Richard J Bodnar

Summary

Affiliation: University of Pittsburgh
Country: USA

Publications

  1. ncbi IP-10 blocks vascular endothelial growth factor-induced endothelial cell motility and tube formation via inhibition of calpain
    Richard J Bodnar
    Department of Pathology, University of Pittsburgh, Pittsburgh, PA 15261 0001, USA
    Circ Res 98:617-25. 2006
  2. ncbi IP-10 induces dissociation of newly formed blood vessels
    Richard J Bodnar
    Pittsburgh Veterans Affairs Medical Center, Pittsburgh, PA 15240, USA
    J Cell Sci 122:2064-77. 2009
  3. ncbi Delayed reepithelialization and basement membrane regeneration after wounding in mice lacking CXCR3
    Cecelia C Yates
    Department of Pathology, University of Pittsburgh, Pittsburgh, Pennsylvania 15261, USA
    Wound Repair Regen 17:34-41. 2009
  4. ncbi Regulation of glycoprotein Ib-IX-von Willebrand factor interaction by cAMP-dependent protein kinase-mediated phosphorylation at Ser 166 of glycoprotein Ib(beta)
    Richard J Bodnar
    Department of Pharmacology, University of Illinois, College of Medicine, Chicago, Illinois 60612, USA
    J Biol Chem 277:47080-7. 2002
  5. ncbi Critical roles for the COOH-terminal NITY and RGT sequences of the integrin beta3 cytoplasmic domain in inside-out and outside-in signaling
    Xiaodong Xi
    Department of Pharmacology, College of Medicine, University of Illinois at Chicago, Chicago, IL 60612, USA
    J Cell Biol 162:329-39. 2003

Research Grants

  1. Chemokine Modulation of Endothelial Cell Behavior
    Richard Bodnar; Fiscal Year: 2005

Collaborators

  • Alan Wells
  • PATRICIA HEBDA
  • Cecelia C Yates
  • Xiaodong Xi
  • Diana Whaley
  • Shveta Hooda
  • Zhenyu Li
  • Xiaoping Du
  • Stephen C-T Lam
  • Stephen C T Lam

Detail Information

Publications5

  1. ncbi IP-10 blocks vascular endothelial growth factor-induced endothelial cell motility and tube formation via inhibition of calpain
    Richard J Bodnar
    Department of Pathology, University of Pittsburgh, Pittsburgh, PA 15261 0001, USA
    Circ Res 98:617-25. 2006
    ..This provides a means by which late wound repair signals limit the angiogenesis driven early in the wound response process...
  2. ncbi IP-10 induces dissociation of newly formed blood vessels
    Richard J Bodnar
    Pittsburgh Veterans Affairs Medical Center, Pittsburgh, PA 15240, USA
    J Cell Sci 122:2064-77. 2009
    ..This is the first direct evidence for an extracellular signaling mechanism through CXCR3 that causes the dissociation of newly formed blood vessels followed by cell death...
  3. ncbi Delayed reepithelialization and basement membrane regeneration after wounding in mice lacking CXCR3
    Cecelia C Yates
    Department of Pathology, University of Pittsburgh, Pittsburgh, Pennsylvania 15261, USA
    Wound Repair Regen 17:34-41. 2009
    ..These studies further establish the emerging signaling network that involves the CXCR3 chemokine receptor and its ligands as a key regulator of wound repair...
  4. ncbi Regulation of glycoprotein Ib-IX-von Willebrand factor interaction by cAMP-dependent protein kinase-mediated phosphorylation at Ser 166 of glycoprotein Ib(beta)
    Richard J Bodnar
    Department of Pharmacology, University of Illinois, College of Medicine, Chicago, Illinois 60612, USA
    J Biol Chem 277:47080-7. 2002
    ..Thus, PKA-mediated phosphorylation of GPIbbeta at Ser(166) negatively regulates VWF binding to GPIb-IX and is one of the mechanisms by which PKA mediates platelet inhibition...
  5. ncbi Critical roles for the COOH-terminal NITY and RGT sequences of the integrin beta3 cytoplasmic domain in inside-out and outside-in signaling
    Xiaodong Xi
    Department of Pharmacology, College of Medicine, University of Illinois at Chicago, Chicago, IL 60612, USA
    J Cell Biol 162:329-39. 2003
    ....

Research Grants1

  1. Chemokine Modulation of Endothelial Cell Behavior
    Richard Bodnar; Fiscal Year: 2005
    ..This study will provide potential new targets to promote or limit vascular development in tissue and contribute to our ability to engineer tissues with appropriate microvasculature. ..