Gavin Bart

Summary

Affiliation: The Rockefeller University
Country: USA

Publications

  1. ncbi Suppressed prolactin response to dynorphin A1-13 in methadone-maintained versus control subjects
    Gavin Bart
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, New York, NY 10021 6399, USA
    J Pharmacol Exp Ther 306:581-7. 2003
  2. ncbi Increased attributable risk related to a functional mu-opioid receptor gene polymorphism in association with alcohol dependence in central Sweden
    Gavin Bart
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, New York, NY 10021, USA
    Neuropsychopharmacology 30:417-22. 2005
  3. ncbi Nalmefene induced elevation in serum prolactin in normal human volunteers: partial kappa opioid agonist activity?
    Gavin Bart
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, 1230 York Avenue, New York, NY 10021, USA
    Neuropsychopharmacology 30:2254-62. 2005
  4. ncbi Altered levels of basal cortisol in healthy subjects with a 118G allele in exon 1 of the Mu opioid receptor gene
    Gavin Bart
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, New York, NY 10021, USA
    Neuropsychopharmacology 31:2313-7. 2006
  5. ncbi Prodynorphin gene promoter repeat associated with cocaine/alcohol codependence
    Tanya J Williams
    The Laboratories of the Biology of Addictive Diseases and Statistical Genetics, The Rockefeller University, USA
    Addict Biol 12:496-502. 2007
  6. ncbi Pharmacogenetics and human molecular genetics of opiate and cocaine addictions and their treatments
    Mary Jeanne Kreek
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, Box 171, 1230 York Avenue, New York, New York 10021, USA
    Pharmacol Rev 57:1-26. 2005
  7. doi Markers for hepatitis A, B and C in methadone maintained patients: an unexpectedly high co-infection with silent hepatitis B
    Gavin Bart
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, NY, NY 10021, USA
    Addiction 103:681-6. 2008
  8. ncbi Corticotropin-releasing factor testing reveals a dose-dependent difference in methadone maintained vs control subjects
    James H Schluger
    Laboratory of the Biology of Addictive Diseases, The Rockefeller University, 1230 York Avenue, New York, NY 10021 6399, USA
    Neuropsychopharmacology 28:985-94. 2003
  9. ncbi Evolving perspectives on neurobiological research on the addictions: celebration of the 30th anniversary of NIDA
    Mary Jeanne Kreek
    Laboratory of the Biology of the Addictive Diseases, The Rockefeller University, 1230 York Avenue, Box 171, New York, NY 10021, USA
    Neuropharmacology 47:324-44. 2004
  10. pmc Mu-opioid receptor A118G polymorphism in healthy volunteers affects hypothalamic-pituitary-adrenal axis adrenocorticotropic hormone stress response to metyrapone
    Elizabeth Ducat
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, New York, NY 10065, USA
    Addict Biol 18:325-31. 2013

Detail Information

Publications11

  1. ncbi Suppressed prolactin response to dynorphin A1-13 in methadone-maintained versus control subjects
    Gavin Bart
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, New York, NY 10021 6399, USA
    J Pharmacol Exp Ther 306:581-7. 2003
    ..Whether methadone-maintained subjects also have decreased dopaminergic response to dynorphin and other kappa-opioid receptor ligands in mesolimbic-mesocortical and nigrostriatal dopaminergic systems cannot be determined from this study...
  2. ncbi Increased attributable risk related to a functional mu-opioid receptor gene polymorphism in association with alcohol dependence in central Sweden
    Gavin Bart
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, New York, NY 10021, USA
    Neuropsychopharmacology 30:417-22. 2005
    ..1%. There was no difference in A118G genotype between type 1 and type 2 alcoholics. In central Sweden, the functional variant 118G allele in exon 1 of OPRM1 is associated with an increased attributable risk for alcohol dependence...
  3. ncbi Nalmefene induced elevation in serum prolactin in normal human volunteers: partial kappa opioid agonist activity?
    Gavin Bart
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, 1230 York Avenue, New York, NY 10021, USA
    Neuropsychopharmacology 30:2254-62. 2005
    ....
  4. ncbi Altered levels of basal cortisol in healthy subjects with a 118G allele in exon 1 of the Mu opioid receptor gene
    Gavin Bart
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, New York, NY 10021, USA
    Neuropsychopharmacology 31:2313-7. 2006
    ..Subjects with at least one copy of the 118G allele have increased basal levels of cortisol, which may influence the susceptibility to and treatment of the stress responsive dyscrasia...
  5. ncbi Prodynorphin gene promoter repeat associated with cocaine/alcohol codependence
    Tanya J Williams
    The Laboratories of the Biology of Addictive Diseases and Statistical Genetics, The Rockefeller University, USA
    Addict Biol 12:496-502. 2007
    ....
  6. ncbi Pharmacogenetics and human molecular genetics of opiate and cocaine addictions and their treatments
    Mary Jeanne Kreek
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, Box 171, 1230 York Avenue, New York, New York 10021, USA
    Pharmacol Rev 57:1-26. 2005
    ..Variants in genes encoding proteins involved in metabolism or biotransformation of drugs of abuse and also of treatment agents are reviewed...
  7. doi Markers for hepatitis A, B and C in methadone maintained patients: an unexpectedly high co-infection with silent hepatitis B
    Gavin Bart
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, NY, NY 10021, USA
    Addiction 103:681-6. 2008
    ..To determine the prevalence of hepatitis A, B and C viruses in patients attending a methadone maintenance clinic in New York City...
  8. ncbi Corticotropin-releasing factor testing reveals a dose-dependent difference in methadone maintained vs control subjects
    James H Schluger
    Laboratory of the Biology of Addictive Diseases, The Rockefeller University, 1230 York Avenue, New York, NY 10021 6399, USA
    Neuropsychopharmacology 28:985-94. 2003
    ....
  9. ncbi Evolving perspectives on neurobiological research on the addictions: celebration of the 30th anniversary of NIDA
    Mary Jeanne Kreek
    Laboratory of the Biology of the Addictive Diseases, The Rockefeller University, 1230 York Avenue, Box 171, New York, NY 10021, USA
    Neuropharmacology 47:324-44. 2004
    ..g., in the hypothalamic-pituitary-adrenal axis) plays a role in vulnerability and relapse to specific addictive diseases. This atypical responsivity may be drug-induced, environmentally acquired, and/or due to genetic variation...
  10. pmc Mu-opioid receptor A118G polymorphism in healthy volunteers affects hypothalamic-pituitary-adrenal axis adrenocorticotropic hormone stress response to metyrapone
    Elizabeth Ducat
    The Laboratory of the Biology of Addictive Diseases, The Rockefeller University, New York, NY 10065, USA
    Addict Biol 18:325-31. 2013
    ..These findings suggest a relatively greater tonic inhibition at hypothalamic-pituitary sites through the mu-opioid receptor and relatively less cyclical glucocorticoid inhibition in subjects with the 118G allele...
  11. ncbi Clock reset for alcoholism
    Vadim Yuferov
    Nat Med 11:23-4. 2005