A A Azenabor

Summary

Affiliation: University of Wisconsin-Milwaukee
Country: USA

Publications

  1. doi request reprint Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca2+ dependent manner in human macrophages
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Comp Immunol Microbiol Infect Dis 33:513-28. 2010
  2. doi request reprint Chlamydia trachomatis induces anti-inflammatory effect in human macrophages by attenuation of immune mediators in Jurkat T-cells
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Immunobiology 216:1248-55. 2011
  3. doi request reprint Free intracellular Ca2+ regulates bacterial lipopolysaccharide induction of iNOS in human macrophages
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Immunobiology 214:143-52. 2009
  4. pmc Chlamydia trachomatis infection of human trophoblast alters estrogen and progesterone biosynthesis: an insight into role of infection in pregnancy sequelae
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Int J Med Sci 4:223-31. 2007
  5. ncbi request reprint Macrophage antioxidant enzymes regulate Chlamydia pneumoniae chronicity: evidence of the effect of redox balance on host-pathogen relationship
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Enderis Hall, Room 469, 2400 E Hartford Avenue, Milwaukee, WI 53211, USA
    Immunobiology 211:325-39. 2006
  6. ncbi request reprint Elicitation of reactive oxygen species in Chlamydia pneumoniae-stimulated macrophages: a Ca2+-dependent process involving simultaneous activation of NADPH oxidase and cytochrome oxidase genes
    Anthony A Azenabor
    Department of Health Sciences, Enderis Hall, University of Wisconsin, Milwaukee, WI 53211, USA
    Med Microbiol Immunol 194:91-103. 2005
  7. ncbi request reprint Expression of iNOS gene in macrophages stimulated with 17beta-estradiol is regulated by free intracellular Ca2+
    A A Azenabor
    Department of Health Sciences, Univesrity of Wisconsin, Enderis Hall, Room 469, 2400 E Hartford Avenue, Milwaukee, WI 53211, USA
    Biochem Cell Biol 82:381-90. 2004
  8. ncbi request reprint Induction of lipoprotein lipase gene expression in Chlamydia pneumoniae-infected macrophages is dependent on Ca2+ signaling events
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, 2400 E Hartford Avenue, Milwaukee, WI 53211, USA
    Biol Chem 385:67-74. 2004
  9. ncbi request reprint Macrophage L-type Ca2+ channel antagonists alter Chlamydia pneumoniae MOMP and HSP-60 mRNA gene expression, and improve antibiotic susceptibility
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Immunobiology 207:237-45. 2003
  10. ncbi request reprint 17 beta-estradiol induces L-type Ca2+ channel activation and regulates redox function in macrophages
    A A Azenabor
    Department of Health Sciences, Enderis Hall, Room 469, University of Wisconsin, 2400 East Hartford Avenue, Milwaukee, WI 53211, USA
    J Reprod Immunol 59:17-28. 2003

Collaborators

Detail Information

Publications14

  1. doi request reprint Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca2+ dependent manner in human macrophages
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Comp Immunol Microbiol Infect Dis 33:513-28. 2010
    ....
  2. doi request reprint Chlamydia trachomatis induces anti-inflammatory effect in human macrophages by attenuation of immune mediators in Jurkat T-cells
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Immunobiology 216:1248-55. 2011
    ..trachomatis mediates a switch from inflammatory to anti-inflammatory function in macrophages due to downregulation of the regulatory cytokine, IFN-γ in Jurkat cells, culminating in C. trachomatis chronic course...
  3. doi request reprint Free intracellular Ca2+ regulates bacterial lipopolysaccharide induction of iNOS in human macrophages
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Immunobiology 214:143-52. 2009
    ....
  4. pmc Chlamydia trachomatis infection of human trophoblast alters estrogen and progesterone biosynthesis: an insight into role of infection in pregnancy sequelae
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Int J Med Sci 4:223-31. 2007
    ..This defect may impair trophoblast functions of implantation and placentation, and consequently affect pregnancy sequelae...
  5. ncbi request reprint Macrophage antioxidant enzymes regulate Chlamydia pneumoniae chronicity: evidence of the effect of redox balance on host-pathogen relationship
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Enderis Hall, Room 469, 2400 E Hartford Avenue, Milwaukee, WI 53211, USA
    Immunobiology 211:325-39. 2006
    ..pneumoniae. These findings reflect the importance of redox balance modulation on the outcome of C. pneumoniae infection in macrophages, a significant process in the pathogenesis of chlamydial diseases...
  6. ncbi request reprint Elicitation of reactive oxygen species in Chlamydia pneumoniae-stimulated macrophages: a Ca2+-dependent process involving simultaneous activation of NADPH oxidase and cytochrome oxidase genes
    Anthony A Azenabor
    Department of Health Sciences, Enderis Hall, University of Wisconsin, Milwaukee, WI 53211, USA
    Med Microbiol Immunol 194:91-103. 2005
    ..The data reported here imply that macrophages infected with C. pneumoniae produce ROS through membrane-associated NADPH oxidase with oxidative phosphorylation levels depending on Ca2+ influx signals...
  7. ncbi request reprint Expression of iNOS gene in macrophages stimulated with 17beta-estradiol is regulated by free intracellular Ca2+
    A A Azenabor
    Department of Health Sciences, Univesrity of Wisconsin, Enderis Hall, Room 469, 2400 E Hartford Avenue, Milwaukee, WI 53211, USA
    Biochem Cell Biol 82:381-90. 2004
    ..These data show that Ca2+ influx pattern determines macrophage NO elaboration...
  8. ncbi request reprint Induction of lipoprotein lipase gene expression in Chlamydia pneumoniae-infected macrophages is dependent on Ca2+ signaling events
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, 2400 E Hartford Avenue, Milwaukee, WI 53211, USA
    Biol Chem 385:67-74. 2004
    ..pneumoniae-infected macrophages provides a mechanistic insight into early events involving C. pneumoniae in macrophage foam cell formation resulting from LDL uptake...
  9. ncbi request reprint Macrophage L-type Ca2+ channel antagonists alter Chlamydia pneumoniae MOMP and HSP-60 mRNA gene expression, and improve antibiotic susceptibility
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Immunobiology 207:237-45. 2003
    ..pneumoniae susceptibility to antibiotic therapy...
  10. ncbi request reprint 17 beta-estradiol induces L-type Ca2+ channel activation and regulates redox function in macrophages
    A A Azenabor
    Department of Health Sciences, Enderis Hall, Room 469, University of Wisconsin, 2400 East Hartford Avenue, Milwaukee, WI 53211, USA
    J Reprod Immunol 59:17-28. 2003
    ..This study demonstrates that 17 beta-estradiol exhibits a delayed phase of Ca(2+) influx involving L-type channel and regulates macrophage immune redox function...
  11. ncbi request reprint Effective macrophage redox defense against Chlamydia pneumoniae depends on L-type Ca2+ channel activation
    Anthony A Azenabor
    Department of Health Sciences, Enderis Hall, Room 469, University of Wisconsin, 2400 E Hartford Avenue, Milwaukee WI 53211, USA
    Med Microbiol Immunol 192:99-106. 2003
    ..pneumoniae inclusion formation. These findings suggest that Ca(2+) influx signal and redox function in C. pneumoniae-infected macrophages depend on L-type Ca(2+) channel activation...
  12. ncbi request reprint Chlamydia pneumoniae survival in macrophages is regulated by free Ca2+ dependent reactive nitrogen and oxygen species
    A A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    J Infect 46:120-8. 2003
    ..To gain better insights into C. pneumoniae survival mechanisms in macrophages, its growth in regular RAW-264.7 cells (nitric oxide sufficient NO (+)) and RAW-264.7 cells (nitric oxide insufficient NO (-)) were studied...
  13. ncbi request reprint Chlamydia pneumoniae infected macrophages exhibit enhanced plasma membrane fluidity and show increased adherence to endothelial cells
    Anthony A Azenabor
    Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
    Mol Cell Biochem 269:69-84. 2005
    ..The implications of these findings are that macrophages infected with C. pneumoniae have altered membrane physicochemical characteristics which may render them atherogenic...
  14. ncbi request reprint Chlamydia pneumoniae as a respiratory pathogen
    David L Hahn
    Arcand Park Clinic, Dean Medical Center, Madison WI 53704, USA
    Front Biosci 7:e66-76. 2002
    ..pneumoniae infection, and to advance our knowledge and understanding of the role of chronic infection in asthma, chronic bronchitis and COPD...