Samar A Jasser

Summary

Affiliation: Thomas Jefferson University
Country: USA

Publications

  1. ncbi request reprint Light during darkness and cancer: relationships in circadian photoreception and tumor biology
    Samar A Jasser
    Department of Neurology, Light Research Program, Thomas Jefferson University, 1025 Walnut Street, Suite 507, Philadelphia, PA 19107, USA
    Cancer Causes Control 17:515-23. 2006
  2. ncbi request reprint Dim light adaptation attenuates acute melatonin suppression in humans
    Samar A Jasser
    Department of Neurology, Thomas Jefferson University, Philadelphia, PA 19107, USA
    J Biol Rhythms 21:394-404. 2006
  3. ncbi request reprint Melatonin-depleted blood from premenopausal women exposed to light at night stimulates growth of human breast cancer xenografts in nude rats
    David E Blask
    Laboratory of Chrono Neuroendocrine Oncology, Bassett Research Institute, The Mary Imogene Bassett Hospital, Cooperstown, New York 13326, USA
    Cancer Res 65:11174-84. 2005
  4. ncbi request reprint Motif analysis of the tumor suppressor gene MMAC/PTEN identifies tyrosines critical for tumor suppression and lipid phosphatase activity
    Dimpy Koul
    Department of Neuro Oncology, Box 100, The Brain Tumor Center, The University of Texas, M D Anderson Cancer Center, 1515 Holcombe Boulevard, Houston, Texas, TX 77030, USA
    Oncogene 21:2357-64. 2002
  5. ncbi request reprint Epidermal growth factor receptor blockade potentiates apoptosis mediated by Paclitaxel and leads to prolonged survival in a murine model of oral cancer
    F Christopher Holsinger
    Department of Head and Neck Surgery, The University of Texas M D Anderson Cancer Center, Houston, Texas 77030 4009, USA
    Clin Cancer Res 9:3183-9. 2003

Collaborators

Detail Information

Publications5

  1. ncbi request reprint Light during darkness and cancer: relationships in circadian photoreception and tumor biology
    Samar A Jasser
    Department of Neurology, Light Research Program, Thomas Jefferson University, 1025 Walnut Street, Suite 507, Philadelphia, PA 19107, USA
    Cancer Causes Control 17:515-23. 2006
    ..Continued rigorous empirical inquiry into the physiology and clinical implications of these habitual, integrated aspects of life is highly warranted at this time...
  2. ncbi request reprint Dim light adaptation attenuates acute melatonin suppression in humans
    Samar A Jasser
    Department of Neurology, Thomas Jefferson University, Philadelphia, PA 19107, USA
    J Biol Rhythms 21:394-404. 2006
    ....
  3. ncbi request reprint Melatonin-depleted blood from premenopausal women exposed to light at night stimulates growth of human breast cancer xenografts in nude rats
    David E Blask
    Laboratory of Chrono Neuroendocrine Oncology, Bassett Research Institute, The Mary Imogene Bassett Hospital, Cooperstown, New York 13326, USA
    Cancer Res 65:11174-84. 2005
    ..These mechanistic studies are the first to provide a rational biological explanation for the increased breast cancer risk in female night shift workers...
  4. ncbi request reprint Motif analysis of the tumor suppressor gene MMAC/PTEN identifies tyrosines critical for tumor suppression and lipid phosphatase activity
    Dimpy Koul
    Department of Neuro Oncology, Box 100, The Brain Tumor Center, The University of Texas, M D Anderson Cancer Center, 1515 Holcombe Boulevard, Houston, Texas, TX 77030, USA
    Oncogene 21:2357-64. 2002
    ..Thus, Y240A and Y315A are involved in the ability of MMAC/PTEN to dephosphorylate PtdIns and regulate tumor cell growth in vitro and in vivo...
  5. ncbi request reprint Epidermal growth factor receptor blockade potentiates apoptosis mediated by Paclitaxel and leads to prolonged survival in a murine model of oral cancer
    F Christopher Holsinger
    Department of Head and Neck Surgery, The University of Texas M D Anderson Cancer Center, Houston, Texas 77030 4009, USA
    Clin Cancer Res 9:3183-9. 2003
    ..A greater apoptotic fraction of tumor cells was found in tumors of mice treated with paclitaxel and PKI166 as compared with the other treatment groups (136.4 versus 37.8; P = 0.016)...