Richard J Bodnar
Affiliation: University of Pittsburgh
- IP-10 blocks vascular endothelial growth factor-induced endothelial cell motility and tube formation via inhibition of calpainRichard J Bodnar
Department of Pathology, University of Pittsburgh, Pittsburgh, PA 15261 0001, USA
Circ Res 98:617-25. 2006..This provides a means by which late wound repair signals limit the angiogenesis driven early in the wound response process...
- IP-10 induces dissociation of newly formed blood vesselsRichard J Bodnar
Pittsburgh Veterans Affairs Medical Center, Pittsburgh, PA 15240, USA
J Cell Sci 122:2064-77. 2009..This is the first direct evidence for an extracellular signaling mechanism through CXCR3 that causes the dissociation of newly formed blood vessels followed by cell death...
- Delayed reepithelialization and basement membrane regeneration after wounding in mice lacking CXCR3Cecelia C Yates
Department of Pathology, University of Pittsburgh, Pittsburgh, Pennsylvania 15261, USA
Wound Repair Regen 17:34-41. 2009..These studies further establish the emerging signaling network that involves the CXCR3 chemokine receptor and its ligands as a key regulator of wound repair...
- Regulation of glycoprotein Ib-IX-von Willebrand factor interaction by cAMP-dependent protein kinase-mediated phosphorylation at Ser 166 of glycoprotein Ib(beta)Richard J Bodnar
Department of Pharmacology, University of Illinois, College of Medicine, Chicago, Illinois 60612, USA
J Biol Chem 277:47080-7. 2002..Thus, PKA-mediated phosphorylation of GPIbbeta at Ser(166) negatively regulates VWF binding to GPIb-IX and is one of the mechanisms by which PKA mediates platelet inhibition...
- Critical roles for the COOH-terminal NITY and RGT sequences of the integrin beta3 cytoplasmic domain in inside-out and outside-in signalingXiaodong Xi
Department of Pharmacology, College of Medicine, University of Illinois at Chicago, Chicago, IL 60612, USA
J Cell Biol 162:329-39. 2003....
- Chemokine Modulation of Endothelial Cell BehaviorRichard Bodnar; Fiscal Year: 2005..This study will provide potential new targets to promote or limit vascular development in tissue and contribute to our ability to engineer tissues with appropriate microvasculature. ..