R A Battaglino

Summary

Affiliation: The Forsyth Institute
Country: USA

Publications

  1. pmc Spinal cord injury-induced osteoporosis: pathogenesis and emerging therapies
    Ricardo A Battaglino
    The Forsyth Institute, Cambridge, MA 02142, USA
    Curr Osteoporos Rep 10:278-85. 2012
  2. ncbi request reprint Fluoxetine treatment increases trabecular bone formation in mice
    R Battaglino
    Department of Cytokine Biology, Forsyth Institute, Boston, Massachusetts 02115, USA
    J Cell Biochem 100:1387-94. 2007
  3. pmc NHA-oc/NHA2: a mitochondrial cation-proton antiporter selectively expressed in osteoclasts
    R A Battaglino
    Department of Cytokine Biology, Forsyth Institute, 140 The Fenway, Boston, MA 02115, USA
    Bone 42:180-92. 2008
  4. pmc Sclerostin: a candidate biomarker of SCI-induced osteoporosis
    L R Morse
    Department of Physical Medicine and Rehabilitation, Harvard Medical School, Boston, MA, USA
    Osteoporos Int 24:961-8. 2013
  5. pmc Expression analysis of nha-oc/NHA2: a novel gene selectively expressed in osteoclasts
    L Pham
    Department of Cytokine Biology, Forsyth Institute, 140 The Fenway, Boston, MA 02115, USA
    Gene Expr Patterns 7:846-51. 2007
  6. pmc Age and motor score predict osteoprotegerin level in chronic spinal cord injury
    L R Morse
    Department of Physical Medicine and Rehabilitation, Harvard Medical School and Spaulding Rehabilitation Hospital, Boston, Massachusetts 02118, USA
    J Musculoskelet Neuronal Interact 8:50-7. 2008
  7. doi request reprint SNX10 is required for osteoclast formation and resorption activity
    C H Zhu
    Department of Cytokine Biology, Forsyth Institute, Boston, Massachusetts 02142, USA
    J Cell Biochem 113:1608-15. 2012
  8. pmc Osteoporotic fractures and hospitalization risk in chronic spinal cord injury
    L R Morse
    Department of Physical Medicine and Rehabilitation, Harvard Medical School and Spaulding Rehabilitation Hospital, Boston, MA 02118, USA
    Osteoporos Int 20:385-92. 2009
  9. ncbi request reprint Inducible nitric oxide synthase mediates bone development and P. gingivalis-induced alveolar bone loss
    R Gyurko
    Department of Periodontology and Oral Biology, Goldman School of Dental Medicine, Boston University, 100 East Newton Street, Room 107, Boston, MA 02118, USA
    Bone 36:472-9. 2005

Detail Information

Publications9

  1. pmc Spinal cord injury-induced osteoporosis: pathogenesis and emerging therapies
    Ricardo A Battaglino
    The Forsyth Institute, Cambridge, MA 02142, USA
    Curr Osteoporos Rep 10:278-85. 2012
    ..This review summarizes emerging therapeutics including anti-sclerostin antibodies, mechanical loading of the lower extremity with electrical stimulation, and mechanical stimulation via vibration therapy...
  2. ncbi request reprint Fluoxetine treatment increases trabecular bone formation in mice
    R Battaglino
    Department of Cytokine Biology, Forsyth Institute, Boston, Massachusetts 02115, USA
    J Cell Biochem 100:1387-94. 2007
    ..These data suggest that commonly used anti-depressive agents may affect bone mass...
  3. pmc NHA-oc/NHA2: a mitochondrial cation-proton antiporter selectively expressed in osteoclasts
    R A Battaglino
    Department of Cytokine Biology, Forsyth Institute, 140 The Fenway, Boston, MA 02115, USA
    Bone 42:180-92. 2008
    ..NHA-oc/NHA2 displays the expected activities of a bona fide CPA and plays a key role(s) in normal osteoclast differentiation and function...
  4. pmc Sclerostin: a candidate biomarker of SCI-induced osteoporosis
    L R Morse
    Department of Physical Medicine and Rehabilitation, Harvard Medical School, Boston, MA, USA
    Osteoporos Int 24:961-8. 2013
    ..We report that sclerostin is significantly associated with bone mineral content and bone density at all skeletal sites tested. We found no association between bone and any other tested biomarker...
  5. pmc Expression analysis of nha-oc/NHA2: a novel gene selectively expressed in osteoclasts
    L Pham
    Department of Cytokine Biology, Forsyth Institute, 140 The Fenway, Boston, MA 02115, USA
    Gene Expr Patterns 7:846-51. 2007
    ..However, only a subset of cathepsin k-expressing cells is positive for nha-oc/NHA2, suggesting that nha-oc is expressed by terminally differentiated osteoclasts...
  6. pmc Age and motor score predict osteoprotegerin level in chronic spinal cord injury
    L R Morse
    Department of Physical Medicine and Rehabilitation, Harvard Medical School and Spaulding Rehabilitation Hospital, Boston, Massachusetts 02118, USA
    J Musculoskelet Neuronal Interact 8:50-7. 2008
    ..e., walking or using a wheelchair)...
  7. doi request reprint SNX10 is required for osteoclast formation and resorption activity
    C H Zhu
    Department of Cytokine Biology, Forsyth Institute, Boston, Massachusetts 02142, USA
    J Cell Biochem 113:1608-15. 2012
    ....
  8. pmc Osteoporotic fractures and hospitalization risk in chronic spinal cord injury
    L R Morse
    Department of Physical Medicine and Rehabilitation, Harvard Medical School and Spaulding Rehabilitation Hospital, Boston, MA 02118, USA
    Osteoporos Int 20:385-92. 2009
    ..The clinical assessment did not include osteoporosis diagnosis and treatment considerations, indicating a need for improved clinical protocols...
  9. ncbi request reprint Inducible nitric oxide synthase mediates bone development and P. gingivalis-induced alveolar bone loss
    R Gyurko
    Department of Periodontology and Oral Biology, Goldman School of Dental Medicine, Boston University, 100 East Newton Street, Room 107, Boston, MA 02118, USA
    Bone 36:472-9. 2005
    ..These data indicate that iNOS promotes bone resorption during bone development as well as after bacterial infection, and that iNOS is an important signal for normal osteoclast differentiation...