05401, USA
    Am J Ophthalmol 139:944-5. 2005
  7. ncbi request reprint No effect of the prothrombin G20210A mutation on protein C activation in a large kindred with type I protein C deficiency
    Carla Y Vossen
    Department of Pathology, University of Vermont, Burlington, Vermont 05405, USA
    Blood Coagul Fibrinolysis 15:573-6. 2004
  8. ncbi request reprint A review of the technical, diagnostic, and epidemiologic considerations for protein S assays
    Andrew J Goodwin
    Department of Pathology, University of Vermont, Burlington 05405, USA
    Arch Pathol Lab Med 126:1349-66. 2002
  9. ncbi request reprint Sensitivity of a questionnaire for data collection on venous thrombosis
    Carla Y Vossen
    Department of Pathology, University of Vermont, Given Building E208, 89 Beaumont Avenue, Burlington, VT 05405, USA
    Thromb Res 114:259-63. 2004
  10. ncbi request reprint The protein C system in placental massive perivillous fibrin deposition
    Annika M Svensson
    Department of Pathology, University of Vermont, Burlington, Vermont 05401, USA
    Blood Coagul Fibrinolysis 15:491-5. 2004

Collaborators

Detail Information

Publications13

  1. doi request reprint Venous valvular stasis-associated hypoxia and thrombosis: what is the link?
    Edwin G Bovill
    Department of Pathology, University of Vermont College of Medicine, Burlington, 05405, USA
    Annu Rev Physiol 73:527-45. 2011
    ..Thus, the effects of blood flow stasis, hypercoagulability of blood, and the characteristics of the vessel wall within the venous valvular sinus are assessed in turn...
  2. ncbi request reprint Effect of tamoxifen on venous thrombosis risk factors in women without cancer: the Breast Cancer Prevention Trial
    Mary Cushman
    Department of Medicine, University of Vermont, Burlington, VT, USA
    Br J Haematol 120:109-16. 2003
    ..Reductions of antithrombin and protein S, but not protein C or APC resistance, might relate to the increased risk of venous thrombosis associated with tamoxifen treatment...
  3. pmc Imaging aspects of cardiovascular disease at the cell and molecular level
    Douglas J Taatjes
    Department of Pathology, College of Medicine, University of Vermont, 89 Beaumont Avenue, Burlington, VT 05405, USA
    Histochem Cell Biol 130:235-45. 2008
    ....
  4. ncbi request reprint Evaluating chronic venous disease with a new venous severity scoring system
    Michael A Ricci
    Department of Surgery, Vermont College of Medicine, Burlington, 05405 0068, USA
    J Vasc Surg 38:909-15. 2003
    ..However, validation of the VCSS against an objective test is lacking. The purpose of this study was to test the VCSS against abnormalities found on venous ultrasound (US) scans...
  5. ncbi request reprint SELDI-TOF plasma profiles distinguish individuals in a protein C-deficient family with thrombotic episodes occurring before age 40
    Annika M Svensson
    Department of Pathology, University of Vermont College of Medicine, Burlington, Vermont, USA
    Thromb Haemost 96:725-30. 2006
    ..This is the first study to apply SELDI-TOF technology in conjunction with a bioinformatics tool to analyze low-molecular-weight proteomic patterns in patients with venous thrombosis...
  6. ncbi request reprint Normal retinal vasculature despite familial protein C deficiency
    Carla Y Vossen
    Department of Pathology, University of Vermont, 199 Main Street, Burlington, VT 05401, USA
    Am J Ophthalmol 139:944-5. 2005
    ..It is unclear whether protein C deficiency is associated with retinal venous occlusive disease...
  7. ncbi request reprint No effect of the prothrombin G20210A mutation on protein C activation in a large kindred with type I protein C deficiency
    Carla Y Vossen
    Department of Pathology, University of Vermont, Burlington, Vermont 05405, USA
    Blood Coagul Fibrinolysis 15:573-6. 2004
    ..Thus, carriers of the prothrombin G20210A mutation do not have increased protein C activation despite the increased thrombin generation resulting from the higher prothrombin concentrations associated with the G20210A mutation...
  8. ncbi request reprint A review of the technical, diagnostic, and epidemiologic considerations for protein S assays
    Andrew J Goodwin
    Department of Pathology, University of Vermont, Burlington 05405, USA
    Arch Pathol Lab Med 126:1349-66. 2002
    ..As described elsewhere, results of discussions at the College of American Pathologists Conference XXXVI on Diagnostic Issues in Thrombophilia were used to revise the manuscript into its final form...
  9. ncbi request reprint Sensitivity of a questionnaire for data collection on venous thrombosis
    Carla Y Vossen
    Department of Pathology, University of Vermont, Given Building E208, 89 Beaumont Avenue, Burlington, VT 05405, USA
    Thromb Res 114:259-63. 2004
  10. ncbi request reprint The protein C system in placental massive perivillous fibrin deposition
    Annika M Svensson
    Department of Pathology, University of Vermont, Burlington, Vermont 05401, USA
    Blood Coagul Fibrinolysis 15:491-5. 2004
    ..This could imply an underlying defect of trophoblastic protein C activation. Alternatively, it may represent a degenerative change secondary to impedence of oxygen and nutrient supply to the trophoblastic epithelium...
  11. pmc Lipoprotein-associated phospholipase A2 and risk of venous thrombosis in older adults
    Nels Olson
    Department of Pathology, University of Vermont College of Medicine, Burlington, Vermont, USA
    Am J Hematol 83:524-7. 2008
    ..Results were robust to several definitions of low or high Lp-PLA2. While the association of Lp-PLA(2) levels with arterial disease events implies a role for this enzyme in atherogenesis, our findings suggest that it is not prothrombotic...
  12. pmc Human monoclonal antiphospholipid antibodies disrupt the annexin A5 anticoagulant crystal shield on phospholipid bilayers: evidence from atomic force microscopy and functional assay
    Jacob H Rand
    Department of Pathology, Montefiore Medical Center, Albert Einstein College of Medicine, Moses Division Campus, Core Laboratory Office, North 8, 110 East 210th Street, Bronx, NY 10467, USA
    Am J Pathol 163:1193-200. 2003
    ..The aPL antibody-mediated disruption of the annexin A5 anticoagulant shield may be an important prothrombotic mechanism in the aPL syndrome...
  13. pmc Valves of the deep venous system: an overlooked risk factor
    Erin G Brooks
    Department of Pathology, University of Vermont, Burlington, VT 05482, USA
    Blood 114:1276-9. 2009
    ..001; for VWF, P = .01). These data support our hypothesis and suggest that variation in valvular sinus thromboresistance may be an important factor in venous thrombogenesis...