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Genomes and Genes | Theoharis TheoharidesSummaryAffiliation: Tufts University Country: USA Publications
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Perinatal stress, brain inflammation and risk of autism-review and proposalAsimenia Angelidou
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Molecular Physiology and Pharmacology, Tufts University School of Medicine and Tufts Medical Center, Boston, MA 02111, USA
BMC Pediatr 12:89. 2012..Many autism "susceptibility" genes have been identified, but "environmental" factors appear to play a significant role. Increasing evidence suggests that there are different ASD endophenotypes...
Is nasal saline irrigation all it is cracked up to be?Reena Khianey
UMDNJ NJMS, Newark, NJ 07103, USA
Ann Allergy Asthma Immunol 109:20-8. 2012..This review examines the literature regarding nasal saline irrigation in the management of sinonasal disease. We explore the various properties of nasal irrigation solutions and their effects on nasal symptoms...
Contribution of stress to asthma worsening through mast cell activationTheoharis C Theoharides
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Molecular Physiology and Pharmacology, Tufts University School of Medicine, Tufts Medical Center, Boston, MA 02111, USA
Ann Allergy Asthma Immunol 109:14-9. 2012..To review the available evidence linking stress to asthma and to investigate whether mast cells contribute to the effect of stress through activation by corticotropin-releasing hormone (CRH)...
Corticotropin-releasing hormone and extracellular mitochondria augment IgE-stimulated human mast-cell vascular endothelial growth factor release, which is inhibited by luteolinShahrzad Asadi
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Molecular Physiology and Pharmacology, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
J Neuroinflammation 9:85. 2012....
Mast cells in allergic and inflammatory diseasesNikolaos Sismanopoulos
Department of Molecular Physiology and Pharmacology, Tufts University School of Medicine, Boston, MA 02111, USA
Curr Pharm Des 18:2261-77. 2012..Better understanding of these two processes and how mast cells exert both immunostimulatory and immunosuppressive actions could lead to the development of inhibitors of release of specific mediators with novel therapeutic applications...
Mast cells and inflammationTheoharis C Theoharides
Laboratory of Molecular Immunopharmacology and Drug Discovery, Department of Molecular Physiology and Pharmacology, Tufts University School of Medicine, Boston, MA 02111, USA
Biochim Biophys Acta 1822:21-33. 2012..Unraveling selective mast cell secretion could also help develop unique mast cell inhibitors with novel therapeutic applications. This article is part of a Special Issue entitled: Mast cells in inflammation...
Mast cell activation and autismTheoharis C Theoharides
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, MA 02111, USA
Biochim Biophys Acta 1822:34-41. 2012..This article is part of a Special Issue entitled: Mast cells in inflammation...
Neuro-inflammation, blood-brain barrier, seizures and autismTheoharis C Theoharides
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Molecular Physiology and Pharmacology, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA, USA
J Neuroinflammation 8:168. 2011..Treating neuro-inflammation may be useful when anti-seizure medications are ineffective...
Mitochondrial DNA and anti-mitochondrial antibodies in serum of autistic childrenBodi Zhang
Laboratory of Molecular Immunopharmacology and Drug Discovery, Tufts University School of Medicine, Boston, MA, USA
J Neuroinflammation 7:80. 2010..001) as compared to normally developing, unrelated controls (n = 12). Extracellular blood mtDNA and other components may characterize an autistic endophenotype and may contribute to its pathogenesis by activating autoimmune responses...
Neurotensin is increased in serum of young children with autistic disorderAsimenia Angelidou
Laboratory of Molecular Immunopharmacology and Drug Discovery, Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, MA, USA
J Neuroinflammation 7:48. 2010..004). There was no statistically significant difference in the serum levels of β-endorphin or substance P (SP). NT could stimulate immune cells, especially mast cells, and/or have direct effects on brain inflammation and ASD...
Mercury induces inflammatory mediator release from human mast cellsDuraisamy Kempuraj
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts Medical Center, Boston, MA 02111, USA
J Neuroinflammation 7:20. 2010..We, therefore, investigated the effect of mercuric chloride (HgCl2) on human mast cell activation...
Treatment approaches for painful bladder syndrome/interstitial cystitisTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Tufts New England Medical Center, Boston, Massachusetts, USA
Drugs 67:215-35. 2007..Pilot clinical trials suggest that the flavonoid quercetin may be helpful. Lack of early diagnosis and treatment can affect outcomes and leads to the development of hyperalgesia/allodynia...
Human umbilical cord blood-derived mast cells: a unique model for the study of neuro-immuno-endocrine interactionsT C Theoharides
Department of Laboratory of Molecular Immunopharmacology and Drug Discovery, Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, MA, USA
Stem Cell Rev 2:143-54. 2006..The recent use of live cell array, with its ability to study such interactions rapidly at the single-cell level, provides unique new opportunities for fast output screening of mast cell triggers and inhibitors...
The critical role of mast cells in allergy and inflammationTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, MA 02111, USA
Ann N Y Acad Sci 1088:78-99. 2006..Combination of CRH receptor antagonists and mast cell inhibitors may present novel therapeutic interventions...
Corticotropin-releasing hormone and the blood-brain-barrierTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts New England Medical Center, 136 Harrison Avenue, Boston, MA 02111, USA
Front Biosci 12:1615-28. 2007..Brain mast cell inhibition and CRHR antagonists offer novel therapeutic possibilities...
Serum interleukin-6 reflects disease severity and osteoporosis in mastocytosis patientsTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, New England Medical Center, Boston, Mass 02111, USA
Int Arch Allergy Immunol 128:344-50. 2002..Serum tryptase has been increasingly used as a biochemical marker of mastocytosis, but is not always elevated...
The role of mast cells in migraine pathophysiologyTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts New England Medical Center, 136 Harrison Avenue, Boston, MA 02111, USA
Brain Res Brain Res Rev 49:65-76. 2005..A better understanding of brain mast cell activation in migraines would be useful and could lead to several points of prophylactic intervention...
Immunomodulators for treatment of interstitial cystitisTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts-New England Medical Center, Boston, Massachusetts 02111, USA
Urology 65:633-8. 2005
A pilot open label study of Cystoprotek in interstitial cystitisT C Theoharides
Departments of Pharmacology and Experimental Therapeutics, Biochemistry, and Internal Medicine, Tufts University School of Medicine, Boston, MA 02111, USA
Int J Immunopathol Pharmacol 18:183-8. 2005..2 (p < 0.05) and the Problem Index from 13.1 +/- 3.7 to 5.4 +/- 4.0 (p <0.05). These results are very promising and warrant a larger study that may permit further analyses with respect to other, especially atopic, comorbid diseases...
Mast cells as targets of corticotropin-releasing factor and related peptidesTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
Trends Pharmacol Sci 25:563-8. 2004....
Differential release of mast cell mediators and the pathogenesis of inflammationTheoharis C Theoharides
Laboratory of Molecular Immunopharmacology and Drug Discovery, Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Tufts New England Medical Center, Boston, MA, USA
Immunol Rev 217:65-78. 2007..Understanding this process could help develop mast cell inhibitors of selective mediator release with novel therapeutic applications...
Mast cells, T cells, and inhibition by luteolin: implications for the pathogenesis and treatment of multiple sclerosisTheoharis C Theoharides
Department of Pharmacology, Internal Medicine and Biochemistry, Immunology Program, Tufts University School of Medicine, Tufts New England Medical Center, Boston, MA, USA
Adv Exp Med Biol 601:423-30. 2007..Pretreatment with the flavone luteolin totally blocks mast cell stimulation and T cell activation. Mast cells could constitute a new unique therapeutic target for MS...
Mast cells squeeze the heart and stretch the gird: their role in atherosclerosis and obesityTheoharis C Theoharides
Laboratory of Molecular Immunopharmacology and Drug Discovery, Department of Molecular Physiology and Pharmacology, Tufts University School of Medicine, Boston, MA 02111, USA
Trends Pharmacol Sci 32:534-42. 2011....
IL-33 augments substance P-induced VEGF secretion from human mast cells and is increased in psoriatic skinTheoharis C Theoharides
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, and Maternal Fetal Medicine, Tufts Medical Center, Boston, MA 02111, USA
Proc Natl Acad Sci U S A 107:4448-53. 2010....
Luteolin as a therapeutic option for multiple sclerosisTheoharis C Theoharides
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts Medical Center, Boston, MA, USA
J Neuroinflammation 6:29. 2009..An appropriate luteolin formulation that permits sufficient absorption and reduces its metabolism could be a useful adjuvant to IFN-beta for MS therapy...
Autism: an emerging 'neuroimmune disorder' in search of therapyTheoharis C Theoharides
Molecular Immunopharmacology and Drug Discovery Laboratory, Tufts University School of Medicine, Tufts Medical Center, Department of Pharmacology, Boston, MA 02111, USA
Expert Opin Pharmacother 10:2127-43. 2009..There is no reliable screening test, and no definite pathogenesis or curative therapy. Consequently, there is a major gap hampering development of effective treatments...
Urticaria pigmentosa associated with acute stress and lesional skin mast-cell expression of CRF-R1T C Theoharides
Department of Pharmacology, Tufts University School of Medicine, Tufts Medical Center, Boston, MA 02111, USA
Clin Exp Dermatol 34:e163-6. 2009..This is the first documented report to our knowledge of UP worsening associated with acute stress, possibly through activation of skin mast-cell CRF-R1...
Human mast cells stimulate activated T cells: implications for multiple sclerosisTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts Medical Center, Boston, MA 02111, USA
Ann N Y Acad Sci 1144:74-82. 2008..Interactions between mast cells and T cells could constitute a new and unique therapeutic target for multiple sclerosis...
Interstitial cystitis: bladder pain and beyondTheoharis C Theoharides
Tufts University School of Medicine, Department of Pharmacology and Experimental Therapeutics, Experimental Therapeutics 136 Harrison Avenue, Boston, MA 02111, USA
Expert Opin Pharmacother 9:2979-94. 2008..Interstitial cystitis occurs primarily in females including adolescents and its diagnosis is still one of exclusion. It is now recognized as a serious medical condition associated with significant disability...
Impact of stress and mast cells on brain metastasesTheoharis C Theoharides
Laboratory of Molecular Immunopharmacology and Drug Discovery, Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts Medical Center, Boston, MA, USA
J Neuroimmunol 205:1-7. 2008..Stress and mast cells could serve as new targets for drug development to prevent brain metastases, especially since CRH receptor antagonists and brain mast cell inhibitors have recently been developed...
Novel therapeutic targets for autismTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Tufts Medical Center, Boston, MA 02111, USA
Trends Pharmacol Sci 29:375-82. 2008..We make suggestions for new research directives and possible novel therapies from readily available molecules...
Mast cells and mast cell mediators as targets of dietary supplementsTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts New England Medical Center, Boston, Massachusetts 02111, USA
Ann Allergy Asthma Immunol 93:S24-34. 2004..To review the increasing amount of data that support or dispel the use of dietary supplements in the treatment of inflammatory conditions that involve mast cells, such as allergies, arthritis, and chronic pelvic pain syndrome...
Cloning and cellular localization of the rat mast cell 78-kDa protein phosphorylated in response to the mast cell "stabilizer" cromolynT C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, MA, USA
J Pharmacol Exp Ther 294:810-21. 2000..It is hypothesized that conformational changes of moesin, regulated by phosphorylation/dephosphorylation, may lead to positional rearrangements with respect to the membrane/cytoskeleton that could possibly regulate mast cell secretion...
Chondroitin sulphate inhibits connective tissue mast cellsT C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, 136 Harrison Avenue, Boston, Massachusetts, MA 02111, USA
Br J Pharmacol 131:1039-49. 2000..5. Chondroitin sulphate appears to be a potent mast cell inhibitor of allergic and nonimmune stimulation with potential clinical implications...
New agents for the medical treatment of interstitial cystitisT C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
Expert Opin Investig Drugs 10:521-46. 2001....
Mast cell involvement in interstitial cystitis: a review of human and experimental evidenceT C Theoharides
Department of Pharmacology, Tufts University School of Medicine and New England Medical Center, Boston, Massachusetts, USA
Urology 57:47-55. 2001..Activation of mast cells, irrespective of bladder location or degree of mastocytosis, is significant. Mast cell-derived vasoactive and proinflammatory molecules may contribute to the pathogenesis of IC...
Mast cells: the Jekyll and Hyde of tumor growthTheoharis C Theoharides
Departments of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and New England Medical Center, 136 Harrison Avenue, Boston, MA 02111, USA
Trends Immunol 25:235-41. 2004
Acute stress shortens the time to onset of experimental allergic encephalomyelitis in SJL/J miceN Chandler
Surgical Research, Tufts University School of Medicine, New England Medical Center, Boston, MA 02111, USA
Brain Behav Immun 16:757-63. 2002
Critical role of mast cells in inflammatory diseases and the effect of acute stressTheoharis C Theoharides
Department of Pharmacology and Experimental Therapeutics, Tufts New England Medical Center, Boston, MA, USA
J Neuroimmunol 146:1-12. 2004....
Human mast cells express corticotropin-releasing hormone (CRH) receptors and CRH leads to selective secretion of vascular endothelial growth factorJing Cao
Department of Biochemistry, Tufts University School of Medicine, Boston, MA 02111, USA
J Immunol 174:7665-75. 2005..CRH-induced mast cell-derived VEGF could, therefore, be involved in chronic inflammatory conditions associated with increased VEGF, such as arthritis or psoriasis, both of which worsen by stress...
Flavonols inhibit proinflammatory mediator release, intracellular calcium ion levels and protein kinase C theta phosphorylation in human mast cellsDuraisamy Kempuraj
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, MA 02111, USA
Br J Pharmacol 145:934-44. 2005..Flavonols may therefore be suitable for the treatment of allergic and inflammatory diseases...
The mast cell in interstitial cystitis: role in pathophysiology and pathogenesisGrannum R Sant
Department of Urology, Tufts University School of Medicine, Boston, Massachusetts, USA
Urology 69:34-40. 2007....
Acute stress results in skin corticotropin-releasing hormone secretion, mast cell activation and vascular permeability, an effect mimicked by intradermal corticotropin-releasing hormone and inhibited by histamine-1 receptor antagonistsMichael Lytinas
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, MA 02111, USA
Int Arch Allergy Immunol 130:224-31. 2003....
Azelastine inhibits secretion of IL-6, TNF-alpha and IL-8 as well as NF-kappaB activation and intracellular calcium ion levels in normal human mast cellsDuraisamy Kempuraj
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts-New England Medical Center, Boston, MA 02111, USA
Int Arch Allergy Immunol 132:231-9. 2003..CONCLUSION: Azelastine inhibited hCBMC secretion of IL-6, TNF-alpha and IL-8, possibly by inhibiting intracellular Ca2+ ions and NF-kappaB activation. Azelastine may, therefore, be helpful in treating allergic inflammation...
Rupatadine inhibits proinflammatory mediator secretion from human mast cells triggered by different stimuliMagdalini Vasiadi
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts Medical Center, Boston, Mass 02111, USA
Int Arch Allergy Immunol 151:38-45. 2010..We investigated the ability of rupatadine, a potent histamine 1 receptor antagonist that also blocks platelet-activating factor actions, to also inhibit mast cell mediator release...
Corticotropin-releasing hormone-receptor 2 is required for acute stress-induced bladder vascular permeability and release of vascular endothelial growth factorWilliam Boucher
Molecular Immunopharmacology and Drug Discovery Laboratory, Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, MA 02111, USA
BJU Int 106:1394-9. 2010....
Stress-induced interleukin-6 release in mice is mast cell-dependent and more pronounced in Apolipoprotein E knockout miceMan Huang
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
Cardiovasc Res 59:241-9. 2003..The current study was carried out in order to determine any possible difference in stress-induced IL-6 release in atherosclerotic mice and the contribution of mast cells, corticotropin-releasing hormone (CRH) and urocortin (Ucn)...
Corticotropin-releasing hormone and brain mast cells regulate blood-brain-barrier permeability induced by acute stressPamela Esposito
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, New England Medical Center, Boston, Massachusetts 02111, USA
J Pharmacol Exp Ther 303:1061-6. 2002..These results demonstrate that CRH and mast cells are involved in regulating BBB permeability and, possibly, brain inflammatory disorders exacerbated by acute stress...
Interleukin-8 is increased in gingival crevicular fluid from patients with acute pulpitisVirginia Karapanou
Department of Endodontics, Tufts University School of Dental Medicine, Boston, Massachusetts 02111, USA
J Endod 34:148-51. 2008..Another group of patients (n = 7) who had received local anesthesia before sampling had very low IIP CXCL8 levels. GCF CXCL8 levels might be a useful measurement for staging patients with acute pulpitis...
Topical immunopharmacology of ocular allergiesLeonard Bielory
Department of Medicine, UMDNJ, Asthma and Allergy Research Center, New Jersey Medical School, Newark, New Jersey, USA
Curr Opin Allergy Clin Immunol 2:435-45. 2002..To review the histamine-1 receptor antagonists, mast cell blockers and natural agents with such actions that can be used for the topical treatment of ocular allergies...
High levels of intrauterine corticotropin-releasing hormone, urocortin, tryptase, and interleukin-8 in spontaneous abortionsBhuvaneshwari Madhappan
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, Massachusetts 02111, USA
Endocrinology 144:2285-90. 2003..Immunostaining of POC showed significantly more tryptase in group III women. High POC levels of CRH and Ucn under stress in habitual spontaneous abortions may activate uterine mast cells to secrete abortogenic tryptase and IL-8...
Azelastine is more potent than olopatadine n inhibiting interleukin-6 and tryptase release from human umbilical cord blood-derived cultured mast cellsDuraisamy Kempuraj
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and New England Medical Center, Boston, Massachusetts 02111, USA
Ann Allergy Asthma Immunol 88:501-6. 2002..CONCLUSIONS: These results indicate that azelastine and olopatadine can inhibit CHMCs activation and release of IL-6, tryptase, and histamine. On an equimolar basis, azelastine was a more potent inhibitor than olopatadine...
Azelastine's inhibition of histamine and tryptase release from human umbilical cord blood-derived cultured mast cells as well as rat skin mast cell-induced vascular permeability: comparison with olopatadineMichael Lytinas
Departments of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and New England Medical Center, Boston, Massachusetts, USA
Allergy Asthma Proc 23:45-51. 2002..These results indicate that on an equimolar basis, azelastine was a more potent inhibitor than olopatadine of both CHMC and rat skin mast cells activation...
Corticotropin-releasing hormone and its structurally related urocortin are synthesized and secreted by human mast cellsDuraisamy Kempuraj
Department of Pharmacology, Tufts University School of Medicine, Boston, Massachusetts 02111, USA
Endocrinology 145:43-8. 2004..These findings indicate that mast cells are not only the target, but also a potential source of CRH and Ucn that could have both autocrine and paracrine functions, especially in allergic inflammatory disorders exacerbated by stress...
IL-1 induces vesicular secretion of IL-6 without degranulation from human mast cellsKristiana Kandere-Grzybowska
Department of Biochemistry, Tufts University School of Medicine, Boston, MA 02111, USA
J Immunol 171:4830-6. 2003..Selective secretion of IL-6 from mast cells appears distinct from degranulation and may contribute to the development of inflammation, where the importance of IL-6 has been recognized...
Regulation of corticotropin-releasing hormone receptor-2 expression in human cord blood-derived cultured mast cellsNikoletta G Papadopoulou
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, MA 02111, USA
J Mol Endocrinol 35:R1-8. 2005..These results further implicate mast cells and CRH in either initiating or potentiating inflammatory diseases, especially those affected by stress...
Corticotropin-releasing hormone induces vascular endothelial growth factor release from human mast cells via the cAMP/protein kinase A/p38 mitogen-activated protein kinase pathwayJing Cao
Department of Biochemistry, Tufts University School of Medicine, Boston, MA 02111, USA
Mol Pharmacol 69:998-1006. 2006....
Corticotropin-releasing hormone induces skin vascular permeability through a neurotensin-dependent processJill Donelan
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Tufts-New England Medical Center, 136 Harrison Avenue, Boston, MA 02111, USA
Proc Natl Acad Sci U S A 103:7759-64. 2006..These findings suggest that CRH induces skin vascular permeability through NT acting on mast cells and that both peptides should be considered in the pathogenesis of skin disorders exacerbated by stress...
Increased numbers of activated mast cells in endometriosis lesions positive for corticotropin-releasing hormone and urocortinDuraisamy Kempuraj
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine and Tufts-New England Medical Center, Boston, MA 02111, USA
Am J Reprod Immunol 52:267-75. 2004..CONCLUSION: High numbers of activated mast cells are present in endometriosis sites that were strongly positive for CRH/Ucn. CRH and Ucn may activate mast cells and contribute to the fibrosis and inflammation in endometriosis...
Acute stress induces cardiac mast cell activation and histamine release, effects that are increased in Apolipoprotein E knockout miceMan Huang
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
Cardiovasc Res 55:150-60. 2002..These results may possibly help better understand stress-related cardiovascular pathology...
Stress-induced dura vascular permeability does not develop in mast cell-deficient and neurokinin-1 receptor knockout miceKristiana Kandere-Grzybowska
Department of Biochemistry, Tufts University School of Medicine, Tufts New England Medical Center, Boston, MA 02111, USA
Brain Res 980:213-20. 2003..These results may help explain initial events in pathogenesis of stress-induced migraines...
Corticotropin-releasing hormone receptor-1 and histidine decarboxylase expression in chronic urticariaNikoletta Papadopoulou
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Tufts New England Medical Center, Boston, Massachusetts 02111, USA
J Invest Dermatol 125:952-5. 2005..The lichen simplex samples had high expression of CRH-R1, but low HDC. These results implicate CRH-R in chronic urticaria, which is often exacerbated by stress...
Regulation of IL-1-induced selective IL-6 release from human mast cells and inhibition by quercetinKristiana Kandere-Grzybowska
Department of Biochemistry, Tufts University School of Medicine and Tufts - New England Medical Center, Harrison Avenue, Boston, MA, USA
Br J Pharmacol 148:208-15. 2006....
Pentosanpolysulfate (Elmiron) is a potent inhibitor of mast cell histamine secretionGeorge Chiang
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, USA
Adv Exp Med Biol 539:713-29. 2003
Mitochondrial uncoupling protein 2 inhibits mast cell activation and reduces histamine contentMichael Tagen
Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Tufts Medical Center, Boston, MA 02111, USA
J Immunol 183:6313-9. 2009..Our results suggest that UCP2 can regulate mast cell activation...
Niacin-induced "flush" involves release of prostaglandin D2 from mast cells and serotonin from platelets: evidence from human cells in vitro and an animal modelDean Papaliodis
Department of Pharmacology and Experimental Therapeutics, Molecular Immunopharmacology and Drug Discovery Laboratory, Tufts University School of Medicine, Tufts Medical Center, Boston, MA 02111, USA
J Pharmacol Exp Ther 327:665-72. 2008..0008), respectively, in this animal model. These results indicate that niacin-induced flush involves both PGD(2) and serotonin, suggesting that drugs other than ASA are required to effectively inhibit niacin-induced flush...
Corticotropin-releasing factor (CRF) can directly affect brain microvessel endothelial cellsPamela Esposito
Department of Pharmacology, Tufts University School of Medicine, New England Medical Center, 136 Harrison Avenue, Boston, MA 02111, USA
Brain Res 968:192-8. 2003..It is, therefore, possible that CRF may directly regulate BBB permeability, in addition to any effect mediated via brain mast cells...
Mast cells and pancreatic cancerTheoharis C Theoharides
Tufts University School of Medicine and Tufts Medical Center, Boston, USA
N Engl J Med 358:1860-1. 2008
Adverse cardiovascular events temporally associated with ma huang, an herbal source of ephedrineDavid Samenuk
Division of Cardiology, New England Medical Center, Boston, MA 02111, USA
Mayo Clin Proc 77:12-6. 2002....
Acute stress-induced seizures and loss of consciousness in a ten-year-old boy with cutaneous mastocytosisChristos Pehlivanidis
J Clin Psychopharmacol 22:221-4. 2002
Interleukin-16 network in inflammation and allergyPio Conti
Immunology Division, University of Chieti, Via dei Vestini, Chieti, Italy
Allergy Asthma Proc 23:103-8. 2002..In accordance with other data in the medical literature, we conclude that the most important function of IL-16 is the chemoattraction of CD4+ cells...
Interleukin-17 and the interleukin-17 family member networkShu-hai Huang
Guangxi Institute of Occupational Health, Nanning, China
Allergy Asthma Proc 25:17-21. 2004....
Relation of serum levels of mast cell tryptase of left ventricular systolic function, left ventricular volume or congestive heart failureBharathi Upadhya
Cardiology Section, Wake Forest University School of Medicine, Winston-Salem, North Carolina, USA
J Card Fail 10:31-5. 2004..In contrast, the presence of angiographically significant CAD is associated with elevated systemic tryptase levels...
Mast cell tryptase: a new biomarker in patients with stable coronary artery diseaseEfthymios N Deliargyris
Cardiology Section, Wake Forest University School of Medicine, Medical Center Boulevard, Winston-Salem, NC 27157-1045, USA
Atherosclerosis 178:381-6. 2005..Tryptase measurements may emerge as a novel way of identifying asymptomatic patients with CAD, and represent a new biomarker of therapeutic efficacy in patients with CAD...
Skin corticotropin-releasing hormone receptor expression in psoriasisMichael Tagen
J Invest Dermatol 127:1789-91. 2007
Neuroimmunoendocrine circuitry of the 'brain-skin connection'Ralf Paus
Department of Dermatology, University Hospital Schleswig Holstein, Campus Lubeck, University of Lubeck, D 23538 Lubeck, Germany
Trends Immunol 27:32-9. 2006..Thus, the skin and its appendages enable the study of complex neuroimmunoendocrine responses that peripheral tissues launch upon stress exposure, as a basis for identifying new targets for therapeutic stress intervention...
Mast cells and stress--a psychoneuroimmunological perspectiveTheoharis C Theoharides
J Clin Psychopharmacol 22:103-8. 2002
Interleukin-6 and mast cellsPio Conti
University of Chieti School of Medicine, Via dei Vestini, 66100 Chieti, Italy
Allergy Asthma Proc 23:331-5. 2002..IL-6 also up-regulates histamine production rather than increases its storage and is an important inducing factor for the expression of immunoglobulin E (IgE) Fc epsilon RI...
Flavones inhibit proliferation and increase mediator content in human leukemic mast cells (HMC-1)Michael Alexandrakis
Department of Hematology, Medical Division, School of Health Sciences, University of Crete, Heraklion, Crete, Greece
Eur J Haematol 71:448-54. 2003..CONCLUSION: These results indicate that certain flavonoids can inhibit HMC-1 proliferation, induce secretory granule development and the accumulation of mediators...
Neuroimmunology of stress: skin takes center stagePetra C Arck
Biomedical Research Center, Charite, University Medicine Berlin, Berlin, Germany
J Invest Dermatol 126:1697-704. 2006....
Perifollicular and perivascular mouse skin mast cells express corticotropin-releasing hormone receptorJill Donelan
J Invest Dermatol 126:929-32. 2006
Role of mast cells in tumor growthPio Conti
Immunology Division, Department of Oncology and Neuro science, University of Chieti, Via dei Vestini, 66100 Chieti, Italy
Ann Clin Lab Sci 37:315-22. 2007..This review focuses on the strategic importance of mast cells to the progression of tumors, and proposes a revised immune effector mechanism of mast cell involvement in tumor growth...
Neuroendocrine-immune disequilibrium and endometriosis: an interdisciplinary approachNadja Tariverdian
Center of Internal Medicine and Dermatology, Division of PsychoNeurolmmunology, Charite, University Medicine Berlin, Berlin, Germany
Semin Immunopathol 29:193-210. 2007..Finally, we introduce the concept of endometrial dissemination as result of a neuroendocrine-immune disequilibrium in response to high levels of perceived stress caused by cardinal clinical symptoms of endometriosis...
Advances in therapy with antileukotriene drugsGraziano Riccioni
Department of Biomedical Sciences, University G D Annunzio, Chieti, Italy
Ann Clin Lab Sci 34:379-87. 2004....
Depressive symptoms in urticaria patients treated with first- or second-generation histamine 1 receptor antagonistsDimitris Kalogeromitros
J Clin Psychopharmacol 27:404-6. 2007
Environmental and occupational stress and autoimmunityPaolo Boscolo
Occupational Medicine, Department of Medicine and Aging, Medical School, University of Chieti Pescara, Italy
Autoimmun Rev 7:340-3. 2008..Such a threat impacts the policy of health surveillance of the workers and requires dedication of further studies to the relationship between occupational stress and autoimmunity...
Panic disorder, interstitial cystitis, and mast cellsTheoharis C Theoharides
J Clin Psychopharmacol 24:361-4. 2004
IL-10, an inflammatory/inhibitory cytokine, but not alwaysPio Conti
Immunology Division, University of Chieti, School of Medicine, Chieti, Italy
Immunol Lett 86:123-9. 2003..Here, we report for the first time that IL-10 fails to inhibit tryptase and IL-6 from human mast cell-1 (HMC-1) and human umbilical cord blood-derived mast cells...
Chronic fatigue syndrome, mast cells, and tricyclic antidepressantsTheoharis C Theoharides
J Clin Psychopharmacol 25:515-20. 2005
Research Grants
- STRESS INDUCED SKIN MAST CELL ACTIVATION & VASODILATIONTheoharis C Theoharides; Fiscal Year: 2010..This is an important and under investigated area of skin pathophysiology that has potential applicability to understanding the pathogenesis of skin diseases and screening compounds that may develop into novel and effective treatments. ..
- STRESS INDUCED SKIN MAST CELL ACTIVATION & VASODILATIONTheoharis Theoharides; Fiscal Year: 2009..This is an important and under investigated area of skin pathophysiology that has potential applicability to understanding the pathogenesis of skin diseases and screening compounds that may develop into novel and effective treatments. ..
- STRESS INDUCED SKIN MAST CELL ACTIVATION & VASODILATIONTheoharis Theoharides; Fiscal Year: 2007..This is an important and under investigated area of skin pathophysiology that has potential applicability to understanding the pathogenesis of skin diseases and screening compounds that may develop into novel and effective treatments. ..
- Mast cells, antidepressants and chronic fatigue syndromeTheoharis Theoharides; Fiscal Year: 2007..Future studies will build on these findings to develop in vitro and in vivo models of CFS and lead to clinical trials with select antidepressants or other molecules that inhibit brain mast cells. ..
- Restraint stress-induced neurogenic bladder inflammationTheoharis Theoharides; Fiscal Year: 2006..Our findings may be relevant to the pathophysiology of IC and may suggest new therapeutic approaches. ..
- Stress Induces Skin Mast Cell Activation & VasodilationTheoharis Theoharides; Fiscal Year: 2005..They will also help investigate the pathophysiology of skin syndromes exacerbated by stress, such as atopic dermatitis or psoriasis, especially since CRH and CRH receptors have been identified in human skin. ..
- CORTICOTROPIN RELEASING HORMONE INDUCED DURA MAST CELL ATheoharis Theoharides; Fiscal Year: 2001..They may also help explain some key events in the pathophysiology of migraines, the clinical symptoms of which are often precipitated by stress. ..
- Brain mast cells and Chronic Fatigue SyndromeTheoharis C Theoharides; Fiscal Year: 2010..The proposed research is expected to advance our understanding of how activation of brain mast cells can contribute to inflammation and CFS, as well as to the development of novel and effective treatments. ..
