Research Topics
Species | A A AzenaborSummaryAffiliation: University of Wisconsin-Milwaukee Country: USA Publications
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Detail Information
Publications
Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca2+ dependent manner in human macrophagesAnthony A Azenabor
Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
Comp Immunol Microbiol Infect Dis 33:513-28. 2010....- Chlamydia trachomatis induces anti-inflammatory effect in human macrophages by attenuation of immune mediators in Jurkat T-cellsAnthony A Azenabor
Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
Immunobiology 216:1248-55. 2011..trachomatis mediates a switch from inflammatory to anti-inflammatory function in macrophages due to downregulation of the regulatory cytokine, IFN-γ in Jurkat cells, culminating in C. trachomatis chronic course... - Free intracellular Ca2+ regulates bacterial lipopolysaccharide induction of iNOS in human macrophagesAnthony A Azenabor
Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
Immunobiology 214:143-52. 2009.... - Chlamydia trachomatis infection of human trophoblast alters estrogen and progesterone biosynthesis: an insight into role of infection in pregnancy sequelaeAnthony A Azenabor
Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
Int J Med Sci 4:223-31. 2007..This defect may impair trophoblast functions of implantation and placentation, and consequently affect pregnancy sequelae... - Macrophage antioxidant enzymes regulate Chlamydia pneumoniae chronicity: evidence of the effect of redox balance on host-pathogen relationshipAnthony A Azenabor
Department of Health Sciences, University of Wisconsin, Enderis Hall, Room 469, 2400 E Hartford Avenue, Milwaukee, WI 53211, USA
Immunobiology 211:325-39. 2006..pneumoniae. These findings reflect the importance of redox balance modulation on the outcome of C. pneumoniae infection in macrophages, a significant process in the pathogenesis of chlamydial diseases... - Elicitation of reactive oxygen species in Chlamydia pneumoniae-stimulated macrophages: a Ca2+-dependent process involving simultaneous activation of NADPH oxidase and cytochrome oxidase genesAnthony A Azenabor
Department of Health Sciences, Enderis Hall, University of Wisconsin, Milwaukee, WI 53211, USA
Med Microbiol Immunol 194:91-103. 2005..The data reported here imply that macrophages infected with C. pneumoniae produce ROS through membrane-associated NADPH oxidase with oxidative phosphorylation levels depending on Ca2+ influx signals... - Expression of iNOS gene in macrophages stimulated with 17beta-estradiol is regulated by free intracellular Ca2+A A Azenabor
Department of Health Sciences, Univesrity of Wisconsin, Enderis Hall, Room 469, 2400 E Hartford Avenue, Milwaukee, WI 53211, USA
Biochem Cell Biol 82:381-90. 2004..These data show that Ca2+ influx pattern determines macrophage NO elaboration... - Induction of lipoprotein lipase gene expression in Chlamydia pneumoniae-infected macrophages is dependent on Ca2+ signaling eventsAnthony A Azenabor
Department of Health Sciences, University of Wisconsin, 2400 E Hartford Avenue, Milwaukee, WI 53211, USA
Biol Chem 385:67-74. 2004..pneumoniae-infected macrophages provides a mechanistic insight into early events involving C. pneumoniae in macrophage foam cell formation resulting from LDL uptake... - Macrophage L-type Ca2+ channel antagonists alter Chlamydia pneumoniae MOMP and HSP-60 mRNA gene expression, and improve antibiotic susceptibilityAnthony A Azenabor
Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
Immunobiology 207:237-45. 2003..pneumoniae susceptibility to antibiotic therapy... - 17 beta-estradiol induces L-type Ca2+ channel activation and regulates redox function in macrophagesA A Azenabor
Department of Health Sciences, Enderis Hall, Room 469, University of Wisconsin, 2400 East Hartford Avenue, Milwaukee, WI 53211, USA
J Reprod Immunol 59:17-28. 2003..This study demonstrates that 17 beta-estradiol exhibits a delayed phase of Ca(2+) influx involving L-type channel and regulates macrophage immune redox function... - Effective macrophage redox defense against Chlamydia pneumoniae depends on L-type Ca2+ channel activationAnthony A Azenabor
Department of Health Sciences, Enderis Hall, Room 469, University of Wisconsin, 2400 E Hartford Avenue, Milwaukee WI 53211, USA
Med Microbiol Immunol 192:99-106. 2003..pneumoniae inclusion formation. These findings suggest that Ca(2+) influx signal and redox function in C. pneumoniae-infected macrophages depend on L-type Ca(2+) channel activation... - Chlamydia pneumoniae survival in macrophages is regulated by free Ca2+ dependent reactive nitrogen and oxygen speciesA A Azenabor
Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
J Infect 46:120-8. 2003..To gain better insights into C. pneumoniae survival mechanisms in macrophages, its growth in regular RAW-264.7 cells (nitric oxide sufficient NO (+)) and RAW-264.7 cells (nitric oxide insufficient NO (-)) were studied... - Chlamydia pneumoniae infected macrophages exhibit enhanced plasma membrane fluidity and show increased adherence to endothelial cellsAnthony A Azenabor
Department of Health Sciences, University of Wisconsin, Milwaukee, WI 53211, USA
Mol Cell Biochem 269:69-84. 2005..The implications of these findings are that macrophages infected with C. pneumoniae have altered membrane physicochemical characteristics which may render them atherogenic... - Chlamydia pneumoniae as a respiratory pathogenDavid L Hahn
Arcand Park Clinic, Dean Medical Center, Madison WI 53704, USA
Front Biosci 7:e66-76. 2002..pneumoniae infection, and to advance our knowledge and understanding of the role of chronic infection in asthma, chronic bronchitis and COPD...