A M Soto

Summary

Affiliation: Tufts University
Country: USA

Publications

  1. pmc The E-SCREEN assay as a tool to identify estrogens: an update on estrogenic environmental pollutants
    A M Soto
    Tufts University School of Medicine, Department of Anatomy and Cellular Biology, Boston, MA 02111, USA
    Environ Health Perspect 103:113-22. 1995
  2. pmc Developing a marker of exposure to xenoestrogen mixtures in human serum
    A M Soto
    Tufts University School of Medicine, Department of Anatomy and Cellular Biology, Boston, Massachusetts, USA
    Environ Health Perspect 105:647-54. 1997
  3. pmc Androgen-induced proliferative quiescence in prostate cancer cells: the role of AS3 as its mediator
    P Geck
    Department of Anatomy and Cell Biology, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
    Proc Natl Acad Sci U S A 97:10185-90. 2000
  4. ncbi request reprint Androgen-induced inhibition of proliferation in human breast cancer MCF7 cells transfected with androgen receptor
    J Szelei
    Tufts University School of Medicine, Department of Anatomy and Cellular Biology, Boston, Massachusetts 02111, USA
    Endocrinology 138:1406-12. 1997
  5. ncbi request reprint Identification of human estrogen-inducible transcripts that potentially mediate the apoptotic response in breast cancer
    J Szelei
    Department of Anatomy and Cell Biology, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
    J Steroid Biochem Mol Biol 72:89-102. 2000
  6. ncbi request reprint An updated review of environmental estrogen and androgen mimics and antagonists
    C Sonnenschein
    Tufts University School of Medicine, Department of Anatomy and Cellular Biology, Boston, MA 02111, USA
    J Steroid Biochem Mol Biol 65:143-50. 1998
  7. ncbi request reprint Identification and characterization of membrane estrogen receptor from MCF7 estrogen-target cells
    C E Powell
    Department of Anatomy and Cellular Biology, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
    J Steroid Biochem Mol Biol 77:97-108. 2001
  8. ncbi request reprint Early gene expression during androgen-induced inhibition of proliferation of prostate cancer cells: a new suppressor candidate on chromosome 13, in the BRCA2-Rb1 locus
    P Geck
    Department of Anatomy and Cell Biology, Tufts University School of Medicine, Boston, MA 02111, USA
    J Steroid Biochem Mol Biol 68:41-50. 1999
  9. ncbi request reprint In utero exposure to bisphenol A alters the development and tissue organization of the mouse mammary gland
    C M Markey
    Department of Anatomy and Cellular Biology, Tufts University School of Medicine, Boston, Massachusetts 02111 1800, USA
    Biol Reprod 65:1215-23. 2001
  10. pmc Perinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levels
    B S Rubin
    Department of Anatomy and Cellular Biology, Tufts Medical School, Boston, MA 02111 USA
    Environ Health Perspect 109:675-80. 2001

Collaborators

Detail Information

Publications11

  1. pmc The E-SCREEN assay as a tool to identify estrogens: an update on estrogenic environmental pollutants
    A M Soto
    Tufts University School of Medicine, Department of Anatomy and Cellular Biology, Boston, MA 02111, USA
    Environ Health Perspect 103:113-22. 1995
    ....
  2. pmc Developing a marker of exposure to xenoestrogen mixtures in human serum
    A M Soto
    Tufts University School of Medicine, Department of Anatomy and Cellular Biology, Boston, Massachusetts, USA
    Environ Health Perspect 105:647-54. 1997
    ..quot;..
  3. pmc Androgen-induced proliferative quiescence in prostate cancer cells: the role of AS3 as its mediator
    P Geck
    Department of Anatomy and Cell Biology, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
    Proc Natl Acad Sci U S A 97:10185-90. 2000
    ..Tetracycline-regulated expression of the empty vector control had no effect on cell proliferation. These experiments strongly suggest that AS3 is a mediator of the androgen-induced proliferative shutoff...
  4. ncbi request reprint Androgen-induced inhibition of proliferation in human breast cancer MCF7 cells transfected with androgen receptor
    J Szelei
    Tufts University School of Medicine, Department of Anatomy and Cellular Biology, Boston, Massachusetts 02111, USA
    Endocrinology 138:1406-12. 1997
    ..These data suggest that: 1) the androgen-induced inhibition of cell proliferation (Step-2) is AR-mediated; and 2) the AR may be necessary, but not sufficient, to mediate the androgen-induced proliferative response (Step-1)...
  5. ncbi request reprint Identification of human estrogen-inducible transcripts that potentially mediate the apoptotic response in breast cancer
    J Szelei
    Department of Anatomy and Cell Biology, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
    J Steroid Biochem Mol Biol 72:89-102. 2000
    ..This correlation suggests that E9 may be a mediator of estrogen-induced regression of breast cancer...
  6. ncbi request reprint An updated review of environmental estrogen and androgen mimics and antagonists
    C Sonnenschein
    Tufts University School of Medicine, Department of Anatomy and Cellular Biology, Boston, MA 02111, USA
    J Steroid Biochem Mol Biol 65:143-50. 1998
    ..In addition, it will be necessary to develop methods to measure cumulative exposure to (a) estrogen mimics, (b) antiandrogens, and (c) other disruptors...
  7. ncbi request reprint Identification and characterization of membrane estrogen receptor from MCF7 estrogen-target cells
    C E Powell
    Department of Anatomy and Cellular Biology, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA
    J Steroid Biochem Mol Biol 77:97-108. 2001
    ..We propose that the observed mER actively participates in the estrogen-mediated proliferation of MCF7 cells...
  8. ncbi request reprint Early gene expression during androgen-induced inhibition of proliferation of prostate cancer cells: a new suppressor candidate on chromosome 13, in the BRCA2-Rb1 locus
    P Geck
    Department of Anatomy and Cell Biology, Tufts University School of Medicine, Boston, MA 02111, USA
    J Steroid Biochem Mol Biol 68:41-50. 1999
    ..The AS3 gene seems to be a novel candidate in the regulation of androgen-induced proliferative arrest of human prostate cells...
  9. ncbi request reprint In utero exposure to bisphenol A alters the development and tissue organization of the mouse mammary gland
    C M Markey
    Department of Anatomy and Cellular Biology, Tufts University School of Medicine, Boston, Massachusetts 02111 1800, USA
    Biol Reprod 65:1215-23. 2001
    ..The altered relationship in DNA synthesis between the epithelium and stroma and the increase in terminal ducts and terminal end buds are striking, because these changes are associated with carcinogenesis in both rodents and humans...
  10. pmc Perinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levels
    B S Rubin
    Department of Anatomy and Cellular Biology, Tufts Medical School, Boston, MA 02111 USA
    Environ Health Perspect 109:675-80. 2001
    ..These data indicate an increased sensitivity to BPA during the perinatal period and suggest the need for careful evaluation of the current levels of exposure to this compound...
  11. doi request reprint On physicalism and downward causation in developmental and cancer biology
    A M Soto
    Department of Anatomy and Cellular Biology, Tufts University School of Medicine, Boston, MA 02111, USA
    Acta Biotheor 56:257-74. 2008
    ..We provide an argument for rejecting this assumption. Furthermore, this allows us to reconsider the concept of diachronic emergence...