Michael J Saganich

Summary

Affiliation: The Salk Institute
Country: USA

Publications

  1. ncbi request reprint B-ephrin reverse signaling is required for NMDA-independent long-term potentiation of mossy fibers in the hippocampus
    John N Armstrong
    Molecular Neurobiology Laboratory, The Salk Institute, La Jolla, California 93037, USA
    J Neurosci 26:3474-81. 2006
  2. ncbi request reprint Deficits in synaptic transmission and learning in amyloid precursor protein (APP) transgenic mice require C-terminal cleavage of APP
    Michael J Saganich
    Salk Institute for Biological Studies, La Jolla, California 92037, USA
    J Neurosci 26:13428-36. 2006
  3. ncbi request reprint DPP10 modulates Kv4-mediated A-type potassium channels
    Edward Zagha
    Department of Physiology and Neuroscience, New York University School of Medicine, New York, New York 10016
    J Biol Chem 280:18853-61. 2005

Collaborators

Detail Information

Publications3

  1. ncbi request reprint B-ephrin reverse signaling is required for NMDA-independent long-term potentiation of mossy fibers in the hippocampus
    John N Armstrong
    Molecular Neurobiology Laboratory, The Salk Institute, La Jolla, California 93037, USA
    J Neurosci 26:3474-81. 2006
    ..These results indicate that B-ephrin reverse signaling into the presynaptic mossy fiber bouton is required for the induction of NMDA receptor-independent LTP at this synapse...
  2. ncbi request reprint Deficits in synaptic transmission and learning in amyloid precursor protein (APP) transgenic mice require C-terminal cleavage of APP
    Michael J Saganich
    Salk Institute for Biological Studies, La Jolla, California 92037, USA
    J Neurosci 26:13428-36. 2006
    ..These results indicate that cleavage of APP may play a critical role in the development of synaptic and behavioral dysfunction in APP transgenic mice...
  3. ncbi request reprint DPP10 modulates Kv4-mediated A-type potassium channels
    Edward Zagha
    Department of Physiology and Neuroscience, New York University School of Medicine, New York, New York 10016
    J Biol Chem 280:18853-61. 2005
    ..Chimeras of DPPX and DPP10, but not DPPIV, were able to modulate the properties of Kv4.2 channels, highlighting the importance of the intracellular and transmembrane domains in this activity...