Suzanne S Bohlson

Summary

Affiliation: University of California
Country: USA

Publications

  1. ncbi CD93 is rapidly shed from the surface of human myeloid cells and the soluble form is detected in human plasma
    Suzanne S Bohlson
    Department of Molecular Biology and Biochemistry, Center for Immunology, University of California Irvine, 2419 McGaugh Hall, Irvine, CA 92697, USA
    J Immunol 175:1239-47. 2005
  2. pmc Modulated interaction of the ERM protein, moesin, with CD93
    MingYu Zhang
    Department of Molecular Biology and Biochemistry, Center for Immunology, University of California, Irvine, CA 92697, USA
    Immunology 115:63-73. 2005
  3. ncbi Complement proteins C1q and MBL are pattern recognition molecules that signal immediate and long-term protective immune functions
    Suzanne S Bohlson
    Department of Molecular Biology and Biochemistry, Center for Immunology, University of California, Irvine, CA 92697, USA
    Mol Immunol 44:33-43. 2007
  4. ncbi Generation of inhibitory NFkappaB complexes and phosphorylated cAMP response element-binding protein correlates with the anti-inflammatory activity of complement protein C1q in human monocytes
    Deborah A Fraser
    Department of Molecular Biology and Biochemistry, Center for Immunology, University of California, Irvine, California 92697, USA
    J Biol Chem 282:7360-7. 2007
  5. ncbi C1q and MBL, components of the innate immune system, influence monocyte cytokine expression
    Deborah A Fraser
    Department of Molecular Biology, University of California, Irvine, 92697, USA
    J Leukoc Biol 80:107-16. 2006
  6. ncbi CD93 interacts with the PDZ domain-containing adaptor protein GIPC: implications in the modulation of phagocytosis
    Suzanne S Bohlson
    Department of Molecular Biology and Biochemistry, University of California, Irvine, CA 92697, USA
    J Leukoc Biol 77:80-9. 2005
  7. pmc Murine low-density lipoprotein receptor-related protein 1 (LRP) is required for phagocytosis of targets bearing LRP ligands but is not required for C1q-triggered enhancement of phagocytosis
    Anna P Lillis
    Center for Vascular and Inflammatory Diseases and Department of Surgery, University of Maryland School of Medicine, Baltimore, MD 21201, USA
    J Immunol 181:364-73. 2008

Collaborators

  • ANDREA TENNER
  • Nenoo Rawal
  • Deborah A Fraser
  • Anna P Lillis
  • MingYu Zhang
  • Irina Mikhailenko
  • Mallary C Greenlee
  • Dudley K Strickland
  • Salvatore V Pizzo
  • Encarnacion Lozano
  • Meenakshi Arora
  • Gail Palmarini
  • Rosemary Rochford
  • Sol Ruiz
  • Nijole Jasinskiene
  • Marisela Dy

Detail Information

Publications7

  1. ncbi CD93 is rapidly shed from the surface of human myeloid cells and the soluble form is detected in human plasma
    Suzanne S Bohlson
    Department of Molecular Biology and Biochemistry, Center for Immunology, University of California Irvine, 2419 McGaugh Hall, Irvine, CA 92697, USA
    J Immunol 175:1239-47. 2005
    ....
  2. pmc Modulated interaction of the ERM protein, moesin, with CD93
    MingYu Zhang
    Department of Molecular Biology and Biochemistry, Center for Immunology, University of California, Irvine, CA 92697, USA
    Immunology 115:63-73. 2005
    ....
  3. ncbi Complement proteins C1q and MBL are pattern recognition molecules that signal immediate and long-term protective immune functions
    Suzanne S Bohlson
    Department of Molecular Biology and Biochemistry, Center for Immunology, University of California, Irvine, CA 92697, USA
    Mol Immunol 44:33-43. 2007
    ....
  4. ncbi Generation of inhibitory NFkappaB complexes and phosphorylated cAMP response element-binding protein correlates with the anti-inflammatory activity of complement protein C1q in human monocytes
    Deborah A Fraser
    Department of Molecular Biology and Biochemistry, Center for Immunology, University of California, Irvine, California 92697, USA
    J Biol Chem 282:7360-7. 2007
    ..Because C1q and other defense collagens have been shown to enhance clearance of apoptotic cells, this regulatory pathway may be beneficial in avoiding autoimmunity and/or resolving inflammation...
  5. ncbi C1q and MBL, components of the innate immune system, influence monocyte cytokine expression
    Deborah A Fraser
    Department of Molecular Biology, University of California, Irvine, 92697, USA
    J Leukoc Biol 80:107-16. 2006
    ..These data support the hypothesis that defense collagen-mediated suppression of a proinflammatory response may be an important step in the avoidance of autoimmunity during the clearance of apoptotic cells...
  6. ncbi CD93 interacts with the PDZ domain-containing adaptor protein GIPC: implications in the modulation of phagocytosis
    Suzanne S Bohlson
    Department of Molecular Biology and Biochemistry, University of California, Irvine, CA 92697, USA
    J Leukoc Biol 77:80-9. 2005
    ..These protein interactions may participate as molecular switches in modulating cellular phagocytic activity...
  7. pmc Murine low-density lipoprotein receptor-related protein 1 (LRP) is required for phagocytosis of targets bearing LRP ligands but is not required for C1q-triggered enhancement of phagocytosis
    Anna P Lillis
    Center for Vascular and Inflammatory Diseases and Department of Surgery, University of Maryland School of Medicine, Baltimore, MD 21201, USA
    J Immunol 181:364-73. 2008
    ....